Gastrointestinal Flashcards

1
Q

Cholecystitis
(how to diagnose & treatment)

A
  • Inflammation of the gallbladder
  • Ultrasound will show an enlarged/irritated gallbladder
  • Tx: chronic antibiotic therapy (amoxicillin and enrofloxacin) or cholecystectomy
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2
Q

Spontaneous rupture of the gall bladder (secondary to? presenting complaint? Treatment?)

A
  • Usually secondary to cholecystitis or biliary mucocele that ruptured
  • Will present with abdominal pain and icterus (bile spilled into peritoneum)
  • Tx: cholecystectomy and aggressive antimicrobial therapy
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3
Q

What is a biliary mucocele?

A

Excessive mucus is secreted in the gall bladder and becomes solid and occludes common bile duct (causing post-hepatic hyperbilirubinemia)

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3
Q

Biliary mucocele treatment

A

Cholecystectomy (ideally before rupture!!)

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4
Q

Canine parvovirus (pathophysiology, prevention and transmission)

A
  • Virus destroys rapidly diving cells (intestinal crypt cells)
  • Prevention via DAPP vaccine
  • Spread fecal oral
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5
Q

Canine parvovirus clinical signs and diagnosis

A
  • leukopenia, fever, severe diarrhea (often bloody), anorexia
  • fecal ELISA
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6
Q

Canine parvovirus treatment

A

IV fluids, antibiotics (IV broad spectrum), gastroprotectants (PPI, H2, sucralfate), anti-emetics

PREVENTION KEY

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7
Q

Constipation treatment

A
  • DSS (docusate sodium/ senna)
  • Soap/water enema
  • KY jelly enema
  • Stool softeners like laxatone, lactulose, polyethylene glycol 3350
  • Increase fiber intake
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8
Q

Persistent deciduous teeth is common in ___ breeds

A

toy/small

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9
Q

How to differentiate uncomplicated vs complicated dental fractures?

A

Uncomplicated- damage is limited to enamel and dentin
Complicated- damage exposes endodontic system and root

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10
Q

Most common location of esophageal foreign bodies

A

thoracic inlet, base of heart, diaphragm (places of esophageal narrowing)

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11
Q

Most common esophageal foreign bodies in dogs vs cats

A

Dogs- bones
Cats- toys, sewing needles

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12
Q

Esophagitis (how to diagnose and treat)

A

Diagnose via endoscopy + biopsy
Treat with sucralfate, H2 antagonist, PPI, Prokinetics (like metoclopramide and cisapride to encourage gastric emptying)

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13
Q

Exocrine pancreatic insufficiency pathophysiology (main cause for dogs vs cats)

A

Inadequate synthesis and secretion of digestive enzymes the pancreas. Results in lack of nutrient absorption.
Dogs- pancreatic acinar atrophy
Cats- chronic pancreatitis

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14
Q

EPI clinical signs

A

Polyphagia, diarrhea, weight loss, yellow/gray feces

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15
Q

EPI diagnosis

A

TLI will be low (less than 5 ug/l)

16
Q

EPI treatment

A

Exogenous pancreatic enzyme supplementation
Low-fiber diet (fiber can interfere with enzymes)

17
Q

Gastric dilation/volvulus signalment (who is most at risk?)

A

Large, deep-chested breeds (german shepherds, great danes, rottweilers)
Fast-eaters

18
Q

GDV pathophysiology

A

Rotation of stomach counter-clockwise resulting in venous compression/congestion and compromise of blood perfusion to the stomach (resulting in necrosis)

19
Q

GDV clinical signs

A

restlessness, pacing, painful, non-productive vomiting, abdominal distention, shock or CV compromise (weak pulses, pale MM, tachycardia)

20
Q

GDV diagnosis

A

Radiographs: look for “popeye arm”
Blood gas: metabolic acidosis (d/t increase lactate)
VPCs on EKG analysis

21
Q

When suspecting a GDV, which position for radiographs should you order?

A

Right lateral abdominal (patient laying on their right, leaving the stomach closest to the beam)

22
Q

When determining GDV prognosis, what should you look at?

A

Lactate >6 = gastric necrosis
Lactate <6 = good prognosis

If patient is in lateral upon presentation that is also a poor prognosis indicator

23
Q

GDV Treatment (stabilization vs surgery)

A
  • TWO large CEPHALIC IV catheters (avoid saphenous)
  • Decompress stomach (OG tube or trocarization)
  • Shock dose of crystalloid fluids 80-90 mg/kg in fractions
  • Monitor BP and EKG

Surgery: decompress and reposition the stomach, perform gastropexy to prevent reoccurence