Gastroenteritis Flashcards

1
Q
A
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2
Q

Define gastroenteritis and classify in terms of length of symptoms.

A
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3
Q

How would you define gastroenteritis in terms of bowel movements?

A

diarrhoea >= 3/day

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4
Q

RFs for developing Acute gastroenteritis.

A
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5
Q

What organisms commonly cause gastroenteritis?
Which organism is specific to the immunocompromised?

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6
Q

Describe the pathogenesis of gastroenteritis of the following organisms:
1. E-coli

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7
Q

Describe the pathogenesis of gastroenteritis of the following organisms:
2. Cholera

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8
Q

Describe the pathogenesis of gastroenteritis of the following organisms:
3. Shigella

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9
Q

What are the S+S of gastroenteritis?

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10
Q

Ddx of gastroenteritis.

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11
Q

Diagnostic investigations for gastroenteritis.

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12
Q

What are the possible EEG findings in acute gastroenteritis?

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13
Q

You are investigating a patient with acute gastroenteritis. The blood results came back with low platelets. What may be the cause of this?

A

HUS, DIC, TTP

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14
Q

How would microangiopathic anaemia look on blood flim?

A

schistocytes

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15
Q

What is the indication for a sigmoidoscopy in an acute gastroenteritis?

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16
Q

What organisms are commonly screened for in a stool culture?

A
17
Q

List and justify the supportive investigations for gastroenteritis.

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18
Q

Outline ALL the investigations for gastroenteritis.

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19
Q

What is fulminant colitis? (include S+S)

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20
Q

Outline the preventative measures to decrease risk of gastroenteritis.

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21
Q

Outline the acute management of gastroenteritis (c diff).

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22
Q

Outline the acute management of gastroenteritis. - non c diff

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23
Q

Outline the FULL management of gastroenteritis.

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24
Q

Describe the complications of gastroenteritis.

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25
Q

What electrolyte disturbances are expected in AKI?

A

In Acute Kidney Injury (AKI), the following electrolyte disturbances are common:

Hyperkalemia: Due to reduced potassium excretion; risk of arrhythmias.

Hyponatremia: Often dilutional from fluid retention; can cause neurological symptoms.

Hyperphosphatemia: Impaired phosphate excretion; can lead to calcium-phosphate imbalances.

Hypocalcemia: Reduced vitamin D activation and phosphate retention; may cause cramps or tetany.

Hypermagnesemia: Decreased magnesium excretion; can lead to bradycardia and hypotension.

Metabolic Acidosis: Accumulation of hydrogen ions; may cause compensatory hyperventilation.

Hypervolemia: Fluid retention; can cause edema and hypertension.

26
Q

What is the triad of HUS?

A