gait

Question 1

in swing phase you have; pre swing

Answer 1

the position limb for swing

Question 2

the initial swing swing is when?

Answer 2

foot clearance of the floor

limb advancement from trailing position

Question 3

what is when the limb advance and foot clears the floor

Answer 3

mid swing

Question 4

a complete limb advancement and preparation for stance

Answer 4

terminal stance

Question 5

in the stance phase what is the inital contact?

Answer 5

position of the limb start to stance

Question 6

when in stance phase what is shock absorption, weight-bearing stability, and forward progression

Answer 6

loading phase

Question 7

what is the progression of COG over BOS and limb and trunk stability?

Answer 7

mid stance

Question 8

what is terminal stance in when there is progression of COG beyond BOS and supporting foot

Answer 8

terminal stance

Question 9

gait is typically impaired following CVA why?

Answer 9

weakness, sensory loss, impaired balance, and loss of confidence

*Important to observe ankles, knees, hips, trunk, UE from all planes of movement
*Time, distance, cadence, velocity should all be measured and recorded

Gait speed is used in predicting a patient’s ability to ambulate in different environments

Question 10

what are common gait deviation post-stroke; abnormal gait?

Answer 10

-Slow speed
-Asymmetrical, uneven step and stride lengths
-Reduced stance time on affected limb
-Decreased push-off force on affected limb
-Use of synergy patterns to advance limb
-Impaired balance with UE and LE posturing
-Reliance on adaptive equipment
-Spasticity requiring compensatory advancement

Question 11

what are some classifications of walking handicaps after a stroke?

Answer 11

-Physiological walker
Walks for exercise only either at home or in therapy

-Limited household walker
Relies on walking for home activities
Requires assist for other walking activities

-Community walker
Unlimited distance outside

look to Power point for chart

Question 12

what abnormal gait:
paresis is a primary contributor to disordered gait?

Answer 12

-Affects the number, type and frequency of motor neurons essential for force production for gait

-Primary impairment after corticospinal pathology

Question 13

why does the Muscles act concentrically to generate movement and force and eccentrically to control the motion?

Answer 13

Ability to propel the body forward and control the body going forward

Question 14

what are two ways spasticity can impact gait?

Answer 14

-Inappropriate activation of a muscle at points during the gait cycle when it’s rapidly being stretched

Produces increased stiffness and the freedom of the muscle to move rapidly

Question 15

Inability to selectively recruit muscles is a contributing factor to abnormal gait why

Answer 15

-Associated with abnormal coupling of muscles resulting in abnormal synergies which manifest as either total extension or total flexion

-Synergistic patterns are associated with reduced stability and poor locomotion

Question 16

Overactivity of muscles unrelated to?

Answer 16

spasticity can affect progression in gait and postural control

HS overactivity is common

Question 17

Inability to time and scale muscle activity during gait can

Answer 17

can give way to ataxic gait which
is
Staggering, veering, irregular stepping, and high steppage
or
Delay in movement of the knee and ankle through the gait cycle

Question 18

Coactivation of antagonist muscles during gait is common post CVA and is

Answer 18

Is seen in both paretic and non paretic limb

Question 19

Impaired reactive balance strategies contributes to

Answer 19

instability in gait and stance (see previous lecture)

*Impaired proactive balance strategies are impaired

Question 20

Decreased ability to modify gait characteristics in response to terrain and slope changes are

Answer 20

-Slower gait speed and increased variability in gait

When walking downhill, people with stroke shorten their step length which causes reduced walking speed

Not able to increase step length when walking up hill

Question 21

what are some abnormal sensory input can result in ataxia

Answer 21

Can be due to peripheral or central proprioceptive pathways

Person has no awareness of where their leg(s) are in space

Reduced modulation of reflex activity throughout the gait cycle

Question 22

Vision is critical to balance strategies during gait how does it leads to abnormal gait

Answer 22

Individuals with vision impairment tend to walk slower and use more auditory cues

Hemianopsia can have an impact on the person’s ability to perceive potential threats to stability

Question 23

Loss of vestibular input in adulthood can produce gait ataxia and difficulty stabilizing the head in space due to?

Answer 23

Longer double stance support

Can cause impaired vision and oscillopsia

Question 24

what can body image deficits cause?

Answer 24

Ipsilateral trunk lean towards the stance leg

Inappropriate foot placement and difficulty in controlling the COM relative to the changing BOS of the feet

Unilateral spatial neglect tend to veer to the opposite side or bump into objects on the affected side

Question 25

Pain can alter the movement pattern used for gait

Question 26

Antalgic gait is a gait pattern that results from pain in the back or Les, compensatory strategies include

Answer 26

Reduce WB time on painful limb (shortened stance of gait) Avoid impact loads Reduce joint excursions (limiting knee flexion during stance) Side bending over a painful hip to bring the COM closer to the joints center of rotation which reduces the need for hip abductor activity

Question 27

what happens with plantarflexors

Answer 27

Studies show reduced activation of the PF is associated with strong hyperextension at the knee in stance and lack of knee flexion for swing Reduced energy during preswing of gait has been largely associated with reduced use of PFs PF recruitment in the paretic limb has been shown to be significantly reduced compared to non-paretic limb or controls Compensation for the reduction in PF recruitment gives increased knee and hip moments of the paretic limb PF paresis is one of the many contributing factors contributing to knee hyperextension Spasticity in the plantar flexors is also a common problem following CVA and neurologic injury.

Question 28

Plantarflexion and abnormal gait

Answer 28

Spasticity primarily occurs in the early party of stance phase of walking secondary to stretching of the gastroc Shortening of the muscle before the body is ahead of the foot causes knee hyperextension Also causes inability for the gastroc to generate enough force for push-off Spasticity is associated with reduced gait speed however the effects are of spasticity are increased with faster speeds Spasticity will impact foot position Limit DF and prevent heel strike at IC -If IC is made with a flat foot = knee hyperextension -Reduced forward foot clearance = toe drag

Question 29

Excessive gastroc and posterior tibilalis activation produce inversion and equinovarus foot position?

Answer 29

Plantarflexors

Question 30

how does the quads affect abnormal Gait?

Answer 30

-Weak quads = difficulty controlling knee flexion during loading and midstance -Compensation is hyperextension of the knee during midstance or forward trunk lean to bring GRF anterior to the knee -If hyperextension continues into preswing, it prevents the knee from moving freely during swing phase = slow progression and toe drag -Spasticity can also give excessive knee extension -During IC, there is a brief knee flexion to absorb shock and can trigger spasticity that can cause hyperextension of the knee -Spasticity can lead to stiff knee gait

Question 31

15.8 The effect of plantarflexor spasticity in gait.

Answer 31

(A) When initial contact is made with a flat foot, the ground reaction force (GRF) vector is anterior to the knee, producing knee extension. (B) Spastic plantarflexors affect forward foot clearance during swing; the subsequent consequence is toe drag.

Question 32

A combination of excessive activity of the TS and posterior tibialis muscle produces

Answer 32

inversion and an equinovarus foot position,

Question 33

Hip flexors and abnormal gait?

Answer 33

-Weakness primarily affects swing phase -Assist in progression to produce a hip flexor moment at the initiation of swing -Knee flexion for swing is lost if there is inadequate hip flexion = toe clearance is reduced of lost and can result in a shortened step -Compensatory strategies used to achieve foot clearance without adequate hip flexion *Posterior tilt of the pelvis and activation if the abdominals *Circumduction (hip hike) *Contralateral vaulting *Leaning the trunk laterally

Question 34

Hip extensors can cause

Answer 34

-Critical to control head, arms, and trunk -Weakness can result in forward trunk lean so to compensate, patient leans backwards to bring the COM behind the hips -TA are active to prevent falling backwards

Question 35

how does Hip abductors vs abnormal gait?

Answer 35

-Weakness can result in contralateral pelvic drop (Trendelenburg) -Common compensation is a lateral shift of the COM over the stance leg with lateral lean of trunk towards stance leg -Weakness results in instability in the frontal plane

Question 36

how does hamstring affect gait?

Answer 36

-Spasticity produces excessive knee flexion which can lead to a crouched-gait pattern -In terminal swing, excessive activation of hamstrings prevents the knee from fully extending -Excessive knee flexion from spasticity can increase the demand on the quads to prevent collapse

Question 37

how does hip adductors affect gait

Answer 37

-Spasticity produces a contralateral drop of the pelvis during stance -Can also result in scissoring gait which can also reduce the BOS and stability

Question 38

TUG

Answer 38

*Pt seated in a standard chair with armrests is instructed to stand and walk toward a line 3 meters (10 feet) away, turn and walk back and return to seated position. *Start on “Go” *Start with back against chair. *May use Assistive Device (AD), but no physical assist. Cutoff Scores: <10-12 Sec normal healthy adult 12-20 sec average frail elderly adult. >14 Sec Fall Risk for Stroke population

Question 39

10 meter walk test

Answer 39

*A clear pathway of at least 10 m (32.8 ft) in length in a designated area over solid flooring is required. * Measure and mark the start and end point of a 10-m walkway. * Add a mark at 2 m and 8 m (identifying the central 6 m which will be timed). -Total time to ambulate is recorded in m/s

Question 40

DGI

Answer 40

*Examines a patient’s ability to perform steady state walking and variations on command. 8 items/conditions 4 -point scale (0-3) 3: no gait dysfunction 0: severe impairment. *Possible score 24 *Strong fall-risk predictor, a score below 19 is indicative of fall Risk. 8 conditions Gait on level surface, change in gait speed, gait with vertical head turns, gait with horizontal head turns, gait pivot and turn, stairs, step over obstacle, step around obstacle

Question 41

FGA

Answer 41

*Used to assess postural stability during walking and assesses an individual's ability to perform multiple motor tasks while walking. *10items/conditions *4 -point scale (0-3) -3: no gait dysfunction -2: Mild impairment -1: Moderate impairment -0: severe impairment. *Possible score 30 *Strong fall-risk predictor, a score below 22 is indicative of fall Risk. *10 conditions ---Gait on level surface, change in gait speed, gait with vertical head turns, gait with horizontal head turns, gait pivot and turns, gait with eyes closed, backwards walking, step over obstacles, gait with narrow BOS

Question 42

6 MWT

Answer 42

*assesses distance walked over 6 minutes as a sub-maximal test of aerobic capacity/endurance. *May stand to rest but cannot sit down. *Test only if appropriate level of activity tolerance