Functional Programming for Stability-Mobility and Movement Flashcards
the ability to maintain or control joint movement or position
joint stability
the range of uninhibited movement around a joint or body segment
joint mobility
T/F: Movement efficiency involves a synergistic approach between stability and mobility where proximal stability promotes distal mobility.
True
the movement efficiency model
- active systems (muscles), passive systems (ligaments, joints), neural systems (proprioceptors, motor units), arthrokinematics contribute to: - sensory input and motor output leads to: - joint stability and mobility leads to: - movement efficiency
the general term for the specific movements of joint surfaces, such as rolling or gliding
arthrokinematics
joints that promote/favor stability
scapulothoracic
lumbar spine
knee
foot
joints that promote/favor mobility
glenohumeral
thoracic spine
hip
ankle
typical movement compensations that occur when mobility is compromised
1) the joint will seek to achieve the desired ROM by incorporating movement into another plane (e.g., in the birddog (sagittal plane movement), lack of flexibility in hip flexors will cause extended leg and hips rotate into the transverse plane)
2) adjacent, more stable joints may need to compromise some degree of stability to facilitate the level of mobility needed (e.g., someone with kyphosis who attempts to extend the thoracic spine may increase lumbar lordosis as compensation for the lack of thoracic mobility)
T/F: Periods of inactivity when joints are held passively in shortened positions result in muscle shortening.
True
T/F: Prolonged periods of sitting without hip extension shortens the hip flexors.
True
cycle of dysfunctional movement
1) muscle imbalances caused by various factors
2) alters muscle physiological and neurological properties
3) compromises the mobility-stability relationship
4) the body subscribes to the law of facilitation
5) dysfunctional movement
6) inevitable breakdowns
the relationship between the contractile proteins (e.g., myosin and actin) of a sarcomere and their force-generating capacity
length-tension relationship
T/F: A slight stretching of the sarcomere beyond its normal resting length is optimal in increasing force-generating capacity.
True
causes/reasons for shifting the length-tension curve to the left (excess sarcomere shortening/overlap)
1) immobilization
2) passive shortening
3) trauma
4) aging
amount of sarcomeres in a typical muscle myofibril
500,000
amount of time it takes for muscles to shorten when held in passively shortened positions without being stretched or used through full or functional ROM
2-4 weeks
force-generating capacity (increased or decreased) of a shortened muscle in various positions (shortened, normal, lengthened)
shortened: increased
normal: decreased
lengthened: decreased
best way (training method) to restore normal resting length and muscle’s force-generating capacity
strengthen the muscle in normal-resting-length positions, not lengthened positions
best method (exercise) to correct a client’s protracted shoulders (lengthened rhomboids and posterior deltoids)
isometric contraction or limited ROM of high-back rows; do not perform the exercise using full ROM as momentum will be carried through the weaker region, decreasing the ability to strengthen the muscle where it needs to be strengthened
through this, muscles function by providing opposing, directional, or contralateral pulls at joint to achieve efficient movement
force-coupling relationships
T/F: Hypertonic muscles decrease the neural drive to the opposing muscle via reciprocal inhibition.
True
When an antagonist muscle is further weakened due to reciprocal inhibition, other muscles at the joint (synergists) will assume responsibility of becoming the prime mover.
synergistic dominance
T/F: An example of synergistic dominance is a tight hip flexor will weaken the gluteus maximus and force the hamstrings to assume a greater role in hip extension.
True
T/F: Synergistic dominance may lead to overuse/overload and increased the likelihood for tightness and injury.
True
pain compensation cycle
1) muscle imbalance
2) altered length-tension and force-coupling relationships
3) altered joint mechanics
4) altered neuromuscular control and function
5) postural misalignments and faulty loading
6) excessive musculoskeletal loading
7) pain, injury, and further compensation
8) further muscle imbalance (cycle starts over)
pre-exercise stretching techniques for deconditioned clients with poor flexibility and muscle imbalance
stability and mobility (restorative flexibility) =>
myofascial release, warm-up, static stretches
pre-exercise stretching techniques for conditioned clients with good flexibility and muscle balance
maintain active flexibility =>
myofascial release, dynamic stretches
pre-exercise stretching techniques for performance athletes with good skill and flexibility
functional flexibility =>
dynamic stretches, ballistic stretches
stretches during exercise
dynamic stretches
post-exercise stretching techniques
myofascial release, proprioceptive neuromuscular facilitation (PNF), static stretches
client performs small, continuous, back-and-forth movements on a foam roller or similar device, covering an area of 2-6 in (5-15 cm) over a tender region for 30-60 seconds
self-myofascial release (SMR)
stretches taken to the point of tension, performing a minimum of 4 reps, and holding each rep for 15-60 seconds
static stretching
a hold-relax stretch, holding the isometric contraction of the agonist for a minimum of 6 seconds, followed by a 10-30 second assisted or passive static stretch
proprioceptive neuromuscular facilitation (PNF)
stretching that is effective for individuals participating in sports that require explosive movement
dynamic and ballistic stretching
beginning routine/phase of a progression model aiming to strengthen weakened muscles
2-4 reps of isometric contractions, each held 5-10 seconds, at less than 50% maximal voluntary contraction (MVC), in a supported, more isolated environment
purpose of the deep (innermost) layer of the core
small muscles that are rich in sensory nerve endings that provide continuous feedback to the brain regarding loading and position of the spine
purpose of the middle layer of the core
provide a solid, stable working foundation from which the body can operate
purpose of the outermost layer of the core
responsible for generating movement and forces within the trunk
muscles contained in the outermost layer of the core
rectus abdominis, erector spinae, internal and external obliques, iliopsoas, and latissimus dorsi
muscles contained in the middle layer of the core
transverse abdominis (TVA), multifidi, quadratus lumborum, deep fibers of the internal oblique, diaphragm, pelvic floor musculature, and the adjoining fasciae
T/F: Delayed activation of the transverse abdominis may inadequately stabilize the lumbar spine during movements of the upper and lower extremities, increasing the potential for injury.
True
percentage of the US population affected by low-back pain
80%
core training phase should begin with what protocol
establishing the stability of the lumbar spine with exercises that emphasize TVA activation and the re-education of faulty motor patterns
terms used to define the activation of the TVA which draws the abdomen inward toward the spine
centering, hollowing, or drawing-in
T/F: Bracing ensures a higher degree of stability than centering (or hollowing, drawing-in).
True
the co-contraction of the core and abdominal muscles to create a more rigid and wider base of support for spinal stabilization
bracing
T/F: Clients should master bracing before being introduced the concept of centering.
False
centering should be mastered first before bracing is introduced
3 stage model for core and balance training
1) core function
2) static balance
3) dynamic balance
focus of core function training
core-activation exercises and isolated stabilization under minimal spinal loading