Fractures Flashcards

1
Q

What is a greenstick fracture?

A
  • incomplete fracture
    • Cortex on the tension side of the fracture fails but the cortex on the compression side of the fracture remains intact
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2
Q

What is a torus fracture?

A

crease of the bone and periosteum

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3
Q

What is plastic deformation?

A

bad bend which stays bent, but isn’t obviously fractured

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4
Q

What is the growth plate?

A
  • a lucency between the epiphysis and metaphysis - can appear similar to a fracture
  • The physis is the weakest part of a developing bone, so is prone to injury
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5
Q

What are Salter-Harris fractures?

A
  • In normal bones, the epiphysis is always centered on the metaphysis
  • In Salter-Harris fractures, the epiphysis is not centered on the metaphysis due to a fracture involving the growth plate
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6
Q

What is a typical history and features of non-accidental injury?

A
  • History that does not match the nature or the severity of the injury
  • Vague parental accounts or accounts that change during the interview - inconsistency
  • Accusations that the child injured him/herself intentionally
  • Delay in seeking help
  • Child dressed inappropriately for the situation
  • Obvious/suspected fractures in a child under 2yrs
  • Injuries in various stages of healing, especially burns and bruises
  • More injuries than usually seen in children of the same age
  • Injuries scattered on many areas of the body
  • Increased intracranial pressure in an infant
  • Suspected intra-cranial trauma in a young child
  • Any injury that does not fit the description of the cause given
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7
Q

What is the general management of children’s fractures?

A
  • Reduce, retain, rehabilitate
  • Children are more amenable to conservative treatment - plaster, traction, less invasive fixation - due to increased modelling potential
  • Exceptions to the rule of conservative management in children - displaced intra-articular fractures, displaced growth plate injuries, some open fractures
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8
Q

What is the plaster of paris?

A
  • Diaphyseal fracture - joint immobilised above and below to prevent rotation
  • Metaphyseal fracture - adjacent joint immobilised
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9
Q

What are the techniques of fixation?

A
  • Diaphyseal - flexible nails
  • Metaphyseal - K wires
  • Epiphyseal - K wires and screws
  • External fixation - try and avoid in children where possible
    • Used in contaminated wounds, acute vascular injury, burns and multiple injuries
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10
Q

What are the symptoms of a fracture?

A
  • Localised bony (marked) tenderness - not diffuse mild tenderness
  • Swelling
  • Deformity
  • Crepitus - from bone ends grating with an unstable fracture
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11
Q

What is found on examination of a fracture?

A
  • Open or closed injury
  • Assessment of distal neurovascular status (pulses, capillary refill, temperature, colour, sensation, motor power)
  • Assess for compartment syndrome
  • Assess the status of the skin and soft tissue envelope
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12
Q

What is the general initial management of a fracture?

A
  • Clinical assessment
  • Analgesia (usually IV morphine)
  • Splintage +/- traction
    • May involve the application of a temporary plaster slab (known as a backslab), a sling, an orthosis or a Thomas splint (for femoral shaft fractures)
  • Imaging - x-ray, CT, MRI
    • Guidelines e.g. Ottawa guidelines for ankle injury assist with selecting patients for x-ray
    • A useful rule is that if a patient cannot weight bear on an injured lower limb, X‐ray of the painful area should be requested
  • If a fracture is obviously grossly displaced, if there is an obvious fracture dislocation (e.g. of the ankle) or if there is risk of skin damage from excessive pressure, reduction of the fracture should be performed before waiting for x-rays
    • X‐rays post reduction should still demonstrate any fractures adequately
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13
Q

How are undisplaced, minimally displaced and minimally angulated fractures managed?

A

considered to be stable are usually treated non‐operatively with a period of splintage or immobilization and then rehabilitation

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14
Q

How are displaced or angulated fractures managed?

A

may require open/closed reduction

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15
Q

How are unstable fractures managed?

A

treated with surgical stabilisation which may involve the use of small percutaneous pins (K‐wires) for small fragments, cerclage wires, screws, plates & screws, intramedullary nails or external fixation

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16
Q

How are unstable extra-articular fractures treated?

A
  • Unstable extra-articular diaphyseal fractures can be fixed with ORIF using plates and screws with the aim of anatomic reduction and rigid fixation leading to primary bone healing
  • In fractures with very swollen soft tissues, where blood supply to fracture site is tenuous (high energy), where ORIF may cause extensive blood loss (e.g. femoral shaft), or plate fixation may be prominent (e.g. tibia), ORIF tends to be avoided and closed reduction with IM nail fixation used instead to encourage secondary bone healing
  • Another option where ORIF is not suitable is external fixation which encourages secondary bone healing, however this carries the risk of pin site infection and loosening
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17
Q

How are displaced intra-articular fractures managed?

A
  • Require anatomic reduction and rigid fixation by way of ORIF using wires, screws and plates
  • Fractures involving a joint with predictable poor outcome may be treated with joint replacement or arthrodesis
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18
Q

How are fracture-dislocations managed?

A

Fractures can occur with dislocations (known as fracture‐dislocation) and these may reduce with closed reduction however ORIF may be required if reduction cannot be achieved, if a bony fragment prevents congruent reduction or if the joint is very unstable

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19
Q

What is the pathophysiology of open fractures?

A

Open fractures can either occur due to a spike of fractured bone puncturing the skin (‘inside-out’ injury) or due to laceration of the skin from tearing or penetrating injury (‘outside-in’ injury)
- The higher the energy of the injury, the amount of contamination, any delay in appropriate treatment and problems with wound closure increases the risk of infection
- The presence of a concomitant vascular injury raises the risk of amputation

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20
Q

What is Gustilo classification?

A

describes the degree of contamination, the size of the wound, whether the would would be able to be closed or require plastic surgery cover, and the presence of an associated vascular injury

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21
Q

What are the investigations of open fractures?

A

X-ray - AP and lateral views

22
Q

What is the immediate management of open fractures?

A
  • Direct pressure if bleeding
  • Reduce dislocation
  • Remove macroscopic debris
  • Photograph and cover with sterile or antiseptic-soaked dressing to prevent further contamination
  • Stabilise
  • Assess neurovascular status before and after reduction
23
Q

What is the prophylaxis management for open fractures?

A
  • Broad-spectrum antibiotics within 3 hours of injury
    • IV flucloxacillin for gram-positive cover
    • IV gentamicin for gram-negative cover
    • IV metronidazole to cover anaerobes if there is soil contamination
  • Tetanus vaccine/immunoglobin
    • If history unknown and uncontaminated, vaccinated >10 years ago (clean injury) or vaccinated >5 years ago (contaminated injury) → vaccine only
    • If contaminated and history unknown/ < 3 prior doses → vaccine and Ig
24
Q

What is the further ongoing management of open fractures?

A
  • Open fractures require fairly prompt surgery
  • Surgical removal of all contamination and excision of non-viable soft tissue - debridement
    • Dead/devitalized tissue can harbour infection as the immune system is unable to access the devascularised tissues
  • Stabilization with internal/external fixation
    • An unstable fracture may produce a haematoma - acts as a culture medium for bacteria and may cause additional necrosis
    • Delayed union more common due to high energy mechanism
    • Frequent wound inspections needed
  • Wound closure
    • If the wound is not grossly contaminated and all remaining skin and muscle is viable, and if the wound can be closed without undue tension on the skin edges, the wound can be closed primarily
      • Any wound tension may result in skin necrosis and wound breakdown
    • Any wound which cannot be closed primary requires either skin grafting, local flap coverage or even free flap coverage from plastic surgery
  • If there is doubt over the viability of the soft tissues or the wound is heavily contaminated, the wound is left open for 48 hours and then patient returned to theatre for further debridement and closure (secondarily or with plastic surgical flap coverage and/or skin grafting)
25
Q

What is compartment syndrome?

A

Increased pressure in the enclosed space of the compartments of the limbs, caused by swelling of tissue or increase in fluid, will affect the functions of the muscles and nerves in the compartment

26
Q

What are the risk factors of compartment syndrome?

A
  • Tibial fractures, especially male 10-35 years
  • Open fractures
  • Forearm fractures
  • IVDAs - comatose
  • Anticoagulation and trivial trauma
  • May not involve a fracture e.g. can occur after burns
27
Q

What is the pathophysiology of compartment syndrome?

A
  • Occurs anywhere in skeletal muscle surrounded by fascia, commonly occurs in anterior and deep posterior compartments of leg and volar compartment of forearm
    • 2-15% of patients develop CS following a tibial shaft fracture
  • Results from interstitial pressure increases in closed osseofascial compartments
  • The rising pressure can compress the venous system → congestion within the muscle and secondary ischaemia as oxygenated arterial blood cannot supply the congested muscle
  • Significant muscle damage at compartment pressures >30 mmHg-40 mmHg or within 10-30 mmHg of diastolic
  • If left untreated ischaemic muscle will necrose resulting in fibrotic contracture (Volkmann’s ischaemic contracture) and poor function
28
Q

What is the presentation of compartment syndrome?

A
  • 4 P’s - (disproportionate) pain, paraesthesia, pallor, pulselessness (late sign)
  • Tensely swollen limb, usually tender to touch
  • Pain on passive stretch of muscles in involved compartment
29
Q

What is the management of compartment syndrome?

A
  • If suspected CS, immediately release all dressings/cast to skin
  • Do not elevate
  • Surgery - emergency fasciotomy to relieve pressure
  • Following surgery, the open wound is left open for a few days before secondary closure
    • Many wounds require split skin grafting
30
Q

What is the pathophysiology of vascular injury with distal ischaemia?

A
  • Distal limb ischaemia risks subsequent amputation
  • Haemorrhage from arterial or venous injury may result in hypovolaemic shock
  • Penetrating injuries can result in transection of a vessel
  • Fractures/dislocations of different bones and joints can affect associated vasculature
    • Knee dislocation → popliteal artery injury
    • Paediatric supracondylar fracture of the elbow → brachial artery injury
    • Shoulder trauma → axillary artery injury
31
Q

What si the management of vascular injury with distal ischaemia?

A
  • Any signs of reduced distal circulation (reduced or absent pulses, pallor, delayed capillary refill, cold to touch) mandates urgent vascular surgery review and emergency surgical management
  • Urgent angiography in theatre may help localize the site of arterial occlusion
  • Temporary restoration of circulation can be achieved with use of a vascular shunt or vascular repair with either a bypass graft or endoluminal stent can be performed
  • Skeletal stabilization with internal or external fixation should be performed to protect the repair from shearing force
  • Ongoing haemorrhage from arterial injury in the pelvis can be controlled by angiographic embolization performed by interventional radiologists
32
Q

What is neuropraxia?

A
  • Occurs when the nerve has a temporary conduction defect from compression or stretch
  • Resolve over time with full recovery (can take up to 28 days)
33
Q

What is axonotmesis?

A
  • Occurs from either a sustained compression or stretch or from a higher degree of force
  • Although the nerve remains in continuity and the internal structure (endoneurial tubes) remain intact, the long nerve cell axons distal to the point of injury die in a process known as Wallerian degeneration
  • The axons then regenerate along the endoneurial tubes at a rate of 1mm per day
  • Longer peripheral nerves therefore take longer to recover
  • Recovery is variable and full power or sensation may not be achieved
  • Recovery can be predicted by nerve conduction studies from around a month from the time of injury
34
Q

What is neurotmesis?

A
  • Complete transection of a nerve - rare in closed injuries but can occur in penetrating injuries
  • No recovery will occur unless the affected nerve is surgically repaired
  • Recovery is variable
35
Q

What is the management of nerve injury in open fractures?

A
  • Nerve grafting
  • Tendon transfers
36
Q

How does skin breakdown occur in open fractures?

A
  • With higher energy injuries or with fragile skin (e.g. due to age, steroids, rheumatoid arthritis) an injury may jeopardise the viability of the overlying skin
  • A protruding spike of bone or tension on the skin from deformity can lead to devitalisation and necrosis with skin breakdown
  • In the situation where a fracture is causing excessive pressure on skin, as manifested by tenting of the skin and ‘blanching’, the fracture should be reduced as an emergency (under analgesia +/‐ sedation) to avoid subsequent necrosis
37
Q

What is de-gloving?

A
  • Avulsion of the skin from its underlying blood supply as a result of a shearing force on the skin
  • Can result in skin ischaemia and necrosis
  • The skin will not ‘blanch’ on pressure and may be insensate
  • Underlying haematoma may also increase pressure on the skin occluding capillaries
  • The de-gloved area may take a few days to declare itself (demarcate)
  • May require skin grafting or flap coverage
38
Q

How does swelling, bruising and blistering occur in open fractures?

A
  • The amount of soft tissue swelling and bruising (contusion) is usually a reflection of the energy of the injuries involved
  • Fracture blisters can occur due to inflammatory exudates causing lifting of the epidermis of the skin (like a burn)
  • A surgical wound through swollen and contused skin and soft tissues is not advisable as the wound may not be able to be closed which would leave a route for infection, or excessive tension on the wound may lead to necrosis and wound breakdown
39
Q

What are the signs of problems with fracture union?

A
  • Resolution of pain and function
  • Absence of point tenderness
  • No local oedema
  • Resolution of movement at fracture site
  • Ongoing pain
  • Ongoing oedema
  • Movement at the fracture site
40
Q

What are the investigations of fracture union problems?

A
  • Bridging callus may be seen on x-ray
  • CT scans can confirm or exclude bridging callus where their is doubt on bony union
41
Q

How does delayed union occur?

A
  • Different fractures heal at different rates, and healing is dependent on the energy of the injury as well as the age and overall health of the patient
  • The tibia is one of the slowest healing bones - fractures typically take around 16 weeks to heal and can take over a year to unit
  • Femoral shaft also typically take 3-4 months to heal
  • Metaphyseal fractures tend to heal more quickly than cortical fractures
  • A delayed union is a fracture that is not healed within the expected time
  • Infection can result in a delayed union
    • If infection is diagnosed, healing can occur if the infection is suppressed with antibiotics however the fracture may go on to an infected non-union
42
Q

How does non-union occur?

A
  • Hypertrophic non-union can occur due to instability and excessive motion at the fracture site
  • Atrophic non-union can occur due to rigid fixation with a fracture gap, lack of blood supply to the fracture site, chronic disease or soft tissue interposition
  • Infection can also lead to atrophic or hypertrophic non-union
  • Some fractures are particularly prone to problems with healing due to poor blood supply:
    • Scaphoid waist fractures
    • Fractures of the distal clavicle
    • Subtrochanteric fractures of the femur
    • Jone’s fracture of the fifth metatarsal
  • Some intra-articular fractures may not unite due to synovial fluid inhibiting healing if a fracture gap exists (intracapsular hip fracture, scaphoid fracture)
43
Q

What is malunion?

A

Fracture has healed in a non-anatomic position sufficient to cause pain, stiffness, loss of function and deformity

44
Q

What is post-traumatic arthritis?

A
  • Can occur due to intra-articular fracture, ligamentous instability or fracture malunion
  • Treatment may involve analgesia, bracing/splinting, arthrodesis or joint replacement
45
Q

What is chronic regional pain syndrome?

A

An exaggerated pain response after injury

46
Q

What is the aetiology of chronic regional pain syndrome?

A

It can be caused by a peripheral nerve injury (type 2) but often is not (type 1)

47
Q

What is the presentation of chronic regional pain syndrome?

A

Characteristics are variable but include constant burning or throbbing, sensitivity to stimuli not normally painful (allodynia) including cold or light touch, chronic swelling, stiffness, painful movement and skin colour changes

48
Q

What is the management of chronic regional pain syndrome?

A
  • Management is difficult and requires specialist pain services
  • Pharmacological agents including analgesics, antidepressants (amitriptyline), anticonvulsants (gabapentin) and steroids may help
  • TENS machines, physiotherapy, lidocaine patches and sympathetic nerve blocking injections may also help
49
Q

What is Volkmann’s Ischaemic Contracture?

A

Permanent contracture of forearm muscles resulting from missed compartment syndrome of the forearm

50
Q

What is the management of osteomyelitis or fixed fracture infection?

A
  • For infections present for longer than a few weeks, fracture healing may still be accomplished with antibiotic suppression but the metalwork will need later removal
  • If the infection cannot be suppressed and becomes too problematic (sepsis, discharging sinus), surgery is required with removal of all implants and debridement of infected bone
  • For medullary infection from an infected intra‐medullary nail, the medullary canal can be reamed out (cored out with a flexible drill) and a new nail implanted
  • Alternatively, an external fixator can be applied to give stability with the advantage of some systems of being able to compress the bone ends and gradually lengthen the bone if it has been substantially shortened
  • This would also be appropriate for infected non‐union of a plate and screw fixation
51
Q

What are the early systemic complications of a fracture?

A
  • Hypovolaemia
  • Fat embolism - respiratory failure, neurologic abnormalities and a petechial rash
  • ARDS
  • SIRS
  • Death
52
Q

What are the late systemic complications of a fracture?

A
  • DVT
    • Can occur particularly after a pelvic or major lower limb fractures within a period of immobility
    • Prophylaxis should be given (usually LMWH) to all ‘at-risk’ patients
    • Suspected DVT requires Duplex scanning and anticoagulation
  • PE
    • Tends to occur several days to weeks after injury but can occur much sooner (occasionally within a day)