Foundations Exam 2 Flashcards
components of the integumentary system include…
hair, skin, nails, sweat glands
body’s largest organ system
skin
functions of the skin
waterproof, protective barrier body temperature regulation vitamin D synthesis sensory perception non verbal communication & identity wound repair waste excretion
three main layers of the skin
epidermis (most superficial), dermis, subcutaneous tissue (deepest)
components of the epidermis
most superficial layer
thin (just a few cells thick)
avascular
two layers: outer (dead keratinized cells) and inner (cell layer where melanin, keratin formed)
components of dermis
vascular
connective tissue
sebaceous and sweat glands
hair follicles
components of subcutaneous tissue
adipose/fatty tissue
directly below dermis–loose later (inc. mobility over underlying structures)
fat for: energy, temp regulation, cushioning
4 pigments that contribute to normal skin colors
melanin, carotene, oxyhemoglobin, deoxyhemoglobin
melanin
brownish pigment
amount genetically determined
amount inc. with more sun exposure
carotene
golden yellow pigment
in subQ fat and heavily keratinized areas (palms, soles)
oxyhemoglobin
bright red pigment
predominates @ arteries, capillaries
blood flow through skin: more (reddening), less (pale pallor)
deoxyhemoglobin
darker bluish pigment
increase amount @ cutaneous vessels: bluish color (cyanosis)
abnormal skin pigments
jaundice, cyanosis
jaundice
yellow color pigment
bilirubin deposits @ skin
secondary to liver disease, biliary duct obstruction, and increased destruction of RBCs
first seen: hard/soft palate junction, sclera of eyes, then @ skin over rest of body
best way to assess jaundice?
in direct natural sunlight
where is jaundice first seen?
junction of hard and soft palate and sclera of eyes
cyanosis
bluish color pigment
nonspecific sign
peripheral (dec. cutaneous blood flow, normal arterial O2 levels–possibly normal response to cold, anxiety)
central (arterial O2 levels low)
hard to assess in people with darker pigmented skin
how do you identify decreased oxygenation in lighter vs darker pigmented persons?
lighter: cyanosis
darker: rely on clinical signs of dec. oxygenation @ brain
skin turgor is….. and it measures…
skin elasticity (ability to promptly return to place when released), hydration status.
sign of decreased skin turgor
tenting when pinched
causes of decreased skin turgor
age (thinning of dermis, reduced elastin)
dehydration
examination of skin, hair, nails is _________ throughout the rest of the comprehensive examination
integrated…not a separate step!
major components of skin assessment:
color, color change, temperature, moisture, thickness, texture, mobility/turgor, edema, lesions, pressure ulcer risks, skin cancer
skin color assessment
general pigmentation, skin tone even, consistent with genetic background, increase/loss of pigmentation, color change (pallor, redness, central/peripheral cyanosis, jaundice, carotene)
high levels of carotene will present as a _______ pigment in the _______ (areas of the body)
yellow color; palms, soles, face
how do you assess skin temperature?
use the backs of hands
check bilaterally
skin should be warm and equal bilaterally
how do you assess skin moisture?
look & touch
should be dry to touch without flaking or cracking
perspiration/ diaphoresis
check mucosal membranes for dehydration (usually smooth, moist)
normal skin texture
smooth and firm with an even surface
a callous forms in response to…
excessive pressure exerted on certain areas (palms, soles)
arterial insufficiency leads to what skin findings?
very thin, shiny (atrophic) skin
skin mobility
the ease with which it lifts
skin turgor
the speed with which it returns to place
where do you usually assess skin turgor?
anterior chest under clavicle (preferred)
back of the hand
pitting edema
fluid in interstitial spaces is displaced when pressure is applied to the surface area with a finger, leaving a depression or “pit”.
usually assessed along the shin
nonpitting edema
cause by plasma proteins “leaking” out of capillaries and into tissues, drawing water with them
see with burns, localized reaction (bee sting, for example), or trauma
scale for pitting edema + corresponding depression depth: 1+ 2+ 3+ 4+
1+ = 2mm (mild, disappears rapidly) 2+ = 4mm (moderate; disappears 10-15s) 3+ = 6mm (moderate-severe; lasts 1+min) 4+ = 8mm (severe; lasts 2+ min)
characteristics of lesions to note
anatomical location, distribution (localized, generalized), patterns, shapes, types, color, elevation
macule
flat, non-palpable skin color change (brown, white, tan, purple, red)
patch
flat, non-palpable skin color change (brown, white, tan, purple, red)
> 1 cm, may have irregular border
papule
elevated, palpable, solid mass with circumscribed border
plaques
elevated, palpable, solid mass with circumscribed border
> 0.5 cm
e.g. psoriasis, actinic keratosis
nodule
elevated, solid, palpable mass that extends deeper into the dermis that a papule
- 5-2 cm and circumscribed
e. g. lipoma, squamous cell carcinoma, poorly absorbed injection, dematofibroma
tumor
elevated, solid, palpable mass that extends deeper into the dermis that a papule
> 1- 2 cm and doesn’t always have sharp borders
e.g. larger lipomas, carcinoma
vesicle
circumscribed, elevated, palpable mass containing serous fluid
bulla
circumscribed, elevated, palpable mass containing serous fluid
> 0.5 cm
e.g. pemphigus, contact dermatitis, large burn blisters, poison ivy, bullous impetigo
wheal
elevated mass, transient borders, often irregular, size/color vary, caused by serous fluid moving into dermis, doesn’t contain free fluid
e.g. hives (urticaria), insect bites
pustule
pus-filled vesicle or bulla
e.g. acna, impetigo, furuncles, carbuncles
cyst
encapsulated, fluid-filled or semi-solid mass located in the subcutaneous tissue or dermis
e.g. sebaceous cyst or epidermoid cyst
primary vs. secondary skin lesions
primary skin lesions are original lesions arising from previously normal skin.
secondary skin lesions originate from primary lesions.
erosion
loss of superficial epidermis
does not extend to dermis
depressed, moist area
e.g. ruptured vesicle, scratch marks, aphthous ulcer
ulcer
skin loss extending past epidermis
necrotic tissue loss
bleeding, scarring possible
e.g. stasis ulcer (venous insufficiency), pressure ulcer
scar
(aka cicatrix)
skin mark left after healing of wound, lesion
injured tissue replaced by connective tissue
young = red, purple; mature = white, glistening
e.g. healed wound or surgical incision
fissure
linear cracks in skin
may extend to dermis
e.g. chapped lips or hands, athlete’s foot
skin and mobility: discuss
patients with decreased mobility are at an increased risk for pressure ulcers, skin breakdown. pressure because not moving + circulation problems.
stage 1 pressure ulcer
non-blanching redness
stage 2 pressure ulcer
involves epidermis + dermis
stage 3 pressure ulcer
involves subcutaneous tissue
stage 4 pressure ulcer
involves muscle, bone, tendons
assessment tool for determining risk for skin breakdown, ulcers?
braden scale. Categories: sensory perception, moisture, activity level, mobility, nutrition, friction/shear. Lower score (6) = higher risk. Higher score (23) = lower risk.
three major types of skin cancer
basal cell carcinoma, squamous cell carcinoma, melanoma
basal cell carcinoma
arises in the basal layer of epidermis
80% of skin cancers
appear pearly white, translucent. grow slowly and rarely metastasize. mostly in sun-exposed areas @ head, neck
squamous cell carcinoma
arise in upper later of epidermis
16% skin cancers
appear crusted, scaly, red, inflamed/ulcerated
can metastasize
melanoma
arise from melanocytes in epidermis (produce melanin)
4% skin cancers, but most lethal type (cause 75% of deaths)
can spread rapidly
ABCDEs of examining moles for melanoma
A: asymmetry
B: borders (irregular)
C: color variation/change (esp black, blue)
D: diameter 6+mm or diff from others
E: evolving (diff from rest, changing over time)
risk factors for melanoma
sun exposure (UV radiation) hx previous melanoma older than 50 years male 1-4 atypical moles light colored eyes, skin severe blistering as child (2nd degree)
most commonly recommended screening method for skin cancer
self-examination
skin cancer prevention strategies
avoid sun exposure(10a-2p), wear sun screen, wear long sleeves/pants/hat, regular self exam
what’s included in hair assessment?
hair color, quantity, distribution, texture, scalp, body, axillae, pubic hair
hair color
results from melanin production. graying occurs with reduction of melanin.
hair quantity
varies from person to person, decreases with age in both men and women. male patterned baldness = normal change. abnormal = patchy or uneven balding
hair texture
fine, thick, straight, curly, kinky
hair distribution
may be diminished in certain populations (asian). look for normal hair distribution @ genital region.
scalp assessment
look for lesions, flaking, parasites
body/axillae/pubic hair assessment
look for changes, unusual patterns, ask pt what is normal, what is different for them
loss of hair on legs is an indication of…
peripheral artery disease
hirsutism
excessive facial hair on women due to elevated testosterone levels
two types of hair
vellus: short, fine, covers body (“peach fuzz”)
terminal: coarser, thicker, conspicuous. scalp, armpits, eyebrows, pubic hair)
function and parts of fingernails
protect distal ends of fingers and toes, prevent infection.
nail plate, lanula, cuticle
how to assess nails?
inspect, palpate
look @ color, shape, any lesions
Beau’s lines
horizontal ridges
indicate acute illness
early & late clubbing
180 degrees + @ nail bed. indicates oxygen deficiency. More clubbing = more chronic hypoxia.
spoon nails
indicates iron deficiency anemia
pitting @ nails
indicative of psoriasis
paronychia
swelling proximal to cuticle. indicative of local infection.
sebaceous glands
oil glands that lubricate skin, hair
reduces water loss @ skin
sweat glands
2 types
eccrine: widely distributed, open directly to surface, body temp control
apocrine: axillary and genital areas, stimulated by emotional stress, body odor
describe the flow of blood @ heart, lungs, body
from body, low O2 blood travels through vena cavas into right atrium. from right atrium through the tricuspid valve and into right ventricle. from right ventricle through pulmonic valve to pulmonary arteries to lungs where blood is oxygenated and returned to left atrium via pulmonary veins. from left atrium through bicuspid/mitral valve to the left ventricle. from left ventricle through aortic valve to the aorta and out to body to deliver O2 and collect waste.
describe the pericardial sac/pericardium
a double walled sac containing the heart and roots of the great vessels. consists of two layers (serous and fibrous) with pericardial fluid between to prevent friction/rubbing.
if pericardial fluid is limited/diminished, what sound might you hear @ auscultation?
pericardial rub
upper chamber, lower chamber, and septum @ heart (describe)
upper: right and left atria, thinner walls
lower: right and left ventricle, thicker walls
septum: separates right and left sides
AV valves
tricuspid and mitral valves. closing of these creates the S1 heart sound.
semilunar valves
pulmonic and aortic valves. closing of these creates the S2 heart sound.
location of the heart
under the sternum between the 2nd and 5th intercostal space (ICS).
Aprox. 2nd ICS = base of heart
Aprox. 5th ICS = apex of heart
pericordium
area of the anterior chest over the heart and great vessels
detrocardia
heart is oriented the wrong way/ on the opposite side in the thorax. aka “flipped heart”
electrical activity @ heart
SA node (60-100) > AV node (40-60) > Bundle of His > Purkinje fibers