Forensics - Biological explanations Flashcards
atavistic form, genetic & neural + evaluation
Historical approach: Atavistic from
Lombroso - criminals are less evolved with innate features that indicate criminality in different ways
Lombroso’s research
- Examined features of around 4000 criminals & skulls of 400 dead criminals
- Shared common characteristics inc:
+ Pronounced jaw
+ Large ears
+ High cheekbones
Strengths of atavistic form
+ Scientific (uses theory of evolution & biological findings over morality -> coined term criminology & started further research)
+ Predict behaviours (interventions can be put into place to prevent criminality)
Limitations of atavistic form
- Racism (features were commonly attributed to people of African descent -> suggested they were criminals and boosted eugenic attitudes)
- Contradictory evidence (Goring 3k offender/non-offenders -> no evidence of distinct features)
- Methodological issues (no control group in Lombroso’s study -> issues with confounding variables)
Genetic explanation
Predisposition to crime inherited from genes from a family member (considers diathesis-stress model, adoption, twin and candidate genes)
Christiansen’s twin study (genetic)
- 3500 twin pairs with offending behaviour in Denmark born between 1880-1910
Findings (concordance rates):
MZ twins: 35% M, 21% F
DZ twins: 13% M, 8% F
Genes must be involved for MZ to have higher rates
Crowe’s adoption study (genetic)
Adoptees with biologically criminal mothers had a 50% of being criminals by 18 (compared to 5% in control group)
Genes linked to criminality (genetic)
MAOA -> warrior gene, linked to aggression, violence & impulsivity, breaks down neurotransmitters using enzymes i.e. dopamine & serotonin (MAOA-L -> unregulated neurotransmitters)
CDH13 -> links to substance abuse & ADHD, development & function of nervous system
Neural explanation
Behaviour is predisposed by the nervous system & brain structures -> anti-social personality disorder
Raine et al’s study of brain activity (neural)
PET scans of 41 murderers & control of 41 non-murderers
Findings: significant differences in certain areas
- Prefrontal cortex: lower activity = lower self control (impulsivity)
- Limbic system: amygdala lacks fear & hippocampus increased aggression (violence)
- Corpus callosum: problems with controlling/expressing emotions (regulation)
Increased risk of committing violence (predispositions)
Mirror neurons (neural)
React to actions & express empathy if there is an understanding of the intention (research into APD - sporadic empathy)
Strengths of genetic & neural explanations
+ Study supports
Genetic: Christiansen, Crowe, Mednicke
Neural: Kandel & Freed (frontal lobe damage = inability to learn from mistakes, impulsivity, emotional instability -> all predispositions)
+ Biological determinism (predictions are able to be made & create interventions [predictive validity])
Limitations of genetic & neural explanations
- [HOWEVER] Biological determinism (cannot be held responsible for their actions -> Mobley defense)
- Correlational (mutations on MAOA gene does not always guarantee criminality)
- Complex (Farrington et al -> high APD scoring individuals had other risk factors such as early experiences - not just biology at play)
- Issues with studies (twin/adoption - treated identically/later in life adoptions -> can genes be the only thing?)