FND Flashcards

1
Q

What is FND

A

Functional neurological disorders is a medical condition in which there is a problem with the functioning of the NS and how the brain/body sends and/or receives signals rather than a structural disease process. Conventional tests such as MRI brain scans and EEGs are usually normal in patients with FND.

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2
Q

Cause of FND

A

Many different predisposing factors can make patients more susceptible to FND such as having another neurological condition, experiencing chronic pain, fatigue or stress. However, some people with FND have none of these risk factors. At the time FND begins, studies have shown that there may be triggering factors like a physical injury, infectious illness, panic attack or migraine which can give someone the first experience of the symptoms. Psychological disorders and stressful life events, both recent and in childhood, may be risk factors for developing the condition in some patients

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3
Q

Diagnosis

A

There is emerging literature on structural imaging and functional MRI’s of the brain in people with conversion disorder. Studies have shown that those with conversion disorder tend to have reduced volumes of right and left basal ganglia and right thalamus compared to others without conversion disorder

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4
Q

DSM-V diagnostic criteria for FND

A

A. One or more symptoms or deficits affecting voluntary motor or sensory function suggest neurological or other general medical condition.
B. Physical findings provide evidence of incompatibility between the symptoms and recognized neurological or medical conditions.
C. The patient is not feigning or intentionally producing his or her symptoms or deficits.
D. The symptom or deficit causes clinically significant distress or impairment in the social, occupational, or other important areas of functioning or warrants medical evaluations

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5
Q

Hoovers sign- diagnostic test

A

involuntary extension of the unaffected leg occurs when flexing the contralateral leg against manual resistance. However, if the physiotherapist does not feel the unaffected leg’s heel pushing down as the patient flexes the hip of the affected limb, then this suggests functional weakness

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6
Q

S and S FND- Cardiac and gastrointestinal

A

Cardiac- SOB, palpitations, chest pain
Gastrointestinal= vomiting, abdominal pain, difficulty swallowing, nausea, bloating, diarrhoea

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7
Q

S and S FND- MSK and neuro

A

MSK- pain in legs, back/joint pain
Neurological- headaches, dizziness, amnesia, vision changes, paralysis

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8
Q

FND- Medical management- other

A

hypnosis, psychotherapy, pharmacotherapy, transcranial magnetic stimulation

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9
Q

physical therapy management- FND

A

The aim of physiotherapy is to prevent secondary complications, it may help the process of remission. Examples of treatment: for a patient presenting with paralysis or weakness, consider working on pre-gait activities such as sit-to-stands, weight-shifting in multiple planes, mini-squats, lunge matrices, balance activities, etc. Gait activities should include stair management, dual-task activities, adjustment of gait mechanics, etc. Next, it is important to incorporate activities that will help the patient with participation, especially with activities of daily living.§

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10
Q

What is complex regional pain syndrome

A

A term for a variety of clinical conditions characterised by chronic persistent pain and are subdivided into type I and type II. It is a condition that can develop after a limb trauma and appears mostly in one or more limbs.

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11
Q

CRPS- causes

A

Can affect any part of the body- occurs most in wrist post distal radial fracture. Central sensitisation is most common cause. Onset is mostly associated with trauma, immobilisation, injections, or surgery. No relationship between the severity of the injury and the following syndrome. A stressful life and other psychological factors may be potential risk factors,

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12
Q

Etiology

A

Sprains and strains, surgeries, fractures, contusions, crush injuries, nerve lesions, stroke.

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13
Q

CPRS- Clinical presentation- sensory disturbances

A

Allodynia and hyperalgesia, hypoesthesia and hypoalgesia, hypersensitivity of the skin

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14
Q

CPRS- Clinical presentation- autonomic disorders

A

swelling and oedema, changes of sweating, abnormal skin BF, colour/temp changes, swelling,

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15
Q

Clinical presentation- motor dysfunction

A

weakness of muscles, inability to move the extremity, stiffness, tremor, decreased ROM, severe impairment of complex movement, atropgy, inability to intiate movement, dystonia

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16
Q

CRPS- stage 1

A

Acute inflammation: denervation and sympathetic hypo-activity- 10 days to 3/6 months- pain more severe than expected/ oedema- soft and localised/ vasomotor changes- warmer

17
Q

CPRS- stage 2

A

stage 2 dystrophic paradoxic sympathetic hyperactivity- 3-6 months start and last 1 year- pain- constant pain, oedema=hard causing joint stiffness, vasomotor- no change, skin= thin, glossy and cool due to vasoconstriction

18
Q

CPRS- stage 3

A

atrophic- lasts for years- pain spreads proximally and occasionally entire body, may plateau, oedema- hardening, vasomotor- decreased SNS control

19
Q

Physio management

A

Aquatic therapy, mirror therapy, desensitisation, gradual WB, stretching, fine motor control, strengthening exercises

20
Q

Type 1 and 2 CPRS

A

CRPS is subdivided into 2 categories: type I and type II CRPS. In CRPS type I, there are no nerve injuries or lesions identified. CRPS type I is also known as “reflex sympathetic dystrophy,” and it comprises about 90 percent of all cases of CRPS. CRPS type II (causalgia), on the other hand, is diagnosed when there is evidence of nerve damage.