Fluid Balance Flashcards

1
Q

Homeostasis

A

how the body works to keep itself balanced

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2
Q

negative feedback

A

the body bringing itself back to normal
ie blood sugar goes up, insulin goes up, sugar goes down

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3
Q

positive feedback

A

causes further instability, like temperature going up when someone is hot instead of down

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4
Q

feed forward regulation

A

anticipatroy ques, like the body pumping the heart rate becuase it knows its going to be running

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5
Q

who is the most susceptible for fluid imbalances

A

the elderly, the very young, preoperative pateints

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6
Q

why are babies at risk for fluid imbalnaces

A

they have more water content than an adult (75%), have different skin, and cannot function independently when getting water

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7
Q

why are the elderly at risk for fluid imbalances

A

less water content-55%, they are already at a disadvantage if they lose water, less lean body mass
Decreased thirst mechanisms
increased drug/drug interaction

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8
Q

why are preoperative patients at risk for fluid imbalances

A

restrictions, blood/fluid loss, surgery stress

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9
Q

what is the best measure of water loss

A

body weight

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10
Q

intracellular fluid compartments

A

inside cells, holds about 2/3rd of body water and 40% of body weight

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11
Q

extracellular fluid compartments

A

area outside cells- like vascular space or interstitial space
20% of body weight

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12
Q

Interstitial space

A

where cells ar flowing, the 3rd space

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13
Q

difference between interstitial and plasma fluid

A

nearly identical, plasma just has more protein

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14
Q

electrolytes def

A

substances whose molecules split into iones when placed in water
ion: electrically charged
cations: positively charged
anions: negatively charged

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15
Q

ECF primary electrolyte

A

sodium

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16
Q

ICf primary electrolyte

A

potassium

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17
Q

Simple diffusion

A

particles become widely dispesed and reach union concentration
moving from areas of high concentration to lower until net flux is equal

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18
Q

facilitated diffusion

A

moving of bigger things that may need a transport protein, like glucose needing insulin
Does not require ATP

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19
Q

Active Transport

A

uses ATP to move molecule against a concentration gradient, from low to high concentration
like sodium potassium pump

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20
Q

what is hydrostatic pressure

A

the pressure pushing water out of the cell, against the capillary
the BP generated by heart contraction

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21
Q

Oncotic pressure

A

pressure that keeps thing in the cell

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22
Q

capillary hydrostatic pressure

A

movesd water out of the capillaries

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23
Q

plasma oncotic pressure

A

moves fluid into the capillaries

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24
Q

interstitial hydrostatic pressure

A

moves fluid out of interstitial space

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25
Q

interstitial oncotic pressurew

A

moves water out of the capillaries

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26
Q

hydrostatic/ oncotic pressure on vascular system

A

in atrial end capillary hydrostatic pressure exceed oncotic pressures so fluid can move into tissue
when it gets to the venous end, oncotic pressure exceed oncotic pressure so fluid flows back into capillary

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27
Q

what can happen with an increase in venous hydrostatic pressure

A

edema- inhibits fluid movement back in the capillary
fluid overload, heart failure, liver failure, other venous problems

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28
Q

Osmosis

A

across a semipermeable water will go to the area with higer concretation

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29
Q

osmotic pressure

A

amount of pressure required to stop osmotic flow of water

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30
Q

osmolarity

A

comes from plasma in vascular space
millimoles/L of solution

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31
Q

osmalality

A

measure the number of milliosmoles/kg of water

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32
Q

what does osmalitly over 295 mean

A

there is greater particles and less water, they are dehydrated and
fluid surrounding cells is hypertonic

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33
Q

what does osmolality of less than 275 mean

A

more, less particles
fluid overload
fluid surrounding cells is hypotonic

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34
Q

hypertonic

A

fluid with more concentrated solutes than within
will cause cell to shrink as fluid will travel to area with more concentrate

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35
Q

hypotonic

A

solution in which their is less solute concentration than inside the cells
will cause cells to swell as fluid flows to area of higher concentration, inside the cell

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36
Q

isotonic fluid

A

balanced fluid, will cause fluid shift

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37
Q

first spacing

A

normal distribution of fluid in ICF and ECF compartment

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38
Q

second spacing

A

abnormal accumulation of interstitial fluid

39
Q

third spacing

A

excess fluid collects in nonfunctional area between cells
causes ascites, edema

40
Q

insensible water loss

A

invisible vaporization from lungs and skin
is only water loss
regulates body temp

41
Q

sensible

A

excess sweating
w/ fever, excercise, high temps
loss of water and electrolytes

42
Q

If a patient has greatly increased capillary hydrostatic pressure that exceeds the pressure exerted by capillary colloid osmotic pressure, where will the fluid move into?

A

interstitial space
The interstitial space. Hydrostatic pressure pushes fluid out of vascular space, osmotic pressure pulls fluid into vessels. If hydro > osmotic = fluid pushed out into interstitial space.

43
Q

The health care provider orders an I.V. solution of 5% dextrose in 0.45% (D5.45) sodium chloride solution for a post-operative patient. What category of fluid is this solution?

A

Hypertonic
When a patient is under stress, they retain fluid. Therefore we give hypertonic fluid to shift fluids out of the cells and into the vascular space so it can be excreted through the kidneys.

44
Q

During administration of a hypertonic IV fluid solution, the mechanism involved in equalizing the fluid concentration between ECF and the cells is

A

osmosis
While osmosis and diffusion is similar, diffusion is the movement of particles to create a union concentration, osmosis is the movement of water to reach the union concentration.

45
Q

Dextrose 5% in 0.45% NaCl (D5.45)

A

Hypertonic

46
Q

0.9% NaCl

47
Q

Dextrose 5% in Water (D5W)

48
Q

0.45% NaCl

49
Q

Lactated Ringer’s (LR)

50
Q

Dextrose 5% in 0.9% NaCl (D5.9)

A

Hypertonic

51
Q

3% sodium

A

Hypertonic

52
Q

Dextrose 5% in Lactated Ringer’s (D5LR)

A

Hypertonic

53
Q

How does the hypothalamus regulate fluid balance

A

works as the thirst center: BOfy fluit deficcit or an increase in plasma osmlality activateds receptors in the hypothalamus that stimulate thirst and release of ADH from posterior pituitary gland

54
Q

How does ADH work with fluid balance

A

acts on distaal tubules of kidney sby making them more water permeable, allowing increased water reabsorption from tubular filtrate into the blood and decreased excretion into the urine

55
Q

What factors infleunce ADH secretion/thirst

A

decreased BP, nausea, pain, hypoglycemia, hypoxemia stimulate ADH release

56
Q

effect of surgery on ADH

A

stress response, analgesics, anathesia ewual ADH release and decreased osmolality

57
Q

aldosterone with fluid balance

A

holds on too sodium, allows retaining of fluid
released by adrenal cortex

58
Q

REnal fluid balance regulation

A

helps maintain normal plasma osmolality, electrolyte balance, blood volume, and acid-base balance
if impaired: edema, potassium/phosphate retention, acidosis, electrolyte imbalances

59
Q

ANP/ BNP

A

atrial natriuretic peptide and B-type natriuretic peptide
Cardia peptides, promote excretion of Sodium and water, decreasing BLood volume/BP

60
Q

Cardiac system with fluid balance

A

increased atrial pressure= high serum sodium levels= increase ANP/BNP to allows water to be gotten rid of
why with heart disease/kidney failure, water retention

61
Q

GI Regulation of Fluid balance

A

Intake/output
Diarrhea, vomiting preventing GI reabsorption of secreted fluid cna cause significant fluid/electrolyte loss

62
Q

Lymphatic system regulation of fluid balance

A

normally drain of excess fluid at ends of venous capillaries, but if removed fluid not drained as easily

63
Q

what does the body do when dehydrated

A

you are hyperosmolar
pituitary stimulates antidiuretic to hold on to flui and lower plasma osmolality
REnal perfusion decreases and hormones renin, angiotensin and aldosterone stimulate/increased

64
Q

Extracellular Fluid volume deficit (ECFVD)

A

Dehydration, loss of fluid from vascular space

65
Q

what can cause extracellular fluid volume deficit

A

Diabetes insipidus, vomiting/diarrhea, NG suction, diuretics, hemorrhage, hyperglycemia/ ketoacidosis, osmotic diuresis, insensible water losses like fever, heatstroke, burns

66
Q

Manifestations of ECFVD

A

sluggish cap refill, dry mucus membranes, skin tenting, orthostatics, thready pulse, tachycardia, decreased blood pressure, weight loss, tried, dizzy, lightheaded, constipation, increased respiratory rates, decreased urine output with more concentrated urine

67
Q

Labs with ECFVD

A

osmolality above 295
plasma sodium above 145
blood urea nitrogen above 25
hematocrit above 55
urine specific gravity above 1.030

68
Q

ECFVD intervention

A

I/Os, daily weights, ORtho BP
LAbs: BUN/CR, HRt, urine specific gravity
Fluid per md order: Oral or IV
REduce risk of falls, skin integrity problems

69
Q

What fluid are good bad for PO rehydration

A

good: water, decaf herbal teas
Bad: Coffee, any caffeine, sugary sodas

70
Q

Isotonic fluids used for

A

Filling vascular space, like .9 normal saline, lactated ringers
Give first with dehydration+low BP

71
Q

Hypotonic fluid used for

A

shifting from vascular space into cells
.45, D5W (IV tap water)
Lower BP, so use after Iso fluids w/ severe dehydration

72
Q

Hypertonic fluid used for

A

Shifting water from cells to vascular space
Dehydrating cells with fluid overload/excess
D545

73
Q

What fluid should be used with ECFVD

A

Isotonic first to fill vascular space and increase BP, then hypotonic to shift fluid into cells

74
Q

Intracellular fluid volume deficit (ICFVD)

A

Cells shrink
Rare, but may be in older adults with acute water loss
hyperosmolar in vascular space, water is pulled from the cells

75
Q

Manifestations of ICFVD

A

Thirst, oliguria, CNS changes from effect on neural cells

76
Q

ICFVD management/intervention

A

hypotonic fluid to push fluid back into cells if BP is ok
address underlying cause

77
Q

Extracellular fluid volume excess: intravascular hypervolermia

A

Fluid overload, failure to excrete, increased total body sodium
increased hydrostatic pressure and decreased oncotic pressure

78
Q

causes of ECFVE (intravascular)

A

long term corticosteroid use
cushing syndrome
heart/renal disease/failure
primary polydipsia
SIADH- syndrome of inappropriate antidiuretic hormone secretion
stress
organ failure

79
Q

Manifestations of ECFVE (intravascular)

A

Bounding pulse, increased BP, pulmonary and peripheral overload crackles, edema
Bloated, swollen, SOB, fatigue
low urine output, pale cold extremities

80
Q

Lab indicators of ECFVE

A

Osmolality < 275
sodium <135
hematocrit < 45%
specific gravity of urine < 1.010
blood urea nitrogen <8

81
Q

ECFVE intervention/management

A

Monitoring skin, daily wieghts, I/Os, fluid restriction
Oxygenation and monitoring of crackles
Elevate legs and mobilize fluid
Promote Urinary w/ diuretic like furosemide, HCTZ, Spironolactone

82
Q

K-wasting diuretics

A

Increase urination and potassium loss, Furosemide, HCTZ

83
Q

k-sparing diuretics

A

increase urination without promoting potassium loss
spironolactone

84
Q

ECFVE: third spacing

A

tissue injury or protein malnutrition leading to fluid shift
increasedf cap permeability, decreased serum protein/albumin levels
obstrcted lymphatic pressure
increased capillary hydrostatic pressure

85
Q

ECFVE 3rd manifestations

A

Weak pulse, hypotension, pallor, oliguria, Decreased LOc, elevated BUN, hematocrit, urine specfic gravity, edema, ascites

86
Q

ECFVE 3rd management/interventions

A

weight, vitals I/os, skin intergity
interstitial to vascular with hypertonic, diuretics to get it out

87
Q

Intracellular fluid volume excess (ICFVE

A

Water intoxication
water excess or solute deficit
can cause brain cell swelling
intracellular shift of water soward sodium inside cells

88
Q

ICFVE manifestations

A

increased intracranial pressure, altered LOC
hemodilution: plasma sodium< 125, decreased hematocrit

88
Q

ICFVE management/interventions

A

safety (ie seizure, fall risk) fluid restriction, sodium administration, intervention to prevent further increase in ICP ( stool softeners, antiemetics)

88
Q

The patient is admitted to a nursing unit from a long-term care facility with a hematocrit of 58% and a serum sodium level of 152mEq/L. Which condition is a cause for these findings?

A

Dehydration
Dehydration = hemoconcentration. More solutes and less fluid would manifest in elevated hematocrit and sodium levels.

88
Q

A patient is admitted with shortness of breath, bibasilar crackles on auscultation, and the B/P is 150/90 mmHg. The nurse suspects fluid volume overload. Which laboratory result would be consistent with this nursing diagnosis?

Serum osmolality - 320 mOsm/L

Blood urea nitrogen (BUN) – 32

Serum sodium - 130 mEq/L

Serum potassium – 4.0 mEq/L

A

Serum sodium
Fluid excess = hemodilution. Less solutes and more fluid would manifest in decreased sodium levels.

89
Q

A patient comes to the clinic reporting frequent, watery stools and dizziness for the past 2 days. Which action should the nurse take first?
Obtain baseline weight

Check the patient’s blood pressure

Draw blood to check serum electrolyte levels

Ask about extremity numbness and tingling

A

Check blood pressure
Patient is at risk for dehydration and is exhibiting signs of fluid deficiency (dizziness). Always assess first, and vitals are vital! In this case, dizziness comes from loss of fluid which leads to vascular depletion and low BP.