Fluid and electrolytes 1 Flashcards
Hypotonic solution is to
Dilute ECF and rehydrate cells of hypertonic fluid imbalances.
Hypotonic solution
< 250 mEq/L
Solutions of hypotonic
D5W, 2.5% dextrose in water
Hypotonic solutions do what to cells
Cells swell
Do not give hypotonic solution to
Hypotension pt., infants, or patients with head injury
Isotonic solutions normal range is
250-375
Lactated Ringer’s and 0.9% NS is
Isotonic solutions
Isotonic solutions are for
Fluid rewsuscitation, keep vein open, dilute mess, expand volume
Hypertonic solutions is greater than
375
D10W, D5LR, and D51/2NS are all
Hypertonic solutions
Hypertonic solutions do what to cells
Shrink
Na is for
Volume replacement of hypertonic solutions
D51/2NS is fo
Severe hyponatremia and cerebral edema
Hypertonic solutions have to be infused
Slow
Be aware for checking patient that is given hypertonic solutions
BP,HR, lung sounds, urine output
Hyponatremia is
Low sodium in the cells to where water shifts for ECF to ICF
Euvolemic hyponatremia
Water increases, Na+ levels stays same
No edema
Na+ is diluted due to increase H2O levels
Hyponatremia causes
SIADH (syndrome of inappropriate antidiuretic hormone), DI ()diabetes insipidus), adrenal insufficency
Hyponatremia
Serum Na+ < 135mEq/L
Hyponatremia results from
Excess of water or loss of Na+
In Hyponatremia, water shifts from
ECF into cells
Hyponatremia you will have
No edema
Hypervolemia hypernatremia
Na+ and h20 levels increase in the body. Fluid volume overload.
Hypervolemia hypernatremia
Na+ and h2o levels are regulated differently and independently of each other in the human body
Causes of hypervolemia hypernatremia
CHF, kidney failure, excessive infusion of saline solution or liver failure.
Sodium-Na+ Range is
135-145 mEq/L
Is sodium a major cation or anion
Cation
Chief electrolyte of extra cellular fluid is
Na+
Cation is a positive or negative charge
Postive
Sodium regulates
Volume of body fluids by maintaining osmotic pressure
Sodium is needed for
Nerve impulse and muscle fiber transmission (Na/K pump)
Major muscle fiber that would be affected by sodium
Heart muscle
Sodium is regulated by
Kidneys/hormones
Sodium is
Outside of the cell in the ECF
Most common electrolyte disturbances is
Hyper and Hyponatremia
Hyper and Hyponatremia is
Most abundant in extracellular fluid and therefore more prone to fluctuation
Osmosis
Movement of water from an area of lesser to one of greater concentration through a semi-permeable membrane
Diffusion
Passive movement of electrolytes or other particles down the concentration gradient (from higher to lower concentration)
Filtration
Movement across a membrane, under pressure from a higher to lower pressure
Filcilatated diffusion
Carrier from higher to lower
Active transport
Metabolic energy is expended, movement from less concerntated solution to more concentrated one
Osmotic pressure
An inward-pulling force caused by particles in the interstitial and intracellular fluids
Hydrostatics pressure
The major force that pushes water out of the vascular system at the capillary level and into interstitial fluid
Extracellular fluid
Found outside the cells and accounts for about 1/3 of total body fluid
Intracellular fluid
Found within the cells of the body
Intravascular fluid
Plasma, accounts for approx 20% of the ECF
Interstitial fluid
Surrounds the cells, 75% of the ECF
Cation
Positively charged ion
Anion
Negatively charged ion
Causes of hyponatremia
“NO NA+”
“No NA+” N is
N-Na+ excretion increased with renal problems, NG suctioning, DI, aldosterone secretion, diuretics, sweating
“NO NA+” O is
Overload of fluids-CHF, hypotonic IVF, liver failure, will dilute sodiumm
“NO NA+” N is
Low intake of Na+, low Na+ diet, NPO, elderly
NO NA+” A is
Antidiuretic hormone over secreted
Signs and symptoms of Hyponatremia
“SALT LOSS”
” SALT LOSS” STANDS FOR
S-seizures and stupor
A-abdominal complaint, attitude change (confusion)
L-lethargic
T-tendon reflexes diminished, trouble concentration
L-loss of urine, appetite
O-orthostatic hypotension, overactive bowel sounds
S-shallow breathing late sign (shallow breathing, skeletal muscle weakness)
S-spasm of muscles
Nursing interventions for hyponaterima
Monitor cardiac, respiratory, neuro, renal and GI systems
Hypovolemic hyponaterima
Give IVF to restore balance of fluids and sodium (hypertonic solution; 3%), given too fast fluid volume overload.
Hypervolemia hyponatremia
Restrict fluids, possibly diuretics, renal failure-dialysis
SIADH
Restrict fluids, antidiuretic hormone antagonist; declomycin-not given with food especially dairy. Also conserves lithium when sodium levels are increased.
For sodium loss
Instruct patients to consume sodium rich foods. (Canned foods, bacon, table salt, processed foods)
What are some medical conditions that may cause a dilution all hyponatremia
Heart disease, renal disease, adrenal insufficiency
What are some conditions that might cause actual loss of sodium from the body
GI losses- nasogastric suctioning, vomiting, diarrhea
Certain diuretic therapies
Polydipsia
Permanent neurological damage can occur when serum Na levels fall below 120 mEq/L. Why?
Hypotonic environment swells cells, increasing ICP-brain damage
Hypernatremia Serum level Na+
> 145mEq/L
Hypernatremia results from
Na+ gained in excess of water Or Water is lost in excess of Na+
Hypernatremia water shifts from
Cells to ECF
S/S: hyperosmolity of ECF
Cellular dehydration, thirst dry mucous membranes and lips Oliguyria Increased temp and pulse Flushed skin Restlessness
Treatment of hypernatremia
IV therapy; hypotonic or isotonic solution
Diet
Causes of hypernatremia- HIGH SALT
H-hypercortisolism overproduction of cortisol (Cushings disease and hyperventilation)
I-increased salt (IV and oral)
G-GI tube feedings without adequate water supplements
H-hypertonic solutions (3% saline)
S-sodium excretion decrease (ex. Corticosteroids)
A-aldosterone problems (^ reabsorption)
L-loss of fluid (dehydration) fever, seweating
T- thirst impairment (access to clean water)
S/S of hyernatremia-No “FRIED” foods for you
F-fever, flushed skin R-restless, really agitated I-increased fluid retention E-edema, extremely confused D-decreased urine output, dry mouth and skin
Nursing interventions for hypernatremia
Restrict Na+
Safety
MD order hypotonic or isotonic IVF (0.45% Na+)-give slowly (hydrating cells)-many cause cerebral edema
Educated patient about diet and S/S of increase sodium levels.
Potassium K+ serum level
3.5-5.0 mEq/L
Chief electrolyte of intracellular fluid is
Potassium K+
Potassium is the major mineral in
All cellular fluids
Potassium AIDS in
Muscle contraction,
nerve and electrical impulse conduction,
regulates enzyme activity,
Regulates intracellular fluid H2O content
Assists in acid-bade balance
Potassium is regulated by
Kidneys/hormones
Potassium is inversely proportional to
Na+- Sodium, too much or to little is deadly
Hypokalemia serum level
<3.5 mEq/L
Hypokalemia results from
Decreased intake, loss via GI/Renal and potassium depleting diuretics f
Hypokalemia is life threatening to
All body systems affected
S/S of Hypokalemia
Muscle weakness and leg cramps
Decreased GI motility
Cardiac Arrhythmias
Treatment of Hypokalemia is
Diet
Supplements
IV therapy-
Causes of hypokalemia “your body is trying to “DITCH potassium”
D-drugs (laxatives, diuretics, corticosteroids)
I-inadequate so sumptuous of Potassium (NPO, anorexia)
T-too much water intake (dilutes the potassium)
C-cushing’s syndrome (high secretion of aldosterone)
H-heavy fluid loss (NG suction, vomiting, diarrhea, wound drainage, sweating)
-(other causes: when the potassium moves from the extracellular to the intracellular with alkalosis or hyperinsulinism (this is where too much insulin in the blood and the patient will have symptoms of hypoglycemia).
7 L’s for hypokalemia
- LETHARGY (confusion)
- LOW, SHALLOW RESPIRATIONS (due to decreased ability to use accessory muscles for breathing)
- LETHAL CARDIAC dysrhythmias
- LOTS of urine
- LEG cramps
- LIMP muscles
- LOW BP and Heart
Nursing interventions for hypokalemia
Watch heart rhythm (place on cardiac monitor…most already on telemetry), respiratory status, Nero, GI, urinary output and renal status (BUN and creatinine levels)
> watch other electrolytes like Magnesium (will also decrease…hard to get K+ to increase if Mag is low), watch glucose, sodium, and calcium all go hand-in-hand and play a role in cell transport
lad minister oral supplements for potassium with doctor’s order: usually for levels 2.5-3.5…give with food can cause GI upset
>IV potassium for levels less 2.5*****NEVER GIVE POTASSIUM VIA IV PUSH OR BY IM OR SUBQ ROUTES!!!!!!!!!!!
>Give according to the bag instruction don’t increase the rate…has to be given SLOW…patients given more than 10-20 mEq/HR should be on a cardiac monitor and monitored for EKG changes
lac use phlebitis or infiltrations
>don’t give LASIX, demanded, or thiazides (waste more Potassium) or Digoxin (cause digoxin toxicity) if potassium level low…notify MD for further orders)
>physician will switch patient to a potassium sparing diuretic Dpironolactone (Aldactone), Dyazide, Maxide, Triamterene.
For hypokalemia instruct patient to eat
"Potassium" rich foods P-POTATOES, PORK O-ORANGES T-TOMATOES A-AVOCADOS S-STRAWBERRIES S-SPINACH I-fIsh U-mUSHROOMS M-MUSK MELONS: CANTALOUPE A-ALSO INCLUDED ARE: CARROTS, RAISINS, BANANAS
Potassium is dangerous when
It is too low! Serum K+ < 3.5mEq/L
What are some medical conditions that may cause hypokalemia
Renal disease,
Heart failure
Metabolic alkalosis
What are some conditions that might cause actual loss of potasaasium from the body
GI losses-nasogastric suctioning, vomiting, diarrhea
Certain diuretic therapies
Cardiac arrest may occur when serum K levels fall below 2.5 mEq/L. Why?
Increased cardiac muscle irritability leads to PACs and PVCs, then AF
Hyperkalemia serum level
> 5 mEq/L
Hyperkalemia results from
Excessive intake, Trauma Crush injuries Burns Renal failure Adrenal insufficiency Acidosis
S/S of hyperkalemia
Muscle weakness
Cardiac changes
N/V
Paresthesia of face/fingers/tongue
You treat hyperkalemia with
Diet
Med’s
IV therapy
Possible dialysis
Causes of hyperkalemia: the body “CARED” too much about potassium
C-cellular movement of potassium from intracellular to extracellular (burns, tissue damages, acidosis)
A- adrenal insufficiency with Addison’s Disease
R-renal failure
E-excessive potassium intake
D-drugs (potassium-sparing drugs like Aldactone (spiraled acetone), Triamterene, ACE inhibitors, NSAIDS (good at retaining)
S/S: “MURDER”
M-muscle weakness
U-urine production little or none (renal failure)
R-respiratory failure (due to the decreased ability to use breathing muscles or seizures develop)
D-decreased cardiac contractile the (weak pulse, low blood pressure)
E-early signs of muscle twitches/cramps…late profound weakness, flaccid
R-rhythm changes: Tall peaked T waves, flat p waves, widened QRS and prolonged PR interval
Hyperkalemia interventions;
Monitor cardiac, respiratory, neuromuscular, renal, and GI status > stop IV potassium if running and hold any PO potassium supplements > initiate potassium restricted diet and remember foods that are high in potassium; "POTASSIUM" -potatoes, pork Oranges Tomatoes Avocados Strawberries Spinach Fish Mushrooms Musk melons: cantaloupe --Also carrots, raisins, bananas
For hyperkalemia instruct patient not to eat
Potassium rich foods, “POTASSIUM-A”
SIGNS of hyperkalemia
Muscle twitches-> cramps-> paresthesia Irritability and anxiety Decreased BP EKG changes Dysrhythmias-irregular rhythm Abdominal cramping Diarrhea
What are some medical conditions that may cause hyperkalemia
Renal disease
Burns trauma
Metabolic acidosis
What are some conditions that might cause potassium levels to rise in the body
Certain diuretics
Excessive intake
Cardiac arrest may occur when serum K levels rise above? MEq/L. Why?
Decreased electrical impulse conduction leads to bradycardia and eventual a systole.
When administering IV potassium:
Monitor the IV sites for phlebitis
Place on cardiac monitor if > 10 mEq
Assure of adequate mixing of K in solution
Monitor for elevated K levels
Monitor for decreased Na levels
NEVER-administer potassium by slow IV push method
Calcium Ca+ total serum levels:
9-11 mg/deciliter, ionized calcium (in serum unbound) 4.25 to 5.25 mg/dL.
Calcium is most abundant
In the body but: 98-99% in teeth and bones
Calcium is needed for
Nerve transmission
Vitamin B12 absorption
Muscle contraction and blood clotting
Calcium has a inverse relationship with
Phosphorus
Calcium is needed for Vitamin D for
Gut
Calcium is needed for magnesium for
Bones
Hypocalcemia serum Ca level
< 9 mg/dL
Hypo alchemical results from
Low intake, Blood transfusions Loop diuretics Parathyroid disorders (decreased PTH) Hypoalbuminemia
S/S: of hypocalcemia
Osteomalacia EKG changes Numbness/tingling in fingers, Muscle cramps/tetany Seizures Chovstek sign and Trousseau Sigh