Finals Flashcards
Associated with congenital heart disease and pierre robin syndrome
Congenital micrognathia
Postnatal in origin
Acquired micrognathia
Severe retrusion of the chin
Mandibular micrognathia
Hereditary condition and only occurs in mandible
Pierre robin syndrome or anomalad
Triad of PRS
Micrognathia
Cleft palate
Glossoptosis
Continuous enlargement of the jaw particularly of the maxilla
Paget’s or osteitis deformans
Form of gigantism associated with hyperpituitarism
Acromegaly
Enlargement of tongue
Macroglossia
Overbite and overjet is zero
Edge to edge
Heterotopic collection of sebaceous gland seen as yellow spots
Fordyce’s granules
Malformation of the lip associated with oral cleft
Congenital and commisural pits and fistula
Fold of excess tissue on the inner mucosal aspect of the lip
Double lip
Frenal fibers are attached up to mucogingival junction
Mucosal frenal attachment
Fibers are inserted within attached gingiva
Gingival frenum attachment
Frenal fibers accross the alveolar process and extend up to palatine papilla
Papilla penetrating frenal attachment
Indication for removal of frenal attachment
Closure of midline diastema
Restrict lip movement
Causes speech problems
Causes gingiva recessions
How to correct midline diastema
Initial closure by orthodontic treatment
Remove the frenum
After eruption of permanent canine
Frenectomy should be delayed until after alignment and space
Scalloping of the tongue
Crenated tongue
Tongue tied or fusion of the to tongue the floor of the mouth
Ankyloglossia
Small furrows and grooves prone to candidiasis, reddish in color and devoid form of filiform papillae
Fissured tongue
Red patch with white keratotic border aka geographic tongue or wandering rash
Benign migratory glossitis
Elongated filiform papillae
Hairy tongue
Exciting factors of hairy tongue
Candida albicans
Systemic factors of hairy tongue
Anemia
Gastric ulcers
Initiating factors of hairy tongue
Antibiotic
Corticosteroid
Oxygenating mouth rinse
Smoker
Radiation therapy
Rhomboid reddish patch due to failure of tuberculum impar to retract
Median rhomboid glossitis
Occurs the lesion above the waist
HSV 1
Occurs lesion below waist, genitalia
HSV 2
Why the virus is neurotropic
Infects the peripheral nerve
Migrates to regional ganglion
Remain dormant
Undetected by immune system
Activation result in migration along the nerve axon
Precipitating factors that suppressed the immune system
Emotional stress
Trauma
Cold
Fever
Patient’s saliva and secretion are highly contagious
Initial stage or vesicular
Rapture of the vesicle
Ulcer
After the ulcer heal
Scab or crust
Lesion is healed and dormant again
Complete resolution
Incubation period of HSV
1-2 weeks
First time has the lesion and occur in children
Primary herpetic gingivostomatitis
Multiple small, punctuate and shallow ulcers
Mild form of primary herpetic gingivostomatitis
Large, diffuse, whitish ulcers with scallaped border
Severe form
Fever and lymphadenopathy, muscle soreness and inability to eat that last 2-10 days
Systemic manifestation
Burning sensation or pain
Recurrent oral herpes simplex
Fluid filled and resolve as crusted brownish lesion
Recurrent herpes labialis
Wet and fragile mucosa, punctuate with red and white lesion
Recurrent intraoral herpes
Mothers experiencing primariy HSV infection
Neonatal HSV
Pustular surrounded by a wide sone of erythema
Herpetic whitlow
Inhalation of droplets, painful, pruritic vesicles
Varicella
Stages of varicella
Vesicle, ulcer and crust
Unilateral multiple ulcers, second part of varicella
Herpes zoster or shingles
Occurs in children has fever and sore throat caused by coxsackie virus
Herpangina
Highly contagious caused by paramyxovirus
Measles
Has fever, respiratory symtomps and rash caused by togavirus
German measles
Has petechiae of the soft palate caused by beta hemolytic streptococcus
Pharyngitis and tonsilitis
Acute pustular eruption on perioral skin caused by streptococcus pyogenes and staphylococcus aureus
Impetigo
Caused by myobacterium tuberculae spread by airborne droplets
Tubercolosis
Chronic bacterial disease and has swelling of the mandible caused by actinomyces israeli
Actinomycosis
Painfuo elevated plaques that can be scraped off
Candidiasis
Fungal colonies on inflammed mucosa
Acute candidiasis
Sore mouth
Denture stomatitis
Form of hyperkeratosis
Hyperplastic
Co-infected with staphylococcus aureus
Perleche
Continuous denture wearing day and night
Inflammatory papillary hyperplasia
Pinpoint hyperemia
Type 1 IPH
Diffuse erythema
Type 2 IPH
Exophilic red and nodular
Type 3 IPH
Surgical approach of IPH
Mucoabrasion
Electrosurgery
Causes of white lesion
Thick surface layer of keratin
Acanthotic epithelium
Edematou epithelial cells
Exudates and adherent surface debris
What are the epithelial alterations
Hyperorthokeratosis
Hyperparakeratosis
Acanthosis
What are the changes in connective tissue
Decrease in vascularity
Increase in collagen content
Execessive thickened layer of the stratum corneum
Hyperkeratosis
Keratin layer without residual
Hyperorthokeratosis
Keratin layer with residual
Hyperparakeratosis
Thickened layer of the spinous layer of squamous epith
Acanthosis
Produce white lesion that cannot be easily removed by rubbing
Leukoplakia
Viral in nature and caused by epstein-barr virus
Hairy leukoplakia
Intracellular accumulation of fluid within spinous layer
Leukoedema
Thick and spongy consistency
White sponge nevus
Caused by pipe and cigar and has red dots surounded by white elevated keratotic ring
Nicotine stomatitis
Cause is unknown and may be autoimmune disease
Lichen planus
Most common oral form of lichen planus
Reticular form
Painful form
Erosive
Rare form
Atropic,papular and plaque types
Cheek and lip chewing and has keratotic surface
Morsication buccarum
Frequent and prolonged contact with spices
Oral submucous fibrosis
Caused by carcinoma in situ and epithelial dysplasia and a clinical term for red patch
Erythyoplakia
Bright red, soft with straight scalloped
Homogenous form
Soft red lesion with irregular outline
Speckled erythroplakia
Vitamin C deficiency
Scurvitic gingivitis
Anticonvulsant
Dilantin sodium
Antihypertensive
Nifedipine
Immunosuppresant
Cyclosporin
Other name of ANUG
Vincent’s infection or trench mouth
2 types of suprabony
Gingival or pseudo pocket
Periodontal or true pocket
Base is still the same and above bone level
Gingival or pseudo pocket
Apical migration of the base and has horizontal bone loss
Periodontal pocket or true pocket
Deepening of the sulcus and has vertical bone loss
Infrabony and infracrestal pocket
Oral manifestation of skin disease in gingivosis
Pemphigus
Intense redness and desquamation of the surface epitheliuk
Gingivosis or
Chronic desquamative gingivitis
Rapid loss of alveolar bone about more than 1 tooth
Juvenile periodontitis
Rapidly aggresive periondontitis
Periondontosis
Proliferation of fibrous connective tissue
Peripheral fibroma
Consisting of reactive hyperplasia with coalesced calcified structure
Peripheral ossifying fibroma
Excessive proliferation of granulation tissue from calculus
Pyogenic granuloma
Granuloma gravidarum
Extraosseous nodule composed of mononuclear and mononucleated giant cells. 0.5 to 1.5mm
Peripheral giant cell
Proliferation of granulation tissue from extraction
Epulis granulomatosum
Enlargment of lips
Macrocheila
Form of fibrous dysplasia and enlargement of maxilla
Leontiasis ossea
Non healing ulcer just like oral TB
Gumma
Disease of treponema pallidum
Syphilis and congenital syphilis
What stage is gumma or glossitis
Tertiary
Maculopapular rash
Secondary
Triad of hutchinson
Dental anomalies , mulberry
8th sense deafness
Interstitial keratitis
Burst of of blood veseels caused by eipstein barr virus
Petechiae
Bleeding larger in size
Ecchymoses
Nevus in basement membrane
Abtropfung effect