finalllllll Flashcards

1
Q

Somatic symptom disorder/anxiety illness disorder

A

Excessive thoughts, feelings, or behaviors related to somatic symptoms including: persistent thoughts about the seriousness of the symptoms, persistent anxiety about the symptoms, excessive time and energy focused on the symptoms.

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2
Q

How many months must somatic symptom disorder be present for?

A

at least 6 months

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3
Q

Comordities of somatic somatic/anxiety illness

A

depression, anxiety, panic disorder “DAP”

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4
Q

Somatic/illness implication for function

A

Mild to moderate in most cases, work deficits with absenteeism, distraction as a result of worry, may be cognitive and emotional regulation deficits, and issue with self identity and self image

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5
Q

Conversion disorder

A

one or more symptoms of altered motor function (weakness, paralyzed, slurred speech, swallowing issues) or sensory function (visual, olfactory, auditory)

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6
Q

Psychological factors affecting medical conditions

of somatic/illness

A
  • presence of a medical symptom or condition other than a mental disorder.
  • Psychological or behavioral factors affect the medical condition: affect the course of the medical condition, interfere with treatment of the medical condition, increase health risk for the person, and influence the pathophysiology, cause, exacerbation of the disorder or the need for medical treatment.
  • Diagnostic category used for situations where medical condition (cancer) is associated with depression or anxiety.
  • Need to address psychological symptoms to improve medical outcomes and quality of life.
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7
Q

Somatic/illness lifespan considerations

A
  • n children, somatic symptoms may be expression of anxiety
  • Children with somatic symptoms are likely to have dysfunctional educational experiences (school absence) and involved in the juvenile justice and welfare systems
  • In later life, somatic symptoms often express anxiety or depression
  • May present in later life with some degree of cognitive confusion
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8
Q

Pica - diagnostic criteria

A

persistently eating non food items

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9
Q

to be diagnosed with pica, u have to show symptoms for at least?

A

1 month

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10
Q

Pica Comorbitities (Alvina Ingests Socks)

A

autism, intellectual and schizophrenia

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11
Q

Pica OT implications

A
Self-care associated with cooking/eating always affected
Physical health (malnutrition, intestinal blockage, toxin), cognitive function and processing
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12
Q

Ot behavioral intervention

A

Establish strategies with positive direction/ outcome
Focus on providing other occupations, meal preparation
Environmental modifications

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13
Q

Anorexia Nervosa DC

A

Restriction of caloric intake leading to significant low body weight
Intense fear of gaining weight or being fat
Disturbance in body image, excessive concern about body weight or shape, or lack of recognition of the seriousness of the current low body weight

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14
Q

Comorbidities Anorexia

A

depression and anxiety

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15
Q

Anorexia treatment

A

Medical management to address nutritional deficits
Psychodynamic therapy- control issues
CBT
Nontraditional antipsychotic medications- address dopamine dysfunction
Combination probably most effective

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16
Q

Anorexia OT implication

A

Occupational engagement
Therapeutic alliance- a cooperative working relationship between client and therapist; goal setting
Focus on acceptable leisure pursuits that de-emphasize food
Social skills training
Stress management; Self-expression opportunity
Support and education for caregivers and families

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17
Q

Bulemia DC

A

Repeated episodes of binge eating
Inappropriate mechanisms for compensating for overeating (vomiting, laxatives)
Binging and purging
Self-image is not excessively influenced by bodyweight

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18
Q

Bulemia DC how long

A

At least once a week for 3 months

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19
Q

DCT (dialetctical behavior therapy) treatment _____ and used in _____

A

focuses on awareness of problems and choices, mood regulation techniques, and coping skills”
Invalidating environment:

bulemia

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20
Q

Invalidating enviroment

A
  • bulemia
  • “its tendency to respond inappropriately to private experiences independent of the validity of the actual behavior”
  • The environments create a sense of uncertainty, unpredictability, or hostility
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21
Q

Bulemia OT implications

A

Coping skills training focused on mood regulation, managing social situations, and interaction with the environment
Practice avoiding triggering situations
Meaningful occupations that reduce urge to binge (e.g. yoga)

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22
Q

Binge Disorder DC

A

Repeated episodes of binge eating: in a limited time, eats an amount much larger than is considered normal; lacks control over eating during each episode
The episodes are associated with: eating too quickly, eating until uncomfortable, eating large amounts when not hungry, eating alone because of guilt or depression, feeling disgusted with oneself, distress about binge eating.
Not associated with anorexia or bulimia

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23
Q

Binge Disorder DC Length

A

Average once a week for at least 3 months

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24
Q

Binge Disorder treatment

A

Integrative response therapy, a group-based guided self-help treatment focused on affect regulation.
Cognitive behavioral therapy with accompanying medication
Medication

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25
Q

Binge Implications for OT

A

Wellness efforts focused on changing behaviors
Emphasis on healthy occupations that address eating habits
Health promotion/prevention activities with children

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26
Q

Eating Disorders lifespan considerations

A

Pica and rumination disorder occur primarily in children
Anorexia and bulimia most often emerge in adolescence. Consider family therapy
For older adults, physiological regulation of appetite changes
Changes in neurotransmitter production often cause decrease in appetite in older adults which can lead to malnutrition
Anorexia of aging is qualitatively different from that of younger adults because it Is not typically associated with conscious choice

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27
Q

Encropese DC

A

once a month 3 months, 4 years

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28
Q

Insomnia DC

A

Dissatisfaction with quality or amount of sleep:
Difficulty falling asleep
Difficulty maintaining sleep (frequent waking)
Early morning waking
*At least three nights a week For at least three months
In spite of adequate opportunities for sleep
Distress or dysfunction

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29
Q

Insomnia Comorbidities

A

Anxiety, Trauma, Depression

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30
Q

Narcolepsy DC

A

Repeated intense need to sleep, falling asleep, or napping within the same day.
At least three times per week for at least three months
At least one of:
Cataplexy at least a few times per month:
Brief episodes of sudden loss of muscle tone triggered by sudden, strong emotions- laughing/ joking, fear, anger or excitement while remaining conscious [a cat that has low tone]
Spontaneous grimaces or global hypotonia
Hypocretin deficiency- regulate sleep and energy
Rapid eye movement (REM) sleep less than 15 minutes

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31
Q

Obstructive Sleep Apnea DC

A

One of the following:
Polysomnography of at least 5 obstructive apneas or hypopneas per hour of sleep with:
Nighttime breathing disturbances like snoring or breathing pauses during sleep
Daytime sleepiness or fatigue in spite of adequate opportunities for sleep
Or 15 or more episodes of apnea (no breathing @ all) or hypopneas (abnormally slow breaths) per hour of sleep without other symptoms

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32
Q

Obstructive Sleep Apnea Etiology

A
Facial structure (shape of jaw, nasal septum) & amount of upper airway soft tissues
Overweight or obesity
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33
Q

Circadian Rythym Disorder DC

A

Sleep disruption due to alteration of circadian system or a mismatch between the person’s rhythm and the requirements of the social or work environment
Excessive sleepiness or insomnia or both
Distress or dysfunction

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34
Q

Circardian Rythym Disorder Etiology

A

Degeneration or decreased neuronal activity of suprachiasmatic nucleus neurons (melatonin)
Decreased responsiveness of the body’s internal clock to signals such as light and activity
Decreased exposure to bright light and structured social and physical activity during the day
These may occur as a result of shift work or jet lag

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35
Q

Sleep Cycle Stage 1

A

Entering sleep, light (1-7 minutes)

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36
Q

Sleep Cycle Stage 2

A

Light sleep, heartbeat and breathing starts to slow down, muscles start to relax (10-60 minutes)

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37
Q

Sleep Cycle Stage 3

A

Deep sleep, more slow and relaxed (20-40 minutes)

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38
Q

Sleep Cycle Stage 4

A

REM- Increase brain activity, increase heart rate & BP, dreams, muscles paralyzed (10-60 minutes)

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39
Q

Nightmare Disorder

A

Frequent troubling dreams that are well-remembered

40
Q

Non-rapid eye movement disorder

A

Sleep walking, sleep terrors

41
Q

Rapid eye movement sleep behavior disorder

A

Repeated periods of arousal during sleep with vocalization and/or complex motor behaviors, occurring during REM sleep
Ex: Think of it as a next level nightmare disorder. You are legit physically reacting to your nightmare. “Moaning or screaming during nightmare”

42
Q

Restless Leg Syndrome

A

Frequent urge to move the legs, with uncomfortable or unpleasant sensations during sleep, frequently in REM

43
Q

Sleep Disorders Implication for Function

A

Performance may be affected in work, leisure, play, education, social participation due to daytime fatigue or excessive sleepiness
Skills affected include cognition (executive function, concentration, attention span), emotional regulation
Self esteem and confidence

44
Q

Sleep Disorders Treatment

A

Behavioral strategies with focus on sleep hygiene
Short or long term medication use
Continuous Positive Airway Pressure (CPAP) machine (only for obstructive sleep apnea)
Surgical options for obstructive sleep apnea
Treat medical conditions

45
Q

Sleep Disorders Implication for OT

A

FOCUS ON SLEEP HYGIENE
Establishing a calm bedtime routine
Using bed only for sleep and sex
Avoiding naps if possible, one nap only at 30 min
Avoiding vigorous exercise too close to bedtime
Minimize or avoid screen time 1 hour before bed
Make sure bedroom is dark and comfortable temperature for sleep
Minimize liquid in evening
Daily exercise (safely)
Mindfulness meditation
Body positioning for comfort- restless leg
Managing and routinely using CPAP
Work simplification/energy conservation to address fatigue until sleep improves
Childhood narcolepsy- parent education
Family education for safety- sleep walking, acting out dreams

46
Q

Strategies to improve circadian rhythm

A

Sunlight exposure in morning

Going to sleep and getting up at the same time

47
Q

AOTA Fact Sheet

A

*Cognitive or behavioral therapy interventions, or strategies to address sensory avoiding or sensory seeking behaviors

48
Q

Older Adults in Long Term Care Sleep

A

Daytime activity programs, including exercise, foster socialization and facilitate arousal, engagement, and decreased involuntary daytime napping.

49
Q

Genito-Pelvic Pain/ Penetration Disorder DC

A

Recurrent difficulties with:
Vaginal penetration during sex
Pain during vaginal intercourse
Anxiety about pelvic pain
Tensing of the pelvic floor in anticipation of pain
**Symptoms persist for at least six months
Symptoms cause distress

50
Q

Genito-Pelvic Pain/ Penetration Disorder Etiology

A

Psychological: Anxiety (anticipation of pain), stress, and tension
Physical: Abdominal abnormalities
Mixed: Childbirth- inadequate healing following childbirth; anxiety about resuming sexual relations

51
Q

Genito-Pelvic Pain/ Penetration Disorder Treatment

A

Treatment of any underlying medical conditions
Behavioral interventions
Estrogen for peri- or post-menopausal women

52
Q

Delayed Ejaculation Disorder DC

A

One or both on most occasions when intercourse is attempted:
Significant delay in ejaculation
Absence of ejaculation
At least six months
Symptoms are distressing to the individual

53
Q

Delayed Ejacualtion Disorder Etiology

A

Physiological/ physical factors- illness, injury, drug side effects, and lifestyle
Psychological factors- distress, anxiety, depression relationship distress
Combined

54
Q

Gender Dysphoria DC

A

Incongruence between self-perceived gender and assigned gender, for at least six months, with at least two of:
Incongruence between experienced gender and sex characteristics
Desire to be rid of sex characteristics because of this incongruence
Wish to have the sex characteristics of the other gender
Wish to be and to be treated as the other gender
Has romantic and sexual feelings or reactions of the other gender
Symptoms are associated with distress

55
Q

Gender Dysphoria Etiology

A

Not well understood, but almost certainly biological- complicated genetic and hormonal events.

56
Q

Gender Dysphoria Prognosis

A

Variable
Depends on the degree of support in his/her social network
Individual must decide what course of action to take, gender reassignment being the most extreme but in some cases, offering the best outcome

57
Q

Gender Dysphoria IFF

A

Affects most performance areas due to psychological distress, particularly social occupation
Self-esteem, self-identity, role
May affect academic for school-aged individuals
May affect emotional regulation, cognition

58
Q

Gender Dysphoria Treatment

A

Psychotherapy

Gender reassignment with accompanying therapy to assist in adaptation to new gender

59
Q

Gender Dysphoria Lifespan Considerations

A

Cultural differences in gender roles and acceptable sexual expression
Cultural differences in willingness to seek help
Level of acceptance and support network
Gender dysphoria often emerges in childhood or adolescence
Potential for victimization, bullying
Social isolation

60
Q

Gender Dysphoria OT Implications

A

Address issues of self-concept, self-esteem, social interaction
Identify performance areas that are particular strengths and assist to enact them
Provide opportunities for emotional expression through non-verbal mechanisms (e.g. creative arts)
For paraphilias, redirect to socially acceptable occupations
For gender dysphoria, assist in adjustment to new gender role
None of these is well-documented in the OT literature
Trauma informed approach notes
Experience/trauma of LGBT youth

61
Q

Trauma Informed Approach

A

systematic approach to ensure youth to prepare to respond to trauma in school organization

62
Q

Oppositional defiant disorder (ODD) DC

A

A pattern of angry or irritable mood with argumentative, defiant, or vindictive behavior, lasting at least six months, with:
Angry/irritable mood
Often loses temper, is easily annoyed and/or is angry or resentful
Argumentative/defiant behavior
Often argues with authority figures
Defies or refuses to comply with rules or requests
Blames others for his misbehavior
Vindictiveness
Has been spiteful or vindictive at least twice in six months
Persistence and frequency of these behaviors is markedly greater than normal behavior and occurs with others who are not related
Symptoms are associated with distress in the individual or in others

63
Q

ODD differential diagosis and comorbidities: angry demo

A

ADHD

Developmental disorders

64
Q

ODD Etiology

A

Genetic/biological
Reduced cortisol reactivity to stress
Reduced amygdala reactivity to negative stimuli
Altered serotonin and noradrenaline neurotransmission
Abnormalities in the amygdala and frontal cortex
Environmental
Prenatal maternal cigarette smoking, alcohol use, or viral illness
Maternal stress and anxiety
Low birthweight
Early neonatal complications
Parental stress
Dysfunctional parenting (can be the reason, but not in most cases)
Early deprivation or adoption all are implicated in development of the disorder

65
Q

ODD prognosis

A

Strong predictor of adult antisocial personality disorder
Strong predictor of conduct disorder and depression
Worse prognosis if early onset
More severe ODD predicts personality disorder

66
Q

ODD Treatment

A

nterventions focused on social learning
Psycho therapies including cognitive-behavioral therapy, parent training and family therapy
Collaborative problem solving has shown some modest success
Develop skills in problem solving, flexibility, frustration tolerance

67
Q

ODD imlications for OT

A

Implications for OT
Parent training- understand behavior & social learning
Behavioral interventions emphasizing engagement in meaningful and acceptable forms of activity, with emphasis on improved social awareness and skill
Teach problem solving, decision making; train frustration tolerance and attention
Clear expectations and consequences are essential
Consistent with classroom rules, house rules

68
Q

Conduct Disorder DC

A

Diagnostic Criteria
Persistent pattern of behavior with serious violation of the rights of others or rules of conduct:
Aggression to people and animals
Bullies, threatens or intimidates others and/or initiates physical fights
Has used a weapon that can cause serious harm
Physical cruelty to people or animals
Destruction of property by fire or in other ways
Deceitfulness or theft
Has broken into someone else’s house or car, has stolen
Lies to obtain something or avoid obligations
Stays out late despite parental rules (younger than 13)
Is often truant from school
Symptoms cause dysfunction
At least 3 episodes in one year, with symptoms in past six months

69
Q

Conduct Disorder Comorbidities (SAIL)

A

Substance use disorders “SAIL”
ADHD
Intellectual disabilities
Learning disabilities

70
Q

Conduct Disorder Etiology

A

Subtle neurological deficits like poor verbal abilities and inhibitory control
Gray matter volume reductions in the areas that process socioemotional stimuli
Family histories of antisocial behavior and either harsh or inconsistent parenting - controversial
Adolescent form less severe

71
Q

Conduct Disorder Prognosis

A

*Poor with associated alcohol or substance abuse
*High probability of developing adult antisocial or borderline personality disorders and encountering financial and legal difficulties
Early onset of the behavior predicts a worse outcome, including a high potential for violence and criminal behavior over time
Worst with callous/unemotional traits

72
Q

Conduct Disorder IFF

A

Negatively affects school, social, and work performance
Difficulty in areas of interpersonal skills, emotional regulation
Difficulty transitioning to adult roles- work & family life
Can contribute to legal difficulties which further affect performance

73
Q

Conduct Disorder IFOT

A

Channel energy to more appropriate activities
Reinforcement for acceptable behavior
Consistent expectations
Engagement in recreation- provide experience of success
Opportunities for appropriate expression of emotion – movement, music, art, play; build self-concept
Anger management, coping strategies
Parent training for the above
Treatment of ADHD and other co-existing conditio

74
Q

Opioids DC

A

Problematic pattern of use with significant impairment or distress, with at least two of the following:
Taken in larger amounts or over a longer time than intended
Desire or unsuccessful efforts to control use
Significant time spent obtaining, using, or recovering from use
Craving or urge to use
Recurrent use interferes with major role obligations
Recurrent use leads to social or personal problems
Important activities given up because of use
Use in situations that are physically hazardous (e.g. driving)
Use continues despite knowledge of problem
Tolerance
Withdrawal- hallucinations, anorexia, depression, insomnia, vomiting

75
Q

Opioids Prognosis

A

*Respiratory suppression with overdose,

poor outcome as logn as 20 years after diagnosis

76
Q

CAGE

A

Cut down
Annoyed
Guilty
Eye-opener

77
Q

CELLPS

A
Complex Attention
Executive Function
Learning and memory 
Langauge 
Perceptual motor 
social cognition
78
Q

Major vs Mild cognitive disorders

A

Decreased memory may have difficulty with certain IADL’s but no problem with ADLS, trouble with money management, writing a check but no problem taking care of self: MILD
Deficits do not interfere with independence in daily activities, but greater effort, compensatory strategies or accommodations may be required
No basic self care, warm up food, dress themselves, trouble remembering names fam members, etc.: MAJOR
Deficits interfere with independence in daily activities
Alzheimers

79
Q

Alzheimers Etiology

A

Combination of genetic and environmental factors
Genetic: *apolipoprotein “PAPOU” is the most important genetic risk factor
Excessive amounts of two proteins undergo synaptic dysfunction, oxidative stress, loss of calcium regulation, and inflammation
Sociodemographic factors include educational level and physical fitness also factors
Most significant risk factor identified to date is *aging

80
Q

Alzheimers DC

A

Evidence of a causative genetic mutation
Evidence of decline in memory and learning
Progressive, graduate decline in cognition
No evidence of other or mixed etiology
Behavioral symptoms: agitation, aggressiveness, sundowning (confusion when sun is going down)
Not a diagnostic criterion, often develop visual processing issues

81
Q

Parkinsons

A

Motor symptoms: hand-resting tremors, pill-rolling, tremors at wrist, shuffling gait, bradykinesia, hypokinesia, akinesia, kyphotic posture, speech and swallowing problems
Can cause Neurocognitive Disorder although symptoms do not appear in every case
NCD symptoms can include
Mood. sleep and autonomic function changes
Impairments in cognition and perception

82
Q

Parkinsons Etiology

A

unclear

83
Q

Huntingtons

A

Autosomal dominant affecting HTT gene
Results in *chorea (jerky and involuntary movements), behavioral disturbances, and dementia/NCD
Diagnostic criteria
Onset is insidious and progression is gradual
Based on family history or genetic testing
HD before age of 40 with a young family
Difficult to manage bc autosomal dominant
50% chance one has a gene if parent is carrier
50% chance of passing to offspring

84
Q

Vascular Disease

A

Characteristics: lesions affect cortical regions important for memory, cognition and behavior
**Often found in conjunction with alzheimers and other causes of dementia making it difficult to classify
Risk factors: high BP (blood pressure), vascular disease, late-life depression

85
Q

VD Etiology

A

One or more cerebrovascular events

Notable deterioration of complex attention and frontal-executive function

86
Q

TBI

A

Unpredictable because depends on area of cortex
Pre-injury function is important
Inflammation and white matter damage can persist for years even after a single event

87
Q

TBI DC

A
one or more of the following:
Loss of consciousness 
Posttraumatic amnesia 
Disorientation
Neurological signs
Chronic effect of TBI can possibly lead to other NCDs
88
Q

NCD IFOT

A

*Compensatory strategies - external memory aids
Caregiver support
Occupation-based program to maximize procedural memory
Just-right challenge - activity that is just slightly above what a person is currently able to easily do. It is an activity the person is able to do, but it requires a little bit of a stretch.
Activities tailored to their abilities and interests
Activities that client enjoyed prior to cognitive deficits

89
Q

Cluster A

A
Cluster A: characterized by odd or peculiar behavior
Schizotypal 
Mild form schizophrenia
May not hallucinations or delusions
If they do its milder and more insight 
Not as frequent/severe
Most severe 
self-centered
90
Q

CLuster B

A

flamboyant or dramatic behavior
antisocial, borderline
Characterized by emotional instability, disruptive and erratic interpersonal relationships, restricted affect, and lack of empathy and insight
Thought to be deficient or abusive parenting
Evidence for inherited component
Borderline PD
Unstable interpersonal relationships for at least 3 years at 18
Failure to conform to lawful behavior; deceitfulness
Self-image problems - uncertainty
Inappropriate affect - anger/impulsivity
Efforts to avoid abandonment
suicidal or self-harm
Depression, bipolar, must be ruled out
Comorbidities

91
Q

CLuster B comorbidites

A

Depression
PTSD
Substance abuse

92
Q

Cluster C

A

characterized primarily by anxiety or fear
Avoidant, dependent, OCD
Highly correlated with major depressive disorder and anxiety disorders
Theories: attachment difficulties, traumatic life events, psychosocial stress and dysfunction
Prognosis

93
Q

Personality Disorders Etiology

A

Genetic factors: serotonin system & stress reactions
Deficits in function of prefrontal cortex & structures related to emotion and impulse control
Structural and functional deficits especially in limbic and paralimbic brain areas, and the cognitive-executive brain regions
Parental influences (anhedonia)
Childhood trauma
Exaggerated defense mechanisms
Avoidant, borderline, histrionic, paranoid, schizoid, and schizotypal strongly comorbid depression

94
Q

Personality Disorders Comorbidity

A

Major depression, anxiety disorder, substance use disorders: worse outcome

95
Q

Personality Disorder OFOT

A

Underlying issues:
Inaccurate perceptions of self and others
Inadequate social skills
Poorly developed personal values and goals
Poor self-esteem
Participate in team:
Psychotherapy
Group therapy
Behavioral approaches
CBT
Medications - evidence is weak
Group/cooperative activities:
Planning social event: social skills training
Interpreting accurately what other people say
Developing empathy, consistent, clear, non judgemental feedback from therapist and other group members

96
Q

PD IFOT Cluster A, B, C

A
Cluster A 
Sensory integrative interventions
Cluster B
Behavioral approach
Occupational engagement 
Sports, self-care/meal preparation 
Self appraisal, build self-esteem
Behavior modification
Cluster C
Social skills training