Final Exam Study Guide Flashcards

1
Q

What are Semantic Paraphasias?

A

Semantically related word is substituted

e.g., for for spoon, sweater for jacket

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2
Q

What are phonemic paraphasias?

A

Sound errors; sound tranpositions in words (e.g., shooshbruss for toothbrush)

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3
Q

What are neologisms?

A

made up words (e.g., dubeid for shoe)

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4
Q

What is jargon?

A

a string of neologisms into a phrase/sentence (e.g., emo donned oh yah beige)

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5
Q

What is anomia?

A

word-finding difficulties, the inability to name objects or to recognize the written names of objects

very common in the aphasias

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6
Q

What are agrammatic utterances?

A

Also known as telegraphic speech. Speech in which content words are used but most function words (articles, prepositions, conjunctions) are missing

A common characteristic of speech for adults with Broca’s Aphasia

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7
Q

What are paragrammatic utterances?

A

grammatical errors-such as wrong tense markers

misuse of pronouns among range of syntactical constructions

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8
Q

What is tangential speech?

A

the speaker wanders and shows a lack of focus, never returning to the initial topic of conversation

often associated with RHS & wernicke’s

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9
Q

What is disorientation?

A

loss of one’s sense of direction, position, or relationship with one’s surroundings

a temporary or permanent state of confusion regarding place, time, or personal identity

an early sign of dementia

geographic and topographical disorientation common in RHS

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10
Q

What is resource allocation?

A

a model of cognitive processing in which mental operations depend on allocatation of processing resources from a limited-capacity pool. Performance deteriorates if the demands for processing resources exceeds the capacity of the pool.

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11
Q

What is ALOC?

A

Altered level of consciousness

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12
Q

What is disinhibition?

A

A lack of restraint manifested in disregard for social convention, impulsivity and poor risk assessment.

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13
Q

What is anosognosia

A

denial of illness (common in RHS)

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14
Q

What is prosopagnosia?

A

inability to recognize faces (common in RHS)

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15
Q

Memory deficits**

A

not sure what to do with that one.. see study guide-Cathy’s portion.

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16
Q

What is Confabulation?

A

s/s of RHS

the production of fabricated, distorted, or misinterpreted memories about oneself or the world without the conscious intention to deceive

person is unaware that the information is false, and are generally very confident about their account of things. It is a memory disturbance

client imagines things bigger than they are

Creating fictitious accounts of past events, believing they are true, to cover a gap in memory

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17
Q

What is circumlocution

A

S/s of dementia & aphasia

intentionally speaking around the target word due to inability to retrieve word; common in context of anomia

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18
Q

What is verbosity?

A

s/s for RHS

Wordiness
Using more words than needed

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19
Q

what are unmodifiable risk factors for stroke?

A

Age, every decade after 55, the risk doubles

Gender, male > female

Race/ethnicity

Family history, genetic tendency + common lifestyle

Previous CVA / TIA

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20
Q

What are modifiable risk factors for stroke?

A

Hypertension (HBP)

Heart Disease (atrial fibrillation)

Diabetes

Atherosclerosis

Blood Cholesterol Levels

High Alcohol consumption

Head and Neck Injuries

Infections (viral and
bacterial infections)

Cigarette Smoking

“Stroke Belt” in U.S

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21
Q

What are some things you can do for stroke prevention?

A

Hypertension – Take Rx as ordered

Identify atrial fibrillation (Afib)–abnormal heartbeat

Stop smoking

Control alcohol use

Treat high cholesterol - > 200

Control/manage diabetes

Exercise

Diet

Treat circulation problems

Recognize/treat TIA (up to 40% of people who experience a TIA may have a stroke)

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22
Q

What are the types of strokes?

A

Ischemic

hemorrhagic

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23
Q

What are the types of ischemic strokes?

A

›80% of strokes are of this type

Large artery
atherothrombotic

›Small artery or lacunar stroke

›Embolic stroke

Dissection stroke

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24
Q

What are predisposing factors to ischemic strokes?

A
›Male
›
Age
›
Mortality rate is about 10% for people under 65 years of age following a stroke.
›
The mortality rate increases with age.
›
Family history of stroke
›Medical conditions such as:
›
hypertension
›
atherosclerosis
›
myocardial infarction
›
diabetes
›
prior transient ischemic attacks (TIAs)
›
smoking
›
excessive alcohol consumption
›
birth control pills
›
obesity

a sedentary life style

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25
Q

What do ischemic strokes generally result from?

A

a thrombus or embolism

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26
Q

What is a thrombus?

A

A collection of fat and blood from the diseased artery that narrows it and blocks the flow of blood going to the brain, causing ischemia. A thrombus is stationary.

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27
Q

What is an embolism?

A

A blood clot containing arterial debris that travels to a smaller artery and “gets stuck”. The blood flow to that area of the brain is now blocked, causing ischemia.

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28
Q

Describe hemorrhagic strokes

A

›Hemorrhagic events comprise 20% of strokes and are identified by location and subtype

›Approximately 20% of all strokes are hemorrhagic.

›The mortality rate for this type is about 50%

if you survive, better outcomes

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29
Q

What are the subtypes of hemorrhagic strokes?

A

›Intracerebral hemorrhage: ›deep hypertensive hemorrhage and lobar hemorrhage
›

Extra-cerebral hemorrhage: subarachnoid hemorrhage and other hemorrhages that do NOT occur within the cerebrum, but within the skull.

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30
Q

What are the general features of a hemorrhagic CVA?

A

›A blood vessel bursts in the brain causing blood to enter the surrounding tissue.

›It can be caused by brain herniation, massive edema, use of cocaine or other illicit drugs.

›It is often due to aneurysms, leukemia, clotting deficiency, diet pills, and brain tumors.

›If hemorrhage is larger than 3cm, surgery is typically performed to evacuate the hematoma.

›The majority are due to a ruptured congenital berry aneurysm.

›Subarachnoid hemorrhage occurs more often in women 50-60 years old.

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31
Q

Describe the Western Aphasia Battery?

A

Assesses the linguistic skills most frequently affected by aphasia, in addition to key nonlinguistic skills, and provides differential diagnosis information

Employs a taxonomic (categorical) approach to classification—
Pts are assigned to diagnostic categories according to their scores on language subtests

The WAB was designed to provide a means of evaluating the major clinical aspects of language function: content, fluency, auditory comprehension, repetition and naming plus reading, writing and calculation

Criterion cut scores:
Aphasia Quotient
Cortical Quotient
Auditory Comprehension Quotient
Oral Expression Quotient
Reading Quotient
Writing Quotient
Bedside WAB–R scores
Two main scores: 
Aphasia Quotient (AQ) score and Cortical Quotient (CQ) score

AQ can essentially be thought of as a measure of language ability, whilst CQ is a more general measure of intellectual ability and includes all the subscales

One apraxia (not AOS) subtest

Yields aphasia syndrome type

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32
Q

Describe the FAS

A

Used to assess verbal fluency–production of individual words under restricting search conditions

Client names as many words as they can that start with F, A, and S in 1 minute (1 min per letter)

The score is the sum of all admissible words for the three letters (proper nouns, wrong words, variations, and repetitions are not counted as correct)

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33
Q

Describe the Aphasia Book club

A

The book club program was initially developed by Berstein-Ellis and Elman at the Aphasia Center of California (ACC) in 1999 in Oakland, CA.

The purpose of Aphasia Book Clubs is to enhance one’s quality of life through social interaction and life participation.

In 2004 the ACC received funding to replicate their program at four sites in North America.

The Aphasia Center of California’s mission is to combine community service, student training and research, centered around individuals living with Aphasia

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34
Q

What are the need for bookclubs?

A

Community need
After a stroke, people continue to improve over a period of time (years). This can be a slow process for both the patient and the family. Individuals living with Aphasia need to learn compensatory strategies for communicating.

Most inpatient rehabilitation is limited to 16-24 days! Outpatient rehabilitation is often only 3-6 months, depending on insurance.

Persons with aphasia, their families, and clinicians are searching for low-cost options in the community that are readily accessible to them!

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35
Q

What are the benefits to aphasia book clubs?

A

Groups promote interaction and variety of communicative functions or speech acts

Groups provide opportunity for wider array of partners = more generalization

Groups promote language improvisation

Groups improve psychosocial functioning – Benefit of Community

Groups are cost effective

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36
Q

What are some strategies for communication partners for the Aphasia book clubs?

A

Increase pause time/decrease interruptions

Do not request known information or ask ‘performance’ questions (e.g., “What’s my name,” or “Tell Mary my name.”)

Utilize gestures, writing, and drawing to facilitate successful communication

Use what works best for each individual (e.g., the mini whiteboard)

Acknowledge that the person knows what he or she wants to say

Rephrase or expand what the person has communicated

Be patient, and verify topic

Acknowledge communication breakdowns (e.g., “That’s not what you meant?”)

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37
Q

What are the procedures for a successful book club?

A

Proxemics! Create a seating chart that strategically arranges book club members and volunteers for maximum ease of communication

Name tags

Start each session by “checking in” with members by reviewing last week’s discussion and taking time for questions

Priority: “Does anyone want to start us off today by telling us about something you found especially interesting in this week’s reading?”

Based our discussion points off of the chapter highlights

PowerPoints or “learning modules” focus on some specifics of the chapter as well as topics that were related to the chapter more broadly

As volunteers and facilitators, try to avoid taking on the role of the classroom teacher

Weekly worksheets to facilitate understanding:
Worksheets completed at home

Choose from worksheet A, B, or C (or all three) depending on the level of difficulty the book club member feels comfortable with

Always look for communication opportunities between members; try to get members talking to each other

“Word of the Day”

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38
Q

What are the Rancho Levels?

A

Level I-no response: total assistance

Level II-Generalized response: total assistance

Level III-Localized response: total assistance

Level IV-Confused/Agitated: Maximal assistance

Level V-confused, inapprpriate, non-agitated: Maximal Assistance

Level VI- Confused, Appropriate: Moderate Assistance

Level VII- Automatic, Appropriate: Minimal Assistance for Daily Living
Skills

Level VIII- Purposeful, Appropriate: Stand-By Assistance

Level IX- Purposeful, Appropriate: Stand-By Assistance on Request

Level X- Purposeful, Appropriate: Modified Independent

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39
Q

Describe Level I of the Rancho Scales

A

Complete absence of observable change in behavior when presented visual,
auditory, tactile, proprioceptive, vestibular or painful stimuli.

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40
Q

Describe Level II of the Rancho Scales

A

Demonstrates generalized reflex response to painful stimuli.

Responds to repeated auditory stimuli with increased or decreased activity.

Responds to external stimuli with physiological changes generalized, gross body
movement and/or not purposeful vocalization.

Responses noted above may be same regardless of type and location of
stimulation.

Responses may be significantly delayed.

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41
Q

Describe Level III of the Rancho Scales

A

Demonstrates withdrawal or vocalization to painful stimuli.

Turns toward or away from auditory stimuli.

Blinks when strong light crosses visual field.

Follows moving object passed within visual field.

Responds to discomfort by pulling tubes or restraints.

Responds inconsistently to simple commands.

Responses directly related to type of stimulus.

May respond to some persons (especially family and friends) but not to others.

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42
Q

Describe Level IV of the Rancho Scales

A

Alert and in heightened state of activity.

Purposeful attempts to remove restraints or tubes or crawl out of bed.

May perform motor activities such as sitting, reaching and walking but without
any apparent purpose or upon another’s request.

Very brief and usually non-purposeful moments of sustained alternatives and
divided attention.

Absent short-term memory.

May cry out or scream out of proportion to stimulus even after its removal.

May exhibit aggressive or flight behavior.

Mood may swing from euphoric to hostile with no apparent relationship to
environmental events.

Unable to cooperate with treatment efforts.

Verbalizations are frequently incoherent and/or inappropriate to activity or
environment.

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43
Q

Describe level V of the Rancho Scales

A

Alert, not agitated but may wander randomly or with a vague intention of going
home.

May become agitated in reponse to external stimulation, and/or lack of
environmental structure.
Not oriented to person, place or time.

Frequent brief periods, non-purposeful sustained attention.

Severely impaired recent memory, with confusion of past and present in
reaction to ongoing activity.

Absent goal directed, problem solving, self-monitoring behavior.
Often demonstrates inappropriate use of objects without external direction.

May be able to perform previously learned tasks when structured and cues
provided.

Unable to learn new information.

Able to respond appropriately to simple commands fairly consistently with
external structures and cues.

Responses to simple commands without external structure are random and nonpurposeful
in relation to command.

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44
Q

Describe CILT

A

Constraint Induced Language Therapy (CILT) aka Constraint Induced Aphasia Therapy (CIAT)

CILT was developed for patients with chronic, stroke-induced Aphasia. It was developed in 2001 from the physical therapy technique of Constraint Induced Movement Therapy. This is where the patient, who tends to overuse their “good arm” due to a hemi, have their “good arm” tied to their body for 90% of the day, to prevent use of it. Therapy is intensive, but much research has shown that it is effective. This concept was taken and employed using a language perspective

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45
Q

What are CILT’s major components?

A

Constraint: ONLY VERBAL communication is allowed. NO compensatory nonverbal communication strategies may be used.

Forced Use: you MUST communicate by only talking

Massed Practice: Therapy occurs 2-4 hours per day

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46
Q

What does CILT tx look like?

A

Therapy basically looks like a modified version of Go Fish

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47
Q

What is Spaced Retrieval therapy?

A

SRT is a memory training procedure in which participants are trained to perform newly taught procedures, recognize newly taught stimuli, or remember to do something at a designated time, with gradually increasing time intervals between training and performance.

Forms of Spaced Retrieval Include:

Semantic memory (remembering facts, names of people, objects, etc…)

Procedural memory (remembering to perform some action–e.g., getting out of a chair, swallowing techniques)

Prospective memory (remembering future appointments, activities, or tasks)

Recent episodic memory (remembering recent personal events–e.g., one’s birthday, a dinner party)

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48
Q

What are SRT’s Major components?

A

to perform SRT several functional targets are selected
then one target is focused on at a time until maintenance level is achieved
the SLP asks a question to elicit the target
if the client answers correctly, increase the time interval and ask the question again.
if the answer is incorrect, provide immediate correction and ask the question again at the last correct time interval.

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49
Q

Who does SRT help, and what is essential for it to be successful?

A

SRT should help a person with dementia recall trained information for up to several months after training.

SRT will not improve generalized memory function.

Selecting specific and functional targets is key for the client to obtain improved independence.

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50
Q

What is the formula for a SMART goal?

A

Pt. will (action) with ___% accuracy with (amount of assistance) by (time frame)

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51
Q

Write a SMART goal If we know they can copy at the single letter stage…

A

Pt. will copy simple single words (e.g., 3-5 letters), with 80% accuracy with min. assistance.

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52
Q

Write a SMART goal if we know they follow 1 step commands at 100%, but 2 step commands at only 50%…

A

Pt. will follow simple/basic 2 step commands (e.g., point up, then touch your nose) with 80% accuracy with min. assistance.

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53
Q

Write a SMART goal if If we know they can read at single sentence step, but they break down at the paragraph level…

A

Pt. will read at the single/compound sentence level, with 80% accuracy with mod. assistance.

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54
Q

Just read: Other examples of SMART goals

A
  • Given a colored photo, Pt. will match single/simple sentences in a field of 3 (f/3), with 90% accuracy given occasional assistance.
  • Given a (novel/complex/abstract paragraph), Pt. will answer multiple choice concrete questions from a field of 3, with 90% accuracy, with min. assistance.
  • Pt. will independently use learned compensatory strategies to engage in two way conversation with an unfamiliar communication partner during six turn-taking events with 80% accuracy with min. assistance.
  • Using the non-dominant (L) hand, the patient will independently formulate and write at the single/simple sentence level with 90% accuracy with minimal assistance by July 2015.
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55
Q

What are the different dementias

A
  • Cortical
  • subcortical
  • mixed
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56
Q

What is cortical dementia?

A

Changes in cerebral cortex

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57
Q

What diseases fall under cortical dementia?

A
  • Alzheimer’s

- PPA (primary progressive aphasia)

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58
Q

What is Alzheimer’s disease?

A
  • Dementia is a syndrome due to disease of the brain, usually of a chronic or progressive nature, in which there is disturbance of multiple higher cortical functions, including memory, thinking, orientation, comprehension, calculation, learning capacity, language, and judgment.
  • Consciousness is not clouded. Impairments of cognitive function are commonly accompanied, and occasionally preceded, by deterioration in emotional control, social behavior, or motivation.
  • Dementia produces an appreciable decline in intellectual functioning, and usually some interference with personal activities of daily living, such as washing, dressing, eating, personal hygiene, excretory and toilet activities.
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59
Q

Early signs of Alzheimer’s

A
  • Memory failure
  • Disorientation
  • Lapses in judgment
  • Difficulty performing activities of daily life
  • Difficulty performing mentally challenging tasks
  • Misplacing things
  • Apathy and loss of initiative
  • Changes in mood
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60
Q

Facts about Alzheimer’s

A
  • fastest growing and most expensive disease
  • Affects more women than men
  • 3 microscopic changes in brain neurons: neurofibrillary tangles, neuritic plaques, and granulovaculoar degeneration
  • Tx: tranquilizers, antidepressants, Cognex and Aricept
  • 1st symptoms: memory lapses, faulty reasoning, poor judgment, disorientation, mood changes
  • Language less affected than cognition
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61
Q

What is subcortical dementia?

A
  • changes in basal ganglia, thalamus, and brain stem
  • Motor impairments (tremors and gait)
  • volitional movement impairments
  • first signs of dementia appear months to year after appearance of motor impairments (as disease progresses)
  • Cortical function impairments occur in the later stages
  • Motor impairments are evident in the early stages
  • Most SDs are delayed consequences of extrapyramidal system disease and reveal volitional movement impairment
  • Assess and tx for speech impairments
  • Screen for cognitive involvement & dysphagia
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62
Q

What diseases fall under subcortical dementia?

A
  • Parkinson’s
  • Huntington’s
  • Progressive Supranuclear Palsy
  • AIDS
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63
Q

Cognitive and communication characteristics of Parkinson’s disease

A
  • Weak voice
  • Reduced vocal loudness
  • Speech rate increases/reduced articulation
  • Rapid, stuttering-like repetitions of syllables, words, and phrases
  • Micrographia is common
  • Drooling/swallowing, middle stages
  • Vocabulary, syntax, and grammar are preserved until late stages
  • Most individuals die within 15-20 years of onset
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64
Q

Facts about Parkinson’s

A
  • Affects more men than women and appears between 50-65
  • Caused by deterioration of dopaminergic neurons in basal ganglia and brain stem
  • 60% of these neurons are destroyed and 1st symptoms emerge: resting tremor, pill rolling, muscle rigidity, slowness, diff. initiating movement
  • Primary treatment: levodopa (L-dopa)
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65
Q

What is Parkinson’s disease?

A
  • disturbances of movement including muscle rigidity, tremor, slowness/abolition of movements, and loss of balance.
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66
Q

Cognitive communication deficits with Huntington’s disease

A
  • Dysarthria
  • Dysphagia
  • Final stages: Mute, incontinent and profoundly demented
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67
Q

What is Huntington’s disease?

A

inherited degenerative neurologic disease

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68
Q

Facts about Huntington’s disease

A

There is no cure, only medication to control movement, emotional, and psychological effects
Appears between 40-60 years
1st symptom: involuntary movements

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69
Q

What subcortical disease resembles Parkinson’s?

A

Progressive Supranuclear Palsy

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70
Q

Facts about Progressive Supranuclear Palsy

A
  • Rare; 1 in 20,000 adults in U.S.
  • Begins between 50 and 80
  • No known tx
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71
Q

What is Progressive Supranuclear Palsy and what is the progression?

A

Caused by neuronal loss, neuronal abnormalities, and proliferation of glial cells through the brain cell and basal ganglia

Progression:
Pt loses vertical and lateral eye movements, limbs become stiff and rigid, dysarthria, and dysphagia

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72
Q

Cognitive and Communication deficits with Progressive Supranuclear Palsy

A
  • dysarthria,
  • speech is slow,
  • repetitions,
  • reduced vocal loudness,
  • language remains intact,
  • mutism is common in very late stages
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73
Q

Personality changes with Progressive Supranuclear Palsy

A

depression & dementia

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74
Q

Early symptoms of Progressive Supranuclear Palsy

A

Paralysis of muscles responsible for downward gaze, rigidity of neck muscles, facial muscle weakness

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75
Q

Facts about AIDS

A
  • Complex signs/symptoms
  • Weakens the immune system
  • AIDS Dementia Complex (HIV encephalopathy)
  • 70% of persons w/ AIDS develop AIDS dementia complex
  • Infection causes pathologic changes in subcortical white matter and basal ganglia (eventually reaching the cortex)
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76
Q

early symptoms of AIDS

A

extrapyramidal pathology (weakness, slowness, rigidity, dyskinesia) and later symptoms reveal cortical involvement (declined perception, memory, intellect, language)

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77
Q

Cognitive and communication deficits with persons with AIDS

A
  • early stages may reveal mildly reduced word retrieval,
  • later speech is dysarthric and labored,
  • eventually reduced to single words.
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78
Q

What diseases fall under mixed dementia?

A
  • Vascular dementia
  • Lewy Body Dementia
  • Frontotemporal dementia (FTD)
    • Behavioral- variant FTD (bvFTD)
    • Pick’s disease (confirming)
    • PPA (Primary progressive aphasia)
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79
Q

Facts about vascular dementia

A
  • Important cause of dementia in adults
  • 2nd only to AD!
  • Dx is complicated and controversial
  • Pure vascular dementia is relatively uncommon; majority have both AD and VD
  • Most common type: Multi-infarct dementia
    • Lacunar state
    • Multiple cortical infarcts
    • Binswanger’s disease (rare; caused by multiple infarcts in subcortical white matter)
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80
Q

Facts about Lewy Body Dementia

A
  • Usually after 75 years of age
  • males > females
  • one of the most common progressive dementias, accounting for 10% yo 15% of all cases, behind Alzheimer’s.
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81
Q

Cognitive and communication deficits of Lewy Body Dementia

A
- visuospatial abilities
language
- attention
- working memory
- executive functions similar to the impairment of person with Alzheimer's dementia, but with memory somewhat better preserved
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82
Q

What is Frontotemporal dementia (FTD)

A
  • There are multiple subtypes
  • Leads to tissue shrinkage and reduced function in the brain’s frontal and temporal loves, which control planning and judgement; emotions, speaking and understanding speech; and certain types of movement
  • FTD was once considered rare, but it’s now thought to account for up to 10 to 15 percent of all dementia cases, but less common than Alzheimer’s disease, vascular dementia and Lewy body dementia
  • In those younger than age 65, FTD may account for up to 20 to 50 percent of dementia cases. People usually develop FTD in their 50s or early 60s, making the disorder relatively more common in this younger age group
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83
Q

What are the categories for FTD?

A
  • Behavioral-variant FTD (bvFTD)
  • Semantic dementia
  • Pick’s disease
  • PPA (primary progressive aphasia)
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84
Q

What are the behavioral symptoms of behavioral-variant FTD (bvFTD)?`

A
  • Hyperoral behaviors include overeating, dietary compulsions, in which the person restricts himself to eating only specific foods (such as a certain flavor of Lifesaver, or eating food only from one fast food restaurant) or attempts to consume inedible objects. Patients may consume excessive amounts of liquids, alcohol and cigarettes.
  • Stereotyped and/or repetitive behaviors can include re-reading the same book multiple times, hand rubbing and clapping, humming one tune repeatedly or walking to the same location day after day.
  • Personal hygiene habits deteriorate early in the disease progression, as the person fails to perform everyday tasks of bathing, grooming and appropriate dressing.
    Hyperactive behavior is exhibited by some patients, and can include agitation, pacing, wandering, outbursts of frustration and aggression.
  • Hypersexual behavior can range from a preoccupation with sexual jokes to compulsive masturbation.
  • Impulsive acts can include shoplifting, impulsive buying and grabbing food off another person’s plate.
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85
Q

What are the emotional symptoms of behavioral-variant FTD (bvFTD)?

A
  • Apathy or indifference toward events and the surrounding environment can be marked by reduced initiative, and lack of motivation.
  • Lack of insight into the person’s own behavior develops early. The patient typically does not recognize the changes in his or her own behaviors, nor do they exhibit awareness or concern for the effect these behaviors have on the people around them, including loved ones.
  • Emotional blunting develops early in the course of the disorder, and is manifested as a loss of emotional warmth, empathy and sympathy, and development of what appears to be indifference toward other people, including loved ones.
  • Mood changes can be abrupt and frequent
86
Q

What are the neurological symptoms of behavioral-variant FTD (bvFTD)?

A

Symptoms similar to those seen in Parkinson disease. Among bvFTD patients, the term “parkinsonism” is used to distinguish the fact that they do not have Parkinson disease, though they do exhibit some of these symptoms. Symptoms include: decreased facial expression, bradykinesia (slowness of movements), rigidity (resistance to imposed movement) and postural instability

87
Q

What is the prominent complaint of people with semantic dementia?

A

Language difficulty.

  • due to the disease damaging the left temporal lobe, an area critical for assigning meaning to words. The language deficit is not in producing speech but is a loss of the meaning, or semantics, of words. At first, you might notice someone substituting a word like “thingy” for more unusual words, but eventually a person with SD will lose the meaning of more common words as well.
  • People speak easily, but their words convey less and less meaning. They tend to use broad general terms, such as “animal” when they mean “cat.” Language comprehension also declines
88
Q

What is Pick’s disease?

A
  • a rare form of progressive dementia, typically occurring in late middle age and often familial, involving localized atrophy of the brain.
  • Changes in personality, and emotion are first signs
89
Q

What is PPA (primary progressive aphasia)?

A
  • a language disorder that involves changes in the ability to speak, read, write and understand what others are saying. It is associated with a disease process that causes atrophy in the frontal and temporal areas of the brain, and is distinct from aphasia resulting from a stroke.
  • slow, insidious onset
  • people lose their ability to generate words easily, and their speech becomes halting, “tongue-tied” and ungrammatical. Ability to read and write also may be impaired.
90
Q

What does CETI stand for?

A

Communication Effectiveness Index

91
Q

What is CETI?

A

Is a rating protocol for communication partner to respond to. Family members rate their loved ones ability in day-to-day situations- either “not at all able to” or “as able as before stroke”. Assesses severely aphasic adults’ functional communication

92
Q

Which aphasias are considered nonfluent?

A
  • Broca’s

- Global

93
Q

What are the speech patterns of persons living with Broca’s aphasia?

A
  • Anomia
  • Phrase length: 0-5 words per breath unit
  • relatively good auditory comp.
  • relatively poor repetition
  • agrammatism/telegraphic speech
  • effortful articulation, disrupted prosody
  • phonemic paraphasias are common
94
Q

What are the speech patterns of persons living with global aphasia?

A
  • Profound anomia
  • virtually no speech output
  • very poor auditory comprehension
  • may have stereotypical utterances (real word or nonsense syllables)
95
Q

Which aphasias are considered fluent?

A

Wernicke’s

Transcortical sensory

Transcortical Motor

Conduction Aphasia

Anomic Aphasia

96
Q

What are the speech patterns of persons living with wenicke’s aphasia?

A

Notable anomia

poor auditory comprehension

Prosody WFL

Articulation WFL

Phonemic, semantic, neologistic paraphasias

empty content

may have jargon

press of speech/logorrhea

97
Q

What are the speech patterns of persons living with transcortical sensory aphasia?

A

Severe anomia

poor auditory comprehension

relatively good repetition skills

reduced flow of output due to anomia but preserved

articulation and prosody

preserved grammar

empty content (e.g., “thing” for target word)

Semantic paraphasias more often than phonemic
paraphasias

perseveration common

98
Q

What are the speech patterns of persons living with transcortical motor aphasia?

A

Impaired initiation of verbal output

anomia

short phrase length

good auditory comprehension

good repetition in comparison to speech output

unable to generate full sentences

99
Q

What are the speech patterns of persons living with conduction aphasia?

A

Anomia

Fluent output with normal average phrase length (e.g., 9 words or more)

Speech may contain delays in word finding, self corrections

auditory comprehension is good

repetition is worse than self-formulated speech

100
Q

What are the speech patterns of persons living with anomic aphasia?

A

Word finding problems across tasks

phrase length could be WNL-but completely empty speech

delays for word retrieval

auditory comprehension and repetition relatively intact

use of nonspecific terms, circumlocutions

paraphasias usually of semantic type

101
Q

What are the speech patterns of persons living with alzheimer’s disease?

A

language is less affected than cognition, memory, and intellect in early stages

102
Q

What are the speech patterns of persons living with primary progressive aphasia disease? (WHY IS THIS IN THE DEMENTIA SECTION OF THE STUDY GUIDE?)

A

slow, insidious onset

Lose ability to generate words easily

speech is halting

“tongue tied” & ungrammatical

ability to read & write also may be impaired

103
Q

What are the speech patterns of persons living with semantic dementia disease?

A

speak easily, but words convey less & less meaning

use broad general terms such as “animal” when they mean “cat”

language comprehension also declines

104
Q

What are the speech patterns of persons living with Parkinson’s disease?

A

weak voice

reduced vocal loudness

speech rate increases/reduced articulation

rapid, stuttering-like repetitions of syllables, words, and phrases

micrographia is common

drooling/swallowing, middle stages

vocabulary, syntax, and grammar are preserved until late stages

105
Q

What are the speech patterns of persons living with Huntington’s disease?

A

Dysarthria

Dysphagia

Final Stages: Mute & profoundly demented

106
Q

What are the speech patterns of persons living with Progressive Supranuclear Palsy disease?

A

dysarthria

speech is slow

repetitions

reduced vocal loudness

language remains intact

mutism is common in very late stages

107
Q

What are the speech patterns of persons living with AIDS disease?

A

Early stages may reveal mildly reduced word retrieval

late speech is dysarthric and labored

eventually reduced to single words

108
Q

What are the speech patterns of persons living with RHS?

A

Diminished Speech Prosody

  • lack normal variability in pitch & loudness
  • speech sounds monotonous/devoid of emotion
  • reduced spontaneity & variety in nonverbal movements that typically accompany speech (e.g., head nod/gestures)
  • slower than normal speech rate
  • reduced emphatic stress in phrases/sentences
  • diminished pitch variability (restricted intonations, failure to distinguish between questions/assertions)
  • Many of the same individuals who fail to communicate emotion via speech prosody also fail to appreciate emotions conveyed by others’ speech prosody and facial expression thereby suggesting an underlying affective impairment

Anomalous Content & organization of connected speech
-excessive, confabulatory, and sometimes inappropriate -connected speech (assess using narrative production, story retell tasks)
connect speech often described as: excessive, rambling, repetitive, irrelevant, tangential, digressive, inefficient
-use more words but produce less information
- narratives are: fragmented, lack cohesion, overall theme or point, focus on incidental details, fail to establish relationships among events, insert tangential comments, permit personal experiences/opinions to intrude into their narratives

Impaired comprehension of narratives & conversations

Pragmatic Impairments

  • turn-taking, topic maintenance, social conventions, and eye contact
  • may begin/end conversations abruptly
  • poor at obtaining and/or maintaining eye contact
  • talk excessively & without regard for their listener
  • difficulty staying on topic
  • Interject irrelevant, tangential, and inappropriate comments
  • Fail to make conversational repairs
    • insensitive to rules governing conversational turn-taking
109
Q

CN I

A

Olfactory
Smell

Assessment:

  • Ask client if s/he has noticed any change in his/her sense of smell
  • Clinician: Use soap, coffee, cloves, vanilla
  • Don’t use a harsh order (e.g., ammonia) as this will stimulate the intranasal pain endings of CN V, rather than assessing CN 1
  • Directions: Have the client/patient close both eyes, clinician occludes (“close”) one nostril/nare, and client should gently inhale to smell the scent.
  • Assess both nares separately.
110
Q

CN II

A

Optic Nerve
Controls central and peripheral vision

Assessment:

  • Test: Visual acuity, color vision, visual fields
  • Test one eye at a time; if client usually wears glasses then use them
  • Obtain a reading sample from client/patient (e.g., hospital menu, newspaper)
  • Ask client to identify colors
  • Have the client count how many fingers you are holding up—6 inches in front of him/her
  • Test peripheral vision one eye at a time.
  • Cover one eye and ask the client to look at your nose
  • Move your index fingers to check the superior and inferior fields one at a time
  • Ask the client to note any movement in the peripheral visual fields
111
Q

CN III

A

Oculomotor Nerve

Controls pupillary constriction
Eye movement

Assessment:

  • Dim the lights, bring the light of the penlight from the outside periphery to the center of each eye, and note the response.
  • Use an mm chart to describe pupil size; descriptions such as “small” or “medium” are too subjective
  • Check where the eyelid falls on the pupil. If it droops, note that the pt has ptsosis.
112
Q

CN IV

A

Trochlear Nerve

Acts as a pulley to move the eyes down—toward the tip of the nose.

Assessment:
-Instruct the patient to follow your finger while you move it down toward his/her nose.

113
Q

CN V

A

Trigeminal Nerve

  • Innervates the forehead, cheek and jaw
  • Sensations of touch, pain, temperature from face, and head, chewing, swallowing

Assessment:

  • Sensory fibers has 3 branches
  • Check sensation in all three areas using soft/dull object (cotton swab):
  • Check both sides (e.g., left and right) / Keep pressure firm and consistent across all trials!
  • Ophthalmic nerve: forehead, eyes, and nose
  • Stroke above eyebrow
  • Maxillary nerve: upper lip, mucosa, maxilla, upper teeth, cheeks, palate and maxillary sinus
  • Stroke the upper lip in upward movement toward cheekbone)
  • Mandibular Nerve: anterior 2/3 of tongue, mandible, lower teeth, lower lip, part of the cheek, and part of external ear
    • Stroke between lower lip and chin in an upward movement toward cheekbone; compare sides of tongue (anterior and medial) for sensitivity
  • Test the motor function of the temporal and masseter muscles by assessing jaw opening strength:
    • Ask client to open mouth, clench teeth (pterygoids), and open jaw against resistance
    • Palpate the area
    • Unilateral paralysis of CN V shows deviation of jaw to side of lesion AND inability to force jaw to side opposite of the lesion
    • Bilateral upper motor neuron lesions result in limited jaw movement (reduced ROM and slow movement)
114
Q

CN VI

A

Abducens Nerve

Controls lateral eye movements

Assessment:

  • Ask the client to look toward each of YOUR ears, one at a time.
  • Have him/her follow your fingers through the six “cardinal fields of gaze”
  • Note any twitching or nystagmus of the eye
  • Definition: “involuntary eye movement”
115
Q

CN VII

A

Facial Nerve

  • Controls facial movements and expression
  • Taste anterior ⅔
  • Somatosensory information from ear
  • Lacrimation & Salivation

Assessment:

  • Assess client’s face “at rest” for facial symmetry
  • Ask him/her to perform the following movements:
  • Wrinkle forehead
  • Close eyes, tightly
  • Smile
  • Pucker lips
  • Show his teeth
  • Puff out cheeks and hold.
  • Are the movements symmetrical?
116
Q

CN VIII

A

Vestibulocochlear

Controls hearing & balance

Assessment:

  • Rub your fingers together by client’s ears; perform one at a time.
  • We do not test vestibular function.
  • If client reports:
    • Tinnitus or dizziness—refer to neurologist and/or audiologist
117
Q

CN IX

A

Glossopharyngeal Nerve

Cranial nerves IX and X, which innervate the tongue and throat (pharynx and larynx) are checked together.

Assess the sense of taste ⅓ posterior portion of the tongue

Somatoseensory information from tongue, tonsils, pharynx

Controls some muscles for swallowing

Assessment:

  • Observe pt’s ability to swallow by noting how he/she handles secretions
  • Ask the client to open his/her mouth and say AHHHHHHHHHH
    • Use a penlight
    • The uvula should be in the midline, and the palate should rise.
118
Q

CN X

A

Vagus Nerve

Autonomic visceral functions, heart rate, glands, digestion

See CN IX for assessment

119
Q

CN XI

A

Accessory nerve

Controls neck and shoulder movement

Assessment:

  • Ask client to raise his shoulders against your hands to assess the trapezius muscle
  • Ask client to turn his head against your hand to assess the sternocleidomastoid muscle
120
Q

CN XII

A

Hypoglossal nerve

Innervates the tongue

Assessment:

  • Ask the client to protrude (“protrude”) his/her tongue;
  • Tongue should be at midline
  • Inspect for signs of atrophy (unilateral LMN lesion; side of lesion will appear shrunken/atrophied)
  • UMN involvement – tongue deviates to the side opposite of lesion (due to contralateral control)
  • Bilateral damage – client is unable to protrude the tongue beyond the lips
  • Look for fasciculations or fibrillations (tiny ripples). WFL – “some ripples may be present”
  • `Check for strength and mobility including lingual protrusion, elevation, lateralization
121
Q

As a stellar medical-based SLP, you are integrating a cranial nerve examination into your oral mech exam. You check for your pt’s lingual strength and mobility by asking her to protrude, elevate, and lateralize her tongue. You are primarily checking for involvement of the:

(a) CN V
(b) CN VII
(c) CN IX
(d) CN XII

A

CN XII

122
Q

As a stellar medical-based SLP, you are integrating a cranial nerve examination into your oral mech exam. You ask your patient to perform the following movements: Wrinkle forehead, close eyes, smile, pucker lips, show teeth, and puff checks/hold. You observe for symmetry in movement and at rest. You are checking on any involvement of the:

(a) CN V
(b) CN VII
(c) CN IX
(d) CN XII

A

CN VII

123
Q

What are the symptoms for TBI?

A
  • Headaches
  • Nausea & vomiting
  • Memory loss
  • Emotional lability
  • Dizziness
  • Vision problems
  • Sleep disturbances
124
Q

What is the etiology for TBI?

A

Moving object strikes head

Moving head strikes object

Penetrating & nonpenetrating injuries

125
Q

What are penetrating injuries?

A

Skull fracture/meninges torn

High velocity: rifle bullets, military projectiles

Carries foreign material into brain

Low velocity: bullets from shrapnel—often less fatal (20-40% cause death)

126
Q

What are non penetrating injuries?

A

Closed Head Injury/Meninges remain intact

Linear acceleration injuries (coup & contracoup)

Shaken baby syndrome, whiplash

Focal damage to meninges and brain tissue

Angular acceleration injuries (blows to the head, causing it to move away from point of impact)

Twisting and shearing tend to be concentrated at boundaries between gray and white matter

Tissue damage, bleeding and swelling affect major nerve fiber tracts

127
Q

What is treatment for TBI dependent on?

A

treatment is dependent on Rancho levels

128
Q

What are the different kinds of treatment programs for TBI?

A

Early Treatment programs: Drill activities stimulate and enhance mental processes

Current Treatment programs: Functionally oriented treatment

Cognitive: rehabilitation-restorative or compensatory

Behavior Management: Control problem behaviors and enhance desired behaviors

Medications: Prescribed to control agitation/restlessness, improve alertness/attention, manage depression, diminish psychotic symptoms, and control seizures.

Sensory Stimulation: Common for comatose patients

Component Training: Repetitive drill activities for attention, memory, problem solving

Crosson’s Model: Intellectual, emergent, and anticipatory awareness

Prigitano & Klonoff: Unawareness (puzzled when told of limitation; direct effect of BI) and denial (angry when told of limitations; coping strategy)

129
Q

What is the etiology for NTBIs?

A
  • Brain tumors
  • Aneurysm
  • Infection/virus
  • Anoxia
  • Surgery
  • Chemo/radiation
  • Substance abuse
  • Meningitis
  • Stroke
130
Q

What are the symptoms for RHS?

A

Highly Variable

Decreased ability to initiate an activity without help

Decreased ability to concentrate and pay attention to what’s important

Decreased orientation

Increased confusion and disorganized thinking

Increased impulsiveness

Decreased memory

Left neglect

Decreased visual perception

Decreased cognition

Denial and defensiveness

Changed personality

Decreased social skills

Decreased clearness of speech

Self-absorbed

Insensitive

Rambling in speech

131
Q

What is the etiology for RHS?

A

Damage to the right parietal lobe (typically after stroke)

132
Q

What is the treatment for RHS?

A

Attentional Activities: Sustained, selective, alternating, and divided attention—drills including paper and pencil tasks (e.g., cancellation, mazes, etc…)

Impulsivity: Use of stop and go signals

Impaired reasoning and problem solving: Identify a problem, think of several possible solutions, evaluate the feasibility and potential consequences of each solution, choose the best solution, apply it, evaluate the results.

Affective communication/Prosody: show individual pictures of faces expressing various emotions, replace by card which carry names of emotions

Reading: Colored vertical lines, colored dots, rulers placed at the left margin, use of finger, verbal cues

Pragmatics: Videotape, cues to improve eye contact, turn-taking rules, structured practice (e.g., games in small groups), PACE

133
Q

What are the symptoms for dementia?

A

Exaggerated forms of:

Memory failure

Disorientation

Lapses in judgment

Difficulty performing ADLs

Difficulty performing mentally challenging tasks

Misplacing things

Apathy and loss of initiative

Changes in mood

134
Q

What is the etiology for cortical dementia?

A

changes in the cerebral cortex (alzheimer’s disease)

135
Q

What is the etiology for subcortical dementias?

A

changes in the basal ganglia, thalamus, and brain stem ( e.g., parkinson’s disease and vascular dementia)

136
Q

What is the etiology for mixed dementias?

A

caused by changes in cortical and subcortical structures

Normal pressure hydrocephalus

Creutzfeldt-Jakob Disease (“Mad cow Disease”)

Late stages of ALS, MS

Brain tumors and chronic SDH if chronic elevation of intracranial pressure

Bacterial or viral infections of the brain (e.g., meningitis, encephalitis)

Hemodialysis (secondary to kidney failure… dialysis dementia)

137
Q

What is the treatment for dementia?

A

It can be managed and slowed with medication, but there isn’t really any treatment

138
Q

Describe pseudodementia

A

Symptoms of depression may mimic symptoms of dementia: cognitive impairments, loss of appetite, difficulty sleeping, social withdrawal, and apathy.

Pseudodementia has an identifiable onset, with rapid symptom development. True dementia has an insidious and gradual onset with slow symptom development.

Persons with pseudodementia make little effort to perform clinical tests. Person with true dementia (in early stages) try hard to prove themselves adequate on tests.

The test performance of person with pseudodementia is highly variable from test to test and day to day, even on test of equivalent difficulty. The test performance of person with true dementia is consistent across tasks and test occasions.

139
Q

Describe Huntington’s Disease

A

A type of subcortical dementia. No cure exists, and all medication is to alleviate symptoms. The first symptom usually seen is involuntary movements. Results in cognition and communication deficits. Dysarthria, and dysphagia are common. In final stages: mute, incontinent, and profoundly demented.

140
Q

What do tangles and plaques do?

A

The formation of amyloid plaques and neurofibrillary tangles are thought to contribute to the degradation of the neurons (nerve cells) in the brain and the subsequent symptoms of Alzheimer’s disease.

141
Q

What are tangles?

A

These are microscoping changes in brain neurons, and primary markers of Alzheimer’s Disease. They are insoluble twisted fibers found inside the brain’s cells. These tangles consist primarily of a protein called tau, which forms part of a structure called a microtubule. The microtubule helps transport nutrients and other important substances from one part of the nerve cell to another. In Alzheimer’s disease, however, the tau protein is abnormal and the microtubule structures collapse.

142
Q

What are plaques?

A

One of the hallmarks of Alzheimer’s disease is the accumulation of amyloid plaques between nerve cells (neurons) in the brain. Amyloid is a general term for protein fragments that the body produces normally. Beta amyloid is a protein fragment snipped from an amyloid precursor protein (APP). In a healthy brain, these protein fragments are broken down and eliminated. In Alzheimer’s disease, the fragments accumulate to form hard, insoluble plaques.

143
Q

Describe PPA

A

o A type of Alzheimer’s, characterized by a slow, insidious onset. PPA begins very gradually and initially is experienced as difficulty thinking of common words while speaking or writing. PPA progressively worsens to the point where verbal communication by any means is very difficult. The ability to understand what others are saying or what is being read also declines. In the early stages, memory, reasoning and visual perception are not affected by the disease and so individuals with PPA are able to function normally in many routine daily living activities despite the aphasia. However, as the illness progresses, other mental abilities also decline.

144
Q

What are the characteristics of Left neglect?

A

Failure to respond to people, sounds, objects on the left side of body

Attend only to right in self care activities

Failure to move or attend to the left side of the body

Bump into walls and doorways on left

Reading only the right side of printed materials

Displaced writing/drawing to the right side of a page

Diminished/poor awareness of deficits

Disinterested/lack of participation in rehab

145
Q

What is disprosody?

A

Short utterances that lack emotional inflection

Lack of prosody

Characteristic of RHS

146
Q

What are primary consequences of TBI?

A

traumatic hemorrhage

subdural hematomas

subarachnoid hematomas

intracerebral hematomas

147
Q

What is traumatic hemorrhage

A

Cuts, bruises, twisting, and shearing forces in the brain cause bleeding (hemorrhages) and accumulations of blood (hematomas).

Auto accidents, falls, sports injuries

148
Q

What are subdural hematomas?

A

Accumulations of blood beneath dura mater (above the arachnoid)

MVA most common

60% or greater mortality rate

149
Q

What are subarachnoid hematomas?

A

Rupture of pial vessels within subarachnoid space

May go for years without overt symptoms

150
Q

What are intracerebral hematomas?

A

Caused by rupture of blood vessels inside the brain

151
Q

What are secondary consequences of TBI?

A

cerebral edema

traumatic hydrocephalus

elevated intracranial pressure

alterations in the blood-brain barrier

152
Q

Describe cerebral edema

A

Accumulation of fluid due to trauma, anoxia, infection, inflammation

153
Q

describe traumatic hydrocephalus

A

Swelling of brain tissues sometimes compresses passages through which CSF circulates among ventricles and into subarachnoid space

154
Q

describe elevated intracranial pressure

A

Most dramatic and deadly consequence of TBI is pressure buildup inside the skull

most frequent cause of death from TBI

Ischemic Brain Damage

Most BI pts sustain ischemic brain damage

Most prominent in severe head injuries

155
Q

describe alterations in the blood-brain barrier

A

Passage of normally excluded substances into the brain may contribute to accumulation of fluid and swelling of brain tissues (cerebral edema)

156
Q

What is concreteness?

A

“loss of the abstract attitude”

Failure to appreciate the abstract or implied meaning of events, situations, language, or visual images.

Difficulties with figurative language, idioms, metaphors, humor, sarcasm, problem solving, and another’s point of view

157
Q

Describe “confusion”

A

normal or slightly lowered levels of consciousness but impaired orientation

can be caused by many factors, including nutritional deficiencies, dementing illness, drug or alcohol intoxication or withdrawal, endocrine disturbances, nutritional disorders, infections, cerebrovascular disorders, head trauma, and psychiatric illness.

Acute confusional states are transitory, but a period of confusion may evolve into a more circumscribed but longer lasting syndrome. For example, a stroke patient may exhibit confusion immediately after the stroke, with the confusion gradually clearing, leaving the patient not confused but with a language specific (or other) deficit.

158
Q

Describe the aphasia diagnostic profiles

A

Comprehensive test used widely in the United States

Has 9 subtests (quick test)

Tells type of aphasia and severity level

159
Q

describe the boston naming test

A

Word retrieval for milder cases of aphasia

Tests confrontation naming

60 items

Not culturally sensitive

Some items are outdated

160
Q

Describe PPA

A

A type of Alzheimer’s, characterized by a slow, insidious onset. PPA begins very gradually and initially is experienced as difficulty thinking of common words while speaking or writing. PPA progressively worsens to the point where verbal communication by any means is very difficult. The ability to understand what others are saying or what is being read also declines. In the early stages, memory, reasoning and visual perception are not affected by the disease and so individuals with PPA are able to function normally in many routine daily living activities despite the aphasia. However, as the illness progresses, other mental abilities also decline.

161
Q

Describe the Ross information processing assessment

A
Enables the examiner to quantify cognitive-linguistic deficits, determine severity levels for each skill area, and develop rehabilitation goals and objectives. The RIPA-2 provides quantifiable data for profiling 10 key areas basic to communicative and cognitive functioning:
•	Temporal Orientation (Recent Memory)
•	Temporal Orientation (Remote Memory)
•	Immediate Memory
•	Recent Memory
•	Spatial Orientation
•	Orientation to Environment
•	Recall of General Information
•	Problem Solving and Abstract Reasoning
•	Organization
•	Auditory Processing and Retention

The study sample included 126 individuals with traumatic brain injury in 17 states. The sample was representative of TBI demographics for gender, ethnicity, and socioeconomic status.

162
Q

Describe reminiscence therapy

A

Often used for patients suffering from dementia. Uses a person’s past experiences and present behaviors. Focus on the remaining abilities of the persons taking into account of the person’s past and present strengths, interests, and difficulties.

163
Q

Describe Semantic Feature Analysis

A

Is used for naming deficits that occur with aphasia

It can be used on PWA and also in the classroom or private clinic (for CH with language deficits)

It enhances lexical retrieval after training with cues; has 6 semantic features for naming pictures:
• Group: what group does it belong to?
• Use: what is the purpose of it?
• Action: what does it do?
• Properties: what are its physical properties?
• Location: where is it found?
• Association: what does it remind you of?

Activities:
• Present picture and ask client to name it
• Ask client to provide verbal response for each semantic feature (fruit, grows on tree, is round, can eat it)
• After naming semantic features, ask client to name object again

164
Q

Describe the CETI: communicative effectiveness index

A

Is a rating protocol for communication partner to respond to. Family members rate their loved ones ability in day-to-day situations- either “not at all able to” or “as able as before stroke”. Assesses severely aphasic adults’ functional communication.

165
Q

Describe the frontal lobe

A

Comprises the area from the central sulcus to the anterior limit of the cerebral cortex just dorsal and posterior to the bone case of the eyes.


The posterior portion of the frontal lobe consists of the precentral gyrus - specialized for control of movement. 


The frontal lobes make us conscious of our actions and thoughts.

166
Q

What are some consequences of frontal lobe impairment?

A

Loss of simple movement

Loss of ability to spontaneously interact

Loss of flexible thinking and problem solving

Perseveration

Inability to focus on a task

Mood Changes

Changes in personality and social behavior

Inability to speak

Inability to sequence complex movements

167
Q

describe the temporal lobe

A

Located inferior to the Sylvain Fissure (the lateral sulcus) and continues back to the Occipital lobe.

The left temporal lobe contains Wernicke’s area, important for language comprehension.

This area has connections to Broca’s area (in left frontal lobe), important for speech production.

The temporal lobes house the primary and secondary auditory cortex and are involved in auditory sensation and perception.

Auditory stimuli are transformed for comprehension of language in the temporal lobes.

Hearing, some visual perception, and categorization skills are dependent, in part, on the temporal lobes.

The temporal lobes are also primary sites for projections from the thalamic nuclei.

168
Q

What are some consequences of temporal lobe impairment?

A

Prospagnosia: Difficulty recognizing faces

Difficulty understanding spoken words

Poor selective attention for verbal and visual information

Short-Term memory loss

Interference with long-term memory

Increased or decreased interest in sexual behavior

Inability to categorize objects

Logorrhea: Persistent talking/press of speech

Increased aggressive behavior

169
Q

Describe the Parietal lobe

A

Located between the occipital lobe and the central sulcus (the Rolandic Fissure) and superior to the temporal lobe

The most anterior region to the post-central gyrus

This is the main area of termination for axons carrying information for the sense of touch

Sensations from the body are represented at various parts of the post-central gyrus

The parietal lobes receive and evaluate most sensory information including touch, pressure, pain, temperature, and taste. They help us identify objects by sensation of touch.

170
Q

what are some consequences of parietal lobe involvement

A

Inability to attend to more than one object at a time.

Alexia: problems with reading

Agraphia: Difficulty writing words

Word Blindness: inability to recognize words

Dyscalculia: difficulty with math and arithmetic concepts

Difficulty drawing objects

Difficulty knowing left from right

Lack of awareness of specific body parts

Unilateral neglect

Inability to focus visual attention

Difficulties with eye-hand coordination

Impaired perception of touch

Difficulty with goal directed movements

Difficulty with the functional manipulation of objects (non-motoric)

171
Q

Describe the occipital lobe

A

Located at the most posterior part of the cerebral cortex.

This lobe is the primary target for projections from the thalamus and receives sensory information from fibers in the eyes.

The retina gets visual input from light, shapes, and shading. This input is then transmitted through the optic nerve to the thalamus and then to the primary visual cortex in the occipital lobe.

If the visual cortex is damaged, it can lead to blindness or a visual field cut (hemianopsia).

Damage to left occipital region impairs vision in the right visual field.

A small area of damage can lead to a small blind area, called a scotoma.

Secondary association areas are important to visual processing and transmit stimuli to other parts of the brain for analysis.

These areas are important to visual processing in order to recognize objects and discriminate features.

172
Q

What are some consequences of occipital lobe involvement

A

Visual field cuts

Difficulty locating objects in the environment

Difficulty recognizing drawn objects

Inability to recognize movement of an object in space

Difficulty identifying colors

Visual illusions or inaccurately seeing objects

Word blindness or inability to recognize words

Difficulties with reading and writing

173
Q

Describe the cerebellum

A

The cerebellum does not initiate or integrate motor activity. It controls and performs online correction of planned movements.

The cerebellum regulates balance and posture and coordinates movements for fine-motor tasks such as speaking.

174
Q

Describe the central fissure

A

aka Central Sulcus

Persons with fluent aphasia have damage to the central sulcus and they speak smoothly and with little effort

The central sulcus is a prominent landmark of the brain, separating the parietal lobe from the frontal lobe and the primary motor cortex from the primary somatosensory cortex.

175
Q

describe the brainstem

A

The medulla is an enlargement of the top of the spinal cord.

It contains both ascending and descending nerve tracts and nuclei of cranial nerves that are important for speech motor control and swallowing.

These include phonation, articulation, and velopharyngeal closure.

176
Q

describe the pons

A

The pons is located superiorly to the medulla and inferiorly to the midbrain.

It contains nuclei for the trigeminal (V) abducens (VI), facial (VII), and vestibulocochlear (VIII) nerves. It is the ‘bridge’ to the cerebellum.

It helps control breathing and sleep.

It is part of the reticular activating system which is important for alertness and arousal.

177
Q

describe the midbrain

A

Medulla and pons contain cells of the reticular activating system–crucial for arousal and sleep.

The brainstem connects the cerebral cortex to the spinal cord and regulates primary life functions including:

Breathing, respiration, swallowing, blood pressure, and eye movements.

Regulation of heart rate, vomiting, salivation, sneezing, coughing, and gagging.

178
Q

describe the MCA

A

The middle cerebral artery is the largest cerebral artery

It supplies most of the lateral cortex and outer brain surface, internal capsules, and most of the basal ganglia.

The superior branch supplies the orbitofrontal and prefrontal cortex, and the inferior branch supplies most of the temporal regions

179
Q

Which rancho scale does this represent?

Complete absence of observable change in behavior when presented visual,
auditory, tactile, proprioceptive, vestibular or painful stimuli.

A

Level I

180
Q

Which rancho scale does this represent?

Responds to repeated auditory stimuli with increased or decreased activity.

Responds to external stimuli with physiological changes generalized, gross body
movement and/or not purposeful vocalization.

A

Level II

181
Q

Which rancho scale does this represent?

Alert and in heightened state of activity.

Purposeful attempts to remove restraints or tubes or crawl out of bed.

A

Level IV

182
Q

Which rancho scale does this represent?

Alert, not agitated but may wander randomly or with a vague intention of going
home.

May become agitated in reponse to external stimulation, and/or lack of
environmental structure.
Not oriented to person, place or time.

A

Level V

183
Q

Which rancho scale does this represent?

Demonstrates withdrawal or vocalization to painful stimuli.

Turns toward or away from auditory stimuli.

A

Level III

184
Q

Which rancho scale does this represent?

Demonstrates generalized reflex response to painful stimuli.

Responds to external stimuli with physiological changes generalized, gross body
movement and/or not purposeful vocalization.

A

Level II

185
Q

Which rancho scale does this represent?

May perform motor activities such as sitting, reaching and walking but without
any apparent purpose or upon another’s request.

Very brief and usually non-purposeful moments of sustained alternatives and
divided attention.

Absent short-term memory.

A

Level IV

186
Q

Which rancho scale does this represent?

Frequent brief periods, non-purposeful sustained attention.

Severely impaired recent memory, with confusion of past and present in
reaction to ongoing activity.

Absent goal directed, problem solving, self-monitoring behavior.

Often demonstrates inappropriate use of objects without external direction.

A

Level V

187
Q

Which rancho scale does this represent?

Blinks when strong light crosses visual field.

Follows moving object passed within visual field.

Responds to discomfort by pulling tubes or restraints.

A

Level III

188
Q

Which rancho scale does this represent?

May cry out or scream out of proportion to stimulus even after its removal.

May exhibit aggressive or flight behavior.

Mood may swing from euphoric to hostile with no apparent relationship to
environmental events.

A

Level IV

189
Q

Which rancho scale does this represent?

May be able to perform previously learned tasks when structured and cues
provided.

Unable to learn new information.

Able to respond appropriately to simple commands fairly consistently with
external structures and cues.

A

Level V

190
Q

Which rancho scale does this represent?

Responses may be significantly delayed.

A

Level II

191
Q

Which rancho scale does this represent?

Responds inconsistently to simple commands.

Responses directly related to type of stimulus.

May respond to some persons (especially family and friends) but not to others.

A

Level III

192
Q

Which rancho scale does this represent?

Unable to cooperate with treatment efforts.

Verbalizations are frequently incoherent and/or inappropriate to activity or
environment.

A

Level IV

193
Q

Which rancho scale does this represent?

Responses to simple commands without external structure are random and nonpurposeful
in relation to command.

A

Level V

194
Q

Describe Rancho level VI

A

• Inconsistently oriented to person, time and place.
• Able to attend to highly familiar tasks in non-distracting environment for 30
minutes with moderate redirection.
• Remote memory has more depth and detail than recent memory.
• Vague recognition of some staff.
• Able to use assistive memory aide with maximum assistance.
• Emerging awareness of appropriate response to self, family and basic needs.
• Moderate assist to problem solve barriers to task completion.
• Supervised for old learning (e.g. self care).
• Shows carry over for relearned familiar tasks (e.g. self care).
• Maximum assistance for new learning with little or nor carry over.
• Unaware of impairments, disabilities and safety risks.
• Consistently follows simple directions.
• Verbal expressions are appropriate in highly familiar and structured situations.

195
Q

Describe rancho level VII

A

• Consistently oriented to person and place, within highly familiar environments.
Moderate assistance for orientation to time.
• Able to attend to highly familiar tasks in a non-distraction environment for at
least 30 minutes with minimal assist to complete tasks.
• Minimal supervision for new learning.
• Demonstrates carry over of new learning.
• Initiates and carries out steps to complete familiar personal and household
routine but has shallow recall of what he/she has been doing.
• Able to monitor accuracy and completeness of each step in routine personal and
household ADLs and modify plan with minimal assistance.
• Superficial awareness of his/her condition but unaware of specific impairments
and disabilities and the limits they place on his/her ability to safely, accurately
and completely carry out his/her household, community, work and leisure ADLs.
• Minimal supervision for safety in routine home and community activities.
• Unrealistic planning for the future.
•Unable to think about consequences of a decision or action.
• Overestimates abilities.
• Unaware of others’ needs and feelings.
• Oppositional/uncooperative.
• Unable to recognize inappropriate social interaction behavior.

196
Q

Describe rancho level VIII

A

• Consistently oriented to person, place and time.
• Independently attends to and completes familiar tasks for 1 hour in distracting
environments.
• Able to recall and integrate past and recent events.
• Uses assistive memory devices to recall daily schedule, “to do” lists and record
critical information for later use with stand-by assistance.
• Initiates and carries out steps to complete familiar personal, household,
community, work and leisure routines with stand-by assistance and can modify
the plan when needed with minimal assistance.
• Requires no assistance once new tasks/activities are learned.
• Aware of and acknowledges impairments and disabilities when they interfere
with task completion but requires stand-by assistance to take appropriate
corrective action.
• Thinks about consequences of a decision or action with minimal assistance.
• Overestimates or underestimates abilities.
Acknowledges others’ needs and feelings and responds appropriately with
minimal assistance.
• Depressed.
• Irritable.
• Low frustration tolerance/easily angered.
• Argumentative.
• Self-centered.
• Uncharacteristically dependent/independent.
• Able to recognize and acknowledge inappropriate social interaction behavior
while it is occurring and takes corrective action with minimal assistance.

197
Q

Describe Rancho level IX

A

• Independently shifts back and forth between tasks and completes them
accurately for at least two consecutive hours.
• Uses assistive memory devices to recall daily schedule, “to do” lists and record
critical information for later use with assistance when requested.
• Initiates and carries out steps to complete familiar personal, household, work
and leisure tasks independently and unfamiliar personal, household, work and
leisure tasks with assistance when requested.
• Aware of and acknowledges impairments and disabilities when they interfere
with task completion and takes appropriate corrective action but requires standby
assist to anticipate a problem before it occurs and take action to avoid it.
• Able to think about consequences of decisions or actions with assistance when
requested.
• Accurately estimates abilities but requires stand-by assistance to adjust to task
demands.
• Acknowledges others’ needs and feelings and responds appropriately with
stand-by assistance.
• Depression may continue.
• May be easily irritable.
• May have low frustration tolerance.
• Able to self monitor appropriateness of social interaction with stand-by
assistance.

198
Q

Describe rancho level X

A

Able to handle multiple tasks simultaneously in all environments but may
require periodic breaks.
• Able to independently procure, create and maintain own assistive memory
devices.
• Independently initiates and carries out steps to complete familiar and unfamiliar
personal, household, community, work and leisure tasks but may require more
than usual amount of time and/or compensatory strategies to complete them.
• Anticipates impact of impairments and disabilities on ability to complete daily
living tasks and takes action to avoid problems before they occur but may require
more than usual amount of time and/or compensatory strategies.
• Able to independently think about consequences of decisions or actions but may
require more than usual amount of time and/or comepensatory strategies to
select the appropriate decision or action.
• Accurately estimates abilities and independently adjusts to task demands.
• Able to recognize the needs and feelings of others and automatically respond in
appropriate manner.
• Periodic periods of depression may occur.
• Irritability and low frustration tolerance when sick, fatigued and/or under
emotional stress.
• Social interaction behavior is consistently appropriate.

199
Q

Which Rancho scale is this?

• Inconsistently oriented to person, time and place.
• Able to attend to highly familiar tasks in non-distracting environment for 30
minutes with moderate redirection.
• Remote memory has more depth and detail than recent memory.
• Vague recognition of some staff.

A

VI

200
Q

Which rancho scale is this?

Able to handle multiple tasks simultaneously in all environments but may
require periodic breaks.
• Able to independently procure, create and maintain own assistive memory
devices.
• Independently initiates and carries out steps to complete familiar and unfamiliar
personal, household, community, work and leisure tasks but may require more
than usual amount of time and/or compensatory strategies to complete them.
• Anticipates impact of impairments and disabilities on ability to complete daily
living tasks and takes action to avoid problems before they occur but may require
more than usual amount of time and/or compensatory strategies.

A

X

201
Q

Which rancho scale is this?

• Independently shifts back and forth between tasks and completes them
accurately for at least two consecutive hours.
• Uses assistive memory devices to recall daily schedule, “to do” lists and record
critical information for later use with assistance when requested.
• Initiates and carries out steps to complete familiar personal, household, work
and leisure tasks independently and unfamiliar personal, household, work and
leisure tasks with assistance when requested.
• Aware of and acknowledges impairments and disabilities when they interfere
with task completion and takes appropriate corrective action but requires standby
assist to anticipate a problem before it occurs and take action to avoid it.

A

IX

202
Q

Which rancho scale is this?

• Consistently oriented to person, place and time.
• Independently attends to and completes familiar tasks for 1 hour in distracting
environments.
• Able to recall and integrate past and recent events.
• Uses assistive memory devices to recall daily schedule, “to do” lists and record
critical information for later use with stand-by assistance.

A

VIII

203
Q

which rancho scale is this?

• Consistently oriented to person and place, within highly familiar environments.
Moderate assistance for orientation to time.
• Able to attend to highly familiar tasks in a non-distraction environment for at
least 30 minutes with minimal assist to complete tasks.
• Minimal supervision for new learning.
• Demonstrates carry over of new learning.

A

VII

204
Q

Which rancho scale is this?

  • Able to use assistive memory aide with maximum assistance.
  • Emerging awareness of appropriate response to self, family and basic needs.
  • Moderate assist to problem solve barriers to task completion.
  • Supervised for old learning (e.g. self care).
A

VI

205
Q

Which rancho scale is this?

• Initiates and carries out steps to complete familiar personal and household
routine but has shallow recall of what he/she has been doing.
• Able to monitor accuracy and completeness of each step in routine personal and
household ADLs and modify plan with minimal assistance.
• Superficial awareness of his/her condition but unaware of specific impairments
and disabilities and the limits they place on his/her ability to safely, accurately
and completely carry out his/her household, community, work and leisure ADLs.
• Minimal supervision for safety in routine home and community activities.

A

VII

206
Q

Which rancho scale is this?

• Initiates and carries out steps to complete familiar personal, household,
community, work and leisure routines with stand-by assistance and can modify
the plan when needed with minimal assistance.
• Requires no assistance once new tasks/activities are learned.
• Aware of and acknowledges impairments and disabilities when they interfere
with task completion but requires stand-by assistance to take appropriate
corrective action.
• Thinks about consequences of a decision or action with minimal assistance.

A

VIII

207
Q

Which rancho scale is this?

• Able to independently think about consequences of decisions or actions but may
require more than usual amount of time and/or comepensatory strategies to
select the appropriate decision or action.
• Accurately estimates abilities and independently adjusts to task demands.
• Able to recognize the needs and feelings of others and automatically respond in
appropriate manner.
• Periodic periods of depression may occur.

A

X

208
Q

which rancho scale is this?

• Able to think about consequences of decisions or actions with assistance when
requested.
• Accurately estimates abilities but requires stand-by assistance to adjust to task
demands.
• Acknowledges others’ needs and feelings and responds appropriately with
stand-by assistance.
• Depression may continue.

A

IX

209
Q

which rancho scale is this?

• Depressed.
• Irritable.
• Low frustration tolerance/easily angered.
• Argumentative.
• Self-centered.
• Uncharacteristically dependent/independent.
• Able to recognize and acknowledge inappropriate social interaction behavior
while it is occurring and takes corrective action with minimal assistance.

A

VIII

210
Q

which rancho scale is this?

• Accurately estimates abilities but requires stand-by assistance to adjust to task
demands.
• Acknowledges others’ needs and feelings and responds appropriately with
stand-by assistance.
• Depression may continue.
• May be easily irritable.
• May have low frustration tolerance.
• Able to self monitor appropriateness of social interaction with stand-by
assistance.

A

IX

211
Q

Which rancho scale is this?

  • Shows carry over for relearned familiar tasks (e.g. self care).
  • Maximum assistance for new learning with little or nor carry over.
  • Unaware of impairments, disabilities and safety risks.
  • Consistently follows simple directions.
  • Verbal expressions are appropriate in highly familiar and structured situations.
A

VI

212
Q

which ranch scale is this?

  • Minimal supervision for safety in routine home and community activities.
  • Unrealistic planning for the future.
  • Unable to think about consequences of a decision or action.
  • Overestimates abilities.
  • Unaware of others’ needs and feelings.
  • Oppositional/uncooperative.
  • Unable to recognize inappropriate social interaction behavior.
A

VII