Final Exam Flashcards

1
Q

Papillon LeFevre inheritance pattern

A

autosomal recessive

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Papillon LeFevre affects which dentition

A

permanent and deciduous

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Papillon Lefevre loss of dentition?

A

yes, without treatment this is inevitable

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Papillon reversible?

A

yes, with aggressive treatment

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

treatment for gingival fibromatosis

A

gingivectomy, selective extractions, OHI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

isolated or generalized collagenous overgrowth of gingiva

A

gingival fibromatosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

describe gingival in gingival fibromatosis (texture, color)

A

firm, normal color

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

drug induced gingival hyperplasia can be treated by

A

removing offending medication
improving plaque control
surgical therapy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

gingival hyperplasia drugs

A

Nifedipine (procardia) and other Ca++ channel blockers
Dilantin (phenytoin)
Cyclosporine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

severity of gingival hyperplasia affected by

A

host susceptibility and level of OHI

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Desquamative gingivitis biopsy?

A

yes, incisional biopsy is indicated to confirm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

“sloughing of the gingival epithelium”

A

desquamative gingivitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

desqu. gingivitis is associated with

A

immune mediated vesiculobullous diseases

  • lichen planus
  • pemphigoid
  • pemphigus
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

desqu. gingivitis a diagnosis?

A

no, it is a clinical description

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

NUG

A
young-mid adults
punched out appearance of papillae
smell, spontaneous hemorrhage,
debride, CHX, OHI, broad spec abx if systemic involved
can spread to adjacent tissues
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

types of angioedema

A
  1. IgE mediated hypersensitivity
  2. ACEi use
  3. C1 INH deficiency
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

how quickly does angioedema resolve?

A

1-3 days

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

erythema with overlying shaggy hyperkeratosis, pain, burning arecharacteristic of

A

cinnamon reaction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

3 types of allergic contact stomatitis

A
  1. dentrifice related sloughing
  2. cinnamon mouth
  3. lichen amalgam reaction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Allergic reactions to systemic drugs - 2 types

A

fixed drug eruption- at site of administration

lichenoid drug reaction - medication induced, looks like lichen planus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Wegener’s Granulomatosis

A

necrotizing granulomatosis with vasculitis
subepithelial hemorrhage
lungs, kidneys, skin, mucosa
generalized, limited, and superficial types

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

generalized , limited, superficial Wegener’s

A

generalized: URT, LRT, kidneys
limited: URT, LRT
superficial: skin and mucosa

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

strawberry gingivitis associated with

A

Wegener’s granulomatosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

oral antral fistula and palatal ulcer seen with

A

Wegener’s granulomatosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Orofacial granulomatosis

A

abnormal immune rxn
NONnecrotizing granulomatous inflammation (Wegener’s is necrotizing)
NT swelling, persistent

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

2 types of orofacial granulomatosis

A
  1. cheilitis granulomatosis (lips only)

2. Melkersson Rosenthal (NT lip swelling, bell’s palsy, fissured tongue)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

characteristics of Melkersson Rosenthal

A

type of orofacial granulomatosis
fissured tongue
Bell’s Palsy
NT lip swelling

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Behcet’s syndrome

A

multisystem disorder
aphthous like ulcers
middle east and Japan
classic triad

29
Q

Behcet’s triad

A

genital ulcerations
oral ulcerations
ocular disease

30
Q

what is usually first manifestation of Behcet’s syndrome

A

oral ulcerations

31
Q

posterior uveitis seen in

A

Behcet’s syndrome ocular disease

32
Q

Behcet’s can involve multiple systems?

A

yes, CNS, GI, CV, etc

33
Q

Tx for recurrent aphthous ulcer

A

topical corticosteroid

34
Q

Herpetiform aphthous ulcers

A

small lesions, most numerous

recurrences likely and close together

35
Q

major aphthous ulcers

A

1-3cm
last longer, scarring
labial mucosa, fauces, soft palate

36
Q

minor aphthous ulcers

A

1-5 painful, non keratinzed mucosa
ERYTHEMATOUS HALO
preceded by erythematous macule w/ prodrome

37
Q

AIDS related Kaposi’s Sarcoma

A

multifocal malignancy of vascular endoth. origin
linked to HHV 8
half have oral involvement on palate or gingiva

38
Q

AIDS aphthous like ulcerations

A

painful, persistent, solitary or multiple

topical corticosteroids

39
Q

AIDS HPV

A

seen intraorally more in HIV patients
exophytic lesions
often multiple lesions

40
Q

parakeratosis and ballooning of cells of upper spinous layers of epithelium =

A

oral hairy leukoplakia

41
Q

oral hairy leukoplakia linked to what virus

A

EBV

42
Q

non removable white plaques on lateral tongue with vertical parallel lines

A

oral hairy leukoplakia

43
Q

Molluscum contagiosum in AIDS

A

caused by poxvirus

immunocompromised develop more lesions that don’t regress

44
Q

Necrotizing stomatitis

A

severe NUG presentation

painful, extensive tissue destruction affecting gingiva and alveolar mucosa, adj soft tissue and deeper bone structures

45
Q

HIV related periodontitis

A

pain, spontaneous gingival bleeding
interproximal necrosis, cratering
edema, erythema
rapid bone loss and soft tissue loss = NO POCKET

46
Q

deep pockets seen in HIV related periodontitis?

A

NO! Soft tissue loss accompanies bone loss = no deep pocket

47
Q

linear gingival erythema

A

gingivitis with unusual linear pattern on margin
may have spontaneous bleeding
no response to improved OHI

48
Q

most common immunocompromised oral fungal infection

A

candidiasis

49
Q

persistent lymphadenopathy is a key trait of

A

HIV

50
Q

AIDS = CD4 count

A

200

51
Q

HFM disease

A

enterovirus caused by coxsackie virus A (and echovirus and enterovirus)
shallow ulcer oral lesions
skin lesions - erythematous macules w/ central vesicle
supportive care

52
Q

Herpangina

A

enterovirus caused by coxsackie virus A, B, echovirus
kids age 1-4
oral ulcers in tonsil pillar / posterior soft palate

53
Q

Herpes Zoster

A

unilateral
older age
reactivation of VZV
painful erythema on trunk

54
Q

VZV

A
direct contact or air borne 
pruritis cutaneous lesions, rupture into crust
fever, malaise
shallow oral ulcers
acyclovir for tx
55
Q

when to give IV acyclovir

A

herpes in immunosuppressed

56
Q

recurrent intraoral herpes

A

uncommon
irritated, rough feeling
cluster of shallow ulcers confined to mucosa over periosteum (hard palate, attached gingiva)

57
Q

few people remember first primary herpes outbreak

A

true

58
Q

primary herpes

A

saliva spread
cervical lymphadenopathy, fever
oral lesions rupture - ulcers - serpentine borders

59
Q

dx primary herpes

A

exfoliative cytology

multinucleate and balloon degeneration of nuclei

60
Q

Tzanck cells

A

herpes

61
Q

neurotropic

A

HSV is neurotropic- spreads via nerves to sensory ganglia = becomes recurrent infection when reactivated from latent stage

62
Q

antral pseudocyst treatment

A

none, radiograph follow up

63
Q

how do you usually find an antral pseudocyst

A

radiographic finding ; floor of maxillary sinus ; infiltrate raises sinus mucosa

64
Q

along lingual mandible mylohoid ridge

A

oral ulceration with cortical bone sequestration

tx by removing dead bone

65
Q

smoker melanosis

A

melanocytes make pigment to protect against smoke

66
Q

foreign body tattoo biopsy?

A

yes, to r/o melanocytic lesion

67
Q

BRONJ more common in which kind of BP treatment

A

IV 90%

oral 10%

68
Q

MRONJ diagnosis

A
  1. current or previous tx with BP
  2. exposed maxillofacial bone for 8 wks
  3. no history of radiation to jaws
69
Q

ionizing radiation to head / neck region leads to

A

radiation mucositis