Final Exam Flashcards

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1
Q

DSM classification broadly identifies mood disorders as either:

A

Unipolar: a single depressive mood experience
Bipolar: involves mania and depression

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2
Q

2 separate categories of mood disorders

A

Depressive disorders
Bipolar and related disorders

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3
Q

Major depressive disorder (MDD) is defined by the presence of: (8)

A
  • one or more major depressive episodes or irritable mood episodes
  • loss of pleasure (anhedonia)
  • change in weight or appetite
  • sleep problems
  • fatigue or loss of energy
  • feelings of worthlessness or guilt
  • difficulty thinking or concentrating
  • thoughts of death or suicidal thoughts and behaviour
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4
Q

DSM diagnostic criteria of MDD

A
  • must have 6 out of 8 symptoms
  • symptoms must be present for at least 3 consecutive days and must last for at least 2 weeks
  • must cause individual clinically significant stress or impairment
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5
Q

Persistent depressive disorder (dysthymia) diagnostic criteria

A
  • same symptoms of MDD but symptoms are less severe and more chronic
  • symptoms must be present for at least 1 year
  • must be clinically significant and cause distress or impaired functioning
  • along with 2 or more of the following symptoms:
    Poor appetite or overeating
    Sleep disturbances
    Low energy or fatigue
    Low self esteem
    Difficulty concentrating or making decisions
    Feelings of hopelessness
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6
Q

When can MDD appear in children

A

Depression can start as early as preschool

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7
Q

What is the most prevalent form of affective disorder among children and adolescents?

A

Major depressive disorder (MDD)

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8
Q

Epidemiology of depression (clinical vs community)

A

-Clinical sample: rates in children range from 80% MDD
-Community sample: rates in children range from 0.4-2.5%
-Lifetime prevalence rates of diagnosable depressive disorders are 20-30%
-Typically occurs more in females than in males after the age of 12

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9
Q

SES, ethnic, and cultural considerations for depression

A
  • research suggests that lower SES associated with higher rates of depression
  • possible influences on income on MDD:
    Chronic stress (on mood and physical symptoms of MDD)
    Family disruption
    Environmental adversities
    Racial and ethnic discrimination
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10
Q

Biological influences on depression (genetics- family history of depression)

A
  • highly heritable but not the only factor
  • higher rates in first degree adult relatives
  • genetic effects may influence personality and temperament
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11
Q

Biological influences of depression (neurochemistry and brain functioning)

A
  • serotonin, norepinephrine, acetylcholine
  • builds on research that finds certain classes of medication are effective as antidepressants
  • low levels of serotonin and norepinephrine are results of too much reabsorption by the neuron and the breakdown of neurotransmitter too efficiently
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12
Q

Social-psychological influences on depression (2)

A

Separation and loss
- can produce adverse circumstances including lack of care, changing in family structure, socioeconomic problems

Cognitive and interpersonal perspective
- interpersonal skills, cognitive distortions, views of self, control beliefs, self regulation, and stress
- the way a person relates to others, is viewed by others, and view themselves contributes to how depression developed and/or is maintained

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13
Q

Impact of parental depression

A

Children from homes with a depressed parent are:
- greater risk of developing MDD and other disorders
- less likely to get treatment

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14
Q

Garber and Flynn (2001): longitudinal study of children with depressed mothers - findings

A

Possible that behaviour of depressed parents may be accompanied by anger, frustration, and hostility
- alters the parent’s ability to parent effectively
- parents may be detached, withdrawn, and inattentive
- depressed behaviour is maintained by parent-child interactions

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15
Q

Assessments of depression

A

Self report measures
- children’s depression index (CDI)
- revised children’s anxiety and depression scale

Parental/teacher measures
- behaviour assessment system for children (BASC)

Observations and clinical judgment

For children, might not use the DSM; would instead use evidence based on that particular child

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16
Q

Treatment of depression (medications and CBT)

A

Medications
- past: tricyclic antidepressants
- present: SSRIs and second-generation antidepressants

CBT and interpersonal psychotherapy
- challenge maladaptive thoughts and negative attributions, teach problem solving/coping skills
- understand interpersonal issues and problem solving

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17
Q

Bipolar disorder definition

A

Involves the presence of mania as well as depressive symptoms

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18
Q

Mania and euphoria definitions

A

Mania: period of abnormally elevated euphoric mood
Euphoria: characterized by inflated self esteem, high rates of activity, speech and thinking, distractibility, exaggerated feelings of physical and mental well being

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19
Q

DSM-5 criteria for manic episode

A

Persistent elevated, and expansive or irritable mood
3 of the following:
- inflated self esteem
- decreased need for sleep
- more talkative than usual
- thoughts racing
- distractibility
- psychomotor agitation
- excessive pleasure seeking

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20
Q

Types of bipolar disorder

A

Bipolar I: involves a history of MDD and mania
Bipolar II: involves a history of MDD and hypomania (euphoric mood that is shorter in duration - about 4 days - and less severe than manic episodes)
Cyclothymic disorder: chronic but less severe fluctuations in mood. Does not meet criteria for depression or BPD

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21
Q

Comorbidity of bipolar disorder and ADHD?

A

60%-90% of children

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22
Q

FIND (frequency, intensity, number, duration) criteria for BD (Kowatch et al. 2005)

A

-Exceedingly happy or silly (no apparent reason)
- Intense outbursts or anger/hostile
-Frequent irritable moods
-Less sleep than usual (full of energy)
-State of grandiose views of their abilities and plans
-Intense concentration on activity but becomes increasingly disorganized
-Rapid/unintelligible and difficult to follow speech
-Flight of ideas
-Poor judgement

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23
Q

Epidemiology of BD - Blader and Carlson (2007)

A

National representative sample (0-19yo) of doctor visits for mental health related issues = 6.67% in 2002-03
Between 1996-2004: children = 1.4 to 7.3 per 10000; teens = 5.1 to 20.4 per 10000

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24
Q

Prevalence of BD

A

0-6% in a community sample of children and adolescents
Distribution of males and females are equal
No significant cultural/ethnic differences
Less prevalent in prepubertal youth

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25
Q

Diagnostic challenges of BD

A

-Same diagnostic criteria are used for adults and children
-Children tend to have very short episodes, very frequent mood shifts, mixed mood, chronic difficulty in regulating moods
-Children present with co-morbid problems
-Mania in adolescents - associated with antisocial behaviour

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26
Q

BD developmental course

A
  • in some children, major depressive disorder may be an early stage of bipolar disorder —> more likely in those youngsters with an earlier onset of depression
  • experience relatively early onset of affective difficulties
    (Median duration for manic episode = 10.8 months; first affective episode = 11.75 years)
  • retrospective and prospective studies suggest that these children continue to show symptoms of affective disorders, social and academic impairment
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27
Q

Risk factors and stoplights of BD

A

Family history of bipolar disorder (genetics)
- biological siblings and parents (immediate family members)
- adult twins and adoption demonstrate a strong genetic component
- multiple genes affecting amygdala and hormonal and neurotransmitter process, but shared with other disorders

Environmental stressors
- stressful life events, family relationships, parenting styles

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28
Q

Assessment of BD

A

Structured diagnostic interview: K-SADS
Mania scales: Young Mania Rating Scales and GBI
Parent/teacher/self-interviews

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29
Q

Treatment of BD

A

Pharmacotherapy is the first and most effective treatment
Family involvement in treatment is very important

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30
Q

Conduct problems listed in the DSM-5

A

Oppositional defiant disorder
Conduct disorder
Intermittent explosive disorder
Antisocial personality disorder
Pyromania and kleptomania

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31
Q

Antisocial personality disorder

A

Pervasive pattern of disregard of and violation of rights of others
Met criteria for CD before the age of 15
ADHD—> ODD —> CD—> APD

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32
Q

Oppositional defiant disorder - DSM-5 criteria

A

A pattern of negativistic, hostile, and defiant behaviour
Under age 5: symptoms have to occur on most days. Over age 5: symptoms have to occur at least once a week
Has to impair functioning (ex. Academically, socially)
At least 3 of these behaviours present for at least 6 months:
- loses temper
- touchy and easily annoyed
- angry and resentful
- argues with adults
- actively defies or refuses to comply with adult request/rules
- deliberately annoys others
- blames others for own mistakes
- is spiteful or vindictive

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33
Q

Conduct disorder - definitions

A

A pattern of repetitive and persistent acts of violence that violates the basic rights of others and goes against societal norms
Mild, moderate, or severe
Limited prosocial emotions (feeling guilty or not)

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34
Q

DSM-5 symptoms of conduct disorder

A

At least 3 of these behaviours are present during the last 12 months with at least one of them present in the last 6 months:
Aggression toward people or animals
- bullies, threatens, or intimidates
- initiates physical fights
- has used a weapon
- is physically cruel to people or animals
- has stolen while confronting a victim
- has forced someone into sexual activity

Destruction of property
- engaged in fire setting with the intent to cause damage
- has deliberately destroyed other’s property

Deceitfulness or theft
- has broken into house, building, or car
- often lies to obtain goods or favours or to avoid obligations
- has stolen items of nontrivial value without confronting victim

Serious violation of rules
- stays out at night despite parent prohibitions
- has run away from home overnight at least twice
- is often truant from school - beginning before age 13

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35
Q

Empirically derived externalizing behaviours associated with CD (2 types of behaviours)

A

Two syndromes:
- aggressive behaviour: argues a lot, destroys things, purposefully disobedient, fights
- rule-breaking behaviour: breaks rules, lies, steals, is truant

Children can exhibit one or both of these problems
Aggressive behaviour carries a higher degree of heritability than rule breaking behaviour

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36
Q

Gender differences in conduct problems

A

Expressed differently in boys and girls
Aggression in boys:
- defined as an intent to hurt or do harm to others (ex. Hitting, pushing, or threatening behaviours)
Aggression in girls:
- more directed to hurting another person’s feelings (relational aggression)
- ex. leaving someone out of play/excluding from peer group, telling a person you won’t like them unless they do as you say, lying about someone so others won’t like her

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37
Q

Epidemiology of conduct problems

A

Rate 1%-15% of ODD
Rate 2%-10% of CD
Comparable across countries

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38
Q

Developmental paths of conduct problems

A

Loeber’s 3 pathways model:
Authority conflict
- stubborn behaviour, defiance, disobedience, truancy
Covert
- property damage, lying, fire setting, theft, burglary
Overt
- physical violence, bullying, and sever forms of violence (rape, attacks, strong arm)

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39
Q

Aggression as a learned behaviour

A

Children can clearly learn to be aggressive by being rewarded for such behaviour
Can also learn through exposure to aggressive models - vicarious learning
Can build the child’s repertoire and lead to disinhibition of aggression
- family violence is a source of modelled aggression
- parents who punish their children physically serve as models of aggressive behaviour

Children who exhibit excessive or antisocial behaviours - likely to have siblings/parents/grandparents with histories of conduct problems and records of aggressive and criminal behaviour

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40
Q

Family variables in conduct disorder

A

Low family SES
Marital disruption
Poor quality parenting
Parental abuse and neglect
Parenting style
Handing down non-effective parenting from generation to generation
Stressors that impact parenting quality
Parental psychopathology: alcoholism or other parental psychopathology can impact parental effectiveness

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41
Q

Biological influences of CD

A

Aggressive and conduct disorder related behaviours tend to run in the family
Longitudinal studies suggest a genetic component to CD
Inherited characteristics (body build, temperament, sensitivity to alcohol, irritability, sensation seeking, impulsivity) make an individual prone to conduct disorders
- mediated by social conditions, family variables, and social learning experiences

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42
Q

Assessment of CD

A

Parent completes Likert scale
36 items for CD

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43
Q

Steps in the cognitive processing of social-emotional cues (CD) (5 steps)

A

Encoding (looking for and attending to aggression)
Interpreting (misinterpreting of own and others’ emotions)
Looking for alternative responses
Selecting a specific response
Enacting the response

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44
Q

Youth with conduct problems display these problematic social-cognitive processes: (5)

A

Poorer problem solving skills
Attribute hostile intent to neutral actions
Generate fewer responses - and those generated are aggressive
Expect that aggressive responses will produce positive outcomes
Label arousal in conflict situations as “anger”

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45
Q

Pharmacological interventions for ODD and CD

A

With co-morbid ADHD: stimulant medications
Mood stabilizers: for extreme aggression and conduct disorder behaviours but works best with parent training and other interventions

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46
Q

Parent training interventions for ODD and CD

A

Best option!
Common features of parent training programs:
- how to give commands
- how to reinforce behaviours
- how to discipline and ignore
- prepare for difficult situations

Incredible Years Training Series
- parenting skills for use with children diagnoses with ODD and CD
- comes with standardized videos to model parenting skills

Cognitive problem solving skills
- targets the interpersonal and social-cognitive aspects of CD behaviour
- used in children 4-8 years of age
- taught to cope with stressful situations
- taped vignettes and discussions
- uses child sized puppets, colouring books, and cartoon stickers and prizes

Parent and child programs are superior in combination

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47
Q

3 types of ADHD

A

ADHD predominantly inattentive subtype
ADHD predominantly hyperactive/impulsive subtype
ADHD combined type

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48
Q

ADHD diagnoses are provided to those who: (5)

A

Symptoms are present before the age of 12
Symptoms are displayed for at least 6 months
Behaviours go beyond that expected developmentally
Impairment identified in social/academic functioning
Symptoms must occur in at least 2 different settings

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49
Q

ADHD primary & secondary features

A

Primary features:
- inattentive
- hyperactive/impulsive

Secondary features:
- motor skills
- intelligence
- cognition (executive functioning skills)
- adaptive functioning skills
- social behaviour
- sleep (lack of sleep)
- accidents (very accident prone)

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50
Q

Parents and teachers often report that children who are inattentive: (5)

A

Jump around from one task to another
Does not attend to what is being said
Is easily distracted
Daydreams
Has difficulty concentrating

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51
Q

Confirmed ADHD children pay less attention to their peers; they have a reduced capacity for: (2)

A

Selective attention
Sustained attention

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52
Q

Parents and teachers often report that hyperactive children are: (3)

A

Restless
Fidgety
Unable to sit still

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53
Q

Objective measures of hyperactivity can be measured using:

A

Actigraphs: little accelerometers that register movements in different planes

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54
Q

What time of day does hyperactivity increase?

A

In the afternoon

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55
Q

Parents and teachers often report that impulsivity in children appears as:

A

Difficulty controlling their own behaviour
Ex. The child might act without thinking, interrupt others, cut in line, engage in dangerous behaviours, appear careless and irresponsible, immature and rude

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56
Q

Motor coordination problems in children with ADHD (4) + how many children diagnosed with ADHD have these problems?

A

Clumsiness
Delay in motor milestones
Poor performance in sports
Difficulties with fine motor coordination and timing (particular difficulty in tasks involving complex movements and sequencing, like dancing)

About 50% for children with ADHD have motor coordination problems

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57
Q

Intelligence and ADHD

A

Overall, children perform worse on intelligence tests
Many have co-morbid learning difficulties/disorders
Reduced academic achievement
56% need tutoring
30% have to repeat a grade
30-40% end up I’m at least 1 special education placement
10-35% fail to graduate high school

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58
Q

Cognition and ADHD

A

Neuropsychological deficits cluster around executive functioning skills (ie. inhibition, working memory, sustained memory, etc.)
Executive functioning skills include those involved in goal-directed behaviour and are involved in planning, organizing, and self-regulation

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59
Q

Adaptive functioning skills and ADHD

A

Deficiencies in everyday adaptive functioning skills (ie. brushing teeth, washing their face, getting ready for school)
Deficits are in doing rather than knowing
Parallel with clumsiness

60
Q

Social behaviour and ADHD

A

-Quickly alienates themselves from peers —> worse if the child is non-compliant and aggressive
-Can correct social behaviours but are disorganized, impulsive, or distracted during social activities
-May not adequately process social emotional cues
-Overrates peer relationships as positive

61
Q

Sleep problems associated with ADHD (3)

A

Problems falling asleep
Needing less sleep
Involuntary movements (leg restlessness, grinding teeth)

62
Q

Accidents and ADHD

A

Children with ADHD tend to have more accidents and injuries (broken bones, bruises, lost teeth, poisonings)
Parents report that children with ADHD are inattentive in risky situations and unaware of consequences of their actions (ie. walking into traffic)

63
Q

ADHD epidemiology

A

Marked increased over the last 20 years
5-9% in school aged children
2-7% in community based samples and similar rates around the world
Assessments using evidence based measures estimate about 20% of children have clinically significant concerns
Presentation changes over time
Diagnosis usually occurs in elementary school aged children and decrease into high school and beyond
Some suggestions of differences in prevalence across social class, ethnicity, and culture

64
Q

ADHD gender ratios (general & clinical population)

A

2:1 or 3:1 (boys:girls) in the general population
4:1 (boys:girls) in a clinical population

65
Q

ADHD presentation in boys and girls

A

Boys: aggressive and antisocial behaviours, excessive running, climbing, escaping, etc.

Girls: inattentive and disorganized
- girls often do not meet diagnostic criteria on DSM
- can go undiagnosed and miss out on early intervention
- girls show many secondary features: executive functioning difficulties, academic problems, negative peer evaluations, higher rates of anxiety, mood disorders, and conduct problems

66
Q

Developmental course of ADHD

A

Symptoms emerge early and children do not often “outgrow” them (but sometimes do)
Community based sample of low income boys between ages 2-10 show 4 developmental pathways:
- chronic
- medium
- moderate
- low

67
Q

What what thought to be the primary cause of ADHD which we now know is not true?

A

Brain damage

68
Q

Which parts of the brain are smaller in children with ADHD? (3)

A

Right frontal corticies
Caudate nucleus
Globes palidus

69
Q

Neurobiological mechanisms of ADHD

A

ADHD involves the under-arousal of frontal cortices
ADHD involves dopamine and norepinephrine deficiencies
ADHD is heterogeneous- there are a number of expressions of symptomology that is reflected in the disruption of different brain regions or circuitry
But the underlying neuropathology is still unclear

70
Q

What is the genetic rate of ADHD in first-degree family?

A

10-35%
Strong genetic component!

71
Q

Environmental factors that influence ADHD

A

Prenatal exposure to maternal smoking and alcohol has been linked to the risk of ADHD in the offspring
Exposure to lead has been implicated in disrupting the development of brain areas associated with the control of attention/activity level and executive functioning skills

72
Q

Assessments of ADHD need to be broad based and include: (4)

A

Developmental/family history
Perinatal period/acquisition of milestones
Examination of home/school environments
Examination of co-occurring disorders

73
Q

Measuring Sustained Attention using the Continuous Performance Test (visual and auditory)

A

Used in the diagnosis of ADHD in children and adults
Involved monitoring a continuous stimulus set for the Occitan ce of a target
Typical results in ADHD: make errors in omission and commission

74
Q

Pharmacotherapy treatment for ADHD

A

In 1937, Bradley and co observed that Benzidine improved the behaviour of children in an inpatient residential care ward
Most commonly used medications are stimulants: methylphenidate, dextroamphetamines, and amphetamine
Ritalin: peaks 2 hours and out of the system within 4.5 hours
Concerta: peaks in 4 hours and out of the system within 8 hours
Adderall: blend of dextroamphetamines and amphetampnes
Strattera: norepinephrine reuptake
Medications have limited time releases

75
Q

Behavioural treatment strategies for ADHD (3)

A

Consequences for behaviour:
- positive behaviour —> lead to desired social or play activities
- negative behaviour —> lead to loss of opportunities, time out, loss of earned reinforcer

Parent training programs:
- teach parents child management techniques (for 4-12 year olds)

Classroom management:
- working with teachers and administrators to set up a code of conduct and consequences

76
Q

ADHD treatment concerns - stimulants

A

Don’t work on 10-20% of cases and behavioural/inattention changes reach optimal levels in only 1/2 of the children who are responders
Biological side effects: sleeping problems, decreased appetite, stomach pains, headaches, irritability, jitteriness, growth suppression
Increase risk of child’s addiction to medication/recreational drugs?

77
Q

Multimodal treatment study of ADHD

A

Ages 6-9
600 kids total
4 treatment groups: medication only, behavioural interventions, combined treatment (medication and behavioural), community care (treatment as normal)
Plus a normal comparison group (no ADHD)
Measured core adhd symptoms at several time points
First 14 months was most intense (weekly check-ins, medication, teacher follow ups)
Findings:
- medication alone and the combined treatment were both better than behavioural alone and community based treatment
- medication is most effective, and the addition of behavioural treatments have additional benefits, but behavioural treatments alone are not as effective
- if a child has both ADHD and anxiety, behavioural treatment was just as effective as medication and combined treatments
- high income families benefited the most from combined treatment

78
Q

Receptive language

A

The comprehension of language used by others

79
Q

Expressive language

A

The production of language

80
Q

Language development is shaped by:

A

The environment (ex. Immigration and bilingualism)

81
Q

Language development - 6 months (receptive & expressive)

A

Receptive: turns to source of sound, startles in response to sudden sounds, watches your face as you talk
Expressive: makes different cries for different needs, imitates coughs or other sounds

82
Q

Language development - 9 months (receptive & expressive)

A

Receptive: responds to their name, understands being told “no”
Expressive: gets what they want through sounds and gestures, plays social games with you, repeats babble sounds

83
Q

Language development - 12 months (receptive & expressive)

A

Receptive: follow simple 1-step directions, look across the room to something you point and look at
Expressive: uses three or more words, uses gestures to communicate - “waves”, gets your attention using sounds, gestures, and pointing while looking at your eyes

84
Q

Language development - 24 months (receptive & expressive)

A

Receptive: follows 2-step directions
Expressive: uses more than 100 words, uses at least 2 pronouns “you” “‘me” “mine”, combines two or more words in short phrases

85
Q

Language development - 30 months (receptive & expressive)

A

Receptive: begins taking short turns with other children in reciprocal communication, shows concern when another child is hurt or sad
Expressive: uses adult grammar - “two cookies” “I jumped”, uses more than 350 words, uses action words “run” “spill” “fall”, produces words with two or more syllables “com-pu-ter”

86
Q

Language and communication disorders are usually associated with one or more of these 6 specific language impairments:

A

Graphemes
Phonemes
Syntax
Grammar
Semantics
Pragmatics

87
Q

The DSM-5 classification of language disorders has a distinction between:

A

Whether the child has deficits in expression and/or receptive language

88
Q

Speech sound disorder

A

Child fails to display developmentally appropriate and dialect appropriate speech sounds
- difficulty articulating speech sounds
- fail to display age-appropriate speech sounds
- course of speech sound delayed
- incorrect speech sounds
- misarticulation
- denotes

89
Q

Receptive-expressive disorder (language)

A

Difficulties comprehending the communication or others and difficulties with expressing language
- difficulties with comprehension and production of language
- may present as silent (mistaken for selective mutism)
- may not respond to speech
- may respond inappropriately to others’ speech

90
Q

Expressive disorder (language)

A

Standardized assessments reveal expressive deficits that fall below nonverbal intelligence and level of receptive language or comprehension
- production of language is abnormal in standardized measures
- limited amount of speech, small vocabulary, parts of sentences may be missing, generate unusual words, make errors is using plurals and verb tenses

91
Q

Epidemiology of language disorders

A

Overall, rates of language impairment range from 3-7% of children in the general population
Boys are reported as having higher rates than girls
Language disorders usually appear by 3-4 years
- but some impairments may only become evident with increased demand in schoolwork and language complexity

92
Q

Types of learning disabilities (4)

A

Language disorders
- impairments in language expression and reception

Reading disabilities
- deficits in the ability to discern the meaning from words in text

Written expression disabilities
- deficits in the ability to transcribe and generate text

Mathematics disabilities
- deficits in numerical understanding, learning, representations, and retrieval of basic arithmetic facts

93
Q

Guidelines for diagnosing if a child’s language and learning skills are below expectations

A

Discrepancy between IQ and academic achievement level
Performance on a measure of general cognitive capacity (IQ) significantly exceeds performance on a specific test of achievement
The discrepancy of 2 or more standard deviations between IQ and achievement scores is required

94
Q

How learning disorders are identified in the school system (3 levels)

A

Core instruction
- 75-90% (level I)

Supplemental
- 10-25% (level II)

Intense
- 2-10% (level III)

95
Q

Learning disorders vs learning disabilities

A

Learning disorders: these disabilities are specific deficits that appear discrepant with intelligence and other abilities
Learning disabilities: refer to specific developmental problems in reading, writing, and arithmetic

96
Q

Learning disorders have the highest comorbidity with ?

A

ADHD

97
Q

DSM-5 classification- reading disorders

A

Involves deficits in the ability to discern the meaning from words in running lines of text
A number of defects in processes are involved:
- visual-perceptual deficits
- phonological processing
- phonological awareness
- irregular word forms
- syntax, semantic components, and working memory

98
Q

Reading disorder prevalence

A

4-10% of school aged children
3:1 or 4:1 (boys:girls)
High comorbidity with ADHD and CD (3.5% with both)
Difficulties can emerge around grade 4
Difficulties can persist into adolescence and adulthood

99
Q

DSM-5 classification- disorder or written expression

A

Children will have difficulty in transcription and text generation or composition
Transcription involves putting ideas into written form
Deficiencies in poor handwriting, spelling, punctuation, capitalization, and word placement
Handwriting: fine motor skills
Spelling: draws on phonetic skills, word recognition, retrieval of learned letters, and words from memory
Text generation: memory for words, understanding sentence structure, higher order executive functioning, and meta cognitive skills

100
Q

Disorder of written expression prevalence

A

6-10% of school aged children

101
Q

DSM-5 classification - mathematic abilities

A

Involves the understanding of numbers and the learning, representation, and retrieval of basic arithmetic facts
In younger children, arithmetic abilities build on skills in understanding number, numerosity, and counting

102
Q

Mathematic disability prevalence

A

5-8% of school aged children

103
Q

Learning disorders- social and emotional problems (4)

A

Children often have difficulties with peers (less popular and face rejection)
Have fewer friends, lower quality friendships, and higher levels of loneliness
Have lower social competence than peers
Have difficulties identifying emotions, understanding social situations, and in social problem solving

104
Q

Learning disorders- self concept and motivation

A

Learning disorders associated with a lowered sense of self worth
Compared to same aged peers, students with learning disorders report more helplessness, lower self esteem, even when their school grades are comparable
Consequences: academic failure —> give up in the face of difficulty —> experience more failure —> reinforces belief in their lack of ability and control —> academic failure…

105
Q

Assessments for language and learning disabilities

A

Need to identify discrepancy between language or learning domain and IQ
Uses standardized testing
- Academic standardized testing
- language specific testing

106
Q

Intellectual disability definition

A

Intellectual disability is characterized by significant limitations both in intellectual functioning and in adaptive behaviour as expressed in conceptual, social, and practical adaptive skills. This disability originates before age 18.
-AAIDD

ID is linked to the social environment and level of supports that should be assessed

107
Q

Intellectual disability levels and approximate IQ range (4)

A

Mild: 50-70
Moderate: 35-50
Severe: 20-35
Profound: below 20

108
Q

Changes in the educational policy of ID have made educators focus more on _____ than _____

A

Focus more on functional descriptions of the child’s needs than IQ

109
Q

Intelligence definition

A

Intelligence involves the knowledge possessed by a person, ability to learn or think, or the capacity to adapt to new situations.
General ability, g, plus other abilities (motor, verbal)

110
Q

Generally, intelligence is measured according to____

A

How individuals process information

111
Q

Measure by IQ is ____ and _____

A

Stable and valid

112
Q

Adaptive functioning definition

A

What people do to take care of themselves/ relate to others in daily living
Conceptual skills, social skills, and practical skills

113
Q

Adaptive functioning skills - younger children

A

Sensorimotor behaviours
Communication
Self help
Primary socialization skills

Ex. Can they: sort cups into different sizes? Organize by colour? Zip up their own zipper? Wave? Use a spoon to feed themselves? Say hi or bye? Follow directions? Ask for help?

114
Q

Adaptive functioning skills - older children

A

Reasoning/ judgements about the environment
Reasoning/ judgement about social relationships

Ex. Can they: determine something looks dangerous? Recognize social norms? Dress themselves? Understand they need a coat because it’s cold out?

115
Q

Developmental outlooks of the 4 ID levels

A

Mild:
- usually develops social and communication skills in preschool
- 6th level grade by late teens
- may need guidance but often successful in the community

Moderate:
- usually develops communication skills in early childhood
- unlikely to progress beyond 2nd grade
- can adapt to supervised community living

Severe:
- may learn to talk at school age
- limited ability to profit from pre-academic training
- can adapt to community living

Profound:
- often has a neurological condition
- sensorimotor impairments
- requires constant supervision

116
Q

Rates of other physiological issues in individuals with ID…

A

May be 2-4x that found in the general population
Most common are ADHD and ODD/CD

117
Q

Difficulties with identifying co-occurring disorders with ID

A

Overshadowing
Cognitive/communication impairments of ID make it difficult to identify
Criteria do not apply well to lower levels of disability

118
Q

Epidemiology of ID

A

1-3% of the population has ID, which is greater than predicted by normal distribution
Prevalence of ID depends on age and severity of ID
Gender and SES also influence rates of ID

119
Q

Pathological influences of ID

A

Biological condition accounting for ID
Genetic processes, prenatal/perinatal/postnatal issues, or a combination of factors

120
Q

Multi genetic influences of ID

A

At least 50% of variance in intelligence due to transmission of multiple genes
More likely for mild ID

121
Q

Psychosocial influences of ID

A

Higher prevalence of ID in lower SES
- possibly due to less access to good nutrition, which is really important for development

122
Q

ID genetic syndromes - Down syndrome

A

Most common (1/800-1200)
Trisomy 21 - error during meiosis
Reduced brain size, reduced number/density of neurons, abnormal dendrites
Moderate to severe disability
Deficits in short-term memory/ auditory processing, delayed speech acquisition; good visual-spatial abilities

123
Q

ID genetic syndromes - fragile X syndrome

A

1/4000 males, 1/6000 females
Moderate to severe ID
Weakness in visual-spatial cognition, sequential information processing, etc.
Many boys also have ASD

124
Q

ID genetic syndromes - Williams syndrome

A

1/777-20000
Small deletions of several genes on chromosome 7
Mild to moderate ID
Abnormal brain activation during tasks involving response inhibition, visual processing, and auditory processing of music/noise
Deficits in short-term visual-spatial memory; short-term verbal memory and verbal ID typically stronger
Excessively outgoing/friendly; generalized anxiety/ specific phobias

125
Q

ID genetic syndromes - Prader-Willi syndrome

A

1/10000-15000
Micro deletion of chromosome material - genomic imprinting
Abnormal functioning of hypothalamus/serotonin
Hyperphagia (eating everything) is the leading cause of death
IQ often >70
Relative strengths in spatial-perceptual organization and visual processing; relative weakness in short-term motor/auditory/visual memory
Skin picking, concerns with exactness/order/cleanliness/sameness in environment (highly co-morbid with OCD)

126
Q

Developmental assessments for ID (2)

A

Bayley scale
- used in children 1-42 months
- sensorimotor focus
- developmental index rather than ID
- identifies developmental delays

Mullens scale of early development
- used in children 0-48 months
- expressive, receptive, gross motor, fine motor, visual reception
- developmental index
- assess early ID
- ex. Putting different sized cups into each other, matching shapes to their places, counting blocks

127
Q

Intelligence tests for ID

A

Wechler scales of intelligence
Stanford Binet 5th edition
- fluid reasoning, general knowledge, qualitative reasoning, visual spatial processing, and working memory
Leiter international performance scale 3rd edition
- completely nonverbal measure of intelligence for nonverbal individuals
- used in cognitively delayed, non-English speaking, hearing impaired, speech impaired, or individuals with ID
- tests 3-75 years

Individuals with ID typically fall below 70 for IQ (well below average)

128
Q

Adaptive functioning assessment domains (Vineland adaptive behaviour scales) (5)

A

Communication domain
- receptive, expressive, written
Daily living skills domain
- personal, domestic, and community
Socialization domain
- interpersonal relationships, play and leisure, coping skills
Motor skills domain
- gross and fine motor
Maladaptive behaviour domain
- maladaptive behaviours index

129
Q

Dual diagnosis

A

Refers to an individual with a mental health disorder and a co-occurring developmental disability
Approximately 1-3% of Canadians have a developmental disability, but prevalence rates of a dual diagnosis are really hard to determine
Individuals with a DD often struggle to articulate symptoms related to a mental health disorder

130
Q

Treatments of IDs

A

No direct treatments to get rid of the disorders, but programs to help build daily living skills that may allow them to live independently

131
Q

Autism core challenges and features (3)

A

Communication and social interaction difficulties
Subscribe to specific interests
Repetitive behaviours

132
Q

Autism - additional challenges that interfere with functioning (3)

A

Sensory difficulties (ex. Loud noises, hates the feeling of jeans on legs)
Cognitive skills (those with higher IQ have fewer autistic symptoms)
Motor skills

133
Q

Prevalence rates of autism

A

1 in 68 children
Comparable rates between socioeconomic, ethnic, and racial groups
1:4 girls to boys (but this is changing)

134
Q

Reasons for increasing prevalence of autism (4)

A

Awareness
Widening criteria for diagnosis
Diagnosis substitution
Environmental factors

135
Q

Historical contest of autism (Leo Kanner and Hans Asperger)

A

Leo kanner:
Worked in a psych ward; found that some individuals (mostly boys) had very similar major characteristics which included communication difficulties, echolalia, language difficulties, high desire for repetition, very reactive to loud noises

Hans Asperger:
- worked in a psych ward; observed boys that had very similar behaviours, including those above, with the differences of average intelligence and average language

136
Q

DSM-5 states that ASD is characterized by:

A

Persistent deficits in social communication and interactions
Presence of restricted, repetitive, and stereotyped behaviours or interests

137
Q

Core diagnostic symptoms of ASD

A

Social communication and interaction deficits (language use, nonverbal behaviours, communicative intent)
Repetitive, restrictive, and stereotyped interests
Inflexible and non-functional routines and rituals
Insistence on sameness
Repetitive motor mannerisms

138
Q

Diagnostic criteria of ASD

A

Impairments must be present in early developmental period
Clinically significant impairments (functional, interferes with quality of life)
A global developmental delay or intellectual disability can co-occur, but not always
- majority of individuals on the spectrum have average or above average IQ

139
Q

Asperger’s in the DSM-4 vs now

A

DSM-4:
No clinically significant delay in language
Higher cognitive ability
Fewer presentations of core symptoms

Now:
No longer recognized by the DSM-5

140
Q

Psychological and behaviour characteristics of ASD (not part of diagnostic criteria) (7)

A

Sensory and perceptual sensitivities (hyper or hypo)
- auditory, visual, smell, touch, synesthesia

Intellectual challenges and strengths
- 40-50% have some associated ID (IQ<70), higher IQ associated with decreased impairments

Emotional and behavioural challenges
- fear, aggression, hyperactivity, self-injurious behaviour

High co-morbidity (70%)
- ADHD, anxiety and depression, OCD, substance use, etc.

Adaptive functioning skills
- daily living skills, gross and fine motor skills, self-help skills, communication and social skills

Splinter skills
- music, mathematic, spelling; 25% with splinter skills

Savant skills (autism savant syndrome)
- remarkable skill/talent, exceeds typical development, 5% display savant skills

141
Q

Developmental pathway of ASD

A

Heterogeneous
- core symptoms may improve (but not disappear)
- features fluctuate
- mental health challenges

142
Q

ASD prevalence

A

1 in 59 children
4:1 boys to girls

143
Q

ASD interventions

A

Pharmacological:
- targets associated behavioural features, but not core features

Behavioural intervention:
- most supported treatment
- targeted (specific deficits) or comprehensive (numerous features)

Discrete trial training (DTT):
- most common behavioural approach
- teaches complex skills by breaking them down into smaller discrete components or skills

Psychosocial interventions:
- focus on improving social skills and emotional functioning

144
Q

At what age are autism symptoms often recognized?

A

Age 2
However, age 4-5 is the average age of diagnosis

145
Q

Neurobiological impairments in ASD

A

Altered brain growth, most implicated in the frontal lobe, temporal lobe-limbo system, and cerebellum