Final CPC Flashcards

1
Q
A

Cleft Lip

Failure of fusion of the medial nasal process and the maxillary process

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2
Q
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Bifid Uvula

Failure of fusion of the palatal shelves

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3
Q
A

Ashers Syndrome

Blepharochalasis, double lip

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4
Q
A

Fordyce Granules

Ectopic sebaceous glands

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5
Q
A

Erythema Migrans

Yellow serpentine boarder

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6
Q
A

Leukoedema

Stretch it to see if it goes away

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7
Q
A

Anklyoglossia

Tongue tied. May not treat because it can tear on its own (This is a good reason to cut it though)

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8
Q
A

Lingual Thyroid

Don’t remomve because it might be the only source of T3 and T4 in the body

Foramen Cecum

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9
Q
A

Condylar Hyperplasia

Deviates to the same side of pathology

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10
Q
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Fissured Tongue (Scrotal Tongue)

Commonly seen with geographic tongue

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11
Q
A

Geographic Tongue

(Benign Migratory Glossitis)

Acute response

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12
Q

Syndrome?

A

Paramedian Lip Pits

Vander Woude Syndrome

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13
Q
A

Lingual Verocosities

Avoid severing

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14
Q
A

Buccal Exostosis

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15
Q
A

Basal Cell Carcinoma

Telangiectasia

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16
Q
A

Calibers Artery

Avoid severing this

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17
Q
A

Condensing Osteitis

Notice the tooth insult

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18
Q
A

Ideopathic Osteosclerosis

Notice there is no tooth pathology

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19
Q
A

Coronoid Hyperplasia

Movement on same side because the coronoid process interferes with the zygomatic bone

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20
Q
A

Torus Palatinus

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21
Q
A

Submandibular Tori

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22
Q
A

Lymphoepithial Cyst

Location on lymphoid tissue

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23
Q
A

Nasopatine Duct Cysy

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24
Q
A

Pierre Robin Sequence

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25
Q
A

Abfraction

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26
Q
A

Errosion (Corrosion)

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27
Q
A

Attrition

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28
Q
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Abrasion

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29
Q
A

Turner’s hypoplasia

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30
Q
A

Dens-en-Dente

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31
Q
A

Taurodontism

Klienfelters Syndrome, Turners Syndrome, Amelogenesis Imperfecta

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32
Q
A

Dentin Dysplasia Type 1

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33
Q
A

Residual Cyst

Notice how there is a missing tooth

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34
Q
A

Ludwigs Angina

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35
Q
A

Cavernous Sinus Thrombosis

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36
Q
A

Herpes

On fixed gingiva

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37
Q
A

Apththous Ulcer

Moveable mucosa

38
Q
A

Mucocele

39
Q
A

Sialolilith

40
Q
A

Necrotizing Sialometaplasia

41
Q
A

Salivary Gland Neoplasm

42
Q
A

Fibroma

43
Q
A

Retrocuspid Papilla

No treatment

44
Q
A

Epulis Fissuratum

Histologically, it is the same as inflammatory fibrous hyperplasia

45
Q
A

Inflammatory Papillary Hyperplasia

46
Q
A

Peripheral Ossifying Fibroma

47
Q
A

Peripheral Giant Cell Granuloma

Can cross the midline

48
Q
A

Pyogenic Ganuloma

Very red and bleeds a lot

49
Q
A

Leukemia

50
Q
A

Langerhan Cell Histiocytosis

Tooth floating in air

Most common in young children

51
Q
A

Cleidocranial Dysplasia

Multiple Osteomas, supernumerary teeth

52
Q

Syndrome?

A

Garnders Syndrom

53
Q
A

Condensing Osteitis

54
Q
A

Ideopathic Osteo Sclerosis

55
Q
A

Traumatic Bone Cyst

Empty

56
Q
A

Ameloblastoma Histo

Notice the reverse polarity

57
Q
A

Ameloblastoma Radiology

  • ​“Soap bubble” – when the RL loculations are larg
  • “Honeycombed” – when the loculations are small
    • **Buccal and lingual cortical expansion is frequently present**
  • Resorption of roots is common
58
Q
A

Sialolilith

Often get trapped in submandibular gland going around the mylohyoid

59
Q
A

Odontogenic Keratocyst

  • Often found near sinus
  • Tend to grow in an anterior-posterior direction
  • Palisading basal lamina 6 to 8 cell layers thick, making it hard to remove and contributing to the 30% recurrance rate.
  • Gorlin’s scyndrome is Nevoid Basal Cell Carcinomas on skin
    • Bifid rib
    • Calcified falx cerebre
    • OKC
    • PAT gen on chromasome 9
60
Q
A

Fibrous Dysplasia

  • Monostotic
    • 80% of cases, Teenagers, Max more than Mand, “Ground glass”
  • Polyostotic
    • Jaffe-Lichtenstein Syndrome
      • Polyostotic fibrous dysplasia
      • Café au lait spots (coast of Maine)
    • McCune-Albright Syndrome
      • Polyostotic fibrous dysplasia
      • Café au lait spots (coast of Maine)
      • Multiple endocrinopathies
61
Q
A

Pagets Disease

“Cotton wool”

Increased head circumference “my dentures don’t fit”

62
Q
A

Focal Cemento Osseous Dysplasia

Mostly in middle age women

Radioleucent rim

Smaller than 1.5 cm

63
Q
A

Periapical Cemento Osseous Dysplasia

No treatment needed. No Biopsy needed.

64
Q
A

Florid Cemento Osseous Dysplasia

No need to biopsy

Extraction is a bad thing because of necrosis

65
Q
A

Cementoblastoma

  • Can’t follow PDL
  • 75% before the age of 30
  • Found in the mandible molar region
  • Surgical extraction
66
Q
A

Metastatic Carcinoma

Notice the widening of PDL “Moth eathen”

Ususally occur in the mandible

Numb-chin Syndrome

67
Q
A

Osteosarcoma

“Sunburst”

Mesenchymal malignancy

bimodal age distribution

68
Q
A

Eruption Cyst

Soft tissue counterpart for a dentigerist cyst

called eruption hematomas

69
Q
A

Mucoepidermoid Carcinoma

Notice location

70
Q
A

Cherubism

Multiple giant cell granulomas present

71
Q
A

Gingival Cyst of the Adult

  • The lateral periodontal cysyt is the hard tissue counterpart
  • Normally between the lateral and the canine teeth
  • Derived from rests of dental lamina (rests of Serres)
  • Painless, dome-like swellings which are less than 5mm
72
Q
A

Lateral Periodontal Cyst

Location between the canine and lateral incisor

Conservative enucleation is treatment

Hard tissue counterpart of gingival cyst of the adult

73
Q
A

Bucal Biforcation Cyst

  • Rests of Seres
  • Apex of teeth tipped lingually
  • Occurs in children, avg age of 10
  • Treated by enucleation
  • No need to extract the tooth
74
Q
A

Dentigerous Cyst

Unilocular, radiolucent and impacted

  • Cyst that originates by separation of the follicle from around the crown of an unerupted tooth
  • Most common developmental cyst
  • Treatment is enucleation of the cyst together with the unerupted tooth
  • Large examples may be treated by marsupialization
  • Usually do not, but can grow to considerable size & expand bone, cause facial asymmetry, etc.
  • That’s why they must be removed (that and to rule out other cysts like OKC)
  • Calcifying epithelial odontogenic tumor, Ameloblastic fibro-odontoma, Calcifying odontogenic cyst and Adenomtoidodontogenic tumor are differetials
75
Q
A

Adenomatoid Odontogenic Tumor

“Snowflake calcifications”

  • *Striking tendency to occur in the anterior jaws
  • *Mx>Md
  • *F:M 2:1
  • Occurs in young patients, ages 10-20
  • *In 75% of cases, the tumor is a circumscribed, unilocular RL involving the crown of an unerupted tooth*
    • Most often a canine tooth
  • Tumor has a thick, fibrous capsule that makes enucleation easy
  • Dentigerous cyst, Calcifying epithelial odontogenic tumor, Calcifying odontogenic cyst and Ameloblastic fibro odontoma are differentials
76
Q
A

Calcifying odontogenic cyst

  • Gorlin cyst
  • Ghost cells
  • 65% are found in the incisor-canine areas; mx=md
  • Posterior mandible
  • Calcifying epithelial odontogenic tumor , Adenomatoid odontogenic tumor and Ameloblastic fibro-odontoma are differentials
77
Q
A

Calcifying epithelial odontogenic tumor

AKA Pindborg Tumor

  • “Driven snow”
  • Amyloid-like extracellular material; positive for Congo red, which exhibits an apple-green birefringence when viewed under polarized light
  • Calcifications with concentric rings form in the amyloid-like areas (Liesegang rings)
  • Calcifying odontogenic tumor, Ameloblastic fibro-odontoma, Adenomatoid odontogenic tumor are differentials
78
Q
A

Ameloblastic Fibro-Odontoma

  • Ameloblastic fibroma –> Ameloblastic fibro-odontoma –> Ameloblastic odontoma
  • Calcifying epithelial odontogenic tumor, Calcifying odontogenic tumor, Adenomatoid odontogenic tumor are differentials
  • Typically associated with an unerupted tooth
  • Treated with conservative curettage with rare recurrence
79
Q
A

CREST

  • Calcinosis cutis
    • Multiple, movable, nontender, subcutaneous, nodular calcifications
  • Raynaud phenomenon
    • Poor blood flow that leads to blue, painful hands when cold
  • Esophageal dysmotility
    • Caused by abnormal collagen deposition in the esophageal submucosa
  • sclerodactyly
    • Fingers become stiff and white from excess collagen
  • telangiectasia
  • Most patients affected are women in their 60s
80
Q
A

White Sponge Nevus

  • Genodermatosis
  • Autosomal dominant
  • Benign condition with no treatment
81
Q
A

Compound Odontoma

  • Most common odontogenic tumor
  • Considered to be developmental anomalies (hamartomas) rather than true neoplasms
  • Divided into compound and complex types
    • Compound – Composed of multiple, small, toothlike structures
    • Complex- Conglomerate mass of enamel and dentin; bears no anatomic resemblance of a tooth
  • Ave age – 15
  • Completely asymptomatic
82
Q
A

Ash Leaf Spots

Found in tuberous sclerosis

  • Mental retardation
  • Seizure disorders
  • Potato-like growths (“tubers”)
  • Facial angiofibromas: Multiple, smooth-surfaced papules occurring primarily on the nasolabial fold area
  • Ungual fibromas: Seen around or under the margins of the nails
  • Shagreen patches: Connective tissue hamartomas
  • Rare tumor of the heart muscle, termed cardiac rhabdomyoma
  • Angiomyolipomas are found on the kidney
  • Oral manifestations:
    • Developmental enamel pitting on the facial aspect of the anterior permanent teeth
    • Multiple fibrous papules
  • Patients have a reduced life span – death is usually via CNS or kidney disease
83
Q
A

Desquamitive Gingivitis

Pemphigus, Pemphigoid, Systemic lupus erythematous, Lichen planus, Hypersensitivity

84
Q
A

Pemphigus

Tzank cells

85
Q
A

Pemphigoid

  • Twice as common as pemphigus
  • Most significant complication is the ocular involvement (up to 25%) symblepharons
    • Scarring can turn the eyelids inward (entropion), which causes the eyelashes to rub against the cornea
    • Scarring can close opening of lacrimal glands
    • After all this happens, the cornea produces keratin as a protective mechanism
    • Autoantibodies are directed against the basement membrane, leading to a subepithelial split (interepithelial)
    • Direct immunofluorescence (DIF) shows C3 and IgG along the basement membrane
    • Send to Opthamologist!!!
86
Q
A

Pemphigus

  • “First to show, last to go”
  • Autoantibodies are directed against the desmosomes. “Intra”
  • A characteristic feature is a positive Nikolsky sign: Bulla can be induced on normal-appearing skin if firm lateral pressure is exerted
  • Cells of the epithelium “fall apart” – termed acantholysis
87
Q
A

LichenPlanus

  • May be due to medications, amalgam, etc. – “lichenoid mucositis”
  • Skin lesions are the 4 Ps:
    • Purple
    • Pruritus
    • Polygonal
    • Papules
  • Degenerating keratinocytes within the epithelium (termed Civatte bodies)
  • Wickam Striae
  • Lesions can be induced with the following:
    • S – Stress
    • T – Trauma
    • A – Advil (any NSAIDs, acetaminophen is ok)
    • Y – Yeast
  • Desquamative gingivitus, Pemphagoid, Pemphigus, Systemic lupus erythematous are differentials
  • DON’T Treat RETICULAR lichen planus
88
Q
A

Systemic Lupus Erythematous

Pemphigus, Pemphigoid, Desquamitive gingivitis, Lichen planus are differentials

89
Q
A

Erythema Multiform

Herpes usually proceeds the condition

90
Q
A

Toxic Epidermolysis Necrosis

Erythema multiform is a precursor

91
Q

DONE!

A