Final Flashcards

1
Q

MUSCULOSKELETAL: bone fractures

A

s/s: Pain and joint tenderness, Pulselessness and Paresthesia distal to fracture, Pallor, Paralysis
tx: cast, surgery for open reduction of fracture; realign and immobiize fractured extremity by traction, ilizarov external fixator

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

*MUSCULOSKELETAL: developmental hip dysplasia/ dislocated hip

A

s/s: asymmetry of gluteal folds, ortolani’s click [hip click], limited hip abduction, shortened femur, trendelenburg sign [pelvis falls instead of rises w/ stepping of the affected leg/ hip], wide waddling gait
tx: relocate femoral head in acetabulum [may wait and watch w/ NB-6 m.o.], bryant’s traction; pavlik harness [keep child strapped as much as possible; assess for ability to kick, swollen feet]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

MUSCULOSKELETAL: bryant’s traction

A

traction will help position the top of the femur into the hip socket correctly
modified extension traction w/ body as counterweight
risks: circulatory insufficiency
NI: neurovascular assessment, ensure safety

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

MUSCULOSKELETAL: osteomyelitis

A

bacterial infection [often staph. aureus] of the bone and soft tissue surrounding the bone, often affected the long bone metaphysis

tx: antibiotics X 4-6 w
- goal is to prevent bone destruction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

MUSCULOSKELETAL: osteogenesis imperfecta

A

characterized by the formation of pathologic fractures resulting from connective tissue and bone defects
- there’s normal Ca and P levels but low collagen formation
risk factors: simple walking > fractures
s/s: multiple fractures, blue sclera, thin soft skin w/ easy bruising, inc’d joint flexibility, short stature, adol. [hypoplastic bluish-grey teeth, conductive hearing loss]
tx: calcitonin to promote healing and biphosphonates to increase bone mass

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

*MUSCULOSKELETAL: scoliosis

A

lateral curvature of spine usually associated

tx: goal is to prevent further curvature or help correct severe curvature
- bracing for curvatures of 20-50 degrees usually worn for 16-23 h/day
- - NI: risk for impaired skin integrity, body image disturbance, ineffective health maintenance
- surgery for curvatures of > 45 degrees
- - NI: pain, risk for injury r/t surgical intervention, altered breathing patterns, impaired skin integrity, altered bowel elimination [place on regime], altered urinary elimination

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

*MUSCULOSKELETAL: duchenne muscular dystrophy

A

common type of inherited muscular dystrophy
s/s: late accomplishment of gross motor milestones, increasing muscle weakness [including resp. muscles], gower’s sign [using legs to push self up from a sitting to a standing position], waddling gait, difficulty climbing, hypertrophied calf “muscles” [fatty and connective tissues], scoliosis and lordosis, contractures
tx: GOAL IS TO MAXIMIZE BREATHING AND MOVEMENT AS LONG AS POSSIBLE; bracing, orthotics, splints, wheelchair; incentive spirometry; low kcal, high fiber, high fluid, high protein diet

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

MUSCULOSKELETAL: juvenile arthritis [JA]

A

autoimmune disease of connective tissue characterized by chronic inflammation of the synovia and possible joint destruction
- episodes recur w/ remissions and exacerbations for a few months up to a life-time
s/s: inflammation around joints; stiffness, pain, guarding at affected joint
tx: reduce stress, climate aggravators; low does corticosteroids or NSAID’s, ROM [delay and prevent damage]; warm compress; splints; preventative eye care, surgery

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

MUSCULOSKELETAL: skull fractures

A

types:

  • linear: str8 line fracture [dura not involved]
  • depressed: bone pressing downward
  • basilar: fracture at base of skull
    • s/s: battle sign [bleeding behind the ear], racoon eyes, rhinorrhea, otorrhea [careful that drainage is not ICF]
  • communicated: fragmentation of bone into many pieces or a multiple fracture line
    tx: AVOID ACTIVITIES THAT WILL INCREASE ICP [constipation, suctioning, lying supine. hyperextension or flexion of the neck, stimulating env’t.], frequent neuro. exam.’s
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

MUSCULOSKELETAL: concussion

A

a transient or reversible neurologic dysfunction w/ instantaneous loss of AWARENESS and RESPONSIVENESS
- there does not have to be a loss of consciousness
s/s: hematomas, cushing’s triad [bradyc., HTN, resp. depression], anisocoria [unequal pupil sizes], abnormal posturinng, SIADH
tx: AVOID ACTIVITIES THAT WILL INCREASE ICP [constipation, suctioning, lying supine. hyperextension or flexion of the neck, stimulating env’t.], frequent neuro. exam.’s

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

MUSCULOSKELETAL: intracranial hemorrhage

A

types:

  • epidural hematoma
    • occurs b/w the skull and the dura w/ brain herniation and compression of the brain stem; life-threatening
  • subdural hematoma
    • occurs b/w the dura and arachnoid dura
    • s/s: H/A, confusion, seizures, coma
      tx: AVOID ACTIVITIES THAT WILL INCREASE ICP [constipation, suctioning, lying supine. hyperextension or flexion of the neck, stimulating env’t.], frequent neuro. exam.’s
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

CV: fetal circulation

A

goal of fetal circulation is to perfuse the fetus w/ highest amount of O2 possible
- placenta works as the lungs; lungs are fluid-filled w/ pulmonary vessels vasoconstricted causing an inc’d pulmonary vascular resistance
blood flows via ductus venosus, foramen ovale, ductus arteriosus
- these shunts close w/i 24-48 h’s p/ birth
upon birth lungs inflate reducing pulmonary resistance > pulmonary artery pressure drops > increasing blood flow to lungs > R-side pressure drops and L-side increases > change in pressures closes foramen ovale, ductus arteriosus > lack of blood flow results in ductus venosus closure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

*CV: care for a child undergoing cardiac catheterization

A
pre-procedure 
- obtain an accurate weight [determines hydration status]
- assess of skin 
- assess and mark pedal pulses 
- O2 sat.
- vital signs [to compare a/ and p/]
- voiding [ensures contrast can get excreted]
-sedation
post-procedure
- assess site/ dressing 
- assess pulses in lower extremities
- lay supine and flat [~4-6 h]
- vital signs [q15m x3, q30m X3]
- monitor catheterization site
- fluid status
- immobolize extremity [may > bleeding 
- home care [area cannot become saturated]
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

*CV: congestive heart failure [CHF]

A

s/s: tachyc., diaphoresis, low urine output, fatigue [from inc’d metabolic needs], restlessness [from dec’d O2], anorexia, poor peripheral circulation, cardiomegaly [b/c of inc’d work], inc’d volume, gallop rhythm, resp. distress, intolerance, anasarca
goals of tx: improve cardiac contractility [digoxin; 0.8-2 ng/mL], preserve energy and decrease metabolic demands, decrease IV fluid volume [diuretics]
NI: strict I/O, daily weights, inc’d HOB [promote diaphragmatic excursion and lung expansion], hi kcal diet, cluster nursing care, decrease risk of infection, monitor electrolytes [Na [decreases fluid overload], K [decreases risk of arrhythmias]; teach parents how to perform CPR and to assess pulses [if child is on digoxin] and provide use and s.e. of digoxin and diuretics

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

CV: classification of congenital heart disease

A

acyanotic
- inc’d pulmonary blood flow
– atrial septal defect, ventricular septal defect, PDA, AV canal
- obstruction to blood flow from ventricles
– coarctation of aorta, aortic stenosis, pulmonic stenosis
cyanotic
- dec’d plmonary blood flow
– tetralogy of fallot, tricuspid atresia
- mixed blood flow
– transposition of great arteries, total anomalous pulmonary venous connection, truncus arteriosus, hypoplactic L-heart syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

CV: atrial septal defect [ASD]

A

opening b/w the wall that divides the R and L atrium causes shunting of blood from LA [higher pressure] to RA [lower] causing mixing of deO and O blood
risks: inc’d volume > inc’d workload
tx: may resolve spontaneously [w/i 18 m], surgery [patch]
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daaily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

CV: ventricular septal defect

A

opening b/w the R and L ventricles causing shunting of blood from LV [higher pressure] to RV [lower] causing mixing of deO and O blood
risks: dec’d volume going to body
tx: may resolve spontaneously [w/i 3 y]
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

CV: atrioventricular canal septal defect [AVSD]

A

endocardial cushions fail to develop [needed to separate the mitral and tricuspid valves] causing a central hole
tx: septal and valve repair
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

*CV: patent ductus arteriosus [PDA]

A

communication b/w the pulmonary artery and aorta [which should’ve closed during the 1st days of life] causing deO blood in the pulmonary artery to enter the aorta and out into the system w/o becoming oxygenated]
s/s: symptomatic; bounding pulses, machinery-like systolic murmur
tx: indomethcin IV w/i 1st 2 weeks of life [inhibits prostaglandin (present in vitro) to encourage closure; s.e.: masks s/s infection, edema, thickens blood]
- if DA needs to remain open, administer prostaglandin [s.e.: apnea [ventilator use], flushing, hTN, bradyc., bleeding]
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

*CV: coarctation of the aorta

A

narrowing in the aortic arch resulting in L-ventricular overload
risks: L-sided CHF, aneuryms [b/c of Hi pressure against narrowing]
s/s: may present w/ other heart deformities; murmurs, change in BP, irritability, tachypnea, poor feedings > no weight gain
tx: PDA [administer prostaglandin] NEEDED UNTIL REPAIR CAN BE MADE; surgery, resection angioplasty
NI: CHECK BP/ PULSE IN ALL 4 EXTREMITIES [BP inc’d in areas a/ narrowing/ and vice versa]
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

CV: aortic valve stenosis

A

obstruction of blood flow from the L-ventricle to aorta > blood back flow into lungs
risks: L-sided HF, delayed development [from dec’d blood getting to brain], bacterial endocarditis [w/ valve replacement]
s/s: angina, dizziness, poor feeding
tx: balloon angioplasty, aortic valve replacement
NI: stress follow-up care for life p/ valve replacement
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

CV: pulmonary valve stenosis

A

obstruction of blood flow from the R-ventricle to the pulmonary artery > blood back flow into system
risks: R-sided heart failure
s/s: lethargic,
tx: PDA or ASD needed until repair can be made; balloon angioplsty, pulmonic valve replacement
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

CV: tetralogy of fallot [TOF]

A

involves 4 defects: RV hypertrophy [inc’d work from pushing blood via stenosed p. valve], overriding aorta [valve present in both ventricular chambers when it should only be in the L-chamber], VSD, pulmonary valve stenosis
s/s: cyanosis [d/o amount of pulmonary stenosis], tet spells
tx: PDA [give prostaglandin] NEEDED UNTIL REPAIR CAN BE MADE; blalock-taussing shunt [anastomosis R-subclavian to pulm. artery to increases pulmonary circulation], cardio-pulmonary bypass
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

*CV: tet spells

A

inc’d constriction of the R ventricular outflow > sudden dec’d oxygenation in the child > cyanosis
may be precipitated by crying, feeding, procedures
s/s: do not occur until p/ the closure of the DA
NI: help child bend knees to chest to increase venous return to the heart [improves oxygenation]; be aware of hypoxia-inducing experiences; O2 therapy may help slightly but not to the full affect until defects are fixed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

CV: tricuspid atresia

A

no communication [no tricuspid valve] b/w the RA and RV > mixing of deO and O blood
risks: hypoplastic RV, under-developed pulm. artery [since no blood flow is going via RV]
tx: PDA or foramen ovale needed until repair can be made
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

CV: pulmonary valve atresia

A

no communication b/w RV and PA > blood cannot leave the RV
- usually presents w/ a VSD and/or ASD [creating mixing of blood so that child. can get some O into system]
tx: PDA needed until repair can be made
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

CV: transposition of the great vessels

A

position of the aorta and pulmonary artery are switched creating 2 separate circulations [R-side pumps to system, L-side pumps to lungs]
tx: PDA, VSD, etc. needed until repair can be made; atrial septotomy
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

CV: truncus arteriosus

A

one major artery leaving the R and L ventricles > mixing of blood
risks: CHF of both sides, pulmonary vascular disease
tx: separation of the pulmonary artery from the aorta
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

CV: hypoplastic L-heart syndrome

A

mitral and aortic valve are not properly formed, ascending aorta is underdeveloped, ASD
s/s: pale, poor perfusion, tachyc., tachyp., metabolic acidosis, poor feeding
NI: vitals [esp. pulse ox.], monitor for sx.’s of CHF, strict I&O, daily weights, hi kcal diet, cluster care, allow for extended rest periods, HOB elevated; post-op. [activity restrictions, incision site care, place on reverse isolation, manage pain, educate for follow-up care]

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

CV: bacterial endocarditis

A

infection of the inner lining of the heart or valves
risk factors: CHD, valvular disease/ repairs
causes: dental work, non-sterile/ indwelling catheterization
s/s: low-grade fever, maliase, wt. loss, H/A, myalgia, splenomegaly, osler nodes [lesions of fingers/ toes], janeway spots [hardened lesions on hands/ feet], mucosal petechiae
dx: CBC, cardiogram
tx: antibiotics
NI: prophylactic antibiotics for certain procedures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

*CV: rheumatic fever

A

a systemic disease caused by post [2-3 w]-group A strep. infection that affects the brain, heart [> weakened contractility], all bony joints
s/s: major [carditis, polyarthiritis, chorea (rapid, jerky movements), erythema marginatum (reddened rashes), subq. nodules], minor [low-grade fever, arthralgia, epitaxis]
dx: CBC, ESR [for inflammation], EKG
tx: med.’s [anti-inflammatory, antibiotics], supportive therapy [bed rest, pain relief]
NI: monitor cardiac status, prevention and prophylactic measures

32
Q

*CV: kawasaki disease

A

self-limiting autoimmune disorder where edema and inflammation > vaculitis of the vessels > impairment > aneurysms
risks: acquired heart disease, thrombocytosis
s/s: acute [abrupt Hi fever, bulbar conjunctivitis w/o drainage, pharyngeal mucosal inflammation, strawberry tongue, edema of hands/ feet, erythema of palms/ soles, myocarditis, cervical lympadenopathy], subacute [continued vessel damage, thrombocytosis, periungual desquamation], convalescent [no sx.’s except coronary complications]
tx: manage sx.’s, med.’s [IVIG, aspirin
NI: monitor cardiac status, steroids monitor for sx.’s of reye’s w/ aspirin use in young child., teach parent CR, delay IV immunoglobulins [MMR, varicella]

33
Q

INTEGUMENTARY: impetigo contagiosa

A

highly contagious [not communicable p/ 48 h on antibiotics] bacterial invasion via skin breakdown or carried in nares or opening in the skin and passed onto skin [usually oon face, neck, limbs]
- incubation time is about 2-5 d
risk factors: immuncompromised pt.’s
causes: group A strep./ staph. aureus
risks: MRSA [rule out] > necrotizing fascitis, glomerular nephritis
s/s: discrete/ grouped vesicles or pustules on an erythematous base > golden, honey colored crusted lesions
NI: hand-wash; mupirocin [bactroban] to lesions

34
Q

INTEGUMENTARY: cellultis

A

acute inflammation involves 3 layers of skin
causes: GABHS, s. aureus
risk factors: trauma, impetigo, otitis media, sinusitis
risks: severe cases > osteomyelitis; infection of the face > meningitis; periorbital cellulitis > optic nerve infection > blindness
s/s: erythema, pain, edema, warmth at site; malaise; lymphadenopathy
tx: PO [mild]/ IV [systemic; esp. if the infection reaches face] antibiotics
NI: warm compresses [for pain and draw pus out], elevate affected limb, pain relief

35
Q

INTEGUMENTARY: tinea corporis [ring-worm]

A

a fungal infection
causes: trichophyton, microsporum
s/s: round/ oval scaling patches w/ central clearing and well demarcated raised borders involving the trunk, hands or face
tx: topical antifungal; PO [for severe cases]
NI: can return to school p/ tx has begun

36
Q

*INTEGUMENTARY: tinea capitis

A

involves the hair shaft > loss of hair
s/s: red, scaly lesion
dx: organism fluorescent under wood lamp exam.
tx: PO griseofulvin, selenium shampoo [reduces contagiousness]
NI: wash sheets/ clothes in hot water

37
Q

*INTEGUMENTARY: atopic dermatitis [eczema]

A

chronic inflammatory skin disease
causes: familial hx., allergic rhinitis, asthma
s/s: infantile [erythematous, weeping vesicles w/ crusting esp. on face/ scalp/ extremities; accompanied by intense pruritus [monitor for 2o infection b/c of scratching], child. [dry, scaly, papular lesions, fissures and lichenification (thick, leathery skin); found on wrists, ankles, flexor aspects of antecubital/ popliteal spaces]
NI: avoid skin irritants, mittens for child. to prevent scratching [benadryl to reduce itch]

38
Q

*INTEGUMENTARY: seborrheic dermatitis [cradle cap]

A

inflammatory, SCALING disorder of face and scalp caused by dysfx. of sebaceous glands [w/i the hair follicle]
s/s: erythematous, greasy salmon-colored oval scaly lesions on scalp/ face
tx: antiseborrheic shampoo [leave-in for 5-10 minutes] softens and loosen scales, remove scales w/ fine brush

39
Q

*INTEGUMENTARY: contact dermatitis [poison ivy]

A

cell mediated response to poisonous oil causing hypersensitivity [T-cell response]
s/s: rash, pruritis, blister formation, oozing and crusting
tx: steroids, benadryl [pruritis]; claritin/ zyrtec [inflammation]
NI: wash exposed area but no scrubbing, wash clothes to rid oil

40
Q

INTEGUMENTARY: scabies

A

occurs from burrowing of adult female mite into epidermis
- incubation form 4-6 w w/o prior exposure and 1-4 d w/ exposure
s/s: linear red rash; INTENSE PRURITUS
tx: permethrin, lindane [not on infants/ child. b/c of risk for neurotoxicity], antipruritics
NI: treat head only in infants and child., tx for family and notification of daycare; treat clothing in hot water

41
Q

INTEGUMENTARY: pediculosis [lice]

A

tx: pedulicilicide [NIX] then COMB HAIR P/ TX TO GET RID OF NITS
NI: child. can go to school p/ tx: treat family if infected; delouse home env’t

42
Q

INTEGUMENTARY: rubeola [red measles, 10-day measles]

A

highly contagious viral infection transmitted via infectious droplets
causes: RNA virus
- incubation: 10-12 d, communicable several days a/ rash and 5 d p/ its appearance
risks: desquamation, resp. complications [otitis media, bronchopneumonia]
s/s: prodrome [a/ lesion appearance] Cold, hacking Cough, Conjunctivitis, photophobia, fever; confluent maculopapular rash; koplik’s spots [red spot w/ white center] on buccal mucosa
dx: elevated IgM antibdy titer
tx: fever control [febrile seizures], acetaminophen, fluids, tepid fluids
NI: protect from light, no school until p/ 5 days of rash appearance,

43
Q

INTEGUMENTARY: rubella [german measles, 3-day measles]

A

viral infection
- incubation: 2-3 w, communicable: 5-7 d a/ rash to 7 d p/ rash
causes: RNA virus
s/s: prodrome malaise, low-grade fever; resp. tract virus; lympadenopathy; pink, maculopapular rah starting on face and spreading rapidly downward over trunk and extremities; trasient polyarthralgia/ polyarthritis [distinguishes rubella from rubeola]
dx: elevated IgM antibody titer
NI: PREVENT EXPOSURE TO PREG. WOMEN [may > birth defects]

44
Q

INTEGUMENTARY: mumps

A

viral infection transmitted via direct contact or droplet

  • incubation: 14-21 days p/ exposure, communicability: a/ and p/ swelling
    causes: paramyxovirus
    risks: meningitis, deafness
    sx: fever, H/A, general malaise, earache [r/t swelling] aggravated w/ chewing, parotitis
    tx: supportive, MMR vaccine for prevention
45
Q

INTEGUMENTARY: erythema infectiosum [fifth’s diease]

A
MOST CONTAGIOUS A/ RASH 
incubation: 4-21 d
causes: human parovirus 
risks: aplastic crisis in child. w/ SCD
s/s: prodrome low-grade fever, H//A, mild URI; fiery red rash on cheeks [slap face] spreads to extremities, may return when skin is traumatized
tx: comfort measures
46
Q

*INTEGUMENTARY: coxsackie virus [hand-foot-mouth]

A

causes: enterovirus
s/s: common cold, pharyngitis, pneumonia; painful papulovesicular lesions on cheeks, gums, hands, feet
NI: cool drinks, PREVENT DEHYDRATION, antipyretics

47
Q

INTEGUMENTARY: varicella zoster [chickenpox]

A

incubation: 2-3 w, communicability: Hi 1st 2 d a/ eruption until ALL LESIONS ARE CRUSTED OVER
causes: resp. herpes zoster virus
risks: shingles [virus remains in nerve ending], pneumonia, encephalitis
s/s: mild fever, general weakness, teardrop lesion
tx: benadryl, acyclovir, oatmeal baths

48
Q

*INTEGUMENTARY: scarlatina [scarlet fever]

A

infection of oropharnyx caused by group A beta-hemolytic strep
- incubation: 1-3 d
risks: AOM, acute glomerulonephritis, rheumatic fever
s/s: predrome sore throat, chills, fever, H/A, vomiting; fine erythematous maculopapular rash w/ sandpaper quality [desquamation occurs when rash fades], beefy red tonsils/ pharynx, strawberry tongue
tx: 10 days PCN, antipyretics
NI: non-infectious p/ 48 h on antibiotics; gargles for comfort; throw out toothbrushes p/ infection

49
Q

NEURO: neurological assessment

A
glasgow coma scale 
LOC
development 
- developmental age; gross/ fine motor development
vital signs
PERRL 
posturing
- decorticate v decerebrate 
-- decor. > decer. indicates worsening of overall status 
reflexes
kernig's sign 
- + indicates meningitis: severe stiffness prevent str8ning of leg when hip is flexed
brudzinski's sign 
- + indicates meningitis; severe neck stiffness > hip and knees to flex when neck is flexed
infant's fontanels 
- bulging indicated inc'd ICP
50
Q

*NEURO: types and sub-types of seizures

A

partial seizures: 1 area of cortex, manifests w/ prodromal [aura] sx.’s
- simple partial: localized to 1 hemisphere, no loss of consciousness
- complex partial: starts in 1 focal area > to both hemispheres, possible loss of consciousness
generalize seizures: both hemispheres
- tonic-clonic: violent body seizures; manifests w/ tonic [stiff] phase, clonic [jerking] phase, postictal [confusion, lethargy] phase
- absent: loss of responsiveness but ability to maintain posture control and not fall and no postictal sx.’s
- atonic/ akinetic: sudden brief loss of muscle tone/ posture, temp. LOC
- myoclonic: sudden, brief cxn.’s of a muscle or muscle groups, can occur in combo. w/ other seizure types
unclassified
- status epilepticus: recurrent or continuous seizures w/o interruption; risk of anorexia > multi-organ damage
- infantile spasms: associated w/ cognitive impairment

51
Q

*NEURO: febrile zeizures

A

causes no neurological damages causes: RAPID rise of temp. to > 102 oF
- only 1 seizure occurs per increase in temp.
occurs in child. ages 6 m to 3 y
NI: FEVER MANAGMENT [tylenol], seizure management

52
Q

*NEURO: nursing care of child. w/ seizures

A

place child on their side when possible to prevent aspiration if they have excess secretions or vomit in their airway
administer antiepilectic as ordered
- pt.’s taking dilantin need frequent oral care
have O2/ resuscitation equipment and suction at the bed side at all times
have side rails raised and padded
document assessment findings during seizure
monitor vitals signs
assess quality of resp.’s an color changes
monitor LOC
note time of onset of seizure and its duration
note any precipitating factors
- plan to avoid these triggers

53
Q

*NEURO: spina bifida I

A

a type of neural tube defect where there is a failure of total enclosure of spinal cord w/i vertebrae/ meninges
risk: neurosensory/ neuromotor dysfx.
s/s: d/o degree of defect and the nerves involved; hydrocephalus; arnold-chiari malformation [downward displacement of portion of brain]; meningitis; UTI’s, bowel obstructions
tx: preventative [FOLIC ACID], post-birth surgical repair, fetal surgery
NI: prevent urinary complications, assess urological status, instruct family self-catheterization techniques; provide colon training, diet high in fiber and low in CHO, laxatives PRN; carefully monitor the skin [due to dec’d sensation] and provide frequent respositioning, PT; provide latex-free env’t; educate about life-long care needs

54
Q

NEURO: hydrocephalus I

A

it is a collection of excess CSF in the brain ventricular system
- a manifestation of an underlying probem [e.g. spina bifida]
types:
- communicating: excess production of CSF due to impaired absorption of CSF
– causes: scarring, congenital anomalies, hemorrhage
- non-communicating: obstruction to flow
– causes: malformations tumors, clots, edema
s/s: infant [enlarged head, thinned cranial bones, separated sutures, bulging fontanels, dilated scalp veins, cranial nerve palsy; poor feeding, vomiting; high-pitched cry], child [H/A, NV, visual changes, ataxia,behavior changes, seizures]

55
Q

NEURO: nursing care perioperatively for a pt. w/ spina bifida

A

preoperatively
- check for leakage of CSF from sac, PREVENT INFECTION AND INJURY; apply moist sterile dressing to defect, avoid fecal contamination to opening; monitor s/s of infection and meningitis; thermoregulation; administer antibiotics; place child in prone position or side-lying; no rectal temp.’s; check for movement sensation of the lower extremities; initiate antibiotic tx, create latex-free env’t
postoperatively
- prevent infection/ injury; MONITOR HC; maintain dry sterile dressing over lesion, examine dressing for CSF leakage, place child in prone or side-lying position [frequent repositioning]; no rectal temp.’s; prevent urinary complications; continue antibiotics
– complications post-op.: hydrocephalus, monitor for sx. on inc’d ICP, measure HC qshift; neurogenic bladder/ bowel; neuromuscular disorders

56
Q

*NEURO: s/s of inc’d ICP

A

infants:
- irritability, high-pitched cry; poor feeding; bulging fontanels, setting-sun sign of the eyes
child.:
- HA, vomiting [w/ or w/o nausea]; seizures; diplopia
behavioral:
- irritability, drowsiness, dec’d physical activity and motor skills; complaint of fatigue; inability to follow commands, memory loss; anorexia
late signs:
- cushing’s triad [systolic HTN w/ widening pulse pressure, bradyc. w/ full pulse, cheyne-stokes resp.’]; dec’d LOC/ motor response to command/ sensory response to painful stimuli; decerebrate/ decorticate posturing; anisocoria

57
Q

NEURO: tx. and NI of a child w/ hydrocephalus

A

tx:
- surgical shunting
- direct removal of obstruction and insertion of shunt
- ventriculostomy: non-shunting procedure to treat child. w/ non-communicating disorder
NI:
- pre-op.: neuro. assessments, assess HC and other signs of inc’d ICP; skin care and assess for breakdown; small, frequent feedings to prevent vomiting; encourage bonding
- post-op.: frequent neuro. checks, monitor fluid balance; position off operative area; pain management; prevent infection [prophylactic antibiotics, monitor incision]

58
Q

*NEURO: meningitis

A

a potentially fatal infection [viral or bacterial] that begins elsewhere in the body [ears, sinuses, upper resp. tract]
risks: cerebral edema, hydrocephalus, cognitive impairment, deafness blindness
s/s: fever, chills; HA; changed LOC; photophobia; nuchal rigidity [child. moves entire upper body instead of just neck]; + kernig and brudzinski signs; petechial rash; septic shock; infants [poor feeding, lethargy; vomiting, opthisotonus]
dx: LP, blood culture
tx: antibiotics, isolation, monitor neuro. fx and s/s inc’d ICP; dec’d sensory input; fever control; seizure precautions

59
Q

*NEURO: cerebral palsy

A

nonprogressive disorders of motor fx.
types: spastic, dyskinetic, ataxic
risk factors: anoxia during labor
s/s: lack of coordination and inc’d spasticity; abnormal motor skills [asymmetry, early handedness, feeding/ swallow problems, persistent toe-walking], altered muscle tone, PERSISTENCE OF PRIMITIVE REFLEXES, abnormalities of cognition, seizures, hearing and vision
tx: PT/ OT/ ST [maintenance of ROM, fx. skills, feeding, language, communication]; orthopedic care; pain relief
NI: handling/ positioning, self-care skills

60
Q

ENDOCRINE: GH deficiency

A

causes: hypopituitarism, infections, trauma, brain tumors
s/s: constitutional delay [delay in growth/ sexual growth]; inc’d amount of fat on waist/ face; psychosocial implications; dec’d muscle mass; delayed skeletal maturation/ onset of puberty/ tooth development; hypoglycemia
dx: GH stimulant test, karotyping
tx: exogenous GH subq. shots [stopped when growth plates fuse]
NI: stress imp. for follow-up appt.’s [dosages may need to change], monitor growth accurately, teach proper technique for shots, have parents call doc. if [H/A, joint pain, rapid weight gain] occur

61
Q

*ENDOCRINE: congenital hypothyroidism

A

dec’d production of T4 [thyroxine] which is essential for growth and development
risks: mental retardation
s/s: large-for-age; puffy face, swollen tongue; hoarse cry; poor muscle tone; cold extremities; persistent constipation; lethargy; little or no growth [despite weight gain]
tx: exogenous T4 [synthroid]
NI: monitor growth and development; assess for retarded physical growth and slow intellectual development; stress imp. of med.’s and to go to follow-up visits [dosages may need to be changed]; never place med. in a whole bottle in case the child doesn’t finish their bottle; teach parents how to take pulse [to monitor for signs of drug overdose]

62
Q

ENDOCRINE: congenital adrenal hyperplasia

A

caused by dec’d enzyme activity required for cortisol [responsible for growth, maintains BP, protects against stress] and, less so, aldosterone [maintains BP, keeps balance b/w Na K and fluids] production in the adrenal cortex
types: salt-losing, simple virilizing
salt-losing form of CAH
- near total deficiency of enzyme > complete inability to produce cortisol/ aldosterone
- risks: hypoglycemia [b/c of x cortisol], Na and H2O loss [b/c of x aldosterone]; inc’d aldos. > masculinization
s/s: males [precocious genital development], females [varying degrees of ambiguous genitalia]
tx: assign sex to child. d/o gentotype; cortisone to balance high secretions of ACTH; reconstructive surgery; exogenous aldosterone; salt-retaining hormone tx.
NI: teach parents s/s of dehydration and alt-losing crises, teach imp. of follow-up care

63
Q

*ENDOCRINE: phenylketonuria [PKU]

A

lack or low levels of an the enzyme phenylalanine hydroxylase (PAH)]; PAH needed to convert phenylalanine to tyrosine
risks: inc’d phenyl. in blood > affects the myelinization process of the CNS > seizures, brain damage, mental retardation
tx: protein restrictions
NI: protein restrictions can be dec’d p/ full brain growth; levels should be measured periodically, child. of mother’s w/ PKU may > congenital defects

64
Q

*ENDOCRINE: type II diabetes

A

s/s:
- hypoglycemia
– trembling, diaphoresis, tachyc., pallor, clammy skin; personality change, irritability, slurred speech, dec’d LOC, seizures
- hyperglycemia
– 3 P’s [poly [uria, dipsia, phagia], fatigue, weight loss, blurred vision, emotional lability, HA, hunger
- ketoacidosis
– s/s of hyperglycemia + abd. pain, chest pain, kussmaul resp.’s N/V, acetone breath, dehydration
NI: teach that an ill child needs extra kcal to keep body from burning fats for fuel and to speed healing
developmental issues:
- infants: erratic eating/ sleeping patterns; very rapid growth
- tot’s: dec’d appetite, temper tantrum v hypoglyecemia rxn.
- pre-school: peer pressure; difficult to find foods that match what siblings/ friends eat
- school-aged: tests independent decision making, fear of being diff. from others
- adol.’s: erratic growth, difficult glucose control, concern about body image, may challenge authority, risk-taking behaviors, substance use [> inc’d complications w/ diabetes]

65
Q

CANCER: leukemias

A

a broad group of malignant diseases of bone marrow and lymphatic system where an unrestricted proliferaton of immature WBCs in the blood-forming tissues of the body crowd our other blood elements w/ resultant anemia
risks: infection [neutropenia], hemorrhage [dec’d plt production], poor growth, CNS/ bone/ testicular involvement; acute forms can > dec’d leukocyte count; anemia; spleen, liver, and lymph glands show infiltration/ fibrosis

66
Q

*CANCER: s/s of bone marrow infiltration

A

anemia
- manifests as pallor, lethargy, dyspnea, inc’d incidence of murmurs
dec’d plt’s
- manifests as bleeding, petechiae, purpura
neutropenia
- manifests as fevers, infections
bone pain
- manifests as limp, dec’d walking, irritability

67
Q

CANCER: nervous system tumors

A

most common childhood solid tumors
s/s: behavior changes; inc’d HC, bulging fontanel, dilated scalp veins; HA, projectile vomiting; pupil, EOM, visual disturbances; motor/ coordination changes; seizures; papilledema; dec’d HR/ RR, wide pulse pressure, temp. instability
NI: monitor for inc’d ICP; developmentally appropriate peri-op. preparation and care; post-op. [minimize activities that increase ICP, perform wound care; neuro/ VS checks; position off surgery site]

68
Q

CANCER: bone tumors

A

types:
- oSteoSarcoma
– most common; found in Shaft of long bones
– tx may include amputation w/ prosthetic replacement
- ewing sarcoma
– arises in marrow and mid-shaft of long bones
- usually able to preserve affected limb
s/s: limp, changed gait, weakness of extremity
NI: pre-op. [address concerns about loss of limb]; manage complications of radiation/ chemo.

69
Q

CANCER: wilm’s tumor

A

a nephroblastoma that originates from immature renoblast cells; associated w/ other syndromes
s/s: unilateral abd. mass [DO NOT PALPATE]; lethargy; weight loss; fever; organ compression; HTN
tx: nephrectomy and adrenalectomy, removal of other involved organs
NI: pre-op. [monitor BP, prepare pt. and family for op., MINIMIZE ABD. PALPATION], post-op. [monitor bowel sounds/ urine output; assess s/s of intestinal obstruction; prevent infection; prevent post-op. pulm. complications]

70
Q

*CANCER: retinoblastoma

A

congenital malignancy
s/s: cat’s eye reflex/ leukokoria [common], strabismus [common]; red, painful eye often w/ glaucoma; blindness [late]
risk: osteogenic sarcoma
NI: educate family on: care fro surgical site/ prosthesis, protective eye gear

71
Q

ABUSE: shaken baby syndrome

A

it is the repetitive violent shaking of an infant while being held by trunk
s/s: manifests as brain injury from cerebral hemorrhage/ cerebral edema [irritability, lethargy, vomiting, seizures, apnea, coma, retinal hemorrhages, rib fractures, hematoma]
NI: recognition of suspicious injuries; obtain supporting evidence while finging alternative diagnosis; report suspected abuse [MANDATED]; remain objective when interacting w/ family and when documenting; advocate for the child

72
Q

DYING/ DEATH: nursing care for the dying child

A

assessment
- physical signs of approaching death: inc’d sleep, dec’d appetite/ need for fluids; loss of tactile sensation/ ovement; loss of bowel/ bladder control; confusion; dysphagia; cheyne-stokes resp.’s
- emotional issues: recall of family events, desire to communicate, reasssure others, visions, withdrawal/ need for closeness
physical needs
- sx./ pain control; mouth/ skin care; positioning; hygiene; D/C unnecessary procedures

73
Q

CANCER: side effect of cancer tx.’s

A
infection/ nutropenia
hemorrhage
anemia
NV
altered nutrition
mucosal ulceration
neuro. alterations
- somnolence
hemorrhagic cystitis
alopecia 
steroid effects [e.g moon face, mood swings]
74
Q

MENTAL: autism spectrum disorders

A

developmental disorder of brain fx. characterized by deficits in intelligence and behavior
s/s: IMPAIRED ABILITY FOR SOCIAL INTERACTIONS/ VERBAL COMMUNICATION
tx: multidisciplinary support

75
Q

MENTAL: attention deficit hyperactivity disorder [ADHD]

A

developmentally inappropriate degrees of inattention, impulsiveness and hyperactivity
s/s: dx when 6 sx.’s of either [inattention OR hyperactivity/ impulsivity AND onset a/ 7 y, impairment in +2 settings, impairment in social, academic, occupational fx., no other pervasive disorder
tx: behavior therapy, biofeedback, structure, med.’s [psychostimulants, ritalin]