FHR Flashcards

1
Q

Rate of fetal blood flow in placenta and 4 things it’s dependent on

A

500ml/min

Cardiac activity, BP, R to L shunts, systemic and pulmonary vascular resistance

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2
Q

Factors that reduce UBF

A
Umbilical cord compression
Maternal hypotension & hypertension
Aortocaval syndrome
Vasoconstriction in uterine vessels (predominantly alpha)
Hypertonic uterine contractions
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3
Q

What is the main determinant of fetal pO2

A

Maternal uterine venous pO2

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4
Q

Fetal metabolic acidosis ABG

A

Ph <7.2, HCO3 < 20 BE -6

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5
Q

Reliability is poor while sensitivity is high

A

FHR monitoring

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6
Q

FHR at 20 weeks and at term

A

155bpm then slows down to 140 bpm as PNS matures

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7
Q

What determines baseline fetal HR

A

Tonic effects on the SA node

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8
Q

What are some causes of fetal bradycardia (<110 BPM for >10 minutes)

A

Maternal hypoxia, drugs decreasing UPP, hypotension, vagal stimuli
Fetal acidosis, asphyxia, cord compression

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9
Q

What are some causes of fetal tachycardia (>160bpm for >10 min)

A

Maternal fever, chorioamnioitis, Drugs (terbutaline, ephedrine, atropine)
Fetal hypoxia, arrythmias

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10
Q

Most reliable sign of fetal compromise d/t fewer false alarms

A

Baseline variability

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11
Q

How is variability reduced

A
CNS depressants (opioids, sedatives, anesthetic agents, barbs, magnesium)
Hypoxemia
Fetal sleep
Acidosis
Anencephaly
Cardiac anomalies
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12
Q

Absent variability is indicative of

A

Metabolic acidosis and depression of fetal brain stem and heart

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13
Q

Periodic accelerations related to fetal movements and uterine pressure

A

Long term variability

Normal is 15-40 an hour

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14
Q

R to R variability

A

Short term
3-6 bpm
Measured by fetal scalp electrode

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15
Q

Treatment of Early decelerations

A

LUD & oxygen

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16
Q

D/t fetal head compression
Benign with contraction & well tolerated by healthy fetus
No risk for fetal hypoxemia

A

Early decelerations

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17
Q

Treatment of late decelerations

A

LUD, O2, IVF, vasopressors, poss C/S

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18
Q

Ominous type of deceleration, indicator of UP insufficiency
Risk for fetal hypoxemia
Gradual decrease of FHR below baseline
Caused by: maternal hypotension, hypovolemia, acidosis, pre-eclampsia

A

Late deceleration

19
Q

This type of deceleration indicates cord compression
Risk for fetal hypoxemia
Abrupt decrease in FHR below baseline
No consistent pattern between FHR and contraction

A

Variable deceleration

20
Q

Treatment of variable deceleration

A

LUD, O2, IVF, amnioinfusion - urgent assessment of fetal status

21
Q

Treatment for prolonged decelerations

A

LUD, O2, IVF, vasopressors, stat c section

22
Q

This type of deceleration is a visually apparent sustained decrease in FHR
Caused by asphyxia, hyperstimulation, examination, supine hypotension
Can be a terminal fetal rhythm

A

Prolonged deceleration

23
Q

Level 1 emergent CS is usually for what type of NRFHR

A

Late decelerations without variability or bradycardia sustained below 70

24
Q

Treatment for NRFHR

A
LUD
O2
Treat hypotension - fluids and pressors
D/c pitocin
Treat uterine hypertony - terbutaline 0.25mg sc
Pelvic exam for cord prolapse
Internal monitoring
Amnioinfusion
25
Q

FHR tracings in category III

A
Absent variability plus
Bradycardia or
Variable decels or 
Late decels or
Sinusoidal pattern
26
Q

FHR tracings in Category II

A

Recurrent variable decels
Tachycardia
Bradycardia without absence of FHR variability

27
Q

What is normal fetal scalp blood gas

A

Ph <7.25, pco2 <50 po2 >20

28
Q

Fetus maintains normal aerobic metabolism until

A

O2 falls to 50% of normal

29
Q

What happens with acute hypoxemia

A

PRIMARY
Maintain BF and O2 in major organs with increased CO and HR
Then FHR slows decreasing o2 consumption

SECONDARY
O2 uptake by fetal hgb becomes less efficient as pH decreases- go to anaerobic metabolism
Will lead to fetal asphyxia

30
Q

Chronic hypoxia is d/t

A

Chronic decreased UPP
- pre eclampsia
Maternal hypertension
Maternal DM

31
Q

What happens to the fetus with chonic hypoxia

A

IUGR and delay in neural development

32
Q

What leads to hypoxic ischemic encephalopathy

A

Surviving severe asphyxia

  • adaptive mechanisms fail
  • arterial O2 falls
  • myocardial depression
  • loss of autoregulation with CBF
33
Q

FHR tracing methods

A

Continuous electronic

Intermittent ausculation

34
Q

Which FHR tracing method is required for neuraxial

A

Continuous electronic

35
Q

Indicators of overall well being

A

Short and long term variability

Accelerations

36
Q

Short term variability represents a

A

Normal functioning autonomic system and reassuring for fetal o2

37
Q

Decrease of long term variability is d/t

A

Fetal sleep, drug effects (opioids, mag, atropine), fetal hypoxia

38
Q

Onset to nadir is >30

Onset to nadir is < 30 seconds

A

Late decelerations

Prolonged and variable

39
Q

Stat C/S indicated with

A

Prolonged & late variability without variability

Sustained brady <70bpm

40
Q

Neonatal resuscitation med doses
Epi
Naloxone
Crystalloid

A

Crystalloid 10ml/kg over 5-10 min
Epi 0.01. To 0.03 mg
Nalaxone 0.1 mg/kg

41
Q

Part A of neonatal resuscitation

A

Term gestation?
Amniotic fluid clear?
Breathing crying?
Good muscle tone?

Provide warmth
Position clear airway
Dry stimulate resposition

42
Q

Apneic or HR <100 for neonatal?

A

PPV

43
Q

HR < 60

A

Provide PPV, chest compressions

And if still HR < 60 give epi and IVF