Fetal wellbeing Flashcards
When assessing fetal wellbeing, what is key on history, examination and investigations?
History: Fetal movements
Examination: Fundal height
Investigations: Ultrasound (blood flow, amniotic fluid) and CTG
What are some parameters looked at on U/S?
Fetal biometry (baby size) > head circumference / abdominal circumference / femur length > absolute and serial measurements > head:abdo ratio Liquor volume > amniotic fluid index Fetal activity > body movements > breathing movements > tone Blood flow (Doppler U/S) > umbilical artery waveform > MCA
After what stage antenatally does CTG become useful?
28 wks, better interpretation from 32 wks onwards
CTG to measure fetal HR is not useful before 28 wks
What are the features of a normal CTG?
“Reactive” = normal
Baseline: 110 - 160 bpm
Variability: 5-25 bpm
Accelerations: at least 2 x 15 bpm in 20 mins
Decelerations: NO ominous delerations
When looking at a CTG, what do some of the concerning changes in variability mean?
Increased variability
- hypoxia
Reduced variability
(= non-reactive)
- sick, sleeping, sedated, submature
Absent
- SEVERE hypoxia
Sinusoidal pattern
- fetal anaemia
What are some of the causes of a non-reactive CTG (reduced variability)?
S ick
> hypoxia
S leeping
S edated
> sedatives / anti-hyptensives / anti-convulsants
S ubmature
> prematurity
When looking at a CTG, what are some concerning decelerations and what can they mean?
Early decelerations: head compression in labour
Late decelerations: fetal hypoxia (appropriate response)
Variable decelerations: cord compression
Prolonged decelerations: sustained hypoxia
What on CTG would warrant investigation antenatally, and what investigations should be done?
Non-reactive
> no accelerations
> reduced variability
** Must think, is it due to hypoxia? **
Investigations:
- Fetal stimulation tests (VAST)
- Doppler US (umbilical artery waveform and MCA peak systolic velocity)
- Biophysical profile (AFI/fetal activity/CTG)
What findings on CTG demonstrate a critical fetal condition and warrant immediate delivery via c-section?
- Late decelerations (fetal hypoxia)
- Absent variability (severe hypoxia)
- Sinusoidal pattern (detal anaemia)
On CTG intrapartum, what changes would be considered critical and warrant immediate delivery (c-s, vaginal delivery)?
Late/prolonged/severe variable decelerations
Absent variability
Sinusoidal pattern
What action should be undertaken if a CTG is non-reactive during intrapartum?
Continuous CTG monitoring
** Could there be narcotics in the mum/baby system? **