Factors Affecting Performance Flashcards
Factors Affecting Performance
- Diet
- CNS Function
- Strength/Skills
- Environment
- Energy Production - anaerobic sources
Diet
- Carbohydrate
2. Water Intake
Energy Production - anaerobic sources
- [PC]
- Glycolysis
Anaerobic Sources
- VO2 max
- Cardiac Output
- O2 delivery
- [Hb]
- PO2
- O2 extraction
- Mitochondria
Environment
- Altitude
- Heat
- Humidity
Strength’Skill
- Practice
- Natural Endowment
- Body type
- Muscle Fiber Type
CNS Function
- Arousal
2. Motivation
Sites of Fatigue
Central and peripheral
Fatigue
Inability to maintain power output or force during repeated muscle contraction
Central Fatigue
CNS
Peripheral Fatigue
- Neural Factors
- Mechanical Factors
- Energetic Contraction
Peripheral Fatigue is
the big picture but central fatigue will cost you the race
Possible sites of fatigue
- Psyche brain (motor unit recruitment/motivation)
- Spinal Cord (reflex drive)
- Peripheral Nerve (Neruomuscular transmission)
- Muscle sarcolemma (Muscle action potential)
- Transverse tubular system (K, Na, excitation)
- Calcium release (activation, energy supply)
- Actin-myosin interaction
- Cross bridge tension and heat
- Force Power output
Central Fatigue listed as a site of fatigue due to
- Reduction in motor units activated
- Reduction in motor unit firing frequency
- CNS arousal can alter the state of fatigue
CNS arousal can alter the state of fatigue
By facilitating motor unit recruitment to increase strength
- increasing motivation (shouting during the exertion)
- Physical or mental diversion (i.e. contracting non-fatiguing muscles during the rest period or doing arithmetic b/w fatiguing bouts.)
Excessive endurance training - overtraining
- Reduced performance, prolonged fatigue, sleep disturbances, loss of appetite,
- Evidence that increases r decreases in brain serotonin during prolonged exercise either hastens or delays fatigue respectively
- New evidence points to ration of serotonin linked to fatigue, sleepiness, depressed mood) to dopamine that either leads to fatigue or arousal
- Brain levels of NE also contribute to fatigue or arousal
Central governor model has some criticism
- Focuses on conscious and subconscious brain not spinal cord or motor unit
- Proposes that the brain regulates exercises intensity in an effort to protect the body from exercise-related damage or by disrupting cellular homeostasis
- The central governor limits/control exercise intensity vy neurally recruiting a certain amount of motor units innervating skeletal muscle
Peripheral Fatigue: Neural Factors
- Majority of evidence for fatigue points to peripheral factors where neural mechanical or energetic events can reduce tension development
Neural Factors
Sarcolemma and transverse tubules - relates to ability of muscle membrane to conduct an action potential
- Inability of Na/K pump to maintain action potential amplitude
- Na/K pump capacity can be improved by training
- Gradual depolarization of sarcolemma can result in action potential block in the T-tubules which signals calcium release from SR
- Reduction in calcium release from SR affects muscle contraction
Neuromuscular Junction
Not a site of fatigue
Peripheral Fatigue Mechanical Factors
- Cross Bridge Cycling and tension development
- High H+ Concentration may contribute to fatigue
- Longer relaxation time is a sign of fatigue
Cross bridge cyclin and tension development depend on
- Arrangement of actin and myosin
- Calcium being available to bind with troponin
- ATP availability needed for activation (causes movement) and dissociation (causes relaxation) of cross bridges
High H+ concentrations may contribute to fatigue by
- Reducing the force per cross-bridge
- Reducing the force generated at given Calcium concentration
- Inhibiting calcium release from SR
Longer Relaxation time is a sign of fatigue
- Relates to time of peak tension development to baseline tension
- Due to slower cross-bridge cycling
Peripheral Fatigue: Energetics of Contraction
- Imbalance between ATP requirements and ATP generating capacity
- Rate of ATP utilization is slowed faster than rate of ATP generation due to cellular fatigue mechanism