Eyes and Vision Flashcards

1
Q

Who is eligible for diabetic eye screening?

A

Diabetic patients
>12 years
Letter every year (unless last 2 have been normal in which case every 2 years).

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2
Q

What is uveitis?

A

Inflammation of the iris, choroid or ciliary body. There may also be secondary inflammation of the retina and optic nerve.

Anterior uveitis: (most common)
Iris / ciliary body

Intermediate uveitis:
Vitreous, ciliary body, retina

Posterior uveitis:
Choroid, retina, optic nerve

Panuveitis:
All parts of the eye

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3
Q

What causes uveitits?

A

50% idiopathic

50%: infection, neoplasia, autoimmune, trauma

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4
Q

What is the difference between acute and chronic uveitis? How does uveitis present.

A

Acute <3 months
Red, aching, watery eye with blurred vision and photophobia. Pain is worse on pupil constriction (near objects/bright light - direct and consensual photophobia).

Chronic > 3 months
Blurred vision and mildly red
Pain and photophobia are milder

Intermediate uveitis may present with floaters and blurred vision. Does’t present with pain or redness typically.

Panuveitis presents as a combination of the above.

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5
Q

What is the difference between direct and consensual photophobia?

A

Direct photophobia is pain in an eye when light is shone into it directly

Consensual photophobia (“True photophobia” is when pain is experienced in the other eye when light is shone into one eye. Occurs in iritis

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6
Q

How is uveitis diagnosed?

A

Slit lamp examination of the anterior segment + dilated fundus exam

Done in secondary care. Uveitis should be managed by an ophthalmologist.

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7
Q

How does diabetic retinopathy present?

A

Gradual progression:
Blurring, patchy vision loss, difficulty seeing in the dark, floaters, fluctuating vision.

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8
Q

What do you expect to see on fundoscopy of an eye with glaucoma?

A

Pathological cupping or pallor of the optic disc

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9
Q

What is the difference between occular hypertension and glaucoma?

A

Occular HTN is simply raised IOP but without signs of glaucoma. It occurs in about 5% of >40 year olds and requires monitoring, as it may progress to glaucoma.

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10
Q

How is glaucoma classified?

A

Age: Congenital, infantile, juvenile, adult

Cause: Primary (unknown), secondary (known)

Rate of onset: Acute, subacute, chronic.

Angle between iris and cornea: open or closed

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11
Q

What is the most common form of glaucoma?

A

Primay, open angle glaucoma (POAG) - usually chronic. Affects 2% of people over 40 years old

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12
Q

How does acute angle closure present?

A

Acutely painful red eye

May also exhibit headache, impaired vision, lights surrounded by haloes, semi-dilated or fixed pupil.

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13
Q

What two conditions are associated with seeing haloes around lights?

A

Glaucoma
Cataracts

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14
Q

How do cataracts present?

A

Cloudy vision
Haloes around lights
Poor night vision
Sensitivity to light

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15
Q

True or false, asian females are more likely to develop acute angle glaucoma?

A

True

Especially if long-sighted and older

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16
Q

True or false, if a patient with acute angle glaucoma cannot be seen same day in secondary care, primary care can administer treatment?

17
Q

How might you manage acute angle glaucoma in primary care if there is a delay in being seen by secondary care?

A

Lie the patient flat, facing up, with no pillows

Pilocarpine drops - 1 drop 2% in blue eyes and 4% in brown eyes

Acetazolamide 500mg PO

Antiemetic + Analgesia

18
Q

True or false, different doses of pilocarpine are required when treating acute angle glaucoma in blue and brown eyed patients?

A

True

1 drop 2% for blue
1 drop 4% for brown

Pilocarpine takes longer to work in brown eyes so higher dose is needed

19
Q

How do pilocarpine and acetazolamide work in the treatment of acute angle glaucoma?

A

Pilocarpine - constructs the pupil (miosis) facilitating drainage of fluid from the eye

Acetazolamide inhibits aqueous humour production thus reducing IOP. It also promotes urinary excretion of salts.

20
Q

How might you recognise orbital cellulitis (post-septal)?

A

Proptosis
Blurred vision
Chemosis (swelling of conjunctiva)
Eye lif oedema
Double vision
Systemic signs e.g. fever

Needs urgent treatment in secondary care

Pre-septal cellulitis is more common and benign. It will have no proptosis or visual change.

21
Q

What is a Chalazion? How are they treated?

A

Meibomian cyst

Chronic, non-infectious, inflammatory granuloma of the meibomian gland ducts (oil gland) of the eye lid.

Most self-limit in weeks to months
Apply warm compress
Lid massage

22
Q

How are Styes different from Chalazions?

A

Styes (hordeola) are infected eye-lid hair follicles (apocrine/sebaceous gland)

Chalazions are non-infectious inflammatory granulomas of meibomian oil ducts.

24
Q

What is macular degeneration?

A

Degeneration of the central part of the retina (macula) in people aged 50 and over

25
Give two risk factors for macular degeneration
Older age Family history of AMD Smoking Genetic
26
What is the difference between wet and dry macular degeneration? Which is more common?
Wet MD occurs when abnormal blood vessels proliferate in the retina and cause exudate of fluid/blood. Wet is more rare but more likely to cause significant impairment. Dry MD is degeneration of the macula with no exudate. Dry is more common and less aggressive.
27
What are Drusen? What condition are they associated with?
Lipid deposits beneath the retinal pigment epithelium (RPE) They are associated with macular degeneration
28
What is the most common cause of severe visual impairment in older adults in the developed world?
Advanced Macular Degeneration
29
What are some features you might expect to find in the retina of patients with macular degeneration?
Retinal Pigment Epithelium (RPE) abnormalities - hypo/hyperpigmentation Drusen - lipid deposits under the RPE Neovascular - leaky blood vessel formation in the choroid Geographic atrophy - loss of colour cells - seen as loss of pigmentation
30
How does macular degeneration present?
Distortion of lines (appear wavy/crooked) Painless loss of central/near central vision Scotoma - patch of grey in centre of vision Difficulty with detailed vision e.g. reading Flickering/Flashing lights Difficulty adjusting from bright to dim light Visual hallucinations (in extreme forms)
31
True or false, in glaucoma, patients typically lose the periphery of their vision but retain central vision?
True
32
How quickly should patients with macular degeneration be seen in secondary care?
Urgent (within a week)
33
True or false, if a person with macular degeneration only affecting one eye, they may not have to inform the DVLA?
True As long as they can meet the visual strandards for driving If AMD affects both eyes, they must inform the DVLA
34
True or false, patients with AMD who are receiving treament do not need a certificate of visual impairment?
False All patients with AMD should be given a certificate of visual impairment, regardless of whether they are receiving treatment.
35
What do "Low vision services" offer patients with AMD?
Advice on how to manage with impaired vision e.g. training with optical aids (magnifiers), appropriate lighting, tactile and electronic aids. They can also receive advice regarding benefits and allowances.
36
What are the treatments for AMD in secondary care?
Anti-VEGF Anti-angiogenic medication which blocks vascular endothelial growth factor. This reduces the leakiness of blood vessels. Laser photocoagulation: This destroys abnormal or leaky blood vessels
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