Exam1 Flashcards
For ANY disease, the patient must be ______ before performing OMT
Stable
New onset of chest pain or shortness of breath is not the time for what?
OMT
Somatic dysfunctions can occur where?
Anywhere!
Sympathetic levels
Parasympathetic levels
Soma (not autonomic related)
Viscerosomatic reflexes occur at?
Sympathetic levels and Parasympathetic levels
Facilitated segments ONLY occur at?
Sympathetic levels
What is the treatment for temporal arteritis?
Steroids
Sometimes, muscle hypertonicity, contraction, and/or spasm can be caused by?
Direct irritation of the muscle from the structure overlying it
If there is a renal lithiasis, it may cause?
It may cause the psoas to become hypertonic & you would have a positive Thomas test
If there is appendicitis, it may cause?
The psoas to become hypertonic and you would have a positive Thomas test
If there are inflamed lymph nodes, it may cause?
The muscle they are touching to become hypertonic, such as the sternocleidomastoid
After obtaining a history, you perform what?
A physical exam
What is the first thing you do in a physical exam?
Observation
A reversible dextroscoliosis or levoscoliosis means?
There is NO Sagittal component present (no flex ion or extension component) so it follows Fryette Type I mechanics
A dextroscoliosis would have the convex side pointing which direction?
Right. Therefore indicating a neutral side-bending left, rotating right pattern for the vertebrae
A dextroscoliosis from T4-T6 would have all the vertebrae?
Neutral, side bent left, rotated right
Left lateral convexity means?
Vertebrae are sidebent right
Right lateral convexity means the vertebrae?
Side-bent
When treating a group dysfunction with OMT, treat what group of the curve
The apex. Example T10-T12, then go for T11
Type II dysfunction would usually occur at?
The apex/middle of the group curve
Translation to the right =
Left side bending
Translation to the left =
Right sidebending
Principle 1 of osteopathic medicine
The body is a unit; the person is a unit of mind, body, spirit.
Example of principle 1
Gastric ulcer causes thoracic tissue texture changes
Principle 2 of osteopathic medicine
The body is capable of self regulation, self healing & health maintenance
Example of principle 2
Healed fracture
Principle 3 of Osteopathic medicine
Structure & function are reciprocally interrelated
Principle 4 of osteopathic medicine
Rational treatment is based upon an understanding of the basic principles of body unity, self-regulation, & the inter-relationship of structure and function
Biomechanical model
Anatomy of muscles, spine, extremities; posture, motion
OMT directed toward normalizing mechanical somatic dysfunction, structural integrity, physiological function, homeostasis
Neurological model
Emphasizes CNS, PNS and ANS that control, coordinate and integrate body functions
Proprioceptive and muscle imbalances, facilitation, nerve compression disorders, autonomic reflex and visceral dysfunctions, brain/CNS dysfunctions
Respiratory/circulatory model
Emphasizes pulmonary, circulatory and fluid (lymphatic, CSF) systems
Lymphatic techniques
Metabolic/nutritional model
Regulates through metabolic processes
Behavioral (psychobehavioral)
Focuses on mental, emotional, social & spiritual dimensions related to health & disease
Post ganglionic sympathetic fibers lead to what?
Tissue texture changes, such as hypertonicity, moisture, erythema, etc…
Dorsal horn of the spinal cord is where somatic and visceral AFFERENTS nerves do what?
Synapse! Giving a viscerosomatic reflex
Somatosomatic reflex
localized somatic stimuli producing patterns of reflex response in segmentally related somatic structures. For example, rib somatic dysfunction from an innominate dysfunction.
Somatovisceral reflex
localized somatic stimulation producing patterns of reflex response in segmentally related visceral structures. For example, triggering an asthmatic attack when working on thoracic spine
Viscerosomatic reflex
localized visceral stimuli producing patterns of reflex response in segmentally related somatic structures. For example gallbladder disease affecting musculature.
A
Viscerovisceral reflex
localized visceral stimuli producing patterns of reflex response in segmentally related visceral structures. For example, myocardial infarction and vomiting.
Old is _____, hot is _____
Cold, not
Signs of acute somatic dysfunction when palpating
Recent history (injury) Sharp or severe localized pain Warm, moist, sweaty skin Boggy,, edematous tissue Erythematous Local increase in muscle tone, contraction, spasm, increased muscle spindle firing Normal or sluggish ROM May be minimal or no somatovisceral effects
Signs of chronic somatic dysfunction when palpating
Long-standing Dull, achy diffuse pain Cool, smooth, dry skin Possible atrophy Fibrotic, ropy feeling tissue Pale/skin pallor Decreased muscle tone, contracted muscles, sometimes flaccid Restricted ROM Somatovisceral effects more often present
Orientation of superior facets
Cervical: BUM
Thoracic: BUL
Lumbar: BM
Orientation of inferior facets
Cervical: AIL
Thoracic: AIM
Lumbar: AL
Fryette Law 1
When side-bending is attempted from neutral (anatomical) position, rotation of vertebral bodies follows to the opposite direction.
Typically applies to a group of vertebrae (more than two)
Occurs in a neutral spine (no extreme flexion or extension) NO SAGITTAL COMPONENT
Side-bending and rotation occur to opposite sides
Side-bending precedes rotation
Side-bending occurs towards the concavity of the curve
Rotation occurs towards the convexity of the curve
Diagnosed as a Type I dysfunction
Fryette Law 2
When side-bending is attempted from non-neutral (hyperflexed or hyperextended) position, rotation must precede side-bending to the same side.
Typically applies to a single vertebra
Occurs in a non-neutral spine (flexion or extension of spine present) SAGITTAL COMPONENT
Side-bending and rotation occur to same sides
Rotation precedes side-bending
Rotation of the vertebra occurs into the concavity of the curve
Diagnosed as a Type II dysfunction
May be described as traumatic injury
If indirect treatment is used, the dysfunction is?
Exaggerated/augmented
If direct treatment is used, the dysfunction is?
The barrier is engaged/dysfunction reversed
Examples of Direct Techniques
Myofascial Release (May also be indirect)
Soft tissue
Articulatory
Muscle Energy
High velocity, low amplitude (HVLA)
Springing
Cranial (may also be indirect)
Still Technique (combined indirect and direct)
Initial positioning of Still Technique set up is indirect
Ending positioning of Still Technique is direct
Stretching
Longitudinal or parallel traction technique in which the origin & insertion of the myofascial structures being treated are longitudinally separated
Kneading
A perpendicular traction technique in which a rhythmic, lateral stretching of a myofascial structure, where the origin and insertion are held stationary and the central portion of the structure is stretched like a bowstring
Inhibition
A deep inhibitory pressure, which is a sustained deep pressure over a hypertonic myofascial structure
Effleurage
Gentle stroking of congested tissue used to encourage lymphatic flow
Petrissage
Involves pinching or tweaking one layer and lifting it or twisting it away from deeper areas
Tapotement
A striking the belly of a muscle with the hypothecate edge of the open hand in rapid succession in order to increase it’s tone and arterial perfusion. A hammering, chopping percussion of tissues to break adhesions and/or encourage bronchial secretions
Examples of indirect techniques
Counterstrain FPR BLT Functional technique Myofascial release Cranial Still technique
Neurologic exam of root C4
Sensation- shoulder
Motor- none
Reflex- none
Neurologic exam root of C5
Sensation- lateral elbow
Motor- biceps
Reflex- biceps
Neurologic exam of root C6
Sensation- thumb, index finger
Motor- wrist extensors
Reflex- triceps
Neurologic exam root of C7
Sensation- mid finger
Motor- triceps
Reflex triceps
Neurologic exam root C8
Sensation- ring finger, pinky
Motor- wrist flexors
Reflex- none
Neurologic exam root of T1
Sensation- medial elbow
Motor- interossi
Reflex- none
Sympathetic level for head and neck
T1-T4
Sympathetic levels heart
T1/T2-T5/T6
Sympathetic levels respiratory
T1/T2-T6/T7
Sympathetic levels esophagus
T2-T8
Sympathetic levels upper GI tract
T5-T9
Stomach, liver, gallbladder, spleen, pancreas, duodenum
Sympathetic levels middle GI tract
T10-T11
Pancreas, duodenum, jejunum, ileum, ascending colon, right transverse colon, kidney, upper ureter, gonads
Sympathetic levels lower GI tract
T12-L2
Left transverse colon, descending colon, sigmoid colon, rectum, prostate, bladder, lower ureter
Sympathetic levels appendix
T10-11
Sympathetic levels kidneys
T10-T11
Sympathetic levels adrenal medulla
T10
Sympathetic levels upper ureters
T10-T11
Sympathetic levels lower ureters
T12-L2
Sympathetic levels gonads
T10-T11
Sympathetic levels bladder
T12-L2
Sympathetic levels uterus & cervix
T10-L2
Sympathetic levels erectile tissue
T11-L2
Sympathetic levels prostate
T12-L2
Sympathetic levels arms
T2-T8
Sympathetic levels legs
T11-L2
Parasympathetic levels
-vagus nerve (OA, AA, C2)
Trachea, esophagus, heart, lungs, liver, gallbladder, stomach, pancreas, spleen, kidneys, proximal ureter, small intestine, ascending colon, & transverse colon up to the splenic flexure
Parasympathetic levels S2-S4
Distal to splenic flexure of the transverse colon descending colon, sigmoid colon, rectum, distal ureter, bladder, reproductive organs, & genitalia
Parasympathetic levels
Ovaries & testes
Vagus nerve, S2-S4
Sympathetic pre-ganglionic T5-T9
Greater Splanchnic
Sympathetic Pre-ganglionics T10-T11
Lesser Splanchnic
Sympathetic Pre-ganglionics T12
Least Splanchnic
Sympathetic Pre-ganglionics L1-L2
Lumbar Splanchnic
Celiac Ganglion (T5-T9) Post-Ganglionic to what structures?
Distal esophagus, stomach (epigastric), liver, gallbladder (cholecystitis), spleen, portions of pancreas, proximal duodenum (foregut)
Superior mesenteric ganglion (T10-T11) post-ganglionic to what structures?
Portions of pancreas, duodenum, jejunum, ileum, ascending colon, proximal 2/3 of transverse colon (midgut); adrenals, gonads, kidneys, upper 1/2 ureter
Inferior mesenteric ganglion (T12-L2) post-ganglionic to what structures?
Distal 1/3 transverse colon, descending colon, sigmoid, rectum (handgun)l; lower 1/2 ureter, bladder, prostate genitalia
Greater splanchnic nerve
Sympathetic innervation
T5-T9
Synapses at the celiac ganglion
Stomach, liver, gallbladder, pancreas, parts of duodenum
Lesser splanchnic nerve (T10-11)
Sympathetic innervation
Synapses at superior mesenteric ganglion
Small intestines & right colon (appendix is found here)
Least splanchnic nerve (T12) & Lumbar splanchnic (L1-2)
Sympathetic innervation
Synapses at the inferior mesenteric ganglia
Innervation the left colon & pelvic organs
Sympathetic nerve supply to the head & neck
T1 – T4
It forms the cervical ganglia (inferior, middle, and superior cervical ganglia), which affects the mid to lower cervical spine
From there, it contributes to other collateral ganglion that govern the sympathetic innervation to the head
Increased sympathetic activity
Increased goblet cells Increased thick, sticky secretions Dries the mucous membranes Pupillary dilation Decreased lymphatic/circulatory drainage Impaired immune response Tinnitus Increased intraocular pressure
Chapman Reflex Points
Liver
Anterior 5th intercostal space near sternum on R
Chapman Reflex Points
Stomach
Anterior 5th intercostal space near sternum on L Stomach acid (think ulcers/NSAID use/steroid use)
Chapman Reflex Points
Liver, gallbladder (think cholecystitis)
Anterior 6th intercostal space near sternum on R
Chapman Reflex Points
Pancreas (think amylase/lipase/blood glucose)
Anterior 7th intercostal space near sternum on R
Chapman Reflex Points
Stomach (peristalsis)
Anterior 6th intercostal space near sternum on L
Stomach peristalsis think emptying time
Chapman Reflex Points
Spleen
Anterior 7th intercostal space near sternum on L
Chapman Reflex Points
Appendix
Anterior tip of right 12th rib
Sympathetic Innervation: Chapman’s Reflexes
5th IC space
Right: Liver
Left: Stomach Acid (gastritis) (may raise red flag to NSAID use)
Sympathetic Innervation: Chapman’s Reflexes
6th IC space
Right: liver, gallbladder (cholecystitis)
Left: stomach peristalsis (may have delayed stomach emptying time, food may not pass quickly through system)
Sympathetic Innvervation: Chapman Reflexes 7th IC space
Right: pancreases (glucose, amylase, lipase)
Left: spleen
Chapman Reflex Points are also known as what?
Ganglioform Nodules or Tissue
CRP sinuses
Anterior points: lie 7 to 9 cm lateral to the sternum on the upper edge of the second rib
Posterior points: upon C2 midway between the spinous process & tip of the transverse process
CRP pharynx
Anterior points: lie upon the first ribs 3 or 4 cm medial to where the ribs emerge from beneath the clavicles
Posterior points: upon C2 midway between the spinous process & the tip of the transverse process
CRP Larynx
Anterior point: lie upon the second ribs, 5 to 7 cm lateral to the sternocostal junction
Posterior points: upon C2 midway between the spinous process & the tip of the transverse process
CPR tonsils
Anterior points: between the first & second ribs adjacent to the sternum
Posterior points: midway between the spinous process **(posterior tubercle) & the tip of the transverse process
CPR Middle Ear
Anterior points: lie upon the superior anterior aspect of the clavicles just lateral to where they cross the first ribs
Posterior points: upon the posterior aspect of the tips of the transverse processes of C1
CPR Eye (retina/conjunctiva)
Anterior points: lie upon the anterior aspect of the humerus at the level of the surgical neck
Posterior points: lie upon the squamous portion of the occipital bone below the superior nuchal line
Parasympathetic cranial nerves
III, VII, IX, X
Ganglion for CN III
Ciliary ganglion
Ganglion for CN IX
Otic ganglion
Ganglion for facial nerve
Pterygopalatine ganglion & submandibular ganglion
Although CN V conveys no presynaptic parasympathetic (visceral efferent) fibers from the CNS, ___ _______parasympathetic ganglia are associated with ___ _____ __ ___ _
The divisions of CN \
Increased parasympathetic activity results in ?
Increased clear, thin, watery secretions of glands
Pupillary constriction
Improved/increased drainage
Dry mouth (Xerostomia)
Can be caused by cranial nerve VII (lacrimal, sublingual and submandibular glands) & cranial nerve IX (parotid gland)
CN VII is associated with what ganglion and exits through which foramen?
Associated with sphenopalantine (pterygopalatine ganglion) & exits through the stylomastoid foramen
CN IX is associated with what ganglion and exits though which foramen?
Otic ganglion and exits through the jugular foramen
Before treating lymphatics, what must you do first?
Thoracic inlet/outlet has to be cleared/opened/treated before any other lymphatic treatment
What are the components of the thoracic inlet/outlet?
Supraclavicular space and 1st rib
Venous drainage
Approximately 85% of the venous drainage from the head occurs via the internal jugular veins
They pass through the jugular foramina, located along the occipitomastoid suture between occipital and temporal bones
Altered temporal bone motion and occipitomastoid compression may impair what?
Venous flow through the jugular foramen and may lead to congestion in the head
Myofascial release to the pteryoid fascia can be used to treat what?
Maxillary sinusitis by stimulating the parasympathetic supply to the nasal mucosa, which is CN VII
Galbreath technique is great for treating what?
Otitis media, fluid in the middle ear, Eustachian tube somatic dysfunction
Galbreath technique
You are pulling traction on the mandible and the goal is decongestion of fluid in the middle ear
CN I dysfunction/entrapment
Anosmia
CN V dysfunction/entrapment
Headache, trigeminal neuralgia
CN VII dysfunction/entrapment
Altered taste, bell’s palsy
CN VIII entrapment/dysfunction
Vertigo, tinnitus, labyrinthitis
CN IX & X dysfunction/entrapment
Poor suckling, failure to thrive
CN III, IV, VI dysfunction/entrapment
pass under the Petrosphenoidal ligament formed by the tentorum cerebelli
Symptoms: blurred vision, diplopia, nystagmus, eye fatigue, HA
CN VI is closest to what?
The dura, most likely affected, results in medial strabismus
CN I through what foramen
Cribiform plate
CN II through what foramen
optic canal
CNIII, IV, V1, VI through what foramen
Superior orbital fissure
CNV2 (maxillary n) through what foramen?
Foramen rotundum
CNV3 (mandibilar n) through what foramen?
Foramen ovale
CN VII & CN VIII through what foramen?
Internal acoustic meatus
CN IX, X, XI through what foramen?
Jugular foramen
CNXII through what foramen?
Hypoglossal canal
Difficulty latching/nursing
CN XII
Colic
CN X
GERD
CN X
Nausea/vomiting
CN X
Torticollis
CN XI
Asthma
CN X
Otitis media/tinnitus/vertigo/labrynthitis
CN VIII
CN I
Anosmia
Cribiform plate through ethmoid bone
CN V
Trigeminal neuralgia/tic deloureaux
May complain of sudden, stabbing severe facial, ear and/or jaw pain
CN VII
Exits stylomastoid foramen
Bell’s Palsy
CN VIII
Labyrinthitis, tinnitus, vertigo
temporal bone is associated with these*
CN X
Exits jugular foramen
Can cause nausea/vomiting
CN XII
Hypoglossal canal
Can cause nursing/latching problems in infants
Jugular foramen is formed by what bones?
Temporal bone and occiput which make up occipitomastoid suture
CN IX, X, & XI exit the jugular foramen
Tension headache description
Bilateral pressure
No aura, nausea
Migraine headache description
Unilateral
Triggers
May have aura, nausea/vomiting, photophobia/phonophobia
Cluster headache description
Unilateral, severe
Trigger point for SCM
Refers pain lateral & behind the eye
Trigger point splenius capitus
Refers pain to the vertex of the head