Exam: Respiratity And Adaptations Flashcards

1
Q

Purpose of the respiratory system

A

Provide O2 to the body for energy production
Remove CO2 from the lungs

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2
Q

Four processes of the respiratory system

A

Pulmonary ventilation (external respiration)
Pulmonary diffusion (external respiration)
Transport of gases via blood
Capillary diffusion (internal respiration)

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3
Q

What is a ventilation

A

Process of moving air into and out of lungs

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4
Q

How does the air travel

A

Transport zone: Nose/mouth πŸ‘ͺ nasal conchae πŸ‘ͺ pharynx πŸ‘ͺ larynx πŸ‘ͺ trachea πŸ‘ͺ bronchial tree

Exchange zone: alveoli

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5
Q

Pleural sacs

A

Lungs suspended by pleural sacs
Parietal pleura lines thoracic wall.
Visceral (pulmonary) pleura attaches to lungs.
Lungs take size and shape of rib cage.

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6
Q

Inspiration

A

aka inhalation
Active part of VE (e.g. lungs, sternum, intercostals, diaphragm)
Pressure of O2 is lower in lungs than atmosphere
Vacuum effect

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7
Q

Expiration

A

aka exhalation
Passive part of VE (e.g. lungs, sternum, intercostals, diaphragm)
Pressure of CO2 is lower in atmosphere than lungs

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8
Q

Alveoli/pulmonary capillaries (external respiration)

A

O2/CO2 exchange
Pressure differences (see diagram on pg 198)
respiratory membrane
Interface btw capillary and alveoli in lungs
Some O2 directly to lungs; also lost via vaporizing, dissolving in CO2
O2 binds to Hb

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9
Q

Capillary/muscle (internal respiration)

A

O2/CO2 exchange
Pressure differences (see diagram on p198)

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10
Q

Pulmonary ventilation (breathing)

A

movement of air into and out of the lungs

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11
Q

Pulmonary diffusion:

A

the exchange of O2 and CO2 between the lungs and the blood

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12
Q

Capillary diffusion

A

the exchange of O2 and CO2 between the capillary blood and the metabolically active tissue

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13
Q

% of co2 dissolves in blood

A

10

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14
Q

% of co2 binds to Hb

A

20

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15
Q

% of co2 is carried originally as carbonic acid, then bicarbonate ion and H ions

A

70

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16
Q

where does cho drive pH

A

down

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17
Q

how is co2 carried around the body

A

as HCO3

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18
Q

The relationship (and problem) between more work and H+

A

More work = more H+ (CO2 and Lactic acid)
Higher acidity = more pain/burning sensation
More H+ = more difficulty in producing and ridding the body of CO2(primary reason: blood flow speed)
More CO2 = increased rate and depth =Fatigue

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19
Q

How breathing is controlled

A

Respiratory Center (brain stem) signals lungs to increase rate and depth due to increase in H+ and CO2 (read p207)
Chemoreceptors (neurons) in aorta and carotid
Stretch receptors (neurons) in bronchioles and alveoli
All involuntary

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20
Q

RR at rest and work

A

respiratory rate 10, 50

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21
Q

TV at rest and work

A

Tidal volume (0.5L, 4L)

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22
Q

VE formula

A

RRxTV

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23
Q

Adaptations to Aerobic Training:
Cardiorespiratory Endurance

A

increased maximal endurance capacity = V*O2max

submaximal endurance capacity:
Lower HR at same submaximal exercise intensity
More related to competitive endurance performance

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24
Q

what is Cardiorespiratory endurance and how is it imporved

A

Ability to sustain prolonged, dynamic exercise
Improvements through multisystem adaptations (cardiovascular, respiratory, muscular, metabolic)

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25
Does an aerobic practice has an impact on aerobic speed
Si
26
Heart size adaptations
With training, 👩 heart mass and LV chamber size – 👩 target pulse rate (TPR) πŸ‘ͺ cardiac hypertrophy πŸ‘ͺ 👩 SV – 👩 plasma volume πŸ‘ͺ 👩 LV volume πŸ‘ͺ 👩 EDV πŸ‘ͺ 👩 SV Volume loading effect SV 👩 after training Resting, submaximal, maximal Plasma volume 👩 with training πŸ‘ͺ 👩 EDV πŸ‘ͺ 👩 preload Resting and submaximal HR 👫 with training πŸ‘ͺ 👩 filling time πŸ‘ͺ 👩 EDV 👩 LV mass with training πŸ‘ͺ 👩 force of contraction Attenuated 👩 TPR with training πŸ‘ͺ 👫 afterload SV adaptations to training 👫 with age
27
Who has the biggest heart
cyclists
28
Stroke volume trained/non-trained
values significantly increase if trained, not as much as if not pre-trained
29
Resting HR adaptations
👫 markedly (~1 beat/min per week of training) – 👩 parasympathetic, 👫 sympathetic activity in heart
30
Submaximal HR
– 👫 HR for same given absolute intensity More noticeable at higher submaximal intensities
31
Maximal HR
No significant change with training – 👫 with age
32
HR–SV interactions
Does 👫 HR πŸ‘ͺ 👩 SV? Does 👩 SV πŸ‘ͺ 👫 HR? HR, SV interact to optimize cardiac output
33
HR recovery
Faster with training Indirect index of cardiorespiratory fitness
34
Cardiac output (Q*)
Little or no change at rest or during submaximal exercise with training Maximal Q* 👩 considerably (due to 👩 SV)
35
Changes in heart rate during recovery after a 4 min, all-out bout of exercise before and after endurance training. (trained/non-trained)
Pre-training, heart rate increases linearly during exercise and plateaus, then slowly declines during recovery but has not fully returned to resting values at minute 8 of recovery. Post-training, heart rate still increases linearly during exercise and plateaus, but **recovers much more rapidly** and is back to resting values by minute 7.
36
Changes in cardiac output with endurance training
Line graph showing changes in cardiac output pre- and post-endurance training. Pre-training, cardiac output starts at about 5 L/min at 5 kph and increases linearly to 20 L/min at 16 kph, then plateaus at this value despite further increases in treadmill speed. Post-training, cardiac output starts at 5 L/min at 5 kph, then increases to 28 L/min at a treadmill speed of 20 kph, then plateaus at this value despite further increases in treadmill speed.
37
blood flow to active muscle
👩
38
capillarization, capillary recruitment
👩
39
blood flow to inactive regions
👫
40
total blood volume
👩
41
name some cardiovascular adaptations
👩 total blood volume * 👫 blood flow to inactive regions 👩 capillarization, capillary recruitment 👩 blood flow to active muscle HR recovery Faster with training Blood volume: total volume 👩 rapidly – 👩 plasma volume via 👩 plasma proteins, 👩 water and Na+ retention (all in first 2 weeks) – 👩 red blood cell volume (hematocrit possibly 👫) – 👫 plasma viscosity
42
Blood volume
👩 rapidly 👩 plasma volume via 👩 plasma proteins, 👩 water and Na+ retention (all in first 2 weeks) – 👩 red blood cell volume (hematocrit possibly 👫) – 👫 plasma viscosity
43
Adaptations to Aerobic Training: Respiratory
Pulmonary ventilation Pulmonary diffusion Arteriovenous O2 difference
44
Pulmonary ventilation adaptations
👫 at given submaximal intensity – 👩 at maximal intensity due to 👩 tidal volume and respiratory frequency
45
Pulmonary diffusion
Unchanged during rest and at submaximal intensity – 👩 at maximal intensity due to 👩 lung perfusion
46
Arteriovenous O2 difference
👩 due to 👩 O2 extraction, active muscle blood flow – 👩 O2 extraction due to 👩 oxidative capacity
47
Adaptations to Aerobic Training: Muscular
Fiber type Capillary supply Myoglobin Mitochondrial function Oxidative enzymes (SDH, citrate synthase)
48
Fiber type
👩 size and number of type I fibers (type II πŸ‘ͺ type I) Type IIx may perform more like type IIa.
49
Capillary supply
👩 number of capillaries supplying each fiber May be key factor in V*O2max 👩.
50
Myoglobin
– 👩 myoglobin content by 75% to 80% Supporting 👩 oxidative capacity in muscle
51
Oxidative enzymes (SDH, citrate synthase)
– 👩 activity with training Continued increase even after V*O2max plateaus Enhanced glycogen sparing
51
Mitochondrial function
– 👩 size and number Magnitude of change dependent on training volume
52
Adaptations to Aerobic Training: Metabolic
Lactate threshold Respiratory exchange ratio (RER) Maximal V*O2 (V*O2max) Long-term improvement Training status and pretraining V*O2max Heredity High versus low responders Sex
53
Lactate threshold
– 👩 to higher percentage of V*O2max – 👫 lactate production, 👩 lactate clearance Allowing higher intensity without lactate accumulation
54
Respiratory exchange ratio (RER)
👫 at both absolute and relative submaximal intensities – 👩 dependent on fat, 👫 dependent on glucose
55
Maximal V*O2 (V*O2max)
Best indicator of cardiorespiratory fitness – 👩 substantially with training (15%-20%) – 👩 due to 👩 cardiac output and capillary density
56
Long-term improvement
12 to 18 months for VO2 Performance continues to 👩 after V*O2max plateaus because lactate threshold continues to 👩 with training.
57
Training status and pretraining V*O2max
Relative improvement dependent on fitness More sedentary individual: greater 👩 More fit individual: smaller 👩
58
Heredity
Finite V*O2max range determined by genetics (V*O2max altered within that range by training) V*O2max more similar for identical than fraternal twins 25%-50% of V*O2max variance due to heredity
59
High versus low responders
genes can determine VO2 training response Physiological mechanisms (cardiac output, expanded blood volume, improved O2 extraction) = LOTS variation in training outcomes with same training!
60
Sex
Untrained female V*O2max < untrained male V*O2max Trained female V*O2max closer to trained male V*O2max Differences in older men versus older women
61
Adaptations to Aerobic Training: Fatigue Across Sports
Endurance training is critical for endurance-based events. It is also important for nonendurance-based sports. All athletes benefit from maximizing cardiorespiratory endurance.
62
Adaptations to Aerobic Training: Aerobic Deconditioning
Bed rest is associated with disease and disability and produces effects like those of weightlessness during space travel. Body weight and lean body mass decline. Loss of cardiovascular fitness is greater if V*O2max was higher at the start of bed rest or weightlessness.
63
Adaptations to Anaerobic Training
Changes in anaerobic power and capacity Anaerobic power and capacity 👩 with training
64
standart amaerobic capacity test
wingateeeeeeeeeee bike test
65
Adaptations in muscle (anaerobic)
👩 in type IIa, IIx cross-sectional area – 👩 in type I cross-sectional area (lesser extent) – 👫 percentage of type I fibers, 👩 percentage of type II
66
ATP-PCr system anaerobic
Little enzymatic change with training ATP-PCr system-specific training πŸ‘ͺ strength 👩
67
Glycolytic system
👩 in key glycolytic enzyme activity with training (phosphorylase, PFK, LDH, hexokinase) But performance gains from 👩 in strength
68
Adaptations to High-Intensity Interval Training (HIIT)
HIIT: time-efficient way to induce many adaptations normally associated with endurance training HIIT stimulates mitochondrial biogenesis and changes in the capacity for carbohydrate and fat transport and oxidation
69
Specificity of Training and Cross-Training
Specificity of training V*O2max substantially higher in athlete’s sport-specific activity Likely due to individual muscle group adaptations
70
Specificity of Cross-Training
Cross-training Training different fitness components at once or training for more than one sport at once Strength benefits blunted by endurance training Endurance benefits not blunted by strength training
72
No long writing in the cumulative final Questions from all exams Define each of 4 types of breathing Change in pulmonary pressures Partial pressure- have a good understanding of that (oxygen moves wherever there is less concentration of oxygen) % of each CO2 travel options Breathing process- multiple choice question (how breathing works)
73
Capillary around a muscle fiber- be able to identify and discuss it (develops more rapidly doing aerobic training than no aerobic training)