exam questions Flashcards

1
Q
  1. What are the stages of Impingement Syndrome?
A

Neer’s stages of impingement syndrome

  • edema and hemorrhage
  • fibrosis and tendinitis
  • bone spurs
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2
Q
  1. What is the difference between a client with shoulder tendonitis versus Calcific tendonitis versus bursitis? Where would they feel pain?
A

refer to individual cards

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3
Q
  1. What is the typical picture of a client with shoulder tendonitis? What are the symptoms and physiology?
A

posture: forward shoulder, forward head.
symptoms: painful arch of motion seen with PROM and AROM. diffuse weakness, edema, spasms in scapular muscles.

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4
Q

***4. Name some common interventions for Tendonitis in the acute and subacute. Know why you would use them.

A

acute phase- conservative therapy
subacute- HEP, periodic eval and progression, 2x/week until sx begin to resolve, patient education to adapt activities, environment is critical, sleep and work positions (body pillows put under shoulder, hug like a teddy bear).

acute- active rest- arm 30 degrees abduction arm up on couch. Use w/o increasing pain and no resistance, not above 90. Out hand in pocket while walk. Ice, sleep supinebody pillow under arm 30 degrees hug like bear.
AAROM pendelum
subacute- do a little bit more start isometrics, massage, cardio, isotonics later

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5
Q
  1. After Rotator Cuff Repair surgery, what is the usual progression of interventions? When do you start a HEP of pendulums (Codmans)? What begins at 4-6 weeks post op as directed by the MD? 6- 8 weeks?
A

Conservative-nonoperative

  • Modalities
    • ultrasound
    • TENS
    • Hot/cold (short term pain relief)
  • Exercise- not one protocol
    • includes PROM, AAROM, AROM, strengthening, manual therapy.
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6
Q
  1. Describe the pathology that occurs with adhesive capsulitis.
A

Frozen shoulder

  • onset usually 50-70 years of age
  • women>men
  • more seen in sedentary workers, diabetics
  • develops slowly with pain and tenderness over deltoid insertion
  • motion aggravates pain
  • decreased P/AROM of shoulder
  • Decreased ADLs and sleep
  • shrugging shoulder motion
  • progresses to very decreased ROM and increased pain

PATHOLOGY:

  • many tissues are involved- such as bursa, glenohumeral capsule, biceps tendon, rotator cuff tendon
  • capsular thickening
  • repair of tissues is delayed or impaired by frequent irritation
  • muscles atrophy
  • 3 phases: freezing, frozen, and thawing phases.
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7
Q
  1. What are the evidence based practice interventions for Adhesive Capsulitis/Rotator Cuff?
A
  • Exercise was demonstrated to be effective in terms of short term recovery in rotator cuff disease and longer term benefit with respect to function.
  • Combining mobilisation with exercise resulted in additional benefit when compared to exercise alone for rotator cuff disease.
  • Laser therapy was demonstrated to be more effective than placebo for adhesive capsulitis but not for supraspinatus tendinitis.
  • Both ultrasound and pulsed electromagnetic field therapy resulted in improvement compared to placebo in pain in calcific tendinitis.

-There is no evidence of the effect of ultrasound in shoulder pain (mixed diagnosis), adhesive capsulitis or rotator cuff tendinitis. When compared to exercises, ultrasound is of no additional benefit over and above exercise alone.

-There is some evidence that for rotator cuff disease, corticosteroid injections are
superior to physiotherapy

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8
Q

***8. What are the classifications for fractures and relationship of fragments? Can you label them if given a picture?

A

-origin
-stress, traumatic, pathological
-anatomical location
-base, midshaft, neck or articular
***ON TEST
fx line
-transverse, oblique, spiral, comminuted (BAD-more than 2 pieces)
relationship of fragments
-closed, open, complete, incomplete, dorsa(not good)l and volar(not good)

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9
Q
  1. What are the various methods of reduction for fracture? Can you label them if given a picture?
A

-closed, open or distraction techniques (cast, immobilized)
-ORIF (open reduction internal fixation)- when closed techniques wont work, use Kirschner wires, interosseus screws, plates with screws
-external fixators- distraction
casting, splinting

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10
Q
  1. What is the normal range for fracture healing in infants, adolescents and adults?
A

a. normal bone is regenerated (fx. bone heals)
rate depends on age, fx characteristics, presence of disease, vascularity of bone segment.
*infants- 4-6 wks
*adolescence- 6-10 wks
adults- 8 weeks and on
(AROM: means you can move safely- wait 2-4 more wks for strengthening).

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11
Q
  1. Examine the progression of intervention for clients with a humeral fracture. Know the general progression so that if an intervention is described, you would know if it was indicated for a client or not.
A

a. Acute
- Pendulum, edema control- wrap it, don’t apply to much pressure (don’t let sh. get stiff, do it soon according to MD).
- Cold to heat modalities- switch quickly to heat
- One handed ADL’s- assistive dressing
- Distal AROM- take arm out of sling 3x a day- move sh., elbow, and wrist- NO ER or IR

3-4 wks

  • gentle AAROM sh, not > than 60 degrees of elevation- pt. is healing based on MD eval.
  • cane, towel on table, pulleys

4-6 wks
- sh. AAROM, isometric scapular protraction/retraction
(lay in bed or chair and push against bed or chair)

8-10 wks
-stretching (bone healing well)

10-12 wks
-sh. isotonics

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12
Q
  1. Describe the pathology that occurs with anterior shoulder dislocation AKA traumatic unidirectional instability (TUBS). What are the clinical signs? What are the usual conservative interventions?
A

Traumatic Unidirectional instability (TUBS)- lax connective tissue usually result of trauma in one direction (anterior, posterior, or inferior)- may damage to RC tendons and glenoid labrum ( anterior are weaker; athletes)
TUBS- (numbness and tingling)
-anterior- usually with a force against the abducted, ER sh. - labrum a problem also.
-posterior instability- forward flexed humerus or FOOSH
-inferior instability- RC weakness

Medical tx- may need surgery- Bankhart repair- use subscpularis to support the anterior capsule- (gives xtra strength)
clinical picture/ func. limitations
-inability to reach or lift at sh. level
-restricted ability in many ADL’s, leisure
-pain when sleeping

(NO AB or ER- avoid lifting arm over 90 degrees.

Interventions (4-8 wks)

  • immobilized in sling for 3 wks, elbow distally needs ex., strike zone mvnts.
  • precautions- no PROM, EX with AB or hyper/
  • do ex rot with arm at side only to 50 degrees
  • strengthening
  • int/ext rotators, int roators and adductors need to be strong to support ant capsule
  • ext rotators strong to cause humeral depression
  • begin with isometric ex with joint at side pain free
  • progress to isotonic ex still limiting
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13
Q
  1. Describe the difference between Rheumatoid Arthritis and Osteoarthritis in the shoulder. List some typical interventions.
A
a. OA- a slowly progressive uniarthicular disorder of unknown cause, occurring late in life, principally affecting the hands and large weightbearing joints and characterized by pain, deformity and limitation of motion and pathologically by focal erosive lesions, cartilage destruction, subchondral sclerosis, cyst formation and large osteophytes at the margins of the joint. very common in adults, 60% of ppl age 60 and over. women> men (breakdown of cartilage, localized to 1 joint, not usually bilateral but can be)
inflammation 
pain at rest and w use 
potential for deformity 
stiffness
INTERVENTIONS:
calm pain and inflammation 1st always! 
bed positioning 
active rest
enegery conservation
joint protection
using sh. in pain free zone
@ home modalities 
AAROM- pendulums, scapular clock, scapular mobility, isometric in pain free range 

RA- systemic connective tissue disease whose onset and progression vary from mild joint symptoms with aching and stiffness to abrupt swelling, stiffness and progressive deformities periods of exacerbation and remissions (don’t get back to where they were before the exacerbations, usually bilateral, visually swollen).
edema at joints
pain w motion
can detect increased temperature at joints
muscle atrophy due to disuse
fatigues easily

INTERVENTION:
joint protection
energy conservation
medications - can take up to 6 months ( help adapt to therapy)
same interventions as OA but need to be MORE CAREFUL w strengthening
have to work on inflammation 1st and ADAPT & MODIFTY ACT. 1st
no repetitive motions
MODAILITES - heat, neutral gentle
adapting daily tasks!!!!
start off cold if pt. joints are hot - move to neutral warmth

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14
Q
  1. Review the general considerations for joint protection on page 459 in Cooper for Arthritis management.
A

a.

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15
Q
  1. Describe the pathology that occurs with Thoracic Outlet Syndrome. What are the clinical signs? What are the usual interventions?
A

a. symptoms are due to compression of blood vessels and nerves by structures in TOS region
COMPRESSION sites- scalenes, first rib, pec minor
contributing factors: laxity in sh., postural variations, respiratory patterns –musicians

CLINCAL SIGNS AND SYMPTOMS
largely neurologica cases- more females
intermittent BP and vascular sx of pain; parathesia, numbness (ring and little finger), weakness, discoloration, and swellings
muscle length imbalances with tightness in anterior and weakness in posterior
faulty postures
poor endurance os postural muscles
shallow respiratory pattern
pt. complains of diff sleeping, inability to hold weight objects on affect side-heaviness
inability to maintain overhead work position or desk work

INTERVENTIONS
increae mobility of tight structures- massage, cardio, stretch (anterior muscles)
increase strength and endurance of weak ms
nerve gliding (flossing)
respiration training
adapt act- driving, purse on sh., bra strap
no excessive breathing –running
modalities for spasm– heat irriated nerves, ultrasound estim
watch pt. at work
safe sleeping positions that avoid stress on nerves/ cx ( on back pillows under arms )

(massage trigger pt/spasms, adapt posture, cardio/ walking, avoid overhead act., stretch muscles and stegthen back muscles, adaptions to take stress off of sh)

TOS exercises
scapular retraction - door way or noodle stretch- sh. need to be hiked to decrease sx
stretch scalenes- chin tuck, straighten neck, side bend away and rotate forward
suboccipital stretch - supine- tuck chin in-gentle turn to side- back to center- isometic -other side
pec stretch
scapular clock- get scap muscles working
modified superman

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16
Q
  1. TOS and Brachial Plexopathy have many interventions in common. What are the differences between the diagnoses?
A

a. Brachial Plexopathy- ( anything that is an irritation to the BP- usually from trauma)
traction neuropathy
components of TOS
avoid stress on nerves-glide- try to move as much as possible
shorten structures that are tight around nerve and promote stretch as tolerated
chronic pain

MAY SEE-
multiple pain sites
negative dx testing
long time dx
vasomotor changes
multiple neural pathologies
sx indicating inflammation around the nerve root , bp, other sites– multiple cruch
may take 24-36 hrs to clam down once exacerbated
tend to have pain all the time with all function
latency of 5-6 hrs after exacerbating activity was performed

same TX as T.O.S- now eights, increase function ( as functional as possible).
active rest
modalities- watch for dependence on passive tx
start at neck
p. ed
go SLOW
long time until resistance ex.

17
Q
  1. Where are the compression sites for TOS?
A

a. Scalenes- BP or subclavian artery cx due to tight or hypertrophied ms
first rib- BP, subclavian artery or vein may be cx as they course under clavicle- heavy purse
pec minor- BP and axillary artery cx against ribs as they course under the pec if tight

(BP may stretch is arm is held in fully elevated position )

18
Q
  1. What are the names of the federal/AMA coding mechanisms used for reimbursement: ICD-9, CPT, DME?
A

Codes- Healthcare Common Procedure Coding Systems (HCPCS)
-Diagnosis- ICD-9 (international classification of Diseases, 0th revision, clinical modification) codes are used to classify illness, injuries, and patient encounters with health care practitioners for services.

-Treatment- CPT (current procedural terminology)

Equipment- L codes- DME

19
Q
  1. What is the difference between the payment sources we discussed in class (and are in your documentation book p. 13-16 related to hand therapy).
A

a

20
Q
  1. What is included in each section of a SOAP note in the evaluation versus the daily note?
A

look at handout pages 2-4 in documentation and reimbursement handout

21
Q
  1. What are the fundamental elements of documentation (doc book page 23-4)?
A

a

22
Q
  1. Be able to look at a goal and determine what is missing.
A

a

23
Q

Tendonitis

A

degeneration caused by nutritional deprivation (ischemia) and mechanical stress.

  • causes debris containing calcium breakdown of fibrils (collagen)
  • microtears of fibrils lead to debris and inflammation
  • posture: forward shoulder, forward head.
  • what muscles in shoulder? :supraspinatus, infraspinatus
  • pain is felt: biceps (long head) results in deltoid tendonitis
    symptoms: painful arch of motion seen with PROM and AROM. diffuse weakness, edema, spasms in scapular muscles.
24
Q

Calcific Tendonitis

A
  • build up of calcium increases and can be seen on X-ray
  • May cause increased pain and further impairment
  • Tends to come on quicker and not respond to treatment as well
25
Q

Bursitis

A

-inflammation of subdeltoid bursa
-overuse or injury to the joint at work or play can also increase a persons risk of bursitis. examples of high-risk activities include gardening, raking, carpentry, shoveling, painting, scrubbing, tennis, golf, skiing, throwing, and pitching. incorrect posture at work or home and poor stretching or condition before exercises can also lead to bursitis.
what do these activities have in common?
-repetitive, 90 degrees above movement, common activities.

26
Q

After Rotator Cuff Repair surgery, when do you start a HEP of pendulums (Codmans)? What begins at 4-6 weeks post op as directed by the MD? 6- 8 weeks?

A

Postop

  • After repair: sling/abd. wedge for 1-6 weeks depending on surgeon
  • Goal: to return patient to full ADL/IADL
  • Treatment:
    • Remove sling for therapy after one week of supervised PROM
    • HEP of cervical, scapular depression-retraction with shoulder at side and distal AROM
    • Massage to scar site once healed

Week 1-3 Pendulums, PROM in limited shoulder planes
4-6 weeks: AAROM with gravity eliminated for abd and ER
6 weeks: AROM
7-8 weeks: light isometrics s

27
Q

***Adhesive capsulitis: What are the usual interventions?

A

medical:
-corticosteroids
-manipulation under anesthesia
athroscopic release- anterior GH ligs, coracohumeral ligament

intervention goals:

  • guide ineffective and efficient functional use
  • prevent motion loss then restore functional motion.
  • don’t increase their pain
    exercise: cardio, zumba, walk,
  • sleep positions- watch their posture