Exam prep Flashcards
How do you evaluate pain? What should you ask the patient in relation to the pain?
PQRST!!!
What are the other risk factors presenting with headache? (8)
- Altered mental state (GCS below 15)
- Fever (Meningococcal infection?)
- Neurologic deficit
- Age above 50 years or below 10 years
- First headache of this kind
- Head trauma in 3 months.
- Continuously increasing intensity
- Skin abnormalities (e.g. petechia) (Meningitis)
What are the risk factors of chest pain? (10)
- Abnormal vital parameters (tachycardia, bradycardia, hypotonia, tachypnea)
- Shortness of breath – dyspnea
- Hypoxia
- Vegetative symptoms – sweating, nausea
- Symptoms of perfusion problem / shock
- Altered mental state
- Asymmetric chest movement
- Paradox pulse (Pericardial Tamponade)
- BP side difference (significant) (Aortic dissection)
- New cardiac murmur (ACS, endocarditis)
What are the Risk factors (Red flags) of abdominal pain? (8)
- Abnormal vital signs– shock signs
- Shortness of breath- dyspnea
- Fever
- Abrupt onset
- Bleeding (hematemesis, melena, hematochezia, hematuria, vaginal bleeding)
- Testicular pain (Testicular torsion)
- Old patient (>75 years)
- Previous abdominal surgery
What are the risk factors (red flags) of Low back pain? (12)
- Abnormal vital signs – shock signs
- Immunosuppressed state (chronic corticosteroid use, chemo, iv drug abuser, HIV)
- Fever
- History of cancer
- Focal neurologic deficit
- Injury / Trauma – high-risk mechanism of injury
- Injury / Trauma – med-risk mechanism of injury but age > 50 y
- Age > 70 years (new onset LBP)
- Pain ongoing > 6 weeks
- Coagulation disorder (acquired – VKA, DOAC ; or inherited)
- Pain at rest
- Pulsating abdominal mass
What are the risk factors of limb pain? (7)
- Abnormal vital signs – shock signs
- Cold, pulseless limb
- Fever
- Immunosuppressed condition
- Trauma – significant deformity
- Focal neurologic deficit
- Major tension, enlargement – compartment signs
What are the risk factors (red flags) of fever? (6)
- Abnormal vital signs – shock signs
- Dyspnea - hypoxia
- Immunocompromised state
- 2 or more SIRS criteria
- Skin abnormality – petechia
- Altered mental state – AMS (GCS below 15)
What are the risk factors (red flags) of AMS (Altered Mental State)? (8)
- Abnormal vital signs
- Low blood glucose (below 3 mmol/l)
- Headache
- Fever
- Trauma – high-risk mechanism of injury
- Focal neurologic deficit
- Acquired or inherited coagulation defect
- Unconsciousness
What are the risk factors (red flags) of focal neurologic deficit? (7)
- Abnormal vital signs – shock signs
- Onset of symptoms (0-4,5 h or 4,5-6h or 6-24h or over 24h)
- Altered mental state (below GCS 15)
- Acquired or inherited coagulation defect
- Fever
- Blood glucose below 3 mmol/l
- Undulating symptoms (on-off), or crescendo symptoms
What are the risk factors of dyspnea? (8)
- Abnormal vital signs – shock signs
- Hypoxia – cyanosis
- Altered mental status (GCS is under 15)
- Stridor
- Breathing work without air movement (frustrane breathing)
- Asymmetric chest wall movement
- Deviating trachea and or unilaterally missing breathing sounds
- Extreme high respiratory rate (over 40/min)
What are the risk factors (red flags) of Syncope (T-LOC)? (8)
- Abnormal vital signs – shock signs
- New onset arrhythmia/ irregular pulse or the major elevation of the frequency of an already diagnosed arrhythmia
- The loss of consciousness did not have a prodrome
- The loss of consciousness happened after the sensation of palpitation or chest pain
- The lot of consciousness happened during physical activity
- Dyspnoe - hypoxia
- Altered mental status (GCS is under 15)
- Fever
What are the risk factors (red flags) of seizures (6)?
- Ongoing seizure
- Abnormal vital signs – shock signs
- Ill detrimental status (GCS is under 15) (Altered mental status)
- Focal neurologic deficit
- Fever
- Headache
What are the 4 main vital signs?
- Body Temperature
- Blood Pressure
- Pulse
- Breathing
What are the shock signs?
- low blood pressure
- nausea or vomiting , sweating
- rapid shallow breathing
- cold, clammy skin
- rapid, weak pulse
- dizziness, fainting or weakness/fatigue
- Pale or ashen skin
- Bluish tinge to lips or fingernails (or gray in the case of dark complexions)
- Enlarged pupils
- Changes in mental status or behavior, such as anxiousness or agitation
What should be looked at in the “quick look” assessment?
- Posture
- Skin - color, wet or dry, temperature, odema, spots and dots.
- Smell
- Breathing
- Mental state
What are the parameters of assessing pain?
In every sort of pain the following parameters should be assessed (PQRST):
- P = Provoke: What provokes and exacerbates, and what alleviates the pain.
- Q = Quality : what is the quality of pain (e.g. stabbing, compressing, burning, colic)
- R = Radiation: in which direction does it radiate
- S = Severity: what is the severity – assess using objective scaling like PPI or VAS. Is is a central or peripheral pain.
- T = Timing: is it acute or chronic (acute = <1 month, or >1 month but changed intensity or quality). Is it continuous or pulsating or does it come-and-go? How fast did it start (pain appeared abruptly or presented gradually)?
When is intraosseous cannulation used?
Intraosseous cannulation is recommended when securing vascular access is crucial and the third attempt of a peripheral vein cannulation fails.
What are the advantages of IO technique?
The IO technique is fast and safe. The IO administration can provide a faster route to the central circulation than a distal peripheral IV line.
Where is the IO inserted?
The most common site recommended for (IO) insertion is the proximal tibia. Alternative sites are the distal tibia, distal femur, sternum, and humerus.
What do you need to avoid when inserting IO cannulation in children?
During IO cannulation of children the growth plate should always be avoided.
What are the contraindications of IO cannulation?
The contraindications are:
- fracture
- infection
- burn of the chosen limb.
- Osteoporosis also can be a relative contraindication.
After an unsuccessful attempt an alternative limb should be chosen.
Why is local anesthesia not needed for IO cannulation?
Idk? Local anaesthesia is not necessary. However Lidocaine is given after the cannulation has been made due to the aspiration pain before infusion is given.
What are the steps of installing a IO cannula?
Insert the IO needle through the skin and subcutaneous tissue. Upon reaching the bone start drilling the needle with constant pressure until the loss of resistance.
What is done after the IO cannula has been placed?
- You remove the inner trocar, attach a special right-angle connector to the needle and push a 5-10ml bolus of isotonic sodium chloride solution through the needle.
- Attach a syringe and aspirate. Obtaining marrow confirms placement. If you are unable to aspirate it does not necessary means that the needle is not in position. The obtained bone marrow can be sent to the lab for initial diagnostic laboratory studies.
- After that you should inject 20-30mg or maximum 1 mg/kg Lidocaine because administering large volumes can be painful.
- Secure the needle with gauze pads and tape.
- Observing the circumference of the calf is important.
- Every IV administrable solution can be injected intraosseously.
How long should a IO cannula be used for?
The IO route can be maintained up to 24 hours but should be immediately removed when it is not needed anymore.
What is important to remember about infusion solutions? What errors can you make if your not careful?
During fluid therapy we can meet a lot of types of infusion solutions therefore knowing there advantages and disadvantages is important. It has been proven that solutions with high chlorine concentrations have adverse effects on the survival of the critically ill patients therefore the solutions containing sodium chloride have lost their priority. The hypochloremic metabolic acidosis was a popular topic over the past decade. Its development is clearly associated with inappropriate or exclusive use of 0.9% sodium chloride solutions. For decades we know that the excess intake of sodium or chloride is harmful for the body. For at least 50 years the so-called balanced solutions are available. With the use of them the hypernatremia and hyperchloremia is avoidable. There are general requirements that every solution must meet. They should be isotonic, isosmotic and its pH should be around neutral. The concentration of positively and and negatively charged ions should be equal. The balanced solutions are the closest to meet these criteria. If we want a solution that has the same components as the plasma than we need 140 mmol of sodium and 103-110 mmol of chloride. This solution would not be isotonic and there would be imbalance between the concentration of the positively and negatively charged ions. Therefore we need to add more cations and if we want to reduce the chlorine concentration we need to add anions as well. This process is called “the balancing” of the solution. We have a lot of cations we can easily and safely add to the solution such as sodium, potassium, calcium, magnesium. Adding anions are much harder. In the plasma the natural anions are chloride, bicarbonate and albumin. They can not be mixed into all solutions, albumin is expensive and bicarbonate is not stable in the presence of other ions. Alexis Hartmann was the one who found a solution. He realised that there are non toxic, negatively charged compounds in the human body. These compounds also can be metabolised quickly to non harmful molecules. He found out that the some components of the citric acid cycle are ideal for “balancing”. These are: lactate, acetate, maleate, citrate. Except the citrate they are non toxic molecules and they are metabolised into bicarbonate which can be useful when there is acidosis or the solution’s pH is low. Lactate was the first to use as a balancing component. In a healthy adult its serum level is under 1-1.5 mmol/l. Its level increases if the perfusion and tissue oxygenation decreases - if the metabolism became anaerobic. In a critically ill patient the rate of its increase correlates with the severity of the condition and with the mortality.
vein cannulas**
The flow rate, thus the possible administered amount of fluid, is determined by the diameter and the length of the cannula. According to Hagen-Poiseuille’s law the flow is proportional to the pressure difference (ΔP) between the ends of the pipe and the fourth power of its radius (r4) but inversely proportional to the fluid viscosity (η) and the tube length (l) [F=(ΔP x π x r4) / (8 x η x l)]. That means that doubling the radius of the cannula results in a 16 times faster fluid flow. Also, the reducing of the cannula length by half will double the flow.
monitoring blood pressure **
When using a non invasive blood pressure measuring equipment the cuff size is determined by the arm circumference. Incorrect cuff size makes the measurement inaccurate or sometimes impossible. In certain cases, such as arrhythmias with changing pulse amplitude (e.g. atrial fibrillation) or the movement or tremor of the upper limb the automatic blood pressure monitor can be inaccurate or sometimes it can not measure the BP. In these cases, the auscultation method which depends on the examiner’s abilities is the only possible option. Normally, however, the automated oscillometric blood pressure measurement is more reliable and more accurate. Positioning the cuff is critical, to obtain a correct measurement the cuff’s sensor should be placed over the brachial artery. The place of the sensor is marked on the cuff. The measuring frequency is based on the patient’s complaints and his or her general status and it varies between one hour to 5 minutes or even less if it is necessary (in these cases usually invasive blood pressure monitoring is indicated). During and after every diagnostic or therapeutic intervention the BP should be measured in every 3-4 minutes until the patient can be considered stable.
ecg monitoring **
The ECG monitoring of a patient with critical or potentially critical condition is obligate and indispensable. For monitoring purposes 3 or 5 electrodes are the most common. If they are positioned correctly 3 electrodes are enough for ECG monitoring and to recognise arrhythmias. Mixing up the order of the electrodes does not affect the quality. Generally, the electrodes should not be placed over bones or the breast tissue what is difficult when the patient is cachexic or thin. When placed over hairy areas the contact is reduced therefore the signal would be impaired. If the electrodes are placed too far from each other e.g. to the extremities the signal strength weakens which can also affect the quality of the monitoring. One electrode should be placed near the right shoulder (red), one near to the left shoulder(yellow) and the third one over the bottom third of the chest wall (can be placed both right or left). It is recommended to choose the lead with the best signal strength on the monitor which is usually a standard bipolar lead. Monitoring with 5 electrodes is a safe way to detect ST changes. The electrodes should be correctly placed: one electrode on the right shoulder (red), one on the left shoulder (yellow), one on the right side of the trunk (green), one on the left side of the trunk (black) and one on the V5 or central position (white). The unipolar V5 lead is the most suitable for monitoring the ST segment. If the ST segment deviation on the previous 12 lead ECG was shown in other leads than V5 then the white electrode should be placed according to that. Keep the cables organised and away from the chest wall so they will not interfere with the possible interventions.
oxygen monitoring **
Judging the patient’s oxygenation in addition to our eyes (searching for cyanosis) we can use a pulse oximeter. It is a non-invasive device, it has no contraindications and it can be used for continuous monitoring. It measures what percentage of haemoglobin is binding oxygen (SpO2). The principle of its operation is the Beer-Lambert Law as it measures the absorption of red and infrared light. The pulse oximeter needs a pulsatile flow to calculate the saturation. Many things can interfere with the measurement. Some of them we can not control (e.g.: carboxyhaemoglobin, intravenous dyes, high serum bilirubin, severe anaemia, hemodilution, low perfusion), but some of them we can (strong light, movements of the device or the extremities, cold or compressed extremities, painted nails). The device is usually calibrated between 70-100% SpO2. The uncertainty of the measurements is usually about +/-2 %. The pulse oximeter can also be used for monitoring the circulation. The pulse pressure and the height of the pulse wave (with correct monitor settings) helps to detect peripheral ischemia and in some cases the changing in the peripheral circulation. The heart rate can be monitored by a pulse oximeter and it is also useful in the detection of pulse deficit but it can not be used to differentiate between arrhythmias. The target SpO2 is 95% or higher. It is important to keep in mind that the changes of the arterial saturation can only be seen on the monitor 30 to 90 seconds later.
Humidification?**
Oxygen therapy without humidification can be harmful for the airways as secretion congestion, mucus plugs, airway obstruction, atelectasis occur. This is especially true for >4L/min and high-flow oxygen therapy methods. In order to maintain mucociliary clearance function and reduce heat-loss caused by ventilation, inspired gas should be warmed to body temperature and humidified. Methods for producing water vapor: A) Active a. Vaporizers: directing air flow over (pass-over vaporizer) or through (pass- through vaporizer) heated water. b. Nebulizers: convert water into tiny water droplets either by ultrasound or high- velocity gas jet B) Passive: heat-moisture exchange (HME) attachment, HME filter (with bacterial filter) The widely used bubble-through (pass-through) humidification method is inappropriate since the cold gas can be humidified much less (the absolute humidity it can reach is low) then body temperature gas, so the humidity of the inspired gas will be insufficient. Also, only vapor particles smaller then 5 μm can reach the bronchioli, which are not created during bubble- through humidification. On the top of that, the water tank is a potential source of infections. Ultrasound nebulization is the best in terms of droplet size, safety and consistency of efficacy.
Fix Performance Systems**
These systems deliver 30-60 L/min oxygen flow which is more then the patient’s Peak Inspiratory Flow (PIF). Thus the patient always gets the FiO2 we set. Two major forms are: - Venturi mask: works on the principle of jet mixing. There are different (color-coded) Venturi masks for different FiO2 targets. Also, there are adjustable Venturi masks where you can set distinct FiO2 values. In general, 0.24-0.6 FiO2 can be reached. - CPAP-systems: Oxygen flow can be even higher, up to 120 L/min. High flow is delivered by a device where FiO2 can be set between 0.21 and 1.0. A well-shaped, well-sealing mask is put on the patient’s face. The mask has a PEEP-valve which maintains a constant 2 – 10 cmH2O positive pressure in the mask.
Variable Performance systems**
These systems work by increasing the functional residual capacity of the patient to 100%, which will increase FiO2 during inspiration. The major drawback of these systems is that in case of elevated minute ventilation or peak inspiratory flow, performance drops because atmospheric air is inhaled besides the ventilation gas, so FiO2 will fall. - Nasal cannula: FiO2 can be 0.3 if oxygen flow is 3-5 L/min and the patient is breathing normally. Higher oxygen flow will not increase FiO2 significantly, but will be very uncomfortable. - Normal mask: Reachable FiO2 is 0.3–0.5. Oxygen flow should be 6-10 L/min. The mask increases the dead space around the face, which is filled with oxygen during the expiratory pause. The drawback is that humidification is difficult and the higher oxygen flow dries the airways. The mask can cause emotional stress (feeling of choking). - Non-rebreathing mask: A reservoir balloon is attached to the mask where the patient exclusively breaths from. The reservoir is completely filled with oxygen during the expiratory pause. The oxygen flow must be set that the reservoir never empties out completely during inspiration but only 30-40%. With 12-15 L/min flow, maximum FiO2~0.8 can be reached (theoretically 1.0 but there is always room air leaking in the system around the imperfect sealing of the mask)
There are two main groups of oxygen delivery systems:**
A) Fix Performance Systems deliver the entire ventilation requirement by providing 40-50 L/min gas through the mask, which is usually enough to meet the total respiratory demand of the patient (gas flow always exceeds peak inspiratory flow). Thus, FiO2 will be constant and not affected by breathing. B) Variable Performance systems deliver only a proportion of the ventilation requirement. They provide slower gas flow, so the patient’s inspiratory flow can exceed the available gas flow, in which case environmental air is inspired to meet the respiratory demand. FiO2 is hence dependent of the breathing pattern of the patient (peak inspiratory flow, minute ventilation [respiratory rate, tidal volume]). The method of oxygen delivery should be decided by the breathing pattern of the patient and the target FiO2. Target FiO2 is decided by the current state of the patient. Oxygen therapy should be guided by continuous monitoring of SpO2 and intermittent blood gas measurements.
oxygen therapy **
There are three phases of breathing: 1) inspiration, 2) expiration, 3) expiratory pause. Expiratory pause has a crucial importance regarding the efficacy of different oxygen therapy methods.
Managing airways **
The first step of oxygen therapy is always airway management. Oxygen has to reach the lungs at first in order to supply the tissues via the blood flow. If the patient has compromised airway, even a properly selected O2-supply method will fail to help. The mental state/consciousness of the patient can give a clue about what airway to expect. Note that a conscious patient can also have compromised airway, so speech, breathing pattern and sounds must be evaluated. Clear auscultation and a speaking patient is a proof of patent airways. You should always expect a compromised airway in a patient in altered mental state. Assess the following: inspect breathing movements (effectivity), listen and auscultate breathing sounds (wheezing, stridor), note increased work of breathing.