Exam prep Flashcards

1
Q

Contamination vs. Pollution

A

Contamination is the naturally occurring presence of elevated concentrations of substances. Is natural and not anthropogenic! E.g. oil seeps along the coast of California.

Pollution is the leakage of anthropogenic substances in the marine environment causing deleterious effects.
(Dissolved nutrients, oil spills, particulates like dredging, coal dust and microplastic, industrial waste like heavy metals, PCBs, organochlorine, and Debris like garbage, ships waste, fishing gear)
Also, hot water from power plants.

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2
Q

What are sublethal effects

A

Deleterious effects caused by pollution that don’t kill the organism, but either deform them or cause them to become infertile etc.

Can be Acute or Chronic

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3
Q

The types of perturbations, release of pollutant

A

Pulse: Release is sudden, big and of short duration. Quite common, like if you tip a bunch of barrels of something into the water column.

Press: Much longer duration, a chronic type of pollution, pollutant keeps “dribbling out”

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4
Q

Types of responses of organisms to perturbations (pollutants) e.g. change in abundance

A

Pulse: Drop off quickly but bounce back, e.g. amphipods

Press: Much longer effect, chronic effect, population not as persistent as the one with the pulse response

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5
Q

What is bioaccumulation?

A

The cummulative accumulation of substances that cannot be excreted in organic tissues. Can cause sublethal effects and when concentration is high enough can be lethal.

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6
Q

Can you draw the response of organisms tolerance and vigour?

A

Graph has vigour on y-axis and concentration of contaminant on the x-axis.
First zone, is the zone of indifference. Vigour is high, accumulation may occur.
Second zone is the zone of stress. vigour drops to zero.
Third zone is the zone of intolerance. Species is absent. Vigour is zero.
(Phillips and Rainbow 1993)

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7
Q

How can we measure pollution?

A

We measure pollution at different levels of biological organisation:

  1. Individual or sub-organismal measures
    - Physiological (looking at anomalies in liver)
    - Biochemical
  2. Whole organism - demographic characteristics
    - Reproductive output
    - Body size
    - Growth
  3. Community and population level studies
    - abundance of populations
    - contribution to food web
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8
Q

Sensitivity to toxins varies..

A
  1. Among species
  2. within species (depending on physiology and nutritional state, also on developmental stage; juveniles are often more sensitive than adults. larval forms are still growing, so the risk of becoming deformed is greater)
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9
Q

Synergistic vs. Antagonistic effects

A

Synergistic effects: multiple toxins increase the effect (a major concern)

Antagonistic effects: multiple toxins decrease the effect

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10
Q

How do we determine if an impact has taken place?

A

Sampling design:
BACI = Before After Control Impact, one control and one impact (Green 1979)

BACIP = Paired and multiple times Before and After, one control, one impact (Oaten 1986)

Beyond-BACI = One impact area, mutliple control sites (Underwood 1991) GOOD OPTION

MBACI = Multiple Before After Control Impact (Keough and Mapstone 1995) IDEAL IMPACT STUDY

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11
Q

Acceptable impact studies and the statistical inferences you can make from them

A

Before and after at control and impact sites can give you temporal and spatial inferences

Sample after impact at control and impact sites can only give you spatial inferences

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12
Q

Not acceptable impact studies

A

Temporal replication with no controls. Can give you temporal inferences, but this is not good.

Spatial replication of impact with no controls. Inferences will be based on experience. Not Good.

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13
Q

How do we measure impacts on planktonic assemblages?

A
  1. Lab experiments - Traditional approach
    (e. g. LD50)
  2. Field studies (Impact studies, MBACI and all that)
  3. Modelling (Oceanography and plankton)
  4. Mesocosms
  5. Monitoring with organisms (Like oysters or jellyfish)
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14
Q

Measures of toxicity

A

Toxicity tests
LT50 - The time it takes for 50 % to be dead

LC50 - Lethal concentration where 50 % are dead

LD50 - Lethal dose where 50 % are dead

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15
Q

What are fish larvae vulnerable to?

A
Metals (Cd, Hg, Pb, Zn, Cu)
pH
Organochlorines (DDT, DDE)
Industrial waste
Chlorine
Temperature
Oils

Effects can occur at very low concentrations

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16
Q

Oceanography and pollutants!

A

Pollutants can concentrate at fronts

Abundance of larvae vs. current speed

17
Q

Bioindicators vs. biomonitors

A

Bioindicators: changes in biochemical, physiological or behavioural attributes of an organism providing a qualitative state of the environment

Biomonitor: Using an organism as a proxy for levels of a xenobiotic in the environment. e.g. oysters and jellyfish

18
Q

How can Climate Change impact plankton and why is it a concern?

A

Changes in the plankton can cause ecosystem wide cascades. Responses can be very quick and affect higher trophic levels.

  • Change size structure
  • Change timing of reproduction
  • Change production
  • Change zooplankton abundance
  • Loss of key species through decalicification (e.g. coccolithophores)
19
Q

The East Australian current is..

A

The fastest warming part of the world