Exam I HENT Flashcards
Evaluation of the parotid glands at
Sup/Post. to Mandible
Submandibular deep to mandible
Stenson’s Duct-Patortid
Wharton’s Duct-Submandibular
Superficial Temporal Artery evaluation
Immediately Ant. to ear
Head evaluation
Hair Distribution-sparce or thickness, infestation
Normocephalic, no lesions, erythema or echymosis
is the superior portion of TM; Is more flaccid and the location of most spontaneous perforations
The Pars Flaccida
Aspect of TM that is usually the most tense
The Pars Tense
where the TM meets the tip of the malleus
Umbo
Vibrations pass through the air and are transmitted through the TM to the ossicles to the cochlea.
Conductive Phase
The cochlea senses and codes the vibrations, and nerve impulses sent to brain through cochlear nerve
Sensorineural Phase
Chronic inflammatory lesion; starts out as painful, tender papule on helix or antihelix, ulcerates and crust
R/O Canrcinoma
Chondrodermatitis Helicis
A deposit of uric acid crystals associated with Gout. Hard nodule on heli/antehelix;
May change tto chalky white crystals through the skin
Tophi
Raised nodules w/ irregular borders; smooth with rolled borders or crusty with scales, telangectatic vessels
Basil Cell carcinoma /Squamous BCC/SCC
Temporal Membrane evaluation
- Color Countour
- Light Reflex
- Landmarks
- Valsalva
Air conduction which falls within range of human speech
Preferred-512-1024 Hz Human Range (300-3000 Hz)
Place tuning fork firmly on the top of the pt’s head or mid forehead.
Weber Test
Conductive Hearing loss is caused by
- Middle ear disease
- Ear compaction= better Bone conduction than A/C
Place the base of the fork on the mastoid bone behind the ear and level with the canal
Rinne Test
Inner ear problems such as loud noise exposure, inner ear infections, aging, congenital and familial disorders
Sensoryneuro Hearing Loss AC>BC
External and middle ear problems such as FOB in canal, otitis media, perforated TM, otosclerosis
Conductive Hearing Loss BC>AC
is a convergence of small fragile vessels located superficially on the anterior superior portion septum;
common source of nosebleeds
Kiesselbach Plexus
unlike the rest of the cavity, is lined with hair-bearing skin, not mucosa
Vestibule
opens onto the buccal mucosa near the upper second molar. Often marked by its own small papillae.
Stenson’s Duct
Starts with softening of the skin at the angles of the mouth, followed by fissuring
nutritional deficiency or overclosure of the mouth, as in people with no teeth or ill-fitting dentures
Angular Cheilitis
Results from excessive exposure to sunlight and affects primarily lower lip.Outdoor Fair-skinned men affected.
Lip loses its normal color and may become scaly, thickened, and slightly everted. R/O Carcinoma
Actinic Cheilitis
Normal sebaceous glands that appear as small yellowish spots in the buccal mucosa or the lips
Fordyce Spots
Early sign of measles (rubeola).Small white specks that resemble grains of salt on a red background.
Usually appear on buccal mucosa near first or second molars
Koplik’s Spots
Small red spots that result when blood escapes from capillaries into the tissues.
Often caused by accidental biting of the cheek, infection, decreased platelets, or trauma.
Petechiae
Yeast infection due to Candida.Thick, white plaques that adhere somewhat to the mucosa.
Causes: prolonged antibiotic or corticosteroid use and AIDS
Oral Canididiasis (Thush)
Thickened white patch on the oral mucosa that cannot be scraped off.
Causes: frequent chewing of tobacco.
Leads to cancer
Leukoplakia
CN evaluation check gag reflex with tongue blade.
CN IX and X