Exam I Flashcards

1
Q

How can we differentiate between a primary muscle disease and neuromuscular disease in our patient?

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2
Q

Patients with generalize muscle disease frequently present with _____.

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3
Q

In addition to weakness, what are some other common clinical signs associated with generalized muscle disease?

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4
Q

A generalized muscle disease can be acquired or inherited. What are some general categories of an acquired muscle disease?

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5
Q

A generalized muscle disease can be acquired or inherited. What are some general categories of an inherited muscle disease?

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6
Q

List some examples of acquired inflammatory myopathies.

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7
Q

What is masticatory muscle myositis (MMM)?

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8
Q

Name the muscles of mastication in the canine.

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9
Q

What is the clinical presentation of a patient with early masticatory muscle myositis?

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10
Q

What is the clinical presentation of a patient with late masticatory muscle myositis?

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11
Q

Your patient presents with clinical signs consistent with masticatory muscle myositis and is painful. What are your differentials?

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12
Q

Your patient presents with clinical signs consistent with masticatory muscle myositis and is non-painful. What are your differentials?

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13
Q

How do we diagnose masticatory muscle myositis?

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14
Q

What are the treatment option for masticatory muscle myositis?

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15
Q

What is the prognosis for masticatory muscle myositis?

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16
Q

What is canine idiopathic polymyositis?

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17
Q

What is the common clinical presentation for a patient with canine idiopathic polymyositis?

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18
Q

How do we diagnose canine idiopathic polymyositis?

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19
Q

What are the treatment options for canine idiopathic polymyositis?

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20
Q

What is the prognosis for canine idiopathic polymyositis?

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21
Q

What are your top differentials for a feline with cervical ventroflexion?

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22
Q

What is feline idiopathic polymyositis?

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23
Q

What is the common clinical presentation of a cat with feline idiopathic polymyositis?

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24
Q

How do we diagnose feline idiopathic polymyositis?

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25
Q

What are the treatment options for feline idiopathic polymyositis?

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26
Q

What is the prognosis for a patient with feline idiopathic polymyositis?

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27
Q

What is the clinical presentation for hypokalemic myopathy?

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28
Q

How do we diagnose hypokalemic myopathy?

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29
Q

What is the treatment for hypokalemic myopathy?

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30
Q

What is dermatomyositis?

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31
Q

What kind of lesions are associated with dermatomyositis?

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32
Q

How do we diagnose dermatomyositis?

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33
Q

What is the treatment for dermatomyositis?

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34
Q

What is the prognosis for a patient with dermatomyositis?

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35
Q

What is fibrotic myopathy?

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36
Q

Describe the common clinical presentation for a patient with fibrotic myopathy.

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37
Q

Identify the anatomy of the medial thigh.

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38
Q

How do we diagnose fibrotic myopathy?

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39
Q

What are the treatment options for a patient with fibrotic myopathy?

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40
Q

What is the prognosis for a patient with fibrotic myopathy?

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41
Q

List the different kinds of endocrine/metabolic acquired myopathies.

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42
Q

What clinical signs might be seen in a dog with postpartum hypocalcemia?

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43
Q

Give a general overview of what tetanus is, the clinical signs and treatment for it.

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44
Q

What is risus sardonicus?

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45
Q

List the different inherited myopathies.

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46
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to inheritance?

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47
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to gender?

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48
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to age of onset?

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49
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to clinical signs?

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50
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to tendon reflexes?

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51
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to creatinine kinase levels?

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52
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to therapy?

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53
Q

How does Labrador retriever myopathy and muscular dystrophy differ when it comes to prognosis?

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54
Q

Provide a brief overview of muscular dystrophy in cats.

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55
Q

What is extraocular myositis?

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56
Q

Animals with a joint disease commonly present with a history of ____ or ____.

A

Lameness ; gait abnormality

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57
Q

Lameness may involve one joint (typically _____ or _____) or multiple joints (_____, _____).

A

Traumatic, developmental ; degenerative or inflammatory

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58
Q

Disorders affecting the joint can either be ____ or ____.

A

Inflammatory or non-inflammatory

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59
Q

Non-inflammatory joint diseases include ____.

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60
Q

Inflammatory joint diseases include _____.

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61
Q

What are some characteristic signs of inflammatory joint diseases?

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62
Q

What are some characteristic signs of non-inflammatory/degenerative joint diseases?

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63
Q

What should you be assesses during your physical exam of a patient with a suspect joint disease?

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64
Q

During your physical exam, you detect palpable pain of the bones. What are your differentials?

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65
Q

During your physical exam, you detect palpable pain in the muscles. What are your differentials?

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66
Q

During your physical exam, you detect palpable pain in the neck. What are your differentials?

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67
Q

During your physical exam, you detect palpable pain in the spine. What are your differentials?

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68
Q

During your physical exam you notice a decreased range of motion, crepitation and joint instability which suggests ______.

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69
Q

Where are some locations in the canine that you can assess and easily detect effusion?

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70
Q

List the different diagnostic imaging modalities we can use to assess for joint pathology.

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71
Q

Describe the use of radiography to assess joint pathology.

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72
Q

Identify the anatomy in this radiography (normal).

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73
Q

Identify the pathology in these radiographs.

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74
Q

Identify the pathology in these radiographs.

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75
Q

Identify the pathology in this CT.

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76
Q

Describe the use of arthrocentesis and synovial fluid analysis as a diagnostic modalities for joint pathology.

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77
Q

What does normal synovial fluid look like? What is its purpose?

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78
Q

Identify the pathology based on the joint fluid appereance.

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79
Q

What is being exemplified in this image relative to the synovial fluid?

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80
Q

What are some cytological characteristics of normal synovial fluid?

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81
Q

Identify the approximate WBC count and % PMN in normal synovial fluid cytology.

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82
Q

Identify the approximate WBC count and % PMN in degenerative synovial fluid cytology.

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83
Q

Identify the approximate WBC count and % PMN in a traumatic synovial fluid cytology.

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84
Q

Identify the approximate WBC count and % PMN in a septic synovial fluid cytology.

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85
Q

Identify the approximate WBC count and % PMN in a non-erosive immune synovial fluid cytology.

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86
Q

Identify the approximate WBC count and % PMN in an erosive arthritis synovial fluid cytology.

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87
Q

Analyze the synovial fluid cytology.

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88
Q

Analyze the synovial fluid cytology.

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89
Q

Analyze the synovial fluid cytology.

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90
Q

Analyze the synovial fluid cytology.

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91
Q

Identify the cells in this cytology slide.

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92
Q

What are some other tests we can use to rule out systemic disease when we have a patient with joint pathology?

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93
Q

Briefly differentiate between non-inflammatory and inflammatory joint disease.

A
94
Q

List some common infectionious joint disease differentials in the canine.

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95
Q

List some common infectious joint disease differentials in the canine.

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96
Q

List some common infectious joint disease differentials in the feline.

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97
Q

What is DJD?

A
98
Q

____, ____, and ____ are the most commonly identified undervaluing causes of DJD.

A
99
Q

What clinical signs are associated with DJD?

A
100
Q

How can we prevent/treat patient with DJD?

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101
Q

How does weight management act as prevention and treatment in patients with DJD?

A
102
Q

Draw out the COX-1 pathway.

A
103
Q

Draw out the COX-2 pathway.

A
104
Q

When using NSAIDs for the treatment of OA, we want to spare ____ and target ____.

A

COX-1 ; COX-2

105
Q

There should be a minimum ____ washout period between NSAIDs.

A
106
Q

List the different NSAIDs and their ability to target COX-2 over COX-1.

A
107
Q

What is the mechanism of tepoxalin?

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108
Q

What is the mechanism of grapiprant?

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109
Q

All NSAIDS have to potential to cause ____.

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110
Q

COX-1 sparing NSAIDs are associated with ___ less GIT ulceration in humans and animal studies have yielded similar results.

A
111
Q

Use should use NSAIDs judiciously in patients with ____.

A
112
Q

Describe the use of managing OA in cats with NSAIDs.

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113
Q

In general, what are the advantages of omega 6 fatty acids in the treatment of OA?

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114
Q

Draw out the omega-6 pathway.

A
115
Q

Draw out the omega-3 pathway.

A
116
Q

List the different oral formulations that can be used as disease modifying OA agents.

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117
Q

List the different injectable formulations that can be used as disease modifying OA agents.

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118
Q

List the different opioids that can be used in the treatment of OA.

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119
Q

List and describe the different novel adjunctives that can be used in the treatment of OA.

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120
Q

Describe joint injections as a therapeutic agent for the OA.

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121
Q

Describe synovetin as a therapeutic agent for the OA.

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122
Q

Describe anti-NGF monoclonal antibodies as a therapeutic agent for the OA.

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123
Q

What are the different routes of infection for septic arthritis?

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124
Q

What is the most common agent(s) for septic arthritis in the canine?

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125
Q

What is the most common agent(s) for septic arthritis in the feline?

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126
Q

Describe the prevalence of septic arthritis in dogs vs. cats and males vs. females.

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127
Q

Describe the prevalence of septic arthritis in dogs vs. cats and males vs. females.

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128
Q

How do we diagnose septic arthritis?

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129
Q

What is the treatment for septic arthritis?

A
130
Q

What etiologic agent is the causative agent of tick borne poly arthritis?

A
131
Q

What are the clinical signs of tick borne polyarthritis?

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132
Q

How do we diagnose tick borne polyarthritis or Lyme?

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133
Q

What is the treatment for tick borne polyarthritis (Lyme)?

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134
Q

What is the treatment for tick borne polyarthritis (Lyme)?

A
135
Q

Describe the use of an ELISA test to confirm tuck borne polyarthritis or Lyme.

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136
Q

What are the different treatment options for tick borne polyarthritis or Lyme?

A
137
Q

What are the different etiological agents responsible for rickettsial polyarthropathy?

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138
Q

What are the most significant clinical signs associated with rickettsial polyarthropathy?

A
139
Q

How do we diagnose rickettsial polyarthropathy?

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140
Q

How do we treat rickettsial polyarthropathy?

A
141
Q

The most common non-inflammatory joint disease of dogs and cats is ____.

A

OA

142
Q

What is the most common inflammatory joint disease of cats?

A
143
Q

What is the most common inflammatory joint disease of dogs?

A
144
Q

Describe feline polyarthropathy.

A
145
Q

What is reactive polyarthropathy? List the causes.

A
146
Q

Idiopathic immune-mediate no erosive polyarthritis is diagnosed only by ruling out other causes of polyarthritis and its the most common form of poly arthritis in the ____.

A

Dog

147
Q

What are the clinical signs associated with idiopathic immune-mediate nonerosive polyarthritis?

A
148
Q

How do we diagnose IMPA?

A
149
Q

How do we treat IMPA?

A
150
Q

____ is the most commonly poly arthritis in the dog.

A
151
Q

List the most common developmental orthopedic diseases in the dog.

A
152
Q

What is canine hip dysplasia?

A
153
Q

What is the pathophysiology of CHD?

A
154
Q

How can we tell that this radiograph is normal?

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155
Q

How can we tell that this radiograph is abnormal?

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156
Q

How can we diagnose early hip dysplasia? Which is the gold standard?

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157
Q
A
158
Q

Describe the medical management of CHD.

A
159
Q

How can we prevent CHD?

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160
Q

List the different diseases that encompass elbow dysplasia.

A
161
Q

What is the etiology and clinical signs associated with elbow dysplasia?

A
162
Q

Describe the pathophysiology of OC/OCD.

A
163
Q

Describe the pathophysiology of MCD (FCP).

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164
Q

Describe the pathophysiology of UAP.

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165
Q

Identify the anatomy.

A
166
Q

Describe the medical management of elbow dysplasia.

A
167
Q

How can we prevent elbow dysplasia?

A
168
Q

What is medial patellar luxation (MPL)?

A
169
Q

Describe the pathophysiology of MPL.

A
170
Q

List some of the common skeletal abnormalities associated with MPL.

A
171
Q

How do we surgically correctly MPLs?

A
172
Q

Describe the medical management of MPL.

A
173
Q

How can we prevent MPLs?

A
174
Q

What is osteochondrosis dessicans (OCD)?

A
175
Q

Describe shoulder OCD.

A
176
Q

What is panosteitis? What are the common predispositions?

A
177
Q

Describe the pathophysiology of panosteitis. What lesions are associated with this disease?

A
178
Q

Interpret the radiograph.

A
179
Q

Describe the medical management of panosteitis.

A