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OPP > Exam I > Flashcards

Exam I Flashcards

1
Q

4 basic principles of osteopathic medicine

A

Principle 1: human being is a unit

Principle 2: structure and function are reciprocally interrelated

Principle 3: the body can self-regulate itself

Principle 4: treatment is based on the above

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2
Q

osteopathic evaluation

A
  • history, PE, observation, palpation, motion testing, special tests, radiology, labs
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3
Q

definition of a primary key lesion

A
  • the somatic dysfunction(s) that maintains a total pattern of dysfunction, including other secondary dysfunctions
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4
Q

secondary key lesion

A
  • somatic dysfunction arising either from mechanical or neurophysiologic response subsequent to or as a consequence of other etiologies
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5
Q

SC levels of greater splanchnic

A
  • T5-9
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6
Q

SC levels of lesser splanchnic

A
  • T10-11
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7
Q

SC levels of least splanchnic

A
  • T12
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8
Q

chapman point at right 5th intercostal space

A
  • liver
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9
Q

chapman point at right 6th intercostal space

A
  • liver and gallbladder
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10
Q

chapman point at left 5th intercostal space

A
  • stomach acid
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11
Q

chapman point at left 6th intercostal space

A
  • stomach peristalsis
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12
Q

purpose of rib raising

A
  • normalize sympathetic tone
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13
Q

functions of the lymphatic system

A
  • Purification/cleansing of tissues

Maintaining fluid balance

Defense/Immunology

Nutrition

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14
Q

definition of lymphangion

A
  • the functional unit with smooth muscle, valves, and intrinsic peristalsis under autonomic (sympathetic) control; can generate enough pressure to account for intravascular lymph movement
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15
Q

autonomic levels to heart/lungs

A
  • Sympathetic (T1-T5)

- Parasympathetic (Vagus)

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16
Q

definition of bioenergetics

A
  • study of how endogenous and exogenous energy sources/forms influence and control living systems and their environment
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17
Q

definition of bioenergy

A
  • energy produced endogenously by living systems
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18
Q

definition of biofield

A
  • complex, extremely weak electromagnetic field of the organism hypothesized to involve electromagnetic bioinformation for regulating homeodynamics
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19
Q

definition of biofield therapy

A
  • any therapeutic modality that interacts and changes the biofield and its manifestations
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20
Q

definition of bioelectromagnetics

A
  • study of the interaction between electromagnetic fields and biological living systems.
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21
Q

definition of bioelectromagnetism

A
  • inherent ability of living cells, tissues, and organisms to produce and emit electrical and magnetic fields, and the response of cells to electromagnetic fields
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22
Q

founder of Dynamical Medicine (fractals)

A

Carlisle Holland, DO

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23
Q

founder of CCP

A
  • Gordon Zink
24
Q

80% well people show this pattern:

A

OA – Left
C-T – Right
T-L – Left
L-S –Right

25
Q

CCP upper cervicle

A

OA/AA RlSr

26
Q

CCP thoracic inlet

A

RrSr

27
Q

CCP thoracolumbar junction

A

– RlSl

28
Q

CCP lower lumbar

A

RrSl

29
Q

CCP piriformis spasm

A

-Right

30
Q

CCP psoas spasm

A
  • Left
31
Q

CCP iliac crest

A

higher on the left

32
Q

Greenmans dirty half dozen

A 
Muscle imbalance (>95%)
Type 2 SD (FRS/ERS 2:1) L-Spine
Pelvic SD Pubes (75% failed backs)
Short leg syndrome (65%)
Innominate Shear (25%)
Extended Sacrums
33
Q

postures with anterior pelvis rotation

A
  • kypholordosis and military
34
Q

postures with posterior pelvis rotation

A
  • swayback and flatback
35
Q

Tonic (Postural) muscles

A

Facilitated
Hypertonic
Shortened

36
Q

Phasic (Fast) muscles

A

Inhibited
Hypotonic
Weak

37
Q

upper cross syndrome

A 
Upper trap, l. scap, scalenes tight
Lower trap/serratus weak
Pecs short/tight
Rhomboids weak
Suboccipital/neck extensors/SCM tight
Deep neck flexors (longus colli, cervicis) weak
38
Q

lower cross syndrome

A

Weak glute max/tight hip flexors
Weak abs
Short e. spinae
Weak glute med, min/short TFL/QL
Anterior pelvic tilt/increased L lordosis
Hypermobility lower T/L spine

39
Q

muscle firing of hip extension

A 
Hamstrings
Gluteus maximus
Contra QL
Ipsi QL
Contra E. spinae
Ipsi E. spinae
40
Q

muscle firing of hip abduction

A

TFL
Gluteus medius
QL
E. spinae

41
Q

founder of FPR

A
  • Stanley Schiowitz
42
Q

how to do FPR

A
  • neutral, compression, ease
43
Q

physiologic theory of FPR

A

-A sudden decrease in load on muscle spindles
Cause the Ia fibers to stop discharging
Which stops the excitatory signal being sent to motor neurons controlling the extrafusal muscle fiber (relaxes)
This resets muscle spindle reflex

44
Q

founder of Still technique

A
  • Richard Buskirk
45
Q

how do to Still

A
  • ease of motion, compression, through restriction
46
Q

SC levels of superior chain ganglia

A
  • C2
47
Q

SC levels of middle chain ganglia

A
  • C4/C5
48
Q

Sc levels of inferior chain ganglia

A
  • C6/C7
49
Q

SC levels of adrenals

A
  • T8-10
50
Q

SC levels of kidneys

A
  • T11-L1
51
Q

right sympathetics to the heart innervate what

A

right heart and SA node

Hypersympathetic activity of these fibers predisposes to supraventricular tachyarrhythmias

52
Q

left sympathetics to the heart innervate what

A

left heart and AV node

Hypersympathetic activity of these fibers predisposes to malignant ventricular arrhythmias.

53
Q

hypersympathetic activity to the heart results in

A

Increased myocardial oxygen demand
Increased heart rate (chronotropy)
Increased contractility (ionotropy)

Increased cardiac irritability

Coronary artery vasospasm

54
Q

right vagus innervates

A
  • SA node

Hyperactivity of these fibers predisposes to sinus
bradyarrhythmias.

55
Q

left vagus innervates

A
  • AV node

Hyperactivity of these fibers predisposes to AV block

56
Q

increased vagal tone results in

A

Slowing of the sinus nodal rate

Predisposition to SA nodal and AV block

57
Q

SC levels associated with SD

A
  • C6T2T6

OPP (1 decks)

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