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NUR 211 > Exam 3 > Flashcards

Exam 3 Flashcards

1
Q

With a SPRAIN in skeletal trauma the __[1]__ structure is affected, and the body parts __[2]__ & __[3]__ are most affected. Usually this is done through __[4]__ & __[5]__, After which you should do what intervention??

A

1: Ligment
2: Ankles
3: Knees
4: Twisting
5: Hyperextension
6: R.I.C.E.

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2
Q

With a STRAIN in skeletal trauma the __[1]__ & __[2]__ structures are affected, and the body parts __[3]__ & __[4]__ are most affected. Usually this is done through __[5]__, After which you should do what intervention??

A

1: Muscles
2: Tendons
3: Lower Back
4: Calves
5: Lifting
6: R.I.C.E.

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3
Q

What is the difference btwn a Subluxation and a Dislocation?

A

A Subluxation is a partial Dislocation

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4
Q

What are the 8 Clinical Manifestations of Skeletal Trauma?

A
  • *Think ABC’s 1st!! ALWAYS assess Neuro!!!
  • edema
  • Pain
  • Contusion/ecchymosis
  • Decreased movement
  • Crepitus

Severe:

  • Shortening or rotation of extremity
  • Inability to bear weight
  • Muscle spasms
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5
Q

What is R.I.C.E.???

A

Rest
Ice
Compression
Elevation

(Immobilize but DO NOT reset joint)
(Remove Restrictive Items)

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6
Q

Fractures described by their “extent” would be described in what 4 ways?

A

Complete
Incomplete
Non-displaced
Displaced

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7
Q

Fractures are also described by their effect on surrounding tissues in what 2 ways?

A

-Closed (simple)
-Open (Compound)
o Skin Penetration
o Antibiotics will need to be used
o immunizations are up to date esp TDAP?

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8
Q

•__[1]__ reduction
o Non surgical
o Traction can be a closed reduction method

•__[2]__ reduction
o Surgical intervention
o ORIF
o immobilization May be used

A

1: Closed
2: Open

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9
Q

What is the Application of a pulling force to the body to provide reduction, alignment, and rest called?

A

Traction

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10
Q

What are the 6 main complications from fractures??

A
  • Infection (to be discussed later)
  • Compartment syndrome
  • Venous thromboembolis (VTE)
  • Fat Embolism Syndrome (FES)
  • Shock
  • Chronic complications: ischemic necrosis (avascular necrosis [AVN]), delayed bone healing or non union of bone
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11
Q

What is Normal Compartment Pressure?

A

0-10mm HG

>30= Bad JUJU

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12
Q

What are the 6 P’s to assess for Acute Compartment Syndrome (ACS)???

A 
o  Paraesthesia – may be first sign
o  Pain
o  Pressure  
o  Pallor
o  Paralysis (Late Sign)
o  Pulselessness (Late Sign/Bad Juju)
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13
Q

When do you NOT use R.I.C.E. when dealing witha skeletal trauma?

A

• ACS: Do NOT elevate extremity in compartment syndrome & Do NOT apply ice

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14
Q

What must you be concerned about when muscle breakdown is occurring?

A

THE KIDNEYS!!

-It causes release of potassium and myoglobins
Which are large particles; They can clog the kidneys and cause renal failure.

-Released K+ can cause arrhythmia’s

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15
Q

What are the 4 main cinical manifestations of a Fat Embolism?

A
  • Acute respiratory distress syndrome
  • Changes in LOC
  • Petechiae; on neck/upper arms/chest/abdomen
  • Sense of impending doom
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16
Q

What are you most concerned about with chest fractures?

A
  • Penetration of lungs, Heart, Or arteries.

- High risk for atelectasis/pneumonia (Shallow Breathing)

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17
Q

What are the 5 main Clinical Manifestations of a Hip fracture?

A
  • External rotation of leg
  • Shortening of leg
  • Severe pain
  • Tenderness/pain in leg or groin area
  • disruption of blood flow -> avascular necrosis of femoral head
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18
Q

What is a Syme amputation?

A

Foot; Just below the ankle

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19
Q

With amputations… How do you treat residual limb pain different from Phantom Limb pain?

A

Residual: Opioids
Phantom: Beta blockers, Antiepileptics, antispasmotics, Nerontin, Lyrica

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20
Q

interventions post amputation?

A

ROM
Trapeze
Firm Mattress
Prone Postion Q3-4hrs/15min

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21
Q

Stump care Post Op?

A

Wash w/soap & Water

Stump Shrinker can be used to help with edema

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22
Q

_________ is a Bone infection caused by bacteria or other germs.

A

Osteomyelitis

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23
Q

What are the 3 steps in the patho of osteomyelitis?

A
  • Initial Infection
  • Site expands; Blocking Blood supply
  • Bone Dies/separates/becomes sequestrum
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24
Q

Clinical Manifestations of Osteomeylitis?

A

Systemic S&S
-Fever, Chills, Restlessness, nausea, malaise
Local S&S
-Pain, swelling, tenderness, and warmth

** Treatment is Longterm Antibiotics(centeral line needed)/surgical debrisment/antibiotic beads/

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25
Q

This autoimmune disease leads to cephalic demyelination Of the nerve fibers of the brain and spinal cord. Occurs in exacerbations and remissions. And ultimately results in permanent loss of function.

A

Multiple sclerosis

-Usually affects women 20-50yo

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26
Q

Why is MS associated with Exacerbations and remissions?

A

Because in early stages the Myelin she is regenerated.

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27
Q

What replaces the Myelin sheath after it can no longer regenerate?

A

a Glial Scar

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28
Q

What drugs are used in acute Exacerbations of MS?

A
  • Adrenocorticotropic hormone (ACTH)

- Methylprednisone/prednisone

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29
Q

PD affects which brain area & structure?

Which neurotransmitter is involved?

A

basal ganglia
substantia nigra
Dopamine (controls movement)

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30
Q

Pro-banthine/Ditropan are what kind of drugs? And used to treat what in MS?

A
  • Anticholinergics
    • Helps Prevent Bladder Spasm
    • For MS Pt’s having issues with frequency
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31
Q

Valium/Lioresal/Dantrium/Zanaflex are what kind of drugs used in the Treatment of MS?

A

-Muscle Relaxants

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32
Q

PD affects which brain area & structure?

A

basal ganglia

substantia nigra

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33
Q

Valium/Lioresal/Dantrium/Zanaflex are what kind of drugs? And used to treat what in MS?

A
  • Muscle Relaxants

- Used to help treat the Rigid/Shakiness of MS

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34
Q

Acetylcholinesterase Inhibitor (Aricept) are what kind of drugs? And used to treat what in MS?

A
  • Muscle Stimulator

- Helps with initiating Movements

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35
Q

(Ampyra) is a Nerve Conduction enhancer are used to treat what in MS?

A

Help improve walking speed.

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36
Q

Phosphate Receptor Modulators like Fingolimod are used to treat what in MS?

A

-Help Prevent Disease progression by preventing T-cells (What attacks the Myelin) from getting to the CNS

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37
Q

Trauma, immunizations, birth, stress, and climate change are all Involved with MS in what way?

A

They are Triggers for Exacerbations

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38
Q

What is Fasiculation?

A

Muscle Twitching

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39
Q

This is an autoimmune disease characterized by symmetrical Ascending polyneuropathic paralysis caused by peripheral demyelination of the cranial Peripheral nerves. It IS Recoverable.
(1.5X more likely in M’s)

A
  • Guillian Barre syndrome
    • Often Triggered by a Virus, Truama, HIV, or
    • IN 30%of cases Campylobacter Jejuni
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40
Q

How is PD Dx ?

What is the ultimate confirmation of PD?

A

2 out of 3 s/s of the Triad
Clinical presentation & Hx
+ve response to antiparkinsonian drugs

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41
Q

This disease results in less motor neurons in the brain stem and spinal cord which cuts communication between the brain and muscles And results in death in 2 to 6 years after diagnosis.
Usually affects people 40-70 y/o and occurs in more M than F.

A

ALS or Lou Gehrig’s disease

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42
Q

Weakness in the upper extremities, dysarthria, dysphagia, muscle twitching/fasiculation, & nasal sounding speech are S&S of what disease.
(Death is usually the result of respiratory infection)

A

ALS or Lou Gehrig’s disease

  • Pt Does remain cognitively intact
  • the Drug Rilotek, is used to Decrease Gluconate in brain
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43
Q

A patient has trouble starting to walk and trouble stopping. Is he most likely to have a Dx of MS, PD, or MG?

A

PD - trouble initiating actions and executing them due to lack of dopamine

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44
Q

List risk factors for Parkinsons

A

exposure to chemicals - especially in rural areas.
Well H2O, pesticides, herbicides, industrial chemicals
Dental fillings: amalgam - lead
Females >40 years old with decreased estrogen
genetic factors

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45
Q 
What is the primary expected outcome for a pt. with PD?
progressive difficulty w/mobility
severe dementia
malnutrition
difficulty w/effective communication
A 
ALL ARE CORRECT
****progressive difficulty w/mobility***** best answer
severe dementia - late stage
malnutrition - late stage
difficulty w/effective communication
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46
Q

What is the primary goal for a pt. w/ PD

A

stay independent with ADLs for as long as possible

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47
Q

PD has 5 stages. Pt. do not necessarily progress through the stages in order. The stages exist to organise Tx.
The goal is to keep pt. in stages A____ - B______

A

The goal is to keep pt. in stages __1_- _3___ bc pt. can complete ADLs
Stages 4 & 5 SNIFs or ATC care

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48
Q 
levodopa
levodopa/carbidopa (Sinimet, Parcopa)
Tx which disease?
Class?
Action?
Tx which Sx's?
Side effects
A

Tx PD
Class: dopamine precursors
Action: cross BBB converted to dopamine in basal ganglia
Tx bradykinesia, tremor, rigidity
SE: ortho hypotension, drowsiness, hallucinations,

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49
Q

Problem with levodopa & protein?

How avoid?

A

lots of dietary protein interferes with action of levodopa.

Give bulk of protein in evening so can digest overnight

50
Q

What would Prednisone a Corticosteroid be used for in dealing with Myethenia Gravis?

A

Helps Suppress the immune response that’s responsible for the disease.

51
Q 
Entacapone (Comtan)
tolcapone (Tasmar)
Tx which disease?
Class?
Action?
A

PD
Class: Catechol O-Methyl Transferase Inhibitors (COMTs)
Action: Block COMT - slow peripheral breakdown of levodopa, protects dopamine so it can reach the CNS

52
Q

—These are all S&S of What Disease???—
Symmetrical weakness of the lower extremities
Paresthesia followed by paralysis
Hypotonia/areflexia (↓Muscle Tone/↓Reflexes)
Orthostatic hypotension
HTN/SIADH
Bradycardia/heart block/asystole
Facial flushing/diaphoresis

A

Guillian Barre Syndrome

53
Q 
Selegiline (Deprenyl, Eldepryl)
Rasagiline  (Azilect)
Tx which disease?
Class?
Action?
AVOID WHAT?
A

PD
Class:Monoamine Oxidase Inhibitors
Actions: Block breakdown of dopamine
AVOID: tyramine - chocolate, wine, cheese (Basically just go ahead and DIE!!!)

54
Q 
benztropine (Cogentin)
procyclidine (Kemadrin)
Trihexyphenidyl (Artane)
Tx which disease?
Class?
Action?
Tx Sx's?
SE?
A

PD
Class: anticholinergics
Action: block cholinergic receptors - balance cholinergic and dopaminergic activity
Tx: Tremor
SE: acute confusion, urinary retension, constipation, dry mouth, blurred vision
(dry things up & slow things down)

55
Q

What is the Patho of Parkinson’s?

Environmental Factors like Pesticides/herbicides/chemicals/metals(Fillings) can ↑possible causes

A

Degeneration of the Dopamine producing neurons, which disrupts the dopamine/Acetylcholine balance. Dopamine is an essential neurotransmitter that assist with motor functions like postural, support, involuntary motion

56
Q 
Amantadine (symmetrel)
Tx which disease?
Class?
Action?
Tx Sx's?
SE?
A

PD
Class: Dopamine agonist (anti viral also)
Action: Blocks reuptake of dopamine into presynaptic neurons
Tx: akinesia, tremors
SE: mood changes, confusion, depression, hallucinations, LE edema, nausea, epigastric distress, urinary retention, headache, visual impairment.

57
Q

What do you assess as a nurse on a PT with Guillian Barre Syndrome?

A 
Respiratory function
ABG's
Swallow reflexes
BP
Heart rate/rhythm (During Acute phase)
**Goal is to support till Pt can Recover**
58
Q 
Selegiline (Deprenyl, Eldepryl)
Rasagiline  (Azilect)
Tx which disease?
Class?
Action?
AVOID WHAT?
A

PD
Class:Monoamine Oxidase Inhibitors
Actions: Block breakdown of dopamine
AVOID: tyramine - chocolate, wine, cheese (Basically just go ahead and DIE!!!)

59
Q

Describe the Tremor feature of Parkinson’s?

A
  • Primarily during rest
  • Aggravated with emotional stress
  • Can involve the diaphragm, tongue, lips, and Jaw
60
Q 
---These are S&S of what disease---
Witness in skeletal muscles particularly:
-Eyelids
-Chewing
-Swallowing
-Speaking
-Breathing
(Strength is usually restored after a period of rest)
A

Mysthenia Gravis

61
Q 
benztropine (Cogentin)
procyclidine (Kemadrin)
Trihexyphenidyl (Artane)
Tx which disease?
Class?
Action?
Tx Sx's?
A

PD
Class: anticholinergics
Action: block cholinergic receptors - balance cholinergic and dopaminergic activity
Tx: Tremor

62
Q

How is Myethenia Gravis Dx’d?

A

Hx
Physical
Tensilon Test

63
Q

What is the Tensilon Test?

A

Used to Dx Myesthenia Gravis.

  • If after admin; Pt has ↑Muscle Contractility
    • Dx: MG
  • If after admin; Pt/No improvement
    • Dx: Cholenergic Crisis aka Tensilon O/D
64
Q

What counteracts Tensilon?

A

Atropine

65
Q

The drugs Mestinon/Prostigmin are what type of drug? and used in what disease?

A
  • Anticholinasterase
  • Myesthenia Gravis
  • *O/D can lead to Cholenergic Crisis
66
Q 
diphenhydramine (Benadryl)
Tx which disease?
Class?
Action?
Tx Sx's?
A

PD
Class: Antihistamine
Action: anticholinergic effects
Tx: tremor, rigidity

67
Q 
Amantadine (symmetrel)
Tx which disease?
Class?
Action?
Tx Sx's?
SE?
A

PD
Class: Dopamine agonist (anti viral also)
Action: Blocks reuptake of dopamine into presynaptic neurons
Tx: akinesia, tremors
SE: mood changes, confusion, depression, hallucinations, LE edema, nausea, epigastric distress, urinary retention, headache, visual impairment.

68
Q

What would Prednisone a Corticosteroid be used for in dealing with Myethenia Gravis?

A

Suppresses the immune response that’s responsible for the disease.

69
Q

Why would Immunosuppressive agents like Imuran, Cellcept, and Sandimmune be used for Myesthenia Gravis?

A

Helps Suppress the immune response that’s responsible for the disease.

70
Q

What are the 2 main Sx’s for those w/Parkinson’s?

A

-Deep Brain Stimulation
-Electrode placed in brain;↓’s neuronal activity
;Improves motor function/Dyskinesia

  • Transplantation
    • Of Fetal Neuronal Tissue; Produces Dopamine
71
Q

Which drug Tx for PD might cause the pt. to need to take a drug holiday?
Why?

A

levodopa

Pt. builds up tolerance over time so drug no longer works

72
Q

What Sx’s can be done for Myesthenia Gravis?

What Other Tx’s can be done?

A
  • Thymectomy (for puberty-65y/o)

- Plasmapheresis (Short-term), & IV IgG

73
Q

This disease is a chronic, progressive neurologic disease characterized by slow initiating and stopping of movements ↑muscle rigidity, tremor at rest, and unsteady gait.
(> in M/peaks in 70’s %chance ↑w/age)

A

Parkinsons Disease

74
Q

Acetylcholine helps ___[1]___.

Dopamine helps ___[2]___.

A

1: Initiate Movement
2: Inhibit Movement

75
Q

What are the Clinical Manifestations of the early Chronic phase of HIV?
(T4 Count >500)

A
  • None; Pt is generally Asymptomatic

- Pt at this point the body is able to keep T4 Production ↑ which keeps the person seemingly healthy.

76
Q

What are the Clinical Manifestations of the Intermediate Chronic phase of HIV?
(T4 Count 200-500)

A
  • Persistent fever, night sweats, chronic diarrhea, recurrent headaches, severe fatigue.
  • Opportunistic infections Like Thrush, Shingles, Herpes, Vaginal Yeast infections, Oral Hairy Leukoplakia (Caused by Epstein Barr), Pneumonitis Jiroveci (PCP), Kaposis Sarcoma (Lesions on face), Toxiplasmosis (in CATS)
77
Q

What are the 3 early S&S of Parkinson’s?

A

Mild tremor
Slight limp
↓ Arm Swing

78
Q

What are the 3 Late S&S of Parkinson’s?

A

Shuffling
Propulsive gait w/arms flexed
Loss of postural reflexes

79
Q

Describe the Tremor of Parkinson’s?

A
  • Primarily during rest
  • Aggravated with emotional stress
  • Can involve the diaphragm, tongue, lips, and Jaw
80
Q

Describe the Rigidity feature of Parkinson’s

A
  • ↑Resistance to passive ROM

- Pt may C/o Muscle soreness/achiness/tiredness

81
Q

Depression, anxiety, apathy, fatigue, constipation, impotence, short-term memory impairment, akinesia, dementia, & dysphagia are all Non-motor related S&S of what disease?

A

Parkinson’s

82
Q

How is Parkinson’s Dx’d?

A
  • Hx

- 2 o/o 3 of the Triad MUST be present

83
Q

Levodopa/Carbidopa are what type of drugs?

What do they do for Parkinson’s Pt’s?

A
  • Dopamine Precursors

- Tx the triad

84
Q

Symmetrel/Apokyn are what type of drugs?

What do they do for Parkinson’s Pt’s?

A
  • Dopamine Agonists

- Tx the triad

85
Q

Benadryl is what type of drug?

What do they do for Parkinson’s Pt’s?

A
  • Antihistamine

- Tx Tremor/Rigidity

86
Q

Eldepryl/Carbex/Azilect are what type of drugs?

What do they do for Parkinson’s Pt’s?

A
  • Monoamine Oxidase Inhibitors

- Tx the Triad

87
Q

Artaine/Cogentin/Akineton are what type of drugs?

What do they do for Parkinson’s Pt’s?

A
  • Anticholinergics

- Tx Tremor

88
Q

Parlodell/Mirapex/Requip/Neupro are what type of drugs?

What do they do for Parkinson’s Pt’s?

A
  • Dopamine Receptor Agonists

- Tx the Triad

89
Q

Comtan/Tasmar are what type of drugs?

What do they do for Parkinson’s Pt’s?

A
  • COMT (Catechol O-Methol Transferase Inhibitors)

- Tx: Given With Levodopa; Prolongs action/Prevents Breakdown

90
Q

What are the 2 main Sx’s for those w/Parkinson’s?

A

-Deep Brain Stimulation
-Electrode placed in brain;↓’s neuronal activity
;Improves motor function/Dyskinesia

91
Q

Describe the Patho of HIV in 6 “Steps”.

A
  • Fusion: Uses gp120 to attach to cell; endocytosis
  • Reversetranscriptase: Converts RNA to DNA
  • Integrase: Integrates Viral DNA into Cell DNA
  • Viral Transcription: New Viral DNA is made
  • Protease: Viral DNA Snipped into RNA
  • Exocytosis: Packaged New RNA leaves cell
92
Q

HIV attacks ___[1]___/___[2]___.
After initial infection the host enters a state of __[3]__. This State is known as the __[4]__.
After a few weeks viral load __[5]__ and HIV.
For the next __[6]__ to __[6]__ years the Virus continues to multiply.

A

1: CD4
2: T4
3: Viremia
4: Window Period
5: Drops
6: 10-12 years

93
Q

People who have HIV may never have AIDS if their T4 count Remains above _________.

A

> 200

-Intermediate: 200-500 T4 Count

94
Q

What does ETOH withdrawal involve?

A 
(1st 24-48hrs)
Hyperalert 
jerky movements 
irritability 
startled easily
 C/O "shaking inside"
Might need to Frequently Orient to P/P/T
My suffer from illusions: Misinterpretation of objects in environment
95
Q

What are the Clinical Manifestation of the early Chronic phase of HIV?

A

None; Pt is generally Asymptomatic

  • Pt at this point the body is able to keep T4 Production ↑ which keeps the person seemingly healthy.
    - Considered to be a T4 count >500
96
Q

What are the Clinical Manifestations of the Intermediate Chronic phase of HIV?
(T4 Count 200-500)

A
  • Persistent fever, night sweats, chronic diarrhea, recurrent headaches, severe fatigue.
  • Opportunistic infections Like Thrush, Shingles, Herpes, Vaginal Yeast infections, Oral Hairy Leukoplakia (Caused by Epstein Barr)
97
Q

How is HIV Dx’d?

A

S&S
↓WBC count (Neutropenia common)
Elisa antibody test
Western Blot (Confirms/Definitive)

98
Q

What is ART in reference to HIV?

A

Anti-Retroviral Therapy

  • Multi-drug approach
  • ↓HIV Replication
99
Q 
---What types of Drugs are these? (used for HIV)---
Retrovir
Videx
Zerit
Epivir
Ziagen
Emtriva
Combivir
Trizivir
Epizicom
A

Nucleoside Reverse Transcriptase Inhibitors

100
Q 
---What types of Drugs are these? (used for HIV)---
Viramune
Rescriptor
Sustiva
Intelence
A

Nonnucleoside Reverse Transcriptase Inhibitors

101
Q

—What types of Drugs are these? (used for HIV)—
Viread
Truvada

A

NucleoTide reverse Transcriptase Inhibitors

102
Q

—What types of Drugs are these? (used for HIV)—

Raltegravir

A

Integrase Inhibitors

103
Q 
---What types of Drugs are these? (used for HIV)---
Invirase
Crixivan
Norvir
Viracept
Reyataz
Lexiva
Tipranavir
Darunavir
Kaletra
A

Protease Inhibitors

104
Q

—What types of Drugs are these? (used for HIV)—
Fuzeon
Selzentry

A

Entry Inhibitors

105
Q

_______ Is the habitual use of a substance that falls outside medical necessity/social acceptance in order to alter one’s mood/emotion/LOC.

A

Abuse

106
Q

_______ Is the habitual Psychological/physiological dependence beyond one’s voluntary control.

A

Addiction

107
Q

What are the 3 C’s to addiction?

A

Compulsion (to use)
continued (Use)
Control (The LOSS of it)
-Cognitive impairment (The 4th “C”)

108
Q

There is a __[1]__ link to Predisposition to Addiction.
Those who abuse drugs are @↑Risk of ___[2]___.
The ___[3]___ Neurotransmitter is usually affected by most drugs.

A

1: Genetic
2: Suicide
3: Dopamine

109
Q

Issues with the drug __[1]__ are the most common seen in the Hospital.
The Disorder ____[2]____ ___[2]___ is a result of Thiamine Deficiency and is associated with__[3]__.
The other Disorder associated with alcohol use is __[4]___ ___[4]___, which is characterized by irreversible Amnesia.

A

1: EOTH
2: Wernicke’s encephalopathy
3: EOTH
4: Korsakoff’s psychosis

110
Q

What are the 3 areas of the brain that are affected by and contribute to Substance abuse?

A
  • Brainstem: HR/Breathing/Sleeping
  • Limbic System: “Reward Circuit”/Pleasure
  • Cerebral Cortex: Process Sensory data/Think/Plan/Problem solve/Decisions
111
Q

___[1]___ account for 40-60% of a persons vulnerability to addiction.
__[2]__ acts as a chemical “Present” to areas of brain that regulate motivation, emotion, cognition,learning, & pleasure.

A

1: Genetics
2: Dopamine

112
Q

Opioids work on __[1]__ receptors.

EOTH works on __[2]__ receptors

A

1: Opioid receptors
2: GABA receptors

113
Q

How do they Dx addiction?

A

Cage-aid Questionare
Toxicology Screen
Blood Alcohol Level

114
Q

What does ETOH withdrawal involve?

A 
(1st 24-48hrs)
Hyperalert 
jerky movements 
irritability 
startled easily
 C/O "shaking inside"
115
Q

This is an ETOH emergency and can lead to death that peaks 2-3 days after cessation.

A

ETOH Delirium
-Cause of death is usually Result of sepsis, MI, fat embolism, peripheral vascular collapse, electrolyte imbalance, aspiration, or suicide

116
Q

The drug __[1]__ creates an imbalance w/Dopamine/epinephrine. It stimulates both sexual arousal and violent behavior. It blocks sensory tranmission; Mainly __[2]___ transmission.

A

1: Cocaine
2: Pain

117
Q

Withdrawal from Cocaine S&S include:

A

Depression, paranoia, lethargic, anxiety, constricted/wide eyes, insomnia, nausea, vomiting, sweating, and chills

118
Q

The term Opiate refers to what drugs?

A

Opium, morphine, heroin, codeine, fentanyl, methadone, and meperidine

119
Q

The Term Hallucinogens refer to what drugs?

A

Mescaline (peyote), shrooms, LSD (acid)

120
Q

_______ lessens touch and sensitivity to pain. S&S of it are Acute anxiety, psychosis, violence, ↓coordination

A

PCP

121
Q

Users of this drug tend to be younger (13-17yo) because of their ease of access. S&S are similar to drunkenness.

A

Inhalants

122
Q

This drug is most commonly used in Clubs (13-28yo); S&S include: ↑HR, euphoria, ↑Energy/self-confidence/sociability, and the feeling of “closeness” to people.

A

Ecstacy

- use can lead to HF, Hyperthermia, and kidney failure

NUR 211 (3 decks)

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