Exam 2: Pain, Preoperative Care, Cultural Competency, Spirituality Flashcards
Who can rate pain levels?
Only the patient experiencing the pain
Why should you trust your patient?
People express pain in different ways, so even though they may not look like they are experiencing pain, they are the only ones who can tell you what they are feeling.
What is pain?
An unpleasant sensory and emotional experience associated with actual or potential tissue damage.
OR
Whatever the person says it is, and existing whenever the person says it does.
What can you do is you sense that someone is drug-seeking?
Be honest with your patient, you can say things like: “It seems like your pan level is always a 10, and the opioids aren’t helping you, what would help?” or “I/m wondering if you are needing this or wanting this for more than your pain?”
Noxious Stimulus
Heat, knife cut, extreme cold, chemicals/acid, things that start the physiological process of feeling pain.
What steps are involved in the physiology of pain?
- Transduction: NT release, converts stimulus to electrical impulse.
- Transmission: impulse relayed to dorsal horn in CNS, processed by NT, sent to thalamus, then relayed to cerebral cortex.
- Perception: Pain perceived in cortex (almost right away)
- Modulation: Pathway that inhibits pain.
What is a nociceptor?
Pain neuron, nerve receptor that transmits pain.
Where are nociceptors located?
Everywhere except in the brain, mostly located at the ends of small afferent neurons.
What stimulates nociceptors? (2 bullet points)
-Direct cell damage
-NT including:
*substance P (released with inflammatory process)
*histamine (causes itching)
*bradykinin (vasodilator, releases pro inflammatory chemicals-redness, swelling, tenderness.)
prostaglandins (released by damaged cells, increase sensitivity of pain reception.)
What is nociceptive pain described as and what does it respond to?
Described as aching or throbbing, known as “normal pain.”
Responds to both non opioid and opioid analgesics.
What should you do after administering an analgesic?
After med is given, Re-assess patient.
If anticipated effect is not shown upon re assessment, something is wrong, contact the physician. (May need another med, may have an infection, etc.)
Conduction Velocity: bigger neuron=________ speed.
Faster
Conduction Velocity: More _______=Faster speed.
Myelinated
Characteristics of C fibers
Nociceptor, 1 um diameter, non myelinated, speed 2 m/second (slowest), sensory perception= dull, aching, burning pain; slow onset, longer duration
Characteristics of A-delta Fibers
Nociceptor, 4 um diameter, myelinated, speed 15 m/sec. Felt immediately, sharp localized pain, short duration.
Characteristics of A-beta fibers
Non-nociceptors (afferent nerve endings but not pain receptors) 13 um diameter, myelinated, speed 70m/sec , sensory transmission: touching the skin.
Characteristics of A-alpha fibers
Non-nociceptors (afferent nerve endings but not pain receptors), 20 um diameter, myelinated, speed 120 m/sec, sensory transmission: muscle impulses (massaging muscle)
What is the “Gate Theory” of Pain?
non-painful input closes the “gates” to painful input, which prevents pain sensation from traveling to the central nervous system. Therefore, stimulation by non-noxious input is able to suppress pain. (If A-beta and A-alpha stimulus moves faster than nociceptor stimulus, they will arrive at the gate first and modulate how many pain impulses get through.)
Where is the “gate” located in the gate theory of pain?
At the dorsal horn synapse
What opens the “gate?”
Nociceptor input
Anxiety
Worry
Concentrating on pain
What closes the “gate?”
Non-nociceptor input
positive mood
Concentrating on other than pain
What are our endogenous opioids?
Our own narcotics, serotonin, Norepinephrine, endorphins, enkephalin
What can trigger our endogenous opioids to be released?
Chili peppers Alcohol Exercise Laughing Meditation Listening to music Walking barefoot in the grass
What effect does Naloxin have on opioids? Does it affect our endogenous opioids?
Naloxin used for overdose, reverses effect of opioids and does the same to our endogenous opioids as well.
How does the endogenous analgesia system work? (2 bullet points)
- Pain impulse activates descending nerve fibers, triggers release of NT including endogenous opioids, they bind to opiate receptors on nerve cells, inhibit substance P release, decrease pain transmission to the brain.
- Pleasurable experiences also trigger release
What is acute pain? (7 bullet points)
- Normal, expected, and treatable
- Sudden onset, localized, lasts < 3 months
- Result of tissue injury or inflammation
- Resolves with healing of disorder (LTG: @ this point, pain should be gone)
- Initiates Protective response; facilities healing by making injured area surrounding tissue hypersensitive
- Stimulates fight or flight response: Tachycardia, tachypnea, increased BP, fear, anxiety, etc.
- Therapy aimed at treating underlying cause & interrupting nociceptive signals.
What might you expect during assessment of acute pain?
Tachycardia, tachypnea, increased BP, fear, anxiety in initial pain perception, may be elevated during assessment before pain med administration. Still not absolute as people handle pain differently.