Exam 2 Mutli Choice Flashcards

1
Q

Understand the Fear Circuit

A

Info comes in from the world; to the thalamus; to either amygdala or prefrontal cortex

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2
Q

If fear information goes to the amygdala first then…

A

The stress response starts quickly (mostly unconscious)

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3
Q

If fear information goes to the prefrontal cortex first then…

A

The stress response starts slowly (thinking about it; intentional)

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4
Q

How do the amygdala and prefrontal cortex communicate?

A

The P.C. can tell the amygdala to calm down; the amygdala can tell P.C. to get anxious

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5
Q

How is GABA associated with anxiety?

A

People with GAD have limited GABA; which is released to inhibit “anxious” neurons from firing

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6
Q

Most common class of antiaxiety drugs

A

Benzodiazepines

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7
Q

Characteristics of Benzodiazepines

A

Provides temporary relief, causes drowsiness, anxiety with withdrawal, physical dependence is possible, mixes poorly with other drugs

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8
Q

Most common class of drugs used to treat most anxiety disorders

A

Antidepressants

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9
Q

Typical characteristics of antidepressants

A

Affects serotonin and norepinephrine in the brain, regulates the fear circuit

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10
Q

3 Cognitive New Wave Explanations for Anxiety

A

-Intolerance of Uncertainty
-Avoidance Theory
-Meta Worry

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11
Q

Intolerance of Uncertainty

A

Some people have a higher tolerance to the unknown

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12
Q

Avoidance Theory

A

Some of us are very uncomfortable with the physical experience of stress

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13
Q

Meta Worry

A

Sometimes we judge ourselves for being anxious (worry about worrying)

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14
Q

What is the biological challenge?

A

The induction of physical sensations to cause panic

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15
Q

What is “preparedness?”

A

The idea that we are inherently more prepared to be afraid of some things over others (we are “primed” to be afraid of certain things)

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16
Q

How does stimulus generalization of Specific Phobias help explain GAD?

A

A person is afraid of one thing; they avoid that thing; the stimulus and its responses become more generalized; person becomes generally anxious

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17
Q

Common treatments for Social Anxiety?

A

Antidepressants, CBT, Social Skills Training, Exposure Therapy

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18
Q

What are the two common areas of the brain associated with OCD and Body Dysmorphic DO?

A

-(Overactive) orbital frontal cortex
-(Overactive) left hemisphere

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19
Q

What is thought action fusion?

A

A risk factor for OCD involving a belief that thinking something is the same as doing it (thoughts are equal to behavior)

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20
Q

What are the 4 clusters of symptoms associated with Stress DOs?

A

-Intrusion
-Dissociative
-Avoidance
-Arousal

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21
Q

What two hormones/NTs seem most implicated in producing stress?

A

-Cortisol
-Norepinephrine

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22
Q

What are the 7 common traumas associated with creating Stress DOs?

A

-Combat
-Natural disasters
-Mass shootings
-Terrorism
-Physical victimization
-Sexual assault/trauma

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23
Q

Common reasons why some people develop a Stress DO when exposed to stress and others don’t

A

Differing biological systems, personalities, childhood experiences, social support systems, cultural backgrounds, severity of the trauma

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24
Q

WET Treatment

A

Written Exposure Therapy

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25
Q

EMDR Treatment

A

Eye Movement Desensitization & Reprocessing

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26
Q

What are WET & EMDR Therapies?

A

Different types of exposure therapies for trauma/stress DOs

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27
Q

What has replaced Psychological Debriefing after community traumas & why?

A

Psychological first aid; P.D. has been found to actually cause PTSD in people who would’ve never had it

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28
Q

What are the 2 main categories of symptom dysfunction associated with Conversion DO?

A

Altered voluntary MOTOR or SENSORY function

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29
Q

What personality characteristic is associated with Conversion DO?

A

High suggestibility

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30
Q

4 Treatments for conversion and somatic DOs

A

-Education
-Changes in reinforcement
-Exposure flooding
-Cognitive restructuring

31
Q

What is Localized Amnesia?

A

Inability to remember ALL events occurring within a LIMITED time period

32
Q

What is Selective Amnesia?

A

Inability to remember SOME events occurring within a period

33
Q

What is Generalized Amnesia?

A

Inability to remember extending back in time - loss of identity

34
Q

What is Fugue?

A

A dissociation involving WANDERING/TRAVEL - making a new life (often from generalized)

35
Q

What are the 3 ways DID personalities differ from one another?

A

-Identifying features
-Ability & preference
-Physiology

36
Q

What DO is DID most often comorbid with?

37
Q

Evidence for the Sociocognitive Explanation for DID

A

-DID is NOT a true DO; caused by media/therapist
-Rapid increase in 80s after movies/novels; differences in culture
-Clients already in therapy for other DOs, false memory syndrome; clients rewarded for showing different personalitities

38
Q

What are the 4 areas of the brain in the depression circuit?

A

-Amygdala
-Prefrontal Cortex
-Hippocampus
-Subgenual Cingulate (AKA Broadmann Area 25)

39
Q

What are the 4 common drug groups used to treat Depression?

A

-MAO Inhibitors
-Tricyclics
-SSRIs
-Ketamine

40
Q

How quickly do SSRIs work for mood DOs?

A

10 days-3 weeks before improvement

41
Q

What is the most common and most problematic side effect of SSRIs?

A

PSSD: Post SSRI Sexual Dysfunction

42
Q

What is treatment resistant depression?

A

Failure to respond to 2 types of antidepressants

43
Q

What is Electroconvulsive Therapy?

A

-Treatment for TRD
-Electrodes on scalp, strapped down, stimulating motor cortex, extreme muscle tension
-10 sessions

44
Q

What is Vagus Nerve Stimulation?

A

-Treatment for TRD
-“Battery” connected to the vagus nerve, which is responsible for sending signals to the depression circuit to turn on/off
-INVASIVE
-9 months

45
Q

What is Transcranial Magnetic Stimulation?

A

-Treatment for TRD
-Magnetic waves from a cap with magnets on it, which turn up or down the electric responses of the brain; “flashes”
-LEAST invasive

46
Q

What is Deep Brain Stimulation?

A

-Treatment for TRD
-“Modern Trephination”
-Drill into skull, completely awake, inserts an electrode into Broadmann Area 25, turns on
-Immediately works
-Must destroy brain tissue to do this
-Potential death

47
Q

What is Artifact Theory?

A

Women & men are equally prone to depression; but clinicians often fail to detect depression in men

48
Q

What is Life Stress Theory?

A

Women in most societies experience more stress than men

49
Q

What is Body Dissatisfaction Theory?

A

Women in most societies are taught to seek unreasonable goals that are unhealthy

50
Q

What is Lack-of-Control Theory?

A

Women feel less control over their lives than men

51
Q

What is Rumination Theory?

A

People who ruminate when sad are more likely to become depressed & stay depressed longer

52
Q

What is mania?

A

-Active, powerful emotions, need for excitement, little need for sleep, very active/quick activity, poor judgement
-For at least a week

53
Q

What is Bipolar vs Unipolar Depression?

A

-With Bipolar Depression, one will typically experience both depressive & manic episodes
-With Unipolar Depression, one will typically only experience depressive episodes

54
Q

How does the permissive theory of NTs explain Bipolar vs Unipolar Depression?

A

-Depression: Low serotonin, low norepinephrine
-Mania: Low serotonin, high norepinephrine
-Norepinephrine causes manic episodes

55
Q

What are the most common drug therapies for Bipolar DO?

A

-Lithium
-Antiseizure Drugs
-Second-Generation Antipsychotics

56
Q

When does psychotherapy work for Bipolar DO?

A

When combined with mood stabilizers

57
Q

What new childhood mood DO was created for the DSM-5 and why?

A

Disruptive Mood DO; doctors were diagnosing young children with Bipolar DO

58
Q

What are the SIGNS of suicide?

A

-Sleep Disturbance
-Isolation
-Giving Away Posessions
-No Interest in Anything
-Seeing no Future

59
Q

Five Steps of Assessing Suicide Risk

A

-Pervasiveness of Mood
-Strength of Desire
-Is There a Plan?
-Are Resources Available to Carry out Plan?
-Do they have attachment relationship?

60
Q

What is a binge episode?

A

An often secret episode of eating large amounts of food in a single sitting

61
Q

Common Compensatory Behaviors (Bulimia)

A

Vomiting, laxatives, excessive exercise/fasting

62
Q

How are mood and eating DOs related?

A

Mood DOs “set the stage” for EDs

63
Q

What groups of women are more likely to suffer from EDs?

A

Models, dancers, actors, college athletes

64
Q

What are the two stages in treating Anorexia?

A

-Return to healthy weight
-Improve psychology

65
Q

3 Phases of Maudsley Approach

A

-Restore Weight (Model parents’ uncritical stance)
-Return Control to Sufferer (Address parental concerns)
-Healthy Adolescent Identity (Increase autonomy, negotiate parental boundaries)

66
Q

3 Phases of Interpersonal Therapy (Bulimia)

A

-Identify interpersonal problems (Role disputes/transitions, Interpersonal deficits, Unresolved grief)
-Patient-Led Change (Therapist strongly encourages change)
-Maintenance (Relapse prevention)

67
Q

Between Anorexia & Bulimia who is more likely to receive in-patient treatment?

68
Q

Between Anorexia & Bulimia who is more likely to receive antidepressant therapy?

69
Q

Most Common Depressants

A

-Alcohol
-Sedative-Hypnotic Drugs
-Opioids

70
Q

Most Common Stimulants

A

-Cocaine
-Amphetamines
-Caffeine
-Nicotine

71
Q

What NT does alcohol affect?

A

Helps GABA (an inhibitory messenger) shut down neurons and relax

72
Q

What NT does heroin (opioids) affect?

A

Mimics endorphins; binds to receptors that receive endorphins

73
Q

What NT does cocaine affect?

A

Increases dopamine; prevents its reabsorption

74
Q

What NT does LSD affect?

A

Binds to serotonin receptors; floods brain with serotonin activity