exam 2- muscles Flashcards
tendons
fibrous connective tissues that attaches skeletal mucsles to bone
ligaments
fibrous connective tissue that connects bones to where they meet a joint
strain
tear or rupture in a tendon, may be accompanied by a freature
sprain
tear or rupture of a ligament, may be accompanied by a fracture
avulsion
a complete separation of a tendon or ligament from the bone,
may be accompanied by a fracture
patho repare chain for traumatic injuries to tendons or ligaments
inflammatory exudate developes between torn ends
repair process- granulation tissue
collagen formation (connective tissue)
vascular fibrous tissue fuses tissues into single mass
clinical mani of traumatic injuries to tendons or ligaments
sharp, localized pain and tenderness
soft tissue swelling
treatment for traumatic injuries to tendons or ligaments
RICE
heat to increase healing and comfort (72hr post injury)
total healing time is normally 4-6 weeks
RICE
rest
ice to decrease swelling (1st 72 hrs)
compression
elevation
muscle strains
local muscle damage from sudden, forced motion, causes muscles to be stretched beyond normal capacity
etiology of muscle strains
sports injuries, trauma
patho of muscle strains
hemorrhage into surrounding tissues and inflammation present
clinical mani of muscle strains
pain and bruising
treatment for muscle strains
ice
analgesia
rest
healing = 6 weeks
myglobinuria (rhabdomyolysis)
life threatening comp of severe muscle trauma
etiology of myglobinuria
burns, trauma such as crush, compartment, extreme exertion
patho of myglobinuria
increased pressure within muscle fibers, idema, muscle ischemia and muscle infarction
muscle cell damage 9rupture of sarcolemma) releases myoglobin and intracellular proteins
myogblobin is detected in the urine where it is not normally found
clinical mani of myglobinuria
dark reddish brown urine
can lead to acute renal failure (intratubular obstruction)
treatment of myglobiuria
treat etiology
prevent renal failure : flush kidneys with diuretics and iv fluids
duchenne muscular dystrophy
genetic disorder characterized by progressive muscle degeneration due to no production of the protein dystrophin
etiology of duchenne MD
an x linked recessive disorder (affects 1/3500 male births
patho of duchenne MD
genetic defect caused no production of dystrophin (protein controls Ca for contraction)
decrease of and necrosis of muscle fibers
increase of connective tissue (causing fibrosis)
increase of fat (fatty infiltration which replace muscle fibers (yellowish pink stain)
elevated serum enzymes (muscle cell mem destroyes and enzymes leak out)
onset of DMD
3 yoa
progression od DMD
rapid lower extremity weakness progresses up to chest and head muscles
mani of DMD
waddling delayed sitting and walking (falls) shoulder girdle weaknedd 3-5 years after kyphoscoliosis causes pulm dysfunction cardiac dysfunction common
when do DMD people die
20-30’s due to respiratory and cardiac involvement
treatment of DMD
maintain function of unaffected muscle groups
supportive
genetic counseling
adult forms of MD
deckers
limb/girdle
facioscapulohumeral
myotonic
adult MD are characterized by
later onset
differing patterns of involvement
heredity
rate of progression