Exam 2 GYT Eyes, ENT, Cards, Resp Flashcards

Get you thinking and objectives

1
Q

What is excessive cupping of the optic disc?

A

Is a backward depression of the disc. Caused by IOP which leades to cupping and atrophy.

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2
Q

Bacterial Conjunctivitis.

A

-KEY FINDING-Erythemia in one or both eyes, then moving to both and yellow-green prulent discharge, encrusted and matted eyelids on awakening. -Burning, stinging or itching and feeling of foreign body. -Petechiae on bulbar conjunct. SX of URI, OM and Acute pharyngitis. -Contageous, H.influenzae. -MOST COMMON ORGANISM ISOLATED IN CHILD <7.

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3
Q

Viral Conjunctivitis.

A

-KEY FINDING- Tearing and profuse clear, watery DC, Fever, headache, anorexia, malaise, UR sx(pharyngitis-conjunct-fever triad w/adenovirus. Pharyngitis with enlarged Pre-auri nodes. -Itchy, red swollen conjunc. Hyperemia (increased blood flow) and swollen eyelids. -Photophobia, herpetic vesicles on eyelid and eyelashed. -Caused by Adenovirus or herpes. more common in children >6y.

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4
Q

Allergic Conjunctivitis

A

4 types: Hay fever-associated Conjunct; Vernal Conjunctivitis, Atopic keratonconjunctivitis and giant papillary conjunct. -KEY FINDING-severe itching and tearing. -Hay fever Assoc-mild injection and swelling with environmental allergens, nasal congestion. -Vernal Conj-More severe, peak in 10-12yom, warm weather prevalent. -Atopic conjunctivitis-occurs in pple with atopic dermatitis or asthma affecting the lower tarsal conjunct. Late adolescence. Significant itching, burning and tearing chronic. -Giant papillary-Most often in contact lens wearers. occurs 10 or more times

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5
Q

TX for Viral, Bacterial and Allergic Conjunctivitis.

A

-Viral-(self limiting) warm compress, antihistamine opth solu. if HSV refer to opth specalist. -Bacterial-(self limiting) topical abx in eye, broad spect abx if you MUST. with lowest resistance rate. -Allergic-Prevention is best, avoid allergens. saline solution artifical tears. topical decong, NSAID

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6
Q

What is the abnormal fundoscopic finding of AV Nicking?

A

Vein appears to stop abruptly on either side of the artery. at the AV crossing points the arteries indent and displace veins.

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7
Q

Why is red relex important what is white reflex of the eyes?

A

Red reflex helps to detect the presence of asymmetric refractive errors, strabismic deviations and abnormalitis in the ocular media (cataracts, corneal abn, retinoblastoma). thse disease block the light from entering or exiting the pupil. -White reflex is what you would see in the absence of the red reflex. (abnormal finding). Small children could indicate blindness.

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8
Q

What is uveitis?

A

Uveitis is a form of eye inflammation. It affects the middle layer of tissue in the eye wall (uvea). It consists of the iris, the ciliary body and the choroid and is the soruce of deep blood flow to retina. SX come on quickly and get worse quickly! Eye redness Eye pain Light sensitivity Blurred vision Dark, floating spots in your field of vision (floaters) Decreased vision -If not cared for can cause Glaucoma Cataracts Optic nerve damage Retinal detachment Permanent vision loss

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9
Q

What is corneal infection?

A

Keratitis is an inflammation of the cornea — the clear, dome-shaped tissue on the front of your eye that covers the pupil and iris. Keratitis is sometimes caused by an infection involving bacteria, viruses, fungi or parasites. Noninfectious keratitis can be caused by a minor injury, contaminated water, wearing your contact lenses too long or other noninfectious diseases. -SS Eye redness Eye pain Excess tears or other discharge from your eye Difficulty opening your eyelid because of pain or irritation Blurred vision Decreased vision Sensitivity to light (photophobia) A feeling that something is in your eye -PREVENTION IS KEY!!

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10
Q

What is anterior epistaxis? What usually causes it?

A

Nosebleed in the Anterior aspect of nose where the majority of bleeds occur. Usually caused by trauma, vigorous nose blowing, nasal dryness and foreign body. Less sever than posterior.

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11
Q

What is Menieres Disease?

A

Is vertigo with hearing loss and nystagmus. Sudden onset. Causes sensorineural hearing loss which recurs and progresses. Tinnitus is present and pressure or fullness in the ear, n/v.

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12
Q

How can you assess hearing loss from Menieres?

A

Tuning fork tests: Webber and Rinne. These test for sensorineural loss which is what happens in Menieres disease.

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13
Q

What does the Webber test do?

A

Tests for Lateralization. In unilateral conductive hearing loss sound is heard in (lateralized to) the impaired ear.

In Unilateral Sensorineural hearing loss sound is heard in the good ear.

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14
Q

The Weber tuning test lateralizes to the affected ear.

A

TRUE.

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15
Q

The Rinne test reveals that air exceeds bone conduction?

A

FALSE. In conductive hearing loss bone conduction exceeds air conduction.(BC>AC) In sensorineural hearing loss sound is heard longer through the air. (AC>BC)

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16
Q

Findings in Menieres Disease?

A

The Webber test would be heard in the good ear for sensorineural hearing loss.

The Rinne test sound would be heard longer though the air (AC>BC).

Positive Rombert test-looses balance when eyes closed.

Positive Fukuda marching test when marching in place wiht eyes closed.

Positive Dix-Hallpike test

17
Q

Romberg Test.

A

test of position sense. pt should stand w/feet together and eyes open and then close them for 30-60 seconds without support.if pt looses balance with eyes closed is a POSITIVE TEST.

18
Q

What is the Fukuda marching test

A

The Fukuda Stepping test is one balance and vestibular test that may also be performed during a vestibular and balance exam. The test is used to determine if there is vestibular system weakness on one side of your body.

Pt unable to march in place with eyes closed.

Postitve test in Menieres

19
Q

Dix-Hallpike test

A

Test for room spinning symptoms. MD turns head all the way to the side and has pt lay back really fast. Will mimic them feeling as if room is spinning. This is a posisitve finding.

Used to test for paryoxysmal vertigo and benign vertigo.

20
Q

What is the physiological response causing allergic rhinitis mediated through (antiobodies)?

A
  1. Allergen exposure>sensitization>production of antigen-specific IgE.
  2. Early phse response-within minutes, release of mast cells, basophils, histamine and newly synthesized mediatiors. Mediators produce early SX like sneezing, itching, rhinorrhea, mild congestion.
  3. Late phase response- 2-4h after exposure, mediators promote production of cytokines, chemokines and adhesion olecules and the influx of eosinophils which release more mediators, leading to a chronic inflammatory response with more nasal congestion.
21
Q
  1. What lymph nodes are often affected by pharyngitis, sinusitis or otitis infections?
A

Pharyngitis- tender enlarged anterior cervical lymph nodes.

22
Q

A PMI (Point of maximal impulse) greater than 2.5 cm is evidence of what and why?

A

LVH from hypertension or AS.

23
Q

What is the meaning of Heart Sounds diastolic S3 and S4 in adult age 40 and older?

A

Correlated with heart failure and acute MI.

S3-an abrupt deceleration of inflow across the mitral valve.

S4- increased LVend diastolic stiffness which decreases compliance.

24
Q

Describe the events in the cardiac cycle.

A

Systole is the period of ventricular contraction.

Diastole is the period of ventricular relaxation.

S1 is closure of the mitral valve.

S2 is closure of aortic valve.

p.347

25
Q

What is the cause of splitting heart sounds and what are you listening to and where?

A

Inspiration causes a split of S2 from the aortic and pulmonar valves closing. Is heard in its own area at teh 2nd and 3rd ICS close to the sternum.

S1 has split of earlier mitral and later tricuspid sound. Can be heard at the apex, the softer tricuspid is best heard at the left lower SB. Does not vary with respiration .

26
Q

What does regurgitant murmur mean what does it sound like and where?

A

A murmur caused by retrograde (back flow) flow. Depends on the murmur.

27
Q

Why do NP’s screen for global risk factors what is this?

A

Screens the highest prevalance of disease in a specific area and population. Begin routine screenings at years for individual risk factors for global risk of CVD and for any family history of premature heart disease (<55 in males, <65 females).

28
Q

What is CVD screening if screening abnormal what would make you think this?

A