Exam 2 Flashcards

0
Q

Transcortical sensory aphasia

A

A fluent aphasia with preserved repetition.

  • usually associated with damage to angular gyrus which isolates wernickes area by cutting off association fibers
  • damage is to the watershed regions of upper parietal
  • conversation speech is fluent and filled with neologistic or semantic jargon
  • their ability to repeat words accurately is surprising. No press on speech like with wernickes aphasia
  • comprehension is severely disturbed
  • when they repeat they don’t understand what they are saying (like a parrot talking)
  • naming is defective
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1
Q

What is transcortical motor aphasia?

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A non-fluent aphasia with preserved repetition

  • brain damage is usually in the dorsalateral frontal cortex. Can affect association fibers (white matter) isolates the language center from other areas of the brain
  • repetition is fine but spontaneous speech is impaired
  • utterances may be preservative and not finished
  • comprehension can be functional
  • seem distant and difficult to engage in conversation
  • show little interest in using language
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2
Q

Transcortical mixed aphasia

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A global aphasia with relatively preserved repetition

  • damage involves both anterior and posterior portions do watershed areas
  • primary cause is stenosis (blockage of internal carotid artery)
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3
Q

Define Primary progressive aphasia

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Definition: a language deficit of insidious onset, gradual progression, and prolonged course, in absence of generalized cognitive impairments due to a degenerative disease, predominantly and presumable involving the left perisylvian region of the brain(Duffy 1987)
-gradual progression mirrors degenerative diseases but cognitive abilities are intact, just language is impaired

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4
Q

Primary progressive aphasia clinical presentation

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-these patients rarely deny their deficits
-usually initiate the search for diagnosis and management
-these patients due not observe memory disturbances unrelated to verbal functions
-no personality changes
Word finding issues & other language problems are present
-different from dementia because there is no concern over memory, personality, or orientation
-due to an unknown condition in the language center

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5
Q

Language confusion

A

This is a language impairment that is usually caused by diffuse bilateral trauma to the brain, such as from head injury, toxic conditions, post surgical stress, or related events

  • typically patients show normal syntax, semantics, and phonology, but the context of their language is confused and confabulatory
  • in most cases the onset is sudden
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6
Q

Language confusion vs. aphasia

A
  • patients with LOC have language abilities that are better than their ability to communicate
  • aphasia patients language abilities are worse than their abilities to communicate
  • LOC – language is good their just not in our context/reality
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7
Q

Sub cortical aphasia

A
  • damage to sub cortical structures and white matter
  • associated with damage to the basal ganglia
  • there is controversy about whether us cortical structures like the thalamus have a direct role in our language abilities
  • nevertheless, language deficits have been associated with subcritical brain damage
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8
Q

Aphasia battery tests: BDAE 3

A

Boston Diagnostic Aphasia Examination 3rd edition

  • there are 5 subsections: 1. Conversational speech, 2. Auditory comprehension, 3. Oral expression, 4. Reading, 5. Writing (plus a section on apraxia)
  • it is norm-referenced, standardized test with norms for both aphasic subjects and normal subjects
  • total administration time is about 2 hours +
  • has a separate naming test
  • included supplementary non-language tests such as R/L orientation, and a acalculia
  • provides a profile of patient scores that can help in the classification of major aphasic syndromes (types)
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9
Q

Aphasia tests: Western aphasia battery

A
  1. Similar in content & purpose of BDAE
  2. The first four sections examine oral language abilities
  3. The final four sections examine reading, writing, apraxia, & arithmetic
  4. This test will give a summary score, which is useful in documenting progress in treatment. The most commonly used score is the aphasia quotient score (AQ)
    - the AQ is the summary score for the oral language abilities section of the test
    - the highest AQ score is 100, meaning either normal language performance or only mildest of impairment
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10
Q

Boston naming test

A
  • assesses name retrieval
  • contains 60 items of progressive difficulty
  • ceiling of 8 consecutive errors
  • most useful for patients with mild naming problems
  • usually used as a criterion referenced test
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11
Q

Overview of aphasia treatment

A
  • clinical treatment has its limits
  • treatment is not limited to speech & language
  • aphasia tx must be structured (repetition)
  • no single set of tasks or procedures is adequate tx (should be dynamic)
  • aphasia tx exploits strengths
  • *clients need to be trained to be their own clinicians
  • involve others
  • many patients take their moods from clinicians
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12
Q

3 goals of aphasia treatment

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  1. To assist people to regain as much communication as their brain damage allows, and their needs drive them to
  2. To help them to learn how to compensate for residual deficits
  3. To help them learn to live in harmony with the difference between the way they were and the way they are
    * *the first two can be acquired through clinical practice and reading books
    * *the third is gained through experience and maturity
    * *the overall goal of treatment is to prepare an aphasic patient for a lifetime of aphasia
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13
Q

What do I treat?

A

Relative Level of Impairment

  • Look at testing to determine which areas are strengths and weaknesses
  • According to porch (1981) treat those specific areas that are slight to moderate impairment
  • If scores of subtests are dramatically treat that area until variability decreases and treatment has approached the patients recovery potential
  • Choosing a goal based on how severe patient is
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14
Q

What do I treat? Fundamental Processes Approach

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-Treat those areas that are thought to be the underlying cause of aphasia impairments–> what areas of language will make the most difference in their life
Ex: treating auditory comprehension in aphasia, as it improves so will so will general communicative ability as the improvement generalizes
-choosing a goal that is most functional for patient

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15
Q

What do I treat? Activity participation approach or functional approaches

A
  • views the purpose of intervention as helping the patients overall success with communication in natural settings
  • choose a targeted life activity
    - ex: talking on the phone &treatment specifically targets skills related to this skill.
  • impairment–>word retrieval issues
  • activity–>ordering a cheeseburger
  • participation–> engage in task with other people
  • *what is the final objective of my tx?
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16
Q

What does therapy look like?

A
  • In a more structured therapy session treating specific impairments:
  • *opening–> conversation
  • *accommodation –> familiar tasks, high success
  • *goal directed therapy–> challenging, tx goal oriented tasks
  • *cool down–> easy tasks, high success
  • *closing–> conversation
  • in activity: participation approach, treatment follows similar pattern but it is slightly less structured, clinician coaches patient in more every day activities
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17
Q

Choosing stimuli

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Resource allocation: help patient toward behavior by eliminating process load (mental capacity)
Stimulus manipulation: intensity (strength ex:auditory how loud) vs. salience (how stimulus stands out)
Clarity/Intelligibility: line drawings may be uncertain
Redundancy/context: redundancy increases the performance of brain injured adults (ex: show me the small red cups vs. show me the cup that is small & red) context is the background or setting
Novelty/interest value: patients respond better to more personally interesting items
Cues: hints given by the clinician when a patient produces an incorrect response

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18
Q

Cueing hierarchy (in order of decreasing power)

A
  • Imitation
  • First sound/syllable
  • Sentence completion
  • Word spelled aloud
  • Rhyme
  • Synonym/Antonym
  • Function/Location (drink it at breakfast)
  • Superordinate (it’s something you drink)
19
Q

How else can a clinician control tx difficulty level?

A
  • Manipulate the length & complexity or responses require sentence length, increased syntactic demand, ect
  • Manipulate familiarity & meaningfulness of responses –alter questions making them more or less personal
  • Manipulate delay–adding a time delay may increase difficulty of a task
20
Q

Giving instructions & feedback

A

-Instructions: clear & concise at an adequate rate for the patient using language the patient understands
-feedback: incentive feedback–> can maintain or eliminate behavior (candy/electric shock)
Informative feedback: tells patient about the accuracy of response
full strength praise (great job) vs. diluted negative feedback (close, but lets do it again)

21
Q

Generalization

A

Use of natural maintenance–>hello/goodbye are naturally reinforced by others in daily lives
Teach the same pattern of response in different settings–>office, home, restaurants
Loose training–> teaching with a variety of stimulus items to promote generalization
Sequential Modification–> training targets in every setting generalization is desired
Programming common stimuli–> making the environment you train look like those you wish to generalize
Mediating generalization–> using mnemonic devices visualization a to train the patient to cue themselves
Training generalization–> spontaneous generalization that occurs during tx activities

22
Q

Treatment of Auditory Comprehension

A
  • Single-Word Comprehension
  • Understanding verbal sentences
    • Answering questions
    • Following verbal directions
    • sentence verification (yes/no)
    • Tasking switching drills: point to___, pick up___, which one do you write with? We’re you born in Fresno?
  • Comprehension of Conversation: higher level
    • yes/no questions
    • retelling a conversation
    • open ended questions
    • *results for adults (book for aphasia)
23
Q

Treatment of reading comprehension

A
  • First acquire a literacy history
  • May start with survival reading skills (maps, signs, medicine labels, addresses)
  • Treating mild to moderate reading impairments
    • printed word drills–> orally sound out words/non words, discriminate similar words, supply missing letters
    • printed sentence drills–> patient must interpret sentences with syntactically difficult (passive voice) sentences, move to paragraphs, ect.
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Treatment of speech production
- volitional speech (word level) * sentence completion tasks * word repetition * confrontation naming drills-->show picture "what is this" - Sentence production * sentence imitation drill (not complete imitation but partial) * repetition--> elaboration drill pt answers then repeats * story completion drill--> like sentence but longer * story elaboration--> verbal or with pictures, look at picture & tell story - Connected speech
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Treatment of Writing
- Survivor writing skills - Signing name on checks, forms, ect (1. Copy lets, 2. Combine) - Writing checks - Writing shopping list - writing bday card * *for patient needing more... - progress from letter to word to sentences, ect - spelling commercial available spelling help, materials, computer programs, ect.
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Personalized cueing
- in general, it is a semantically based self cueing approach. The patient generates their own cues for each target word. - indicates more lasting benefits when words are trained using self cueing vs. a cue given by clinician - see hand out
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Other specific tx activities for severe aphasia: PACE
Promoting aphasics communicative effectiveness (PACE) - concentrates on the ideas to be conveyed rather than linguistic accuracy - the clinician & client participate equally as senders & receivers. The speaker has free choice as to which modality is used to convey message - Feedback from the listener concentrates on adequacy of the message
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Semantic feature analysis
- assumes based on research about lexical retrieval that our semantic system is like a web of concepts - semantic feature analysis is a organized method of activating these semantic networks - trying to build a web of concepts by activating semantic networks features to enhance retrieval
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Barrier activities
- these are related to PACE & involve placing an opaque barrier between the client & clinician - typically, both participants have the same materials on their sides of the barrier (wall) - you give directions on where to place items you both have, boards should look the same after - messages are conveyed regarding materials or how to move them, then barriers are removed
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Melodic intonation
- the original program takes non fluent aphasic patients from intoning(singing) simple 2 syllable phrases to more than 5 syllables across tx - the patient is presented with visual cues for the target phrases & phrases are intoned with 2 pitches - the patient uses higher pitch for stressed words & lower pitch for non stressed words - also tapping the left hand along with each syllable
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Consistent yes & no responses
- make it clear to patient what the goal of this activity is - physically assist the patient with five reps of "yes" (head nods) same with "no" - request gestured "yes" response to two ambiguous questions. Do the same for no. (E.g. Is your name ___) - start with simple questions - begin tx of personal, environment, and informational questions.
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Gestures communication
- Begin training with one gesture - when it is intact, add a second - drill until patient can alternate between two gesture - add a third gestures & repeat the process until all 3 are reliable
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Communication boards
- use boards that have pictures and the written word under the picture - try to personalize the pictures - repeated training trials are needed - choose only one picture from board and drill on it - then choose one maximally different foil and request the patient to point to target item, continue to three, four, and more items - you'll probably need to do each of these steps in repeated groups of five
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Visual action therapy
- takes a patient through several steps involving object manipulation in order to train pantomimic gestures - the program begins with perception & recognition of objects - then it moves to gesturing with object in hand - then gesturing without object - **acting out what you would do with object
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aphasia defined
 Aphasia is an acquired impairment of the cognitive system for comprehending (receptive) and formulating language, leaving other cognitive capacities relatively intact (Davis, 1993).  Many times pts know what’s going on they can’t formulate the words. They also know that there is a problem.
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cognitive linguistic process
1. Cognitive process— organizing your thoughts, memories, and emotions in order to say what you want. (TBI) 2. Linguistic process—taking thoughts memories and emotions and finding the verbal symbols that follow the rules of language (aphasia) 3. Motor speech programming— selecting sensory motor programs that activate speech muscles in the correct sequence so that you can smoothly produce the verbal symbols organized in step two. (apraxia) 4. Neuromuscular execution—transmitting the sensorimotor programs through the CNS and PNS correctly so that the speech muscles move with the correct force, direction, and timing to produce spoken words. (Dysarthria)
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Blood Supply to the Brain: | The brain and meninges are supplied blood by three main arteries:
 Internal carotid—Branches from the common carotid artery and travels to the Circle of Willis.  Vertebral-basilar – branches from the subclavian arch, travels inside cervical vertebra to the level of the brainstem. Then left and right portions join to form the basilar artery, which travels to the Circle of Willis.  External carotid— also branches from the common carotid artery; supplies blood to the face and meninges
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The cerebral cortex is supplied blood by the three cerebral arteries. These branch away from the COW.
 Anterior cerebral— branches from COW, supplies the medial surface and very top of frontal lobe. It does penetrate cortex to supply blood to subcortical areas.  Middle cerebral— branches from COW, serves the insula and nearly all the lateral surface of the cerebral hemisphere.  Posterior cerebral— branches from the COW, serves the bottom of temporal lobe, occipital lobe and medial parietal lobe.
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Circle of Willis (COW):
 Is an arterial interconnection between the two internal carotid and the basilar artery  Can provide an alternate blood supply to the cortex if one of the carotid or basilar arteries are blocked  This safety value function can only occur when the blockage is BELOW the COW. The COW is only functional if the blockage is below.
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Connectionist Theory of Language
Primary visual cortex makes a basic analysis of visual sensation from the eye. The primary auditory cortex makes a basic analysis of auditory sensation from what we hear. Signal is then sent to Wernicke's area. Wernicke's has access to semantic memory which contains our knowledge of the world, word meanings, facts, and so forth. Wernicke's has access to lexicon which contains information on what words sound like (phonology) or look like. Wernickes job is to attach meanings to the words we hear and read and construct a neural cod of phonemes and graphemes and communicate it with Broca's. Broca's area then takes the neural code and turns it into a motor code than can be implemented by the speech muscles.
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Wernicke's aphasia
• Little effort needed to produce their speech. • Length of uttered phrases is normal (5-8 words) • There are few problems with either prosody or articulation o aka: Receptive Aphasia • Paraphrasias present: literal, verbal/semantic, neologistic. • Is a fluent type of aphasia • Auditory comprehension is disturbed: o Pt retains the ability to understand some single words, phrases, and sentences. • Can typically do the “hi how are you” o May understand only the first few words spoken. • So pt may have problems following directions depending on how much comprehension they have. o Even words initially understood may not be comprehended if they are retested immediately—rapid fatigue of comprehension. • So they may get the answer right the first time but then the second time they would get the answer incorrect. o They may have problems with topic switching. • Pt may still keep talking about the topic they were on because they can’t comprehend that you’ve changed the subject. • Environmental distractions decrease comprehension. o Reading is impaired and may parallel their problems with auditory comprehension. o Writing is always abnormal. There may be properly formed letters, but many words are meaningless. • Example on page 302. looks like writing and is smooth but many of the words are meaningless. Not really connected, words are there and it’s English but it’s not relevant. • Fluent aphasia with no artic errors, use the term Press of Speech (it just runs on). Because there is nothing telling them that their speech is wrong. Circumlocution (talking around the subject) • commonly won’t see a right-sided hemiparesis compared to broca’s aphasia • Empty speech that doesn’t give us a lot of details.
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Broca's aphasia
 Nonfluent, effortful verbal output  Sparse words, poor articulation possible  Most common in terms of the aphasias  Usually speaks in short phrases  Speech contains more content words compared to the number of function words, more nouns, verbs, less connection words.  Pts with broca’s seem to really look like they are working to talk (how it appears)  Naming is poor—lots of word finding problems.  Prosody and rhythm are distorted  Not really a great rhythm to their speech.  Syntax is "telegraphic.”  Auditory comprehension may be functional for ordinary conversation, but is still impaired  Reading comprehension of single words and short sentences may be functional.  Functional here means enough to do day to day simple activities.  Writing is impaired—oversized letters, poorly formed letters, many misspellings, letter omissions.  Letters aren’t formed properly, the writing mimics the speech.
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Conduction aphasia
This aphasia is characterized by:  Relatively fluent, intelligible speech  Functional comprehension  Poor repetition (HUGE when comparing the Aphasias)  May understand what they are needing to do know they are failing, better at saying what they want to say but not what you want them to say. So spontaneous speech is better than imitation.  Brain damage leading to this type of aphasia most often involves the left lower parietal lobe and upper temporal lobe.  Lower postcentral and supramarginal and the insula  Articulate Fasisculus if the white is damaged the message cannot be sent. You will understand if there is damage but you will not be able to repeat what is said.  There is disagreement over the exact location of the damage in this aphasia.  Is the damage to the subcortical white matter (i.e., arcuate fasciculus) or .....  Is the damage to the cortical areas mentioned in the prior slide?  Always a disagreement as to where the damage is. When looking at groups you can find patterns of lesions and areas that are affected. But when comparing individuals they may have lesions in different parts of their brains.  The mechanics of how this damage results in the symptoms of conduction aphasia have been explained in several ways.  The oldest is the disconnection theory, which states that damage to the arcuate fasciculus “disconnects” Wernicke’s area from Broca’s area.  Another theory states that conduction aphasia is a special type of apraxia  A third theory states that the symptoms are the result of a disturbance of phonemic memory processing.  People with conduction aphasia have a noticeable difference between their ability to name an object and point to it. 1. Pointing is easy and accurate, because of functional auditory comprehension. 2. Naming an object, however, can result in some literal paraphasias. 3. Reading can be functional for simple, day-to-day materials. 4. Writing will have some disturbances. There may be well-formed letters but lots of letter omissions and substitutions.  Tends to mimic speech, writing becomes worse when trying to copy a message and write it out.  IN general the main characteristic of this aphasia is poor repetition.  For reps multisyllabic words are more challenging.
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global aphasia
 "A severe, acquired impairment of communicative ability, which crosses all language modalities, usually with no modality substantially better than any other" (LaPointe, 1997).  In general, this type of aphasia occurs when the area of lesion covers a large portion of the cortex in the left hemisphere.  A type of aphasia that there is no one area that is better than another. A larger lesion site. The aphasia can change or evolve as a pt improves and starts to look like another type of aphasia.  Unlike what is typically seen in Broca’s, Wernicke’s and conduction aphasia, patients with global aphasia will probably show a decrease in nonverbal problem solving abilities.  When the area of damage is larger more cognitive functions are damaged. Although some pts have normal cognitive functions and do okay on nonverbal tests. Global aphasia can be subtyped as acute, evolving, or chronic. Acute global aphasia: is what may occur in the very early stages after a left hemisphere lesion. May have difficulty even answering a simple yes/no questions. A lot of jargon and paraphasias if there is any speech. Can also be nonverbal. May speak in over learned phrases. Evolving global aphasia is a term that is used to describe a patient with severe aphasia who is beginning to recover some language functions, usually found within the first week post onset. When a pt who was globally aphasic is starting to recover some language function. Able to understand, maybe answering simple yes/no questions, some verbal output. Chronic global aphasia describes a patient with a severe aphasia that is not changing. Generally, this stabilization happens from 1 week to 1 month post onset. Has had time to rehabilitate but is not showing any signs of recovering will have a chronic global aphasia.
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anomic aphasia
 This is an aphasia subtype characterized by 1. Mild comprehension deficit 2. Fluent, syntactically coherent utterances, but..... 3. Utterances filled with indefinite nouns and pronouns. 4. Many word finding difficulties  There is some controversy about whether to say that anomic aphasia is a separate aphasia subtype or that it is only a mild type of Broca’s aphasia.  Pts only have difficulty with naming. And their speech is relatively fluent unlike Broca’s Aphasia. These pts have word finding issues.  Anomic aphasia often results from the diverse set of causes. Many different areas of lesion or types of damage can result in word finding difficulties.  These pts may exhibit circumlocution  This diagnosis is for pts that have no other impairments.  Note: All individuals with aphasia will demonstrate anomia (e.g., word finding difficulties). Only a few can be diagnosed as having anomic aphasia.  Damage, unless it hits somewhere in the motor strip, you should have normal motor skills  Damage to the extrasylvian area in the left hemisphere. Can also be seen in right hemisphere strokes sometimes as well.  However, since anomic aphasia can occur after damage to most anywhere in the left hemisphere, the suggestion that it can be a mild Broca’s aphasia probably is not valid.  This type of aphasia is not as severe. Show word finding issues even when all other aspects have been recovered.  Extra Sylvian Aphasia  Conversational language fluent but relatively empty of specific content  Auditory comprehension- good  Repetition of spoken language- good  Naming- very bad (anomia= without naming)  Writing- good  Motor system usually normal