Exam 2 Flashcards

1
Q

What is known about continuity and change of ADHD symptoms overtime? (Franke)

A
  • Typically starts during childhood > follows trait like course
  • Diagnostics and intervention dependent upon dynamic age presentations of ADHD
  • Very young children > externalizing problems (hyperactive-impulsive)
  • Middle childhood > inattentive Sxs
  • Late adolescence and adulthood > inattention persists, hyperactivity declines, emotions easily aroused
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2
Q

What is Franke opinion on adult onset ADHD?

A
  • Diagnosable ADHD syndrome MAY arise in adulthood > but many likely to have undiagnosed ADHD or sub threshold ADHD in youth
  • TBI correlated w/ADHD
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3
Q

What is known about predictors of ADHD co-morbidity overtime?

A
  • Pattern of comorbidity changes substantially
  • ODD and CD most prevalent comorbid conditions
  • SUDs more of a problem during adolescence and adulthood
  • Adults > mood/anxiety, ASP, sleep disorders, somatic comorbidity
  • Children w/ADHD > ASD, Tics, learning disabilit comorbidity
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4
Q

How does ADHD treatment response differ overtime?

A
  • Tx should be multimodal > psychoeducation, medication, disorder specify psycotherapy, skills training, CBT, family/couple therapy
  • Older adults > practical and psychosocial support (daily life skills building, organizational skills, medication, support groups
  • Franke suggest access to support and Tx is significant
  • Adults > medication as primary + psychosocial support (mindfulness promising)
  • Moderate severe cases for children and adolescents > non-pharmacological + medication
  • Mild cases for children and adolescents > non-pharmacological
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5
Q

Does treatment alter trajectories of ADHD overtime?

A
  • ADHD associated w/negative academic outcomes, poor occupational outcomes, increased risk poor social relationships, risk transport accidents
  • Reviews of treated vs untreated ADHD outcomes inconsistent
  • Untreated > poorer long term outcomes
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6
Q

ADHD Prevalence

A
  • 6% in children

- 1-3% in adults

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7
Q

What are barriers to access to ADHD care? Discuss with Brofenbrenner’s ecological systems theory? (Wright)

A
  • Access to care not universal > under diagnosed, misdiagnosed?
  • Child gender, age, ethnicity, social networks, low SES, urban residence > access to care
  • AA and Latino children less likely recognized as having ADHD
  • Parental decisions influenced by views of teachers to recognize ADHD
  • Culture and language barriers more likely for non poor children
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8
Q

Why is early detection of anxiety important? (Mohatt)

A
  • Children > Most common mental illnesss, earlier age of onset, associated w/significant impairment, persist into adulthood > early Tx critical to prevent future disability and avoiding adulthood persistence
  • Early tx mitigates coping and adaptation skill deficits
  • Early tx ‘turns down the volume’ of Sxs
  • Early tx teaches parents how they may be reinforcing maladaptive BX
  • Early tx may teach children/ adolescents how to challenge anxious thoughts
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9
Q

what are the proposed mechanisms by which media affects mood and anxiety? (Hoge)

A
  • Amount of tv viewing > sleep problems
  • Content of media exposure > trauma like Sxs
  • Social comparison, dissatisfaction w/oneself > Attractive people leading exciting lives
  • Digital distraction from distressful emotions
  • Lack of social interaction > increase risk for substance abuse suicide > social anxiety dx
  • Substituted digital communication > depression
  • Constant connection to social networking > anxiety and depression
  • Cyberbullying > suicidal thoughts and self-injurious Bx
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10
Q

What do we know about social media use and anxiety mood overtime?

A
  • Associations between social networking and depression
  • Adults > depression decreases when internet use used for communicating w/friends and family but > More facebook = more satisfaction decreases over time
  • Internet overuse > avoidance of negative emotions
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11
Q

What are some interventions to reduce the impact of media use on youth well-being?

A
  • Pediatrician screening of media exposure (amount and content) > Pts w/anxiety, insomnia
  • Pediatricians guide parents > appropriate media content (ratings, reviews, plot descriptions)
  • Children prone to social anxiety > parents provide ample face to face communication opportunities, limit online communication
  • Parents implement rules about media use
  • Clinicians help youth access social resources > face to face and mediated
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12
Q

(Silverman) what gap in treatment may PCIT-ED (Parent child interactive therapy) fill?

A
  • Prevention of chronic, highly tx resistant, relapsing childhood dx
  • Suicide prevention efforts
  • Moves beyond targeting childhood depressive Sxs > teaching parents to be external emotion regulators and emotion coach
  • Only small effect sizes > CBT
  • Long term safety of medication tx > unknown and practice is on the rise
  • Capitalizes on neuroplasticity > altering of development and trajectory of illness
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13
Q

What is PCIT-ED?

A
  • Parent child interactive therapy > low risk
  • Short term
  • Targeting parent child interaction patterns
  • Emotion development skills > emotional competence, emotion regulation
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14
Q

Considering a family systems perspective and the etiology of dysregulation why do we think PCIT-ED is effective? What are the putative mechanisms?

A
  • Emotion development skills not as outcome but as mediators
  • PCIT-ED is likely effective as it correlates to attachment theory > secure attachments to parents results in intersubjective experience leading to learned emotion regulation in self, co-creation of meaning within ones life
  • Parents model to their children > boundaries, rules, hierarchies, and status quo dependent upon family system patterns > children taught by parents (previous generational patterns)
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15
Q

What is the association between severity of maltreatment and neurobiological outcomes? Timing of maltreatment? Gender effects? Persistence of neurobiological changes?

A

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16
Q

What brain regions are most affected by child maltreatment? What are the primary functions of those brain regions?

A
  • hippocampus: retrieval of memory, autobiographical memory, spatiotemporal representations of place, routes, experiences
  • Female hippocampal volume less vulnerable than male
  • Female hippocampus more affected at age 3-5 than 11-13 (with maltreatment)
  • Both females and males more affected at age 7-14 (with maltreatment)
  • amygdala: implicit emotional memory, responding to stimuli like facial expression and potential threats
  • Early exposure to maltreatment > Initial increase in amygdala volume
  • Early exposure to maltreatment > continued distress > Reduction in amygdala volume
  • Maltreatment associated > reduction of white and gray matter (prefrontal)
  • Exposure to more types of adversity > Increase the risk of critical experience of abuse at critical age
  • Prefrontal cortex: decision making, emotion regulation
17
Q

Why may the brain respond to maltreatment in this way?

A

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18
Q

What is known about the natural course of PTSD symptoms, following trauma?

A

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19
Q

How may these findings relate to DSM-5 stress/trauma related diagnostic categories?

A

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20
Q

How may these findings guide our approach to assessment and intervention?

A

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