exam 2 Flashcards

1
Q

Respiratory complications=life threatening complications

Priority

What are common nursing diagnoses?
ineffective airway clearance
impaired gas exchange
eneffective breathing pattern

A

resp complications

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2
Q

Cessation of breathing while sleeping due to upper airway obstruction
Interferes with ability to get adequate sleep
What else will this affect?
judgement, mood, weight, mental clarity (confusion, difficultly concentrating)

A

obstructive sleep apnea - upper airway

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3
Q

At least 5 obstructive events / one hour of sleep –> hypoxia & hypercapnia –> sympathetic nervous system response
What can cause obstruction?
-Reduced diameter of upper airway or continuous changes in airway
Leads to HTN, MI, CVA, death, arrhythmias, vascular disease
More prevalent in patients with CAD, CHF, metabolic syndrome, DM
-Men
-Older
-Overweight
-Structural changes (inflamed tonsils or airway)

A

osa patho

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4
Q

tachycardia, restlessness, anxious, tachypnea, sweating, cold and clammy

A

sympathetic nervous system response

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5
Q
Snoring	
Breathing cessation for 10 sec or longer, 5 episodes or more/hour, abrupt awakening, blood oxygen levels drop
Need a sleep study + clinical symptoms to diagnose
Treatments
weight loss
oral appliances
CPAP
BIPAP (more inspiration than expiration)
A

s/s and treatment of osa

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6
Q

CPAP
Positive pressure that keeps the alveoli open
Keeps the upper airway and trachea open during sleep
Patient must be able to breathe on own
BIPAP
Provides pressure support ventilation
2 different levels of positive airway pressure
Inspiratory
Expiratory
Used for severe COPD, severe sleep apnea
Hard to tolerate

A

CPAP and BIPAP

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7
Q

Monitor skin - break down & pressure ulcers
Monitor for dryness of nasal passages, nasal congestion
Education
On OSA
On cpap/bipap, oxygen
On weight loss
Avoid alcohol, sedatives

A

nursing management for cpap

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8
Q

Abnormal accumulation of fluid in the lung tissue and/or alveolar space
Due to fluid in alveoli, gas is not exchanged  hypoxemia
Manifestations
Respiratory distress
Anxiety
Frothy sputum - when air mixes with fluid leaked from aveoli, froth forms
Confusion
What will lungs sound like?
CXR-interstitial fluid
Tachycardia - sympathetic ns/ fight/flight increases heartrate
Decreased oxygen saturations

A

pulmonary edema

lower resp tract

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9
Q

Fix the underlying problem!
Oxygen-what form of oxygen may be needed?
Morphine-vasodilator, reduces preload - preload: volume in ventricles at end of diastole (diastole = resting of heart)
Vasodilators - dilate and reflex
Inotropic medications-increase contractility and this increases cardiac output
Diuretics

A

pulmonary edema treatment

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10
Q
Inflammation of the pleurae
Caused from
Pneumonia, URI, TB, chest trauma, PE, cancer
Severe, sharp pain worse on inspiration
Patients will often hold their breath or breath shallow to decrease the pain
Treat underlying cause
Analgesics
Anti-inflammatory medication
Steroids
A

Pleurisy

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11
Q

Pleural effusion-collection of fluid in the pleural space
Complication of
Heart failure - due to poor pump –> backflow
TB
Pneumonia
Viral infections
PE
Tumors
Empyema-collection of pus like fluid within the pleural space (this is a type of pleural effusion)

A

pleural effusion and empyema

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12
Q

Can be clear fluid, bloody fluid, purulent fluid
The size of the effusion will determine the severity of symptoms
Dyspnea
Fever, chills, pain, cough
Patient will have decreased or absent breath sounds over the effusion
Tracheal deviation
Hypoxemia
Hypotension
Tachycardia

A

pleural effusion

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13
Q
Must find underlying cause of effusion
Relieve dyspnea
Thoracentesis - needle in chest 
-Removes fluid
-Ultrasound
-Blind
-- need to assess BP due to fast removal of fluid 
Chest tube
Surgery
What is the nurse’s role? Baseline assessments, immobilize, keep still, sit up, document fluid appearance/amount, set up equipment
A

pleural effusion management

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14
Q

Sudden life-threatening deterioration of gas exchange
PaO2 <50 mmHg on RA
PaCO2 >50 mmHg, pH <7.35
Ventilation/perfusion mismatch: area in lung that receives blood flow or no oxygen or oxygen with no blood flow
*respiratory failure increases CO2, ven/perfus mismatch

A

acute respiratory failure

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15
Q
Manifestations
Restlessness
Fatigue
Dyspnea
Headache
Tachycardia
Tachypnea
Cyanosis
Management
Intubation - for vent/perfus mismatch - give high concentration of oxygen 
Mechanical ventilation
Assess patient: what focused assessment is PRIORITY? - respiratory: work of breathing
Monitor O2 sats continuously
Assess vital signs
Assess underlying event
A

acute respiratory failure

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16
Q

Obstruction of the pulmonary artery or one of it’s branches by a thrombus

Starts somewhere in the venous system or the right side of the heart

3rd leading cause of death in hospitalized patients. WHY? due to being immobile. Puts you at risk for DVT and PE so use SCD’s

What leads to PE?
Trauma
Arrhythmia (atrial fibrillation): irregular heart rate: ventricles pump, atria quivers, doesn’t pump like they should and blood pools - admin anticoagulant
Surgery
Pregnancy
Heart failure - inadequate pump pumping good blood flow –> risk for clots
Prolonged immobility

A

pulmonary embolism (PE)

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17
Q

Thrombus obstructs pulmonary artery or branch –> decreases alveolar dead space –> impaired gas exchange

Clot causes vasoconstriction in surrounding bronchioles and blood vessels –> surfactant decreases –> atelectasis and hypoxemia

Increased pulmonary artery pressures increase workload on heart –> right ventricle fails –> decreased cardiac output –> hemodynamic instability

A

pulmonary embolism (PE) pathophys

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18
Q
Manifestations
Acute dyspnea
Chest pain
Cough
Hemoptysis - coughing up blood/sputum
Palpitations
Tachypnea
Crackles
Tachycardia
Often associated with DVT….so what other physical assessment will you be looking for? - came from somewhere so check legs for swelling, redness, warmth in calf
*severity S/S depends on size/location of embolism
A

pulmonary embolism

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19
Q
Chest X-ray
ABGs
Doppler studies
Spiral or contrast angiogram CT scan (contrast: 1. pt with kidney failure can't do this b/c it goes through kidneys 2. allergy to contrast dye/shell fish)
What patients cannot have CT scan?
D Dimer to detect clotting
A

pulmonary embolism (PE) diagnostic tests

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20
Q
Must prevent DVT!
Leg exercises
Ambulation
TEDS/SCDs
Look at pg. 304 table 10-3
Meds
Heparin SQ
Enoxaparin
Arixtra
A

prevention of PE

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21
Q

This is an emergency! So give Oxygen and Treat hypotension (due to decreased cardiac output)
-IVF, vasopressors, inotropic medications (give fluids)
Monitor EKG
Heparin-IV: look at PTT to ensure PTT is therapeutic. If it is then they’re blood is where we want it to be
Thrombolytics-if hemodynamically compromised
-Alteplase (TPA: clot buster; give if pt is hemodynamically compromised: low BP, increased heart rate)
Inferior vena cava (IVC) filters

A

management of PE

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22
Q
Assess
Control pain
Manage oxygen
Relieve anxiety
Monitor vitals
Monitor for complications

PE: potential complication is right side heart failure - look and prepare for this

A

nursing management of PE

ADPIE and ABCs

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23
Q

Either parietal or visceral pleura punctured positive pressure enters pleural space  lung collapses

Three types
Simple
Traumatic
Tension

A

Pneumothorax

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24
Q

Also called spontaneous
Can happen in ‘healthy’ people
Also happens with emphysema or pulmonary fibrosis

A

simple pneumothorax

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25
Q

Air escapes from a laceration in the lung and enters pleural space or when air enters the pleural space through a wound in the chest wall
Blunt trauma
Penetrating trauma
Diaphragm tear
Lung biopsy
Insertion of subclavian line - they could have nicked it
Barotrauma from mechanical ventilation pressure on chest
Often will result in hemothorax (blood fills pleural space)

A

traumatic pneumothorax and reasons for it

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26
Q

Tension pneumothorax-air into pleural space that cannot escape. This will create a build up of positive pressure and will decrease the venous return to the heart
This is life threatening! Both respiratory and circulatory issues will occur.
Patient will experience immediate shortness of breath
Cardiac output will decrease and cardiac arrest can occur
- PEA: pulseless electrical activity: on heart monitor it looks good, but when you assess them they’re dead (no pump or squeeze)
Trachea will shift toward unaffected side-late sign
What is this called?

A

Tension pneumothorax

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27
Q
Pain
Shortness of breath
Respiratory distress
Anxiety
Increased use of accessory muscles
Tracheal shift (tension pneumo)
Decreased breath sounds/absent breath sounds on affected side
Cyanosis
A

pneumothorax manifestations

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28
Q

Chest tube
Needle decompression - emergency needle decompression: blind stick between ribs into pleural space to drain and allow pressure back in

Nursing management
Respiratory assessment
Oxygen assessment
Assess tracheal alignment

A

pneumothorax management

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29
Q

Chest trauma
-MVA
Often leads to respiratory issues
Often results in tension pneumothorax

A

chest trauma

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30
Q

Used to drain fluid or air
Restores negative pressure needed to re-expand the lung
Suction-will generally see dry suction used at CHI
Gravity
Tidaling: increase with inspiration and decrease with expiration
-No tidaling means 1. lung rexpanded/fixed so take chest xray to verify 2. chest tube stuck on wall so xray to verify

A

chest tubes

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31
Q

Subcutaneous emphysema-air enters the tissues under the skin
Crackling “rice krispies” - can feel/see if it’s bad enough - edema is affected airway or xray
Will spontaneously resolve when air leak stopped

A

chest tube - subcutaneous emphysema

crepetis and subcutaneous air all mean it too

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32
Q

Monitor pain level
Assess respiratory status
Assess oxygen levels
Assess chest tube! look for crepitus, air leak, drainage
Monitor dressing for bleeding and s/s of infection
Monitor for crepitus
Assess insertion site of chest tube
Document! What should you document? All that and is it to gravity/suction, tidaling, location, drainage
What if it accidentally comes out? Cover with occlusive dressing*
Maintain tube patency
Keep below chest level
Do not kink or clamp without MD order
Why? never clamp/kink it due to positive pressure and backflow

A

chest tube nursing management

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33
Q

Fluid and electrolytes for intracellular space

A

2/3rd body fluid
increase concentration of potassium, magnesium and phosphorus
inside the cells

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34
Q

fluid and electrolyte extracellular space

A
  1. intravascular: fluid within blood vessels
    - increased: increase BP and heart rate
    - decreased: decreased BP and heart rate
  2. Interstitial: surrounds cells
    - increased: edema, crackles
    - decreased: dehydration
35
Q

concentration of solutes
Increase: blood increases, body released adh and urine gets concentrated
decrease: blood decreases, suppresses adh and urine is more dilute

A

osmolality

36
Q

movement of water caused by a concentration gradient
lower solute concentration to area of increased solute
equalize solutes of concentration

A

osmosis

37
Q

natural tendency of a substance to move from an area of higher concentration to one of lower concentration
ex: exchange between CO2 and O2

A

diffusion

38
Q

the separation of a liquid from the undissolved particles floating in it
Kidneys

A

filtration

39
Q

located in the cell membrane and actively moves sodium from the cell into the ECF

A

sodium potassium pump

40
Q

movement of molecules across a cell membrane from a region of lower concentration to higher concentration in the direction against a concentration gradient

A

active transport movement

41
Q

measures kidneys ability to excrete/conserve water

  1. 010-1.025
    - larger volume of urine - lower specific gravity
A

urine specific gravity

42
Q

end product of metabolism of protein
10-20
increase due to dehydration, fever, sepsis, diet

A

BUN

43
Q

end product of muscle metabolism
0.7-1.4
best indicator of kidney function

A

creatinine

44
Q
measure volume percentage of RBC in blood
males: 42-52%
females: 35-47%
increase when dehydrated
fluid overload --> decrease
A

hematocrit

45
Q

assess volume status
200 meq/24 hrs
changes with no intake and change in fluid volume
used to diagnose hyponatremia and acute renal failure

A

urine sodium

46
Q

Fluid and electrolyte balance
Functions:
Regulation of volume & osmolality
-decrease in kidney function, volume in body goes up and osmolality goes down
Regulation of electrolyte levels in the ECF
Regulation of pH of the ECF
Excretion of metabolic wastes and toxic substances

A

kidneys

47
Q

Remove approximately 400ml of water daily via exhalation

Acid-base balance

What conditions increase the loss of water from lungs? coughing, rapid respirations

What conditions decrease the loss of water from lungs? Humidity - will decrease water loss

A

lungs

48
Q
posterior pituitary gland
body conserves water
regulates volume and osmolality by conservation of H20
Increased=volume increase
Decreased=volume decrease
A

pituitary: adh

49
Q

Increased secretion: sodium retention & water retention
Decreased secretion: sodium & water loss; K+ retention
regulates Na and K values and regulates BP and fluid balance

A

adrenal: aldosterone

50
Q

Renin-enzyme that converts angiotensinogen to angiotensin I
Angiotensin-converting enzyme (ACE) converts angiotensin I to II.
Angiotensin II-vasoconstrictor (increases arterial perfusion)
Aldosterone released when SNS activated because of increased renin in body
Aldosterone-regulates volume and will increase reabsorption of sodium & water-increasing plasma volume

A

renin angiotensin aldosterone system

51
Q
Expand the ECF volume
Expand intravascular space
Use cautiously in patients with	
Hypertension
Heart failure
Types
D5W-limited ability to expand intravascular volume - treat hypernatremia
0.9% Normal saline-remains in ECF
Lactated Ringers-contains K+, Ca++ - fluid replacement: does have electrolytes so use for GI distress
A

isotonic

52
Q

Provide Na, Cl, and free water
Treat hypernatremia

Types
0.45% NS
Use cautiously-can lead to intravascular fluid depletion due to a fluid shift

A

hypotonic

53
Q

Increase ECF volume
Critical situations-hyponatremia

Administer slowly-can cause volume overload and pulmonary edema
Assess respiratory and BP

Types
3% NS
5% NS
Both are only administered in intensive care

A

hypertonic s

54
Q
Loss of ECF volume exceeds the intake of fluid
Water and electrolytes are both lost at the same proportion
Causes
Inadequate intake
Vomiting
Diarrhea
GI suctioning
Sweating
Diabetes Insipidus
Hemorrhage
Third-space shifts
A

hypovolemia or fluid volume deficit (FVD)

55
Q
Rapid weight loss
Decreased skin turgor
Oliguria (low urine output)
Concentrated urine
Postural hypotension (low bp when stand)
Tachycardia
Cool, clammy skin, dry mucous membranes
Altered LOC
BUN increased not in proportion to creatinine 
Hematocrit increased (rbc get suspended and hemoconcentrated) 
Electrolytes: Sodium and Potassium
Urine specific gravity increased
Urine osmolality increased
A

manifestations of hypovolemia

56
Q

Oral fluids

IV fluids
Isotonic (0.9% NS or lactated ringers)
Rate & volume based on volume loss & hemodynamics
Assessment
I&O
What will the output be? decreased <30 ml/hour
What will the urine look like? very concentrated
Weights
Vital signs
Skin turgor
Mental status

Prevention

A

nursing management of hypovolemia

57
Q

Expansion of the ECF
abnormal retention of sodium and water

Causes
fluid overload
heart failure
renal failure
cirrhosis: liver issue, decrease albumin and unable to excrete urine
excessive salt intake
A

hypervolmia

58
Q
Edema
JVD
Crackles in lungs
Tachycardia, bounding pulse, S3 heart sounds
Hypertension
Increased weight
Increased urine output
Shortness of breath* very important 
BUN decreased
Hematocrit decreased
Urine sodium-increased
Chest X-ray-pulmonary congestion
A

manifestations of hypervolemia

59
Q

Diuretics
Hemodialysis
Sodium restriction and/or Fluid restriction

Assessment
I &amp; O
Weight
Lung sounds
Edema

Prevention
Teaching: fluid/na restriction

A

nursing management of hypervolemia

60
Q

keeps water in extracellular space

135-145

A

sodium

61
Q
Salt loss that is greater than water loss
Causes
Diarrhea
Diuretics
NG tube suctioning
manifestations
-poor skin turgor
-dry mucousa
-headache
-orthostatic hypotension
-nausea
A

cause of hyponatremia

true salt loss / fluid volume deficit

62
Q
Excess of water relative to total body sodium
Causes
CHF
Cirrhosis
Excessive H2O intake
manifestations
-edema
-crackles
-ascites
-JVD
A

cause of hyponatremia

too much water / fluid volume excess

63
Q
Neurological (especially when NA <115)
Altered mental status
Headache
Lethargy
Seizures *
Coma 

Due to water shifting from the ECF into the cell  increased ICF volume  cerebral edema

A

manifestations of hyponatremia

64
Q

why are they?
-true sodium loss = give sodium in diet
-fluid overload = fluid restriction
neurologic symptoms? critically low; give 3%/5% NaCl slowly to prevent volume OD and monitor lung sounds
edema? sodium and fluids are restricted
early detection? I/O, daily weight, assessments, especially confusion with elder, monitor labs

A

nursing management of hyponatremia

65
Q

More sodium than water

Causes
Dehydration
Enteral feeding with no water flushes
Diarrhea
Burns 
manifestations
water shifts from ICS to ESC and leaves cell dehydrated, give hypotonic to make cell swell and decrease NA levels
neurologic S/S: restless, weak, hallucinations in severe
increased deep tendon reflexes 
thirst = first sign
A

hypernatremia

66
Q
excreted via kidneys
3.5-5
98% is in cells
80% excreted via kidney, 20% excreted in stool
poor renal function = trouble excreting
A

potassium

67
Q

cause:
diuretics: especially Thiazide
diarrhea
vomiting
GI suction
NPO: poor nutrition (alcoholics/elderly) = low potassium
insulin: pushes potassium in ICF cell, not to the blood

A

hypokalemia

68
Q
S/S:
Fatigue
Anorexia
Nausea
Vomiting
Muscle weakness
Leg cramps
Arrhythmias*
Decreased tendon reflexes

Replacement

  • Oral
  • IV….be careful b/c burns/hurts at site
    • worse pain in PIH, less in Central
  • Diet: bananas, leafy greens, milk, meats, pickle juice

Watch kidney function*

Assessment!
Cardiac-monitor for arrhythmias
Musculoskeletal-monitor for weakness

A

management of hypokalemia

69
Q

Kidney function
Cardiac arrest

Causes
K+ sparing diuretics: Spironalactone (K paring)
Renal failure: dialysis to help excrete
Acidosis: move K out of cell into ECF and cause increase serum
-S/S: cardiac disturbances so watch EKG

A

hyperkalemia

70
Q

S/S:
-cardiac disturbances

Restricting diet and K+ meds
Meds
Kayexalate: binds K in bowel and pts excrete it in the stool
Calcium Gluconate: regulates cardiac muscles –> lowers effects of hyperkalemia
Sodium Bicarbonate: give to acidosis pt: makes blood alkaline and pushes K back into cell
Regular Insulin: push K intracellular to lower K levels
Diuretics
Dialysis
Assessment
Cardiac
Muscle weakness
Labs

A

management of hyperkalemia

71
Q

Transmits nerve impulses
Regulates muscle contraction and relaxation
Absorbed from foods, excreted in feces & urine
Inverse relationship with Phosphorous (so if Ph is up, Ca is down)
8.5-10.5

A

calcium

72
Q
causes:
Inadequate calcium intake
Acute pancreatitis
Medications
Decrease in Vitamin D
High phosphorous levels

S/S:
Tetany: muscle spasm, tingle on mouth, finger tips and feet. monitor through these tests
-Trousseau’s sign: pump BP all the way, fingers invert
-Chvostek’s sign: tap on facial nerve, twitch
-Chvostek’s Sign and Trousseau’s Sign due to Postoperative Acquired Hypoparathyroidism - YouTube
Seizures: b/c irritability of CNS and peripheral nerves
EKG changes

A

management of hypocalcemia

73
Q
IV Calcium-slow (calcium gluconate to increase absorption of ca)
Vitamin D
Diet
Seizure precautions
Cardiac monitoring
Calcium supplements
Teaching
Encourage exercise to strengthen bones
A

manage hypocalcemia

74
Q

causes:
malignancy: 20% of pts with cancer have increases Ca –> tumor cells secrete PTH and that increases Ca
hyperparathyroidism
immbolization

A

hypercalcemia

75
Q

S/S
Muscle weakness: lower tone in smooth muscle
Incoordination
Constipation, anorexia, nausea, vomiting *
Hypertension
Neurologic effects when Ca >16 (slurred speech, impaired memory, confusion, lethargy)

Treat underlying cause (tumor --> chemo)
IVF to dilute calcium
Furosemide: excrete ca
Calcitonin: lower Ca level by increase urinary excetion and helps bones reabsorb 
Dialysis
Increase mobility
Encourage fluids
Encourage fiber: help constipation 
Monitor for EKG changes
A

s/s management of hypercalcemia

76
Q

1.3-2.3mEq/L, excreted by kidneys
Neuromuscular function
Cardiovascular system
-vasodilation: increase in mag and decrease in BP, causes alcohol withdrawal in hypo magnesium

A

magnesium

77
Q
cause:
alcohol withdrawal: 
NG suction, diarrhea
Tube feed
DKA: acidosis push mag back into cell and mag levels decrease
s/s
Muscle weakness
Tremors
Seizures
Apathy
Depression
Agitation
EKG changes- torsades de pointes, pvc’
NM
diet, oral pills, IV slow
assess swallow r/t muscle weakness, DTR (be hyperactive DTR) and teach
A

hypomagnesaemia

78
Q
cause: renal failure b/c it's excreted in the kidneys and DKA
s/s
Hypotension
Nausea, vomiting, weakness
Lethargy dysarthria
Loss of deep tendon reflexes
Paralysis 
Respiratory depression, cardiac arrest when severely elevated
nm:
Discontinue all magnesium administrations
Loop diuretics if renal function allows
Dialysis

Assess vitals: BP
Assess cardiac & respiratory status: EKG continuous telemetry
Assess deep tendon reflexes: watch for loss
Assess neuro status

A

hypermagnesemia

79
Q

2.5-4.5 mg/dL
Maintenance of acid-base balance
Structural support to bones/teeth

A

phosphorus

80
Q
Causes
Anorexia
Alcoholism
Heat stroke
DKA
Chronic diarrhea
s/s
Neurologic: irritability, fatigue, weakness
muscle weakness: including respiratory
NM:
Phosphorous infusions
-*Sodium phosphorus or potassium phosphorus
Assessment
identify patient early
Prevent infection: hypo can alter grandular sites
Monitor labs
Diet
A

hypophosphatemia

81
Q
Causes
Renal failure *
Chemotherapy
s/s: like decreased calcium
seizures, lethargy(2 tests)
nm:
Phosphate binders
Loop diuretics
Dialysis 
Low phosphorus diet
Recognize s/s of hypocalcemia
Monitor urine output
A

hyperphosphatemia

82
Q

97-107, produced in stomach, inverse relationship with bicarbonate
Found in interstitial fluid, gastric juice, sweat, bile, saliva
Maintains acid-base balance

A

chloride

83
Q
Causes
GI drainage
Severe vomiting, diarrhea
Any cause of volume depletion
s/s: like hyponatremia, hypokalemia (they're lost with chloride)
hypokalemia can cause hypochloremia (watch for cardiac arrhythmias) 
nm
Normal saline
Discontinue loop diuretic
Diet
Monitor I&amp;O: excess water
Monitor ABGs: r/t acid base balance
Monitor labs
Vital signs
Diet teaching
A

hypochloremia

84
Q
Causes
Related to hypernatremia, bicarb loss, metabolic acidosis
Increased intake of chloride
Hyperparathyroidism, renal failure
s/s
Tachypnea
Weakness
Lethargy
Hypertension
Usually also has a high sodium level and fluid retention
nm
Hypotonic IV solutions
IV sodium bicarbonate
Diuretics
Restrict sodium, chloride and fluids

Monitor vitals- respiratory r/t fluid, hypertension, neurological r/t sodium
abgs, I&O
Assess respiratory, neuro and cardiac

A

hyperchloremia