Exam 2 Flashcards
Pulmonary embolism is
Risk factors
Sign of DVT
Sign of fat embolism
Blockage of pulmonary artery by thrombus
- DVT, immobility, obesity, fractured long bone, smoking, CVC, pregnancy
- swelling in one leg greater than the other (homans sign)
- petechiae rash on chest
Virchows triad includes
Causes of each
Endothelial damage - smoking, HTN, surgery, catheter
hyper-coagulability - prothrombin, cancer, chemo, oral contraceptives or HRT, pregnancy, heparin induced thrombocytopenia, dehydration
stasis - immobility, polycythemia
(must have all 3 to form a clot)
Clinical manifestations of pulmonary embolism
Respiratory
Cardiac
Respiratory #1 symptoms- dyspnea, tachypnea, tachycardia, pleuritic chest pain, hemoptysis in severe cases
- acute pulmonary HTN, JVD, systemic hypotension, abnormal heart sounds & ECG
Diagnostic studies
Which is gold standard
If patient can’t have contrast
Arterial blood gasses - PO2 always low
BNP levels go up
CT angiography; requires IV contrast (pre-op considerations)
- pulmonary angiography is gold standard; most sensitive but invasive
- use VQ scan if no contrast
Non surgical management
Nasal cannula range of O2
Venturi mask used for
Partial non-rebreather delivers
Non-rebreather uses & delivery
% of oxygen on RA & per L
NC - 1L to 6L
VM - used for COPD with valves
PNRM - flap stays on, delivers ~80%
NRM - used in emergencies, delivers 100%, reservoir bag filled 2/3 before putting on patient
- oxygen on RA is 21% , each L adds 4%. So 2L NC = 28% O2
Medication management
Heparin & goal
Alteplase tPA
After 3rd day of heparin
Interventions
Anticoagulants (heparin) PTT norm is 25-35 (goal is 1.5-2x so ~ 70 PTT)
- used for emboli 0.5-1mg at clot
- start on warfarin
- assess bleeding, measure abdominal girth, inferior vena cava filter (IVC)
Acute respiratory failure due to
Hypoxemia (PaO2 < 60) or Hypercapnia (PaCO2 > 50) PH < 7.3
Clinical manifestations of acute respiratory failure
Dyspnea interventions
Medications in ARF
1 sign of poor oxygenation is Mental status change; others (dyspnea, tachycardia, cyanosis, ABGs- hypoxemia & hypercarbia)
- oxygen therapy, CPAP or BiPAP (Bi for COPD)
- bronchodilators (Albuterol; tachy side effect), corticosteroids (solumedrol), Benzo or morphine to calm anxiety & pain
Acute respiratory distress syndrome (ARDS) what happens
Highlights
Mortality rate
Main causes
Alveoli fill with fluid & collapse ; decreased surfactant
- persisting hypoxia even with 100% O2, pulmonary infiltrates seen on x-ray “whited-out” or “ground glass”
- 60% mortality in ARDS
- due to underlying causes; #1 reason is due to sepsis - shock
Interventions
Normal lvl vs ppl w ARDS
Normal tidal volume calc
Low Tidal volume in ARDS
Side effects of treatment
PEEP (positive end expiratory pressure) treatment for ARDS
- Normal PEEP is <5, treatment for ARDS starts at >5
- weight in kg x10ml = TV
- weight in kg x4-6ml = TV in ARDS
- BP is #1 side effect of PEEP, check BP before treatment
Important points of ARDS
Purpose of proning patient in ARDS
Alveoli permeability, white-out/ground glass X-ray, PEEP treatment
- proning patient eliminates compression of the lungs by the weight of the heart (full expansion)
ARDS complications of treatment & prevention
Ventilator associated pneumonia
Barotrauma & volutrauma
Stress ulcers
Renal failure
- prevent by proper hand-washing & elevating HOB 45 degrees
- rupture of alveoli due to high pressure ; ventilate with smaller tidal volume , allow hypercapnia
- initiate early enteral feeding, anti-ulcer agents
- due to hypotension & hypoxemia or nephrotoxic drugs (vancomycin)
Endotracheal tube placement time
Before inserting tube
Intubation attempt
Verifying placement
Stabilizing tube
When is sedation contraindicated
Can only be in place for 10-14 days
- hyper-oxygenate
- cannot attempt longer than 30 seconds, less than 15 is ideal
- auscultate or check end tidal CO2 lvls (litmus strip) ; confirm with chest Xray
- mark level of tube at the incisor tooth (gum line if no teeth)
- sedation contraindicated for neuro injuries
Assist control ventilation
Synchronized intermittent mandatory ventilation
CPAP
Rate of ventilator
FiO2 is
- ventilator takes over, allows for rest
- delivers preset rate at preset tidal volume, patient initiates own breath
- cannot use CPAP unless patient is awake; only used to wean off mechanical ventilation
- typically set between 10-14 because it is going straight to lungs
- oxygen level delivered to patient, always set by doctor, determined based on patients PaO2
Peak inspiratory pressure rises (PIP)
ET tube assessment (DOPE)
If patient declining
High RR due to
High pressure due to
Sudden decrease in pressure
- increased if there is resistance in airway or pinched tubing
- dislodgement, obstruction, pneumothorax, equipment failure
- disconnect vent, suction first, bag breathe & call a code
- water in ventilator
- biting tube, mucous plug, pneumo
- vent disconnected
Subcutaneous emphysema is
Steps of extubation
Tear in trachea on intubation causes air to release under skin (bubble wrap)
- hyper-oxygenate patient, suction tube & deflate cuff ; wean off with SIMV, CPAP & T-piece