Exam 2 Flashcards

1
Q

Lameness Exam

A

o Walk

o Slow steady trot
o 2-beat symmetrical gait
o increased stress and concussion
o Smooth, flat, hard surface 

o Straight line and small circles 

o Examine moving toward and away, as well as from the side 

o May need to see during activity where lameness identified

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2
Q

Features of a Lameness Gait

A

o Consistent- pain every time the limb is used
o Regularly irregular
o Decreased stride length

o Decreased concussion

o Occurs the same every stride
o May seem inconsistent when only seen during specific activity, but should be consistent within that activity

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3
Q

Features of a Mechanical Gait

A

o Consistent

o Repeated every stride

o More irregular

o Less subtle gait alteration than a lameness gait

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4
Q

Features of a Neurologic Gait

A

o Inconsistent 

o Irregularly irregular 

o Foot flight, stride length, and concussion may vary with every stride 

o Very common 

o Easy to confuse with lameness 

o Neurologic and lame 


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5
Q

Signs of Forelimb Lameness

A

o Head nod

o Unequal concussion
(louder on sound limb)
o Shoulder drop
(lower when weight on sound leg)
o Decreased stride length


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6
Q

Define: Unequal Concussion, Head Nod, Fetlock Drop

A

Unequal Concussion
* Landing heavier on the sound leg

Head nod
* up when lame leg hits, down on the sound leg

Fetlock drop
* lower on the sound leg
* Shortened cranial phase of stride

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7
Q

Why Work in a Circle

A

o Used to exacerbate mild lameness 

o Usually makes lameness of inside leg more noticeable 

o Increased strain on lateral side of inside leg and medial side of outside leg 

o If pain is equal on both limbs no head nod 


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8
Q

Signs of Rear Limb Lameness

A

o More difficult to recognize
o Cannot tell source by how horse travels
o Unequal concussion
o Hip hike
up when lame leg hits ground
o Fetlock drop lower on sound leg
o Swinging leg in or out
o Shortened stride

o Head nod down on forelimb diagonal to lame hind limb

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9
Q

Nerve Blocks for Lameness Localization

A

o carbocaine(best), lidocaine, bupivicaine -> wait 10 mins
o Can start with any suspect joint 

o If no clues-start distally and work your way up 

o May need to perform additional blocks on another day 

o Localizes lameness to a smaller area 


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10
Q

Common Causes for Forelimb Vs Rear Limb Lameness

A

Common Forelimb Lameness
o Sore feet

o Navicular Syndrome/ Palmar Heel Pain
o Carpal or fetlock arthritis

o Tendinitis
o Suspensory desmitis

o OCD

Common Rear Limb Lameness
o Distal tarsitis

o OCD

o Suspensory desmitis
o Sore feet

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11
Q

What do these look like? Fibrotic Myopathy, Stringhalt, Shivers, Upward Fixation of Patella, Rupture Peroneus Tertius

A

Fibrotic myopathy
o Fibrous band of tissue
o Horse brings foot forward and then slaps down

Stringhalt
o Hyperflexion of one leg when walking

Shivers
o Spastic “shivering” when trying to backup

Upward fixation of patella
* Dragging toe

Ruptured peroneus tertius
* Floppy leg
* Can extend hock while flexing stifle

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12
Q

Objectives of Fracture First Aid

A

o Relieve stress, pain, and anxiety
o Preserve nerves and blood vessels
o Protect soft tissue and prevent bone penetration of the skin
o Prevent further damage to bones
o FIRST THING STABILIZE

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13
Q

Sedation for Fractures

A

Acepromazine:
* Limit use to only animals without signs of shock!
* mares and geldings to decrease ataxia of other sedatives

Butorphanol:
* Good analgesia, may cause excitement if given alone

Xylazine:
* predictable and safer in animal with shock

Detomidine:
* longer duration of action than Xylazine

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14
Q

What to do if Bone is coming out of skin on Fx

A

o CLEAN
o Clip?
o Cover in water soluble dressing & sterile bandage

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15
Q

Ideal Splint for Fracture

A

o Stabilize joint above & below
o Neutralize damaging forces
o Minimally cumbersome
o Easy to apply
o Does NOT require general anesthesia
o Economical/accessible
o PVC, oak boards, etc
o metal is not usually a good choice

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16
Q

Things to Remember about Fractures of the Phalanges & Distal Metacarpus

A

o Biomechanically dominated by the angle of the Metacarpal joint
o Principal bending focus becomes the fracture site rather than the joint
o Splint must include neutralizing both the bending forces of the Metacarpal joint as well as the fracture site

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17
Q

How to: Splint a Fracture of the Phalanges or Distal Metacarpus

A

o Don’t put joint in normal anatomical position
o Dorsal cortices of the bones should be aligned in a straight line ->
o Apply a light bandage (~1/2 inch thick) to the distal limb ->
o Tape splint (PVC or board) to dorsal aspect of distal limb from carpus to toe ->
o Splint should align dorsal cortices of bones & neutralize suspensory apparatus ->
o Apply cast material over splint

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18
Q

Important things to know about Fractures of the Mid-Forelimb

A

o medial aspect of the radius is not protected by muscle
o the skin is easily penetrated by a fracture and must be protected

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19
Q

Splint Fractures of the Mid-Forelimb

A

o Apply a Robert Jones from elbow to ground (3x diameter of limb)->
o Apply a lateral splint extended up the lateral side of the shoulder
o Tape securely to the proximal forelimb at the level of the axilla (prevents the distal limb from abducting)

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20
Q

Important things to know about Fractures Proximal to Cubital Joint

A

o Scapula, humerus and ulna are well protected by muscle -> stabilize the fracture and protect the skin
o disarms the triceps apparatus -> impossible for horse to fix elbow for weight bearing
o splint carpus in extension so horse can use limb for balance & ambulation

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21
Q

Important things to know about Fractures of the Mid & Proximal Metatarsus (Hind Limb)

A

o Splints are placed caudally and laterally over a Robert Jones bandage
o Robert Jones should be less extensive than on the forelimb to help keep the splints in place
o splints extend from the calcaneal tuber to the ground

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22
Q

Important things to know about Fractures of the Tarsus & Tibia (Hind Limb)

A

o Very difficult to adequately stabilize
o Reciprocal apparatus causes tarsus to flex and extend each time the stifle flexes and extends
o Stifle flexion causes fractures of the tibia or tarsus to override
o Splinting can minimize lower limb abduction-adduction and trauma
o Use Robert Jones with a single extended lateral splint that extends over the angle of the hock
o in proximal fractures splint should extend to the point of the hip

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23
Q

Important things to know about Fractures of the Humerus

A

o Do not need external stabilization

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24
Q

Antibiotics & Analgesia for fractures

A

Antibiotics for Open Fractures
o Penicillin + Gentocin
o Cefazolin + Gentocin

Analgesia For Fractures
o Phenylbutazone
o Butorphanol
o IM Detomidine

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25
Q

Transporting Horse w/ Fracture

A

o hay net may be best for keeping horse calm
o Space minimized to support the animal on all sides
o Forelimbs should be transported backwards
o Hindlimbs transported forward
o Head and neck should be left free or loosely tied for balance

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26
Q

Sequestrum; Pathophysiology, Clinical Signs, Diagnosis, Treatment

A

Pathophysiology
* Piece of dead bone that has become separated during the process of necrosis from the sound bone
* Most secondary to trauma and are infected
* Or develop secondary to vascular compromise of the cortex (periosteum)

Clinical Signs
* +/-lameness
* Localized heat and swelling
* Persistent drainage with tract

Diagnosis
* Radiographs at 12 days usually demonstrate separated bone
* Bone usually is not completely separated until 3 weeks

Treatment
* Surgical removal
* May be done standing in selected cases
* Fragment should be removed and the granulation bed débrided to healthy tissue
* +/- Antibiotics

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27
Q

Septic Arthritis/Tenosynovitis; Risk factors, Clinical Signs, Diagnosis

A

Risk Factors
* More common in foals w/ bacteremia/septicemia
* Standardbred
* Intra-articular injections
* Failure of passive transfer
* Systemic sepsis

Clinical Signs
* Lameness
* Transient fever
* Joint effusion

Diagnosis
* Arthrocentesis
* Usually >30,000 WBCs w/ 80% degenerative
* TP > 3
* gram stain slides
* culture
* rads for rule out & determine prognosis

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28
Q

Septic Arthritis/Tenosynovitis; Common Bacteria, Treatment, Prognosis

A

Common Bacteria
* E. coli
* Strep
* Staph (post injection or sx)
* Anaerobes

Treatment
* Broad spectrum antibiotics 2-3 wks post infection
* Joint lavage
* Hyaluronana IV or IA to reduce inflammation & DJD

Prognosis
* Better in adults than foals

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29
Q

Joint Lavage

A

o Indicated in ALL cases of septic arthritis
o Use needle, arthroscopy, or arthrotomy
o Sterile balanced electrolyte solution

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30
Q

When/How to Use Open Drainage of Joints

A

o chronic sepsis or cases that do not respond to joint lavage
o 3-5 cm arthrotomy incisions 
->
o Drain into sterile bandage which is changed 1-2x daily
o When upper joints are involved cross tie to prevent contamination

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31
Q

Regional Perfusion of Antibiotics

A

o Maximize tissue penetration of antibiotic around joint
o Use 1g Gentamicin diluted to 30-60ml
o Place tourniquet above & maybe below target site ->
o Inject antibiotics into vein distal to site or into medullary cavity

32
Q

Polymethylmethacrylated MOA

A

o Used to impregnate Antibiotic into tissue
o Antibiotic release is based on diffusion concentration gradients

33
Q

Epiphyseal Osteomyelitis; Signalment, Pathophysiology, Diagnosis

A

o Part of the polyarthritis or polyosteomyelitis syndrome in young foals
o Usually <2mo old

Pathophysiology
* Pooling of blood in the venous sinusoid at the junction of the epiphyseal bone and cartilage ->
* Sluggish blood flow encourages bacteria to lodge and establish at this site

Diagnosis
* Septic arthritis of adjacent joint
* Arthrocentesis & culture
* Radiographs now and in 7 days
* CT shows early changes
* MRIs best

34
Q

Golden Period

A

o Time it takes for bacteria to multiply 10^6 bacteria per gram of tissue

35
Q

Factors that Favor Wound Infection

A

o Contamination with feces (10^11 bacteria per gram)
o Contamination with soil (as little as 100 microorganisms per gram)
o Virulence of offending bacteria
o Reduced local defense mechanisms due to damaged blood supply, foreign body presence, or necrotic tissue

36
Q

Wound Lavage; What to use, figure out if the joint is involved

A

What to use
* balance between antimicrobial action and tissue cytotoxicity.
* 10ml Betadine 10% per liter of irrigation fluid = 0.1%
* 25ml Chlorahexidine 2% per 975 mL of irrigation fluid = 0.05%
* homemade saline = 8tsp salt/1gallon H2O or 10ml/L
* may use antibiotics but should use before closure

Is the joint involved?
* Do arthrocentesis
* Don’t go through contaminated wound

37
Q

Proud Flesh; What is it? Pathophysiology, Treatment

A

o Wounds at or below carpus & tarsus
o Normal process due to lack of skin, motion, infection
o Must eliminate underlying issues

Pathophysiology
* Fibroblasts & endothelial cells + no nerves ->
* Needed as scaffold for epithelial migration ->
* Resistant to infection

Treatment
* Trim
* Topical corticosteroids

38
Q

Management of Wounds at the Coffin Joint

A
  • Stop bleeding
  • Assess the ENTIRE horse
  • Clean wound
  • Local anesthesia
  • Assess for synovial envolvement
  • Bandage or cast
39
Q

Extensor Tender Lacerations; Clinical Signs, Treatment

A

Clinical Signs
* Knuckiling
* Quickly learn to place correctly

Treatment
* Debride aggressively
* May need splint/cast/shoe if horse is knuckling
* Rarely suture extensor tendon

40
Q

Repairing Comminution

A

o must have 6 cortices proximal and distal to fracture 

o must have 180 degrees of cortices to carry weight 

o P1 fractures must have an intact strut of bone spanning the fetlock and pastern joints for internal fixation

41
Q

Placing Lag Screws

A

o Drill glide hole (outside diameter of the screw) near cortex ->
o Drill thread hole (core diameter of the screw) ->
o Counter sink ->
o Measure ->
o Tap (create threads in far cortex) ->
o Place and tighten screw

42
Q

Locking Compression Plate

A

o Every screw = 1mm fracture compression (up to 4)

43
Q

Open Fractures & Repair

A

o Reduce survival by 50%

44
Q

Indirect Bone Healing

A

o Callus formation due to unstable mechanical environment caused by motion

45
Q

Strain & Mechanisms to Decrease Strain

A

o Strain = ratio of change in gap width over total width of gap
o More change in gap -> less good healing

Mechanisms for reduction
* Fragment end resorption increases width of gap -> decreases ratio of change -> decreases strain
* Number of fracture lines decreases strain on any individual fracture line
* Sequential formation of stiffer tissues in the fracture gap

46
Q

Worst Fractures for Healing

A

Displaced, comminuted fractures of the
* femur,
* humerus,
* proximal tibia
* proximal radius in adult horse

47
Q

Options for Correcting Angular Limb Deformities

A

o Controlled Exercise (wind swept foals)
o Corrective Shoeing (lateral or medial bending)
o Cast Application
o Growth Acceleration
o Growth Retardation
o Deformity of bones

48
Q

Names of Direction of Angular Limb Deformity

A

o ^ valgus
o v verus

49
Q

Treatment Timing for Angular Limb Deformities

A

Fetlock
 Medical – 4wks
 Surgical – 2mo

Distal tibia
 Medical – 4mo
 Surgical – 4-6mo

Distal radius
 Medical – 4-6mo
 Surgical – 6mo

50
Q

Periosteal Stripping

A

o Inverted T-shape in periosteum above physis
o Done on side hoping to accelerate growth
o Controversial

51
Q

Options for Growth Retardation

A

o Both must be CAREFULLY monitored

Bridge
 Screws above and below physis parallel to joint surface
 Retards growth across physis on that side
 Wire must get tight before change

Transphyseal
 Screw bridging physis
 Quicker than bridge

52
Q

Congenital Hyperextension; What is it? Treatment

A

o Hyperextension of fetlock
o Laxity at flexors

Treatment
 Exercise
 Shoes
 Cautious w/ bandaging (do not use rigid)

53
Q

Congenital Contracted Tendons Treatment

A
  • Analgesics
  • Oxytetracycline IV (once hydrated!)
  • Toe extension
  • Splints/casts
  • Surgery for flexor carpi ulnaris & ulnaris lateralis tendons
54
Q

Distal interphalangeal joint contracture; What? Cause, Treatment

A
  • Most common acquired Flexural Deformity
  • Coloqually called club foot

Cause
* Contracture of the deep digital flexor tendon

Treatment
* Transection of the inferior check ligament (desmotomy)
* + concurrent & repeated trimming of hoof to lower the heel

55
Q

Distal interphalangeal joint contracture; Medial Vs Lateral Approach

A

Medial
* Cosmetic

Lateral
* Easier
* Less wrap around of check ligament
* Can result in entrapment of medial palmer artery (bad)

56
Q

Osteochondrosis; Signalment, Most Common Joints, Pathogenesis, Histo Lesions

A

Signalment
* 5-25% of horses affected
* Warmbloods, thoroughbreds, QHs

Most Common Joints
* Tarsocural
* Femoral patellar
* Metacarpalphalangeal

Pathogenesis
* Genetic predisposition +
* Too much dietary energy, high phosphorous, high zinc +
* Biomechanical overload ->
* Vascular compromise to nourishing vessels of epiphyseal cartilage ->
* Failure of ossification & retained growth cartilage ->
* Retained cartilage plugs die

Histilogic Lesions
* Persistent chondrocytes in mid-late hypertrophic zone
* Failure of vascular invasion
* Osteogenesis

57
Q

OCD Lesions Vs Subchondral Bone Cysts

A

OCD
* Flap develops in areas of shear forces

Cysts
* Develop in areas of weight bearing

58
Q

OCD of Tarsocrural Joint; Signalment, Location, Symptoms, Diagnosis, Treatment

A

Signalment
* All ages but mostly <3yo

Location
* Distal intermediate ridge of tibia > lateral trochlear ridge > medial malleolus

Symptoms
* Often present w/ synovial effusion
* Lameness rare

Diagnosis
* Rads of dorsomedial plantarolateral oblique (DMPLO)

Treatment
* Surgical removal if clinical
* May recover on own

59
Q

Osteochondrosis of Femoropatellar Joint; Symptoms, Locations, Prognosis

A

Clinical Signs
* Lesions stabilize by 8mo old & show signs at 3yr old
* Often occur bilaterally
* Joint effusion
* Variable lameness

Common locations
* Lateral trochlear ridge
* Maybe medial trochlear & patella

Prognosis
* Dependent on extent of damage in trochlear groove

60
Q

Subchondral Bone Cysts; Locations, Clinical Signs, Treatment

A

Locations
* Usually medial femoral condyle
* Maybe lateral condyle or proximal tibia
* Usually bilateral

Clinical Signs
* Detected once in training
* Can result from trauma

Treatment
* Sx debridement
* Injection of cyst w/ steroids
* Rest
* Transcondylar Screw across cyst (best)

61
Q

Foot Abscess; Affected Area, Clinical Signs, Causes, Diagnosis

A

Area
 Between sensitive & insensitive laminae

Clinical Signs
 Warm
 Increased digital pulses
 Sensitive to hoof testers
 Severe lameness
 Acute onset

Causes
 Sole bruising
 Penetrating injury of foot
 “hot nail” nail from shoe in wrong place
 Migration of infection from white line

Diagnosis
 May use nerve blocks
 ID of abscess tract

62
Q

Foot Abscess; Treatment

A

 Create small hole to drain & follow tract to completion
 DO NOT expose too much solar corium

If abscess won’t open yet
* Soak in warm water & Mg Sulfate
* Poultice of Epsom salt or Mg Sulfate
* Extensive sole resection & hospital plate

Once Open
* Flush tract w/ iodine ->
* Pack w/ small amount iodine soaked gauze ->
* Soak 1-2x/day for 2d
* Check tetanus status
* NSAIDs

63
Q

Gravel; Pathophysiology, Clinical Signs, Diagnosis, Treatment

A

Pathophysiology
 Similar to hoof abscess
 Ascending infection from white line
 Usually associated w/ previous pathology of white line

Clinical Signs
 Swelling at coronary band
 Lameness
 Heat
 Pain
 Swelling
 Drainage at coronary band

Diagnosis
 Rads

Treatment
 Debridement of hoof wall
 Radical hoof wall resection

64
Q

Laminitis; Pathophysiology, Signalment, Causes, Clinical Signs, Treatment

A

Pathophysiology
 Inflammation or edema of sensitive lamellae ->
 Breakdown & degeneration of union btwn horny & sensitive laminae ->
 Rotation or sinking of P3
 Often affects front > rear

Signalment
 15% of adult horses affected at some point
 Most result in euthanasia

Causes
 Biomechanical
 Ingestion of excess grain
 Grazing of lush pasture
 Excessive exercise/concussion in unfit horse
 Endotoxemia
 Systemic Dz

Clinical Signs
 Increased digital pulses
 Warm/cold feet
 Stance: weight on hind w/ front extended

Treatment
 Stall rest
 NSAIDs, Opioids
 Foam support p[ads
 Ice boots
 Corrective trimming/shoeing
 May do DDF tenotomy but only helps some

65
Q

Lamellar Anatomy

A

 Surface areas = 1sq meter

Epidermal lamellae
* Avascular & aneural

Dermal Lamellae
* Very vascular & neural

Basement Membrane
* Tough sheet of connective tissue at the interface of the lamellar
* Receptor site for growth factors, cytokines, and adhesion molecules

66
Q

Phases of Laminitis

A

Developmental
* 24-48hrs
* You may be able to intervene but rarely seen now

Acute
* First signs of hoof pain

Chronic
* Rotation or sinking of P3

67
Q

Basics of Articular Cartilage

A

o Mostly water
o Other part is type II collagen > proteoglycan > minerals > chondrocytes/lipids
o Avascular, aneural, alymphatic
o Matrix maintenance and turnover regulated by chondrocytes (VERY slow)

68
Q

Pathophysiology of DJD

A

 Disruption of balance between degradation and synthesis of extracellular matrix ->
 Breakdown of the collagen framework ->
 Reduction in proteoglycan content and alteration of proteoglycan structure ->
 Increased water content ->
 Increased degradative enzyme activity ->
 Articular cartilage becomes softer in compression and weaker in tension ->
 DJD
 Cartilage has very limited ability to heal

69
Q

Extrinsic Repair of Articular Cartilage

A

 Partial thickness defects -> rarely completely heal, may not compromise joint function
 Full thickness defects -> repair by in growth of subchondral fibrous tissue -> not affective for normal use
 Non weight bearing heals fast than weight bearing
 Repair tissue is usually Type I collagen

70
Q

Osteoarthritis; Clinical Signs, Rads, Treatment

A

Clinical Signs
o Heat
o Pain
o Lameness
o Effusion
o Crepitus
o Decreased range of motion

OA Rads
o Periarticular osteophyte formation
o Subchondral bone sclerosis
o Subchondral bone lysis
o Narrowing of the joint space
o Ankylosis

Treatment
o CANNOT REDUCE DAMAGE
o Surgery for prevention
o Adequan
o Hyaluronan
o Corticosteroids

71
Q

Adequan for Joints

A

 PSGAG
 May alter progression of DJD
 Used when extensive cartilage loss
 Chondroprotective
 Can prolong bleeding times & potentiate infection
 Give IM or IA w/ Amikacin

72
Q

Hyaluronan for Joints

A

 Hyaluronic acid
 Linear polysaccharide
 May decrease lameness
 Anti-inflammatory
 May induce production of endogenous hyaluronic acid

73
Q

Corticosteroids for OA; Use, Drugs, Adverse Effects

A

Use
* Used to treat various arthopathies
* Onset of action is immediate

Drugs
* Methylprednisolone Acetate (Depo)
* Triamcinolone Acetonide (causes laminitis?)

Adverse Effects
* High doses can damage cartilage matrix metabolism
* Steroid arthropathy
* Post injection flare
* Potentiation of infection

74
Q

Palmer Heel Pain; Signalment, Clinical Signs, Affected Areas, Diagnosis

A

Signalment
o Small feet
o Underrun heels
o Long toes
o Often QHs

Clinical Signs
o Blocks out w/ palmer digital nerve block
o Bilateral
o Chronic
o Progressive forelimb lameness

Areas that can be affected
o Navicular bone
o Navicular bursa
o Coffin joint
o P2, P3
o Impar ligament
o Deep digital flexor tendon

Diagnosis
o Ultrasound
o CT (best value)
o MRI (best image)

75
Q

Palmer Heel Pain; Treatment

A

Medical
 Correct hoof imbalances w/ trimming and/or shoeing
 Allow hoof expansion
 Short-term raise angle of hoof w/ heel wedge
 Phenylbutazone
 Steroid injection in coffin joint or navicular bursa

Surgical
 Palmer digital neurectomy (careful they may hurt themselves)
 Navicular suspensory ligament desmotomy (out of favor)
 Navicular bursoscopy