Exam 2 Flashcards
Cardiac output is
What affects stroke volume
Heart rate x stroke volume
Preload - amount of blood return to right side
Afterload - force of resistance to pump blood
Contractility - ability of LV to contract
Causes of secondary HTN
What is normal glomerular filtration rate
Renal - fluid overload, RAAS
Adrenal/endocrine - regulates HR
Contraceptives, pregnancy, alcohol/drugs
Dyslipidemia - high cholesterol (plaq)
125ml/min. < 60 is risk for HTN
Chronic HTN can lead to
How to treat HTN
Retinal damage, renal damage, MI, cardiac hypertrophy, stroke
ACE inhibitors (-pril) & ARBs block angiotensin & protect kidneys
Diuretics (furosemide/spironolact)
Calcium channel block (amlodipine/diltiazem)
Deep vein thrombosis typically occur
Biggest problem of DVT
Virchows triad for etiology includes
-In lower extremities where blood flow is slow
-Thrombus dislodges & travels to heart resulting in pulmonary embolus
-Hypercoagulability, vascular damage, circulatory stasis
Management of DVT w/
What to monitor & (antidote)
Pharmacological therapy
-Anticoag therapy : heparin (SQ - prophylaxis / IV - existing problem)
-Monitor platelets, PTT (protamine sulf)
-Coumadin (initiate w heparin/ monitor PT INR/ vit K antidote)
Thrombolytic therapy to break clots (alteplase)
Varicose veins are due to
Diagnose with
Treatment options
-Prolonged standing, obesity, chronic disease (valve damage)
-Trendelenburg test (symptoms subside when patient laid back)
- sclerotherapy (inject solution to obliterate vein lumen)
Stripping (pull out vein)
Clinical manifestations of venous ulcers
Management
- Shape & border irregular, typically at ankle, very wet (exudate), surrounding skin is brownish
- promote mobility, debridement, wound dressing, VAC, hyperbaric O2
Most common causes of Peripheral vascular disease
Clinical manifestations
Diagnostic findings
- obese, smokers, Hyperlipidemia
- claudication (leg pain w activity), pain at rest (severe stage), dry skin, no hair on extremities
- ankle brachial index (0.5-.95 lower = more severe)
Clinical manifestations if arterial ulcers
Can lead to
- Borders are defined & circular, ulcer use dry, deep & looks necrotic
- possible amputation
Care for amputation
Elevate stump for first 24hrs (prevent swelling)
Bandage distal to proximal & dont occlude circulation
Compress dressing
Encourage use of prosthesis when getting up
Discourage semi-Fowler for above knee
Buergers disease
Treatment
Raynauds disease
- Inflammation in arteries in veins, only occurs in smokers
- smoking cessation, calcium channel blockers, Thrombolytic, amputation
- Vasospasms in arteries of fingers & toes; due to stress, tobacco, caffeine, cold
Aneurysms are
Types include
AAA is
TAA is
Management
-Outpouching or dilation of artery
-Saccular, berry, fusiform, dissecting
- abdominal aortic aneurysm (abdominal or back pain)
- thoracic aortic aneurysm (chest wall pain)
- control BP, surgical intervention
Heart muscles possess following properties
Automaticity
Excitability
Conductivity
Contractility
- pacemaker ability; beats on its own
- responds to electrical stimulus
- each cells conducts impulses to next
- ability to contract
PQRST complex
EKG strip small square & big square
P-R interval
QRS complex
- P (atrial depo) QRS (ventricle depo, atrial repo) T (ventricle repo)
- small is .04 sec / big is 0.20 sec
- should be 3-5 small squares (0.12-0.20) prolonged can be due to AV block or digitalis
- should be 1-3 small squares (0.06-0.12) if widened can be due to premature ventricular contraction PVC
How to Calculate HR using 300 method
If R-R interval is 2 boxes apart
How to know if rhythm is regular or irregular
Only if rhythm is regular
- Divide 300 by number of big boxes between each peak (R-R interval)
- 300/2 HR is 150
- if R-R interval isn’t the same then rhythm is irregular