Exam 2 Flashcards
timeline of septal development of the heart
conception embryo through utero- delivery
whartons jelly
covers umbilical cord
contracts around vessels (Vein and 2 arteries) when temp dec (exposed room air)
acts as natural cord clamp
creates high resistance
umbilical vein- after delivery
does not transport blood- closed
inferior vena cava/ superior -after delivery
deoxygenated blood fills right atrium/ventricle
shunted to pulmonary arteries
lungs- after delivery
required for ventilation air pushes fluid inside the alveoli out fluid shunted into capillary inc 02 lvl= stim dilation arteriole dec PVR from pulmonary artery into lungs
foramen ovale -after delivery
blood from lungs enters L side heart
dec P in right side
forman ovale flap is pushed shut from pressure of the blood
happens w/in min
ductus arteriosus- after delivery
dec p in pulmonary artery creates high aortic P
sm musc constricts w/ in 02 in blood and dec in prostaglandins from removal of placenta
happens w/in hours
eventually closes completely
umbilical artery- after delivery
branch off common iliac
receives oxygenated blood from desc aorta
high 02, low prostaglandins= constriction
inc PVR- dec blood flow
eventually no blood reaches end of cord
fetal circulation pathway before birth
deoxygenated blood in trhough SVC ductus venosus/IVC to RA
mixes with oxygenated blood in the RV
sent to pulmonary artery, placenta, LA, LV, aorta
patent ductus arteriosus allows mixed blood to flow freely between pulmonary artery and aorta (deoxygenated artery blood mixes w/ oxygenated aorta blood)
Fetal circulation changes after birth
ductus arteriosus closed
ductus venosus closed
fetal circulation- characteristics- blood vessels
constricted pulmonary blood v
dilated systemic blood v w/ low resistance
fetal circulation- characteristics- patency
patent foramen ovale and ductus venosus
fetal circulation- characteristics- arteriosus
large ductus arteriosus
connects w/ aorta and brings mixed blood to lower body
neonatal circulation- blood vessels
dilated pulmonary blood v
neonatal circ- charact
vascular resistance, arterial p and systemic blood vol inc
neonatal circ- charact- ductus arteriosus and venosus
arteriosus- closes 2-5 days after birth
venosus- closes days after birth
neonatal circ- charact- foramen ovale
hole in L atria closes and blood flow reverses
peripheral vascular assessment
color- acrocyanosis (norm up to about 24h) and circumoral pulse cap refill (central and peripherally) skin activity (inc. crying) fluid status pulses (central- femoral) peripheral (brachial) should be equal when compared 2+
cardiac assessment
heart rate (assess all valves) heart rhythm adven. breath sounds heart sounds (S3 common) chest symmetry and circumference
cardiac output- rate factors
pressure
dec p= dec co
babies have high hr and low bp at birth
hr dec and bp inc w/ age
formula for minimal sbp 1-10 yrs
if assessment finding < calculated value= hypotension
2years + 70 mmHg
ex. 4 yr old
24= 8 + 70 + 78
function prostaglandin E
prevents closure patent ductus arteriosus
can also give ibuprofen
indomethacin function
closes Patent DA
inhibits prostaglandins
function of prostaglandins in heart function
prostaglandins inhibit contraction of sm- prevents closure of openings
inotrope funtion
used for heart failure
improves cardiac output, alters force of contractions
congenital heart defects- inc pul blood flow- defect types
patent ductus arteriosus (PDA)
atrial septal defect (ASD)
ventricular septal defect (VSD)
atrioventricular canal
congenital heart defects- inc pul blood flow- manif
tachypnea, freq respir infections, tachycardia, poor weight gain, heart failure
congenital heart defects- DEC pul blood flow- defect types
pulmonic stenosis
tetralogy of fallot
pulmonary atresia
tricuspid atresia
congenital heart defects- DEC pul blood flow- manif
cyanosis, polycethmia (inc number RBC, can cause clotting disorders), poor weight gain
congenital heart defects- obstruction to systemic blood flow- defect types
coarctation of the aorta
hypoplastic left heart syndrome
mitral stenosis
interrupted aortic arch
congenital heart defects- obstruction to systemic blood flow- manif
dimin. pulses, delayed cap refil, poor color, heart failure, pulm edema
congenital heart defects- mixed blood flow- defect types
transposition of the great arteries
truncus arteriosus
double outlet right ventricle
congenital heart defects- obstruction to systemic blood flow- manif
cyanosis
poor weight gain
pulm congestion
heart failure
inc pulm blood flow defect- general
deoxygen blood mixes w/ oxygenated blood
L to R shunting (high to low pressure)
usually more than one type occurs at the same time
inc pulm blood flow defect- additional manif
edema, cardiomegaly, failure to thrive
PDA- patent ductus arteriosus
inc pul blood flow
ductus arteriosus fails to close (links aorta to pulm artery)
can be asymptomatic
use indomethacin/ ibuprofen IV, surgery
ASD- atrial septal defect
inc pul blood flow atrial septal defect manif depend on size of defect can close spontaneously surgical patch blood L atrium shunts back into R
VSD- ventricular septal defect
patch placed 3-12 mo monitor for HF use meds for HF manif depend on size can close on own w/in first yr inc pul blood flow
atrioventricular canal
inc pul blood flow
bidirectional shunting possible
“endocardial cushion defect”
lifelong prophylaxis for infective endocarditis
dental health is extremely important
aortic and ventricular walls not definitively different
surgical repair w/in 1st yr life
dec pul blood flow- general
obstriction of blood to lungs (not complete)
R to L shunting
hypercyanotic spells, hypoxemia and polycethemia
pulmonic stenosis
dec pul blood flow right ventr outflow obstruction subvalvular, valvular or supravalvular lifelong endocarditis prophylaxis hf management, surgery
pulmonary atresia
dec pul blood flow undev pulmonic valve hypoxia and cyanosis if not managed PDA must remain open until surgery use prostaglandins palliative repair used before surgery
tetralogy of fallot
dec pul blood flow
need all components for diagnosis
pulmonic stenosis, right ventr hypertrophy, overriding aorta, and vsd
can also have open foramen ovale or Atrial septal defect
*most common cyanotic defect
hypercyanotic episodes
dec systemic venous flow during spells
surgical repair
tricuspid atresia
dec pulm blood flow imcomp tricuspid valve, right atrium and ventricle not connected asd present prostaglandins immed! HF meds 3-stage surgical repair
Coarctation of the aorta
obstruction to systemic blood flow
Narrowing of aorta.
High blood pressure in upper extremities and low blood pressure in lower.
Preductal/ post ductal bp important
Impaired perfusion when PDA closes. (temporary)
Surgical repair.
hypoplastic left heart syndrome
obstruction to systemic blood flow underdev left side of heart must have pda open can admin prostaglandins before birth surgical repair
aortic stenosis
obstruction to systemic blood flow
narrowing aortic valve
exercise restrictions!!!!
surgical or balloon valvuloplasty
Transposition of the great arteries:
mixed defect
Arteries connected to wrong ventricles.
Must have another defect for blood flow/O2 mixing.
Prostaglandins to keep PDA open until surgical repair.
Truncus arteriosus:
mixed defect
Single great artery with large VSD.
Activity limitations.
Manage heart failure and surgical repair.
Double-outlet right ventricle:
mixed defect
Both great arteries exit from right ventricle.
Always has VSD present (required to survive).
Manage heart failure and surgical repair.
cardiovasc disorders- nursing care
pain interventions- especially post op adequate oxygenation hydration and nutrition promote growth and dev provide emotional/psychosocial support
cardiovasc disorders- oxygenation intervention
dec CO leads to inadeq o2
admin supply o2 and monitor sat
cardiovasc disorders- hydration/nutrition intervention
monitor protein, vitamin intake
use small, freq meals
cardiovasc disorders- grwth/dev intervention
use OT/PT to meet motor milestones
encourage play/socialization w/ peer to promote psychosocial dev
Rheumatic fever- def
autoimmune, inflamm reation to group A strep
can be prevented w/ admin antib for strep pharyngitis
diagnosed using Jones criteria
Rheumatic fever- manif/ trtmnt
chorea (unpredictable movements), arthralgia (joint stiffness), fever, carditis, erythema marginatum (pink/red skin rash on trunk), subcutaneous nodules, polyarthritis, and elevated C-reactive proteins.
CRP- systemic inflammatory marker
trt- antib and anti-inflamm
prolonged hospitalizations
infective endocarditis- def and cause
infection fo endocardium (inner lining heart)
vegetations grow in valves and lining, causing dysfunction of valves
common cause- bacterial pathogens
diagnosed w/ Duke criteria
infective endocarditis- manif/ trtmnt
osler nodules and janeway lesions
erythemic nodes on tips of fingers/toes
trt- antib b4 procedures, and surgeries
oral health!! - directly r/t cardiac health
cardiomyopathy- def, types
dis heart musc resulting in defects
types- dilated and hypertrophic
dilated- ventricles enlarge
results in dec contractility and reduced CO
hypertrophic- cardiac music. thickens
cardiomyopathy- relation to obstruction
obstructions occur after hypertrophic cardiomyopathy
common cause hypertrophic cardiomyopathy
malformation syndromes
common cuase dilated cardiomyopathy
myocarditis (viral)
trt same as HF
kawaksi dis- def
acute, idiopathic
systemic vascular inflamm disorder
common in kids younger than 5
kawaksi dis- phases
lasts several weeks
acute (abrupt fever)
subacute (fever resolution)
risk for coronary aneurysm highest during subacute stage
convalescent (complete resolution)
phase can take 3 months
kawaksi dis- r/t heart dis
kawasaki dis most common cause of acquired heart dis
main- strawberry tongue, fever, erythema hands/feet, cervical lymphadenopathy
kawaksi dis- trtmnt
iv immunoglobulin G therapy
aspirin
effects of adversity during childhood
delay cognitive, language and emotional development
beginning of neurological dev
soon after conception
8 wks brain waves
neurological system dev after birth
rapidly during infant and toddler years
fontanelles and suture function
brain growth
fontan fuse after brain growth slows
skill thickness inc w/ age
pliability dec w/ age
myelination
completed at age 3
cerebral metabolism in kids
increased v adults
have large heads
neuro assessment
hx social interactions visual cranial/skull _ oral head circum measured in infants and toddlers spinal nerve motor dev reflex testing
test for vestibular function
vestibular- provides sense balance and info about body position
whisper word and observe head movement
interventions for neuro disorders
maintain hydration/ nutrition promote safety (helmet/car seat) maintain neuro function ( incl playing and social interaction) prevent/manage infection promote pain relief/comfort admin and manage meds
craniosynostosis- def
premature closure of cranial sutures simple or complex causes inc ICP/ head malformations can dampen brain growth assess sutures of infants at every appointment
craniosynostosis- trtmnt
surgery before 6 mo and post op helmet
deformational plagiocephaly (DP)- def
assymm and flattening of head from external forces
inc w/ back to sleep campaign
skull deformation right occiput more than left occiput
deformational plagiocephaly (DP)- prev/ trt
prev- infants prone 30-60 min / day to dec P on skull
trt- freq repositioning and orthotics (helmet)
microcephaly- def
abdnom small head
primary or secondary
causes- cognitive impairments
head circum 2 standard deviations below mean
microcephaly- trtmnt
supportive care
hydrocephalus- def, cause
buildup of CSF in brain
cause- inc CSF production, dec csf absorption or flow obstruction
hydrocephalus- manif
change in consciousness (comm vs not communicating)
depend on age and severity
dilated scalp veins, bulging gontanel, apnea, irritability, ha and vomiting
hydrocephalus- trtmnt
lumbar punctures (not advised if pt noncommunicating) ventriculoperitoneal shunt
ventriculoperitoneal shunt- considerations
monitor for infection may need replacement as child grows monitor head circum to assess patency position on non-op side keep HOB elevation below 30 degrees *assoc w/ hydrocephalus
meningitis- cause
aseptic (viral)
septic (bacterial)
meningitis- interventions
isolation, seiz precautions, freq neuro assessments and fever management
reyes syndrome- common cause
aspirin to children
seizure- def and classification
electrical disturb in brain
classified by location, severity, manif, freq, duration or etiology
seizure- risks, environmental factors
can cause motor, sensory and cognitive changes
some environm can be triggers- inc risk for seizure
seizure- focal v generalized
focal- one part of brain
generalized- entire brain
simple seizure
partial
have awareness/memory and conciousness
20-60 seconds