Exam Flashcards

1
Q

<p>What is borderline personality disorder? How can it be viewed through the dimensional approach? How common is it? What is its major risk?</p>

A

<p>It is characterized by volatility, instability, and impulsivity relating to oneself, the world, and others. It is characterized by anxiety, fear, and deep insecurity. It can be seen in the dimensional approach as a disturbance in the self-other relationship (lack of clear borders), primarily, with unhealthy self-self relationship as well. The pathological trait at its heart is negative affectivity, and disinhibition. It is the most commonly diagnosed personality disorder: 1.6%-5.9% prevalence. Its major risk is injurious self-harm: suicide and non-suicidal self harm.</p>

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2
Q

<p>What is it important to keep in mind when diagnosing neurocognitive disorders?</p>

A

<p>That the person show deficits that are GREATER than those typically seen at a certain age.</p>

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3
Q

<p>What is the name of the personality disorder characterized by a persistent state of suspcion and mistrust of others without sufficient justification?</p>

A

<p>paranoid personality disorder. Cluster A</p>

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4
Q

<p>What are the causes of antisocial personality disorder? How to treat it?</p>

A

<p>Remember conduct disorder: gene-environment interactions. Strong abnormalities in brain regions associated with processing social information and fear. Underarousal hypothesis: low levels of cortisol leads to sensation ands timulation seeking. Fearlessness hypothesis: higher threshold for experiencing fear. Child-parent conflicts. Best to intervene early in childhood with treatment. CBT and cognitive therapy: learning new behaviours, and correct cognitive disotrtions, recognizing signs that norms are being violated, and teaching them about consequences of violating norms.</p>

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5
Q

<p>What are the difference between acute stress disorder and post-traumatic stress disorder?</p>

A

<p>acute stress disorder is short: symptoms persist for at least 3 days but not longer than 1 month after event, rather than persisting for longer than 1 month, as in PTSD. Also, acute stress disorder requires 9 symptoms from ANY of the symptom clusters, whereas PTSD needs at least 1 symptom from EACH of its clusters.</p>

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6
Q

<p>What are the social and sociocultural risk factors (causes) of trauma/stress-related disorders?</p>

A

<p>They are not having a social support network (especially during childhood), family trauma/conflict/maltreatment, and gender (woman are 2x more likely than men to develop trauma/stress-related disorders, which they think is caused by gender roles/socialization/age of trauamtic event exposure.</p>

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7
Q

<p>What are the main parts of the brain involved in threat-response and what do they do?</p>

A

<p>The thalamus acts as the sensory relay into higher brain regions, the amygdala acts as the alarm bell/highlighter that plays a part in the fast fear response, the hippocampus which is involved in memory and learning (such as retrieving relavent memories), the PFC which is involved in interpretation and planning, and the sensorimotor cortex which is involved in coordinating motor and sense functions needed for behaviour.</p>

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8
Q

<p>How should old age be factored into diagnosis?</p>

A

<p>Presentation of certain disorders can look different in old age. There are cohort effects, such that people experiencing anxiety might describe it in terms that seem to indicate it's much milder than it really is "im concerned" or "i'm fretting". Also, there disorders like depression might have more cognitive symptoms in old age (difficulty concentrating, or thinking), and might overlap with underlying medical conditions like vascular depression, which is cauesd by a restriction of blood flow to the brain.</p>

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9
Q

<p>What is the abstinence-violation effect?</p>

A

<p>It describes the catastrophization that occurs when you violate your abstinence from a drug. Ex: "I'm terrible and I can't do it ever, so I might as well take this since it's impossible."</p>

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10
Q

<p>What are the signs of a physical dependence? What are the signs of a psychological dependence?</p>

A

<p>Physical dependence is seen in 1.) Tolerance (need more to get same "buzz") 2.) Withdrawal (physical symptoms associated with substance being out of your system). Pyschological dependence is seen in 1.) Repeated use despite significant costs 2.) Compulsion (loss of control / desperation) 3.) Returning to the substance even after abstinence / getting clean.</p>

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11
Q

<p>What is the name of the disorder characterized by gaining sexual gratification by targeting prepubescent or early pubescent children?</p>

A

<p>Pedophilic disorder</p>

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12
Q

<p>What is the name of the disorder characterized by furtively touching unsuspecting strangers?</p>

A

<p>Frotteuristic Disorder</p>

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13
Q

<p>What are impulse-control disoders? Give some examples.</p>

A

<p>They addictions, basically, but to behaviours rather than substances. Example: Gambling Disorder, Kleptomania, Pyromania, Internet Gaming Disorder. Research suggests that the etiology is similar to substance-abuse.</p>

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14
Q

<p>What is Mild Neurocognitive Disorder?</p>

A

<p>It is a NEW disorder int he DSM-5, that targets the EARLY STAGES of cognitive decline, which DO NOT YET interfere with INDEPENDENCE in practical day-to-day life.</p>

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15
Q

<p>What is reverse tolerance?</p>

A

<p>It is when some people need less and less of a substance to experience its effects. Can happen in marijuana users.</p>

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16
Q

<p>What is a paraphilia?</p>

A

<p>It describes sexual arousal/gratification that comes from socially unacceptable and/or non-normative objects, situations, or individuals.</p>

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17
Q

<p>What is the name of the disorder characterized by gaining sexual gratification through cross-dressing?</p>

A

<p>Transvestic Disorder</p>

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18
Q

<p>What are the causes of borderline personality disorder? How to treat it?</p>

A

<p>The biological causes are genetic (trait impulsivity), as well as through difference in brain structure and function (there is reduced brain volume in some regeions that are involved with emotion regulation, and high reactivity in the brain in potentially threatening situations - cyberball experiment). The psychological causes are a host of negative cognitions: I am bad. the world sux. nothing is going to change, and I can't change, etc. The social causes invovle childhood abuse and neglect. Treatments involve medication for mood (antidepressents for depression/anxiety, lithium for mood swings, and aytpical antipsychotics maybe), psychotherapy (dialectical beahviour therapy!!! super effective for suicidality, specifically. also dynamic deconstructive therapy and transference-focused psychotherapy, which help people develop a healthy and coherenet sense of self.)</p>

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19
Q

<p>What's the named of the disorder characterized by strange patterns of behaviour addressed at delusionary themes, with normal behaving in other spheres?</p>

A

<p>Delusional Disorder</p>

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20
Q

<p>What is Dialectical behaviour therapy specifically good at addressing? What steps does it use?</p>

A

<p>Suicidality and self harm rooted in poor self image. Pretreatment step: improve therapeutic relationship. Stage 1: create stability in life. Stage 2: address trauma. Stage 3: create self-respect and self-worth.</p>

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21
Q

<p>What causes neurocognitve disorders?</p>

A

<p>All sorts of medical conditions and other conditions: Alzheimers, vascular disease, prion disease, Huntington's disease, HIV infection, Substance abuse, Parkinson's disease, traumatic brain injury, frontotemporal lobar degeneration, Lewy body disease</p>

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22
Q

<p>What does the genetic perspective say about the etiology of schizophrenia?</p>

A

<p>Although no "schizophrenia" genes have been identified, several endophenotypes have been implicated. Furthermore, there is a strong genetic component, as expressed inthe prevalanece rates of schizophrenia among relatives in a family. The closer the familial relationship, the more likely there is a correlation of schizophrenia between them. Additionally, there are environmental/epigenetic causes.</p>

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23
Q

<p>What is the dimensional approach to personality disorders?</p>

A

<p>The dimensional approach says personality disorders are 1) a disturbance in the continuum of a healthy self COMBINED WITH 2) one or more pathological personality traits. The continuum of a healthy self is composed in relation to three relationships: self with self, self with others, self with world. A healthy self-world relationship is seen in: meaningful goal making consistent with identity, that is adaptive, and and self-corrective. A healthy self-self relationship is seen in: an identity that clearly distinguishes between itself, the world, and others, that extents through space and time, and that accurately reflects the truth about what one is. A health self-others relationship is seen in: interpersonal relationships that grow through two-way adaptive process that bring about the realization and flourishing of all parties, involving positive emotions and intimacy.</p>

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24
Q

<p>What is paranoid personality disorder? How can it be viewed through the dimensional approach?</p>

A

<p>It is characterized by a persistent state of suspicion and mistrust of others without sufficient justification because of the expectation of maltreatment or exploitation causing clinically significant distress or dysfunction. Dimensionally, this can be seen as a disturbance in the self-others and and potentially the self-world relationship, and involves the pathological trait of Detatchment and Negative Affectivity (suspiciousness)</p>

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25
Q

<p>What happens in the brain due to Alzheimer's Disease, and where?</p>

A

<p>There are two main problems which cause neurons to die, starting arounding the hippocampus and surrounding area: 1.) Twisted tangles of TAU (neurofibrillary tangles) inside cell eventually lead to death. TAU holds microtubules together, which provide platforms of intracellular transport, and structure to cells. These can even "infect" neighbor cells. 2.) Beta amyloid plaques (neuritic) aggregate in spaces between neurons. These are normally byproducts of metabolism that get cleaned away, but in alzheimers those enzymbes dont' do their job, eventually leading to "choking" the neurons, leading to death.</p>

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26
Q

<p>What's the difference between transgenderism and gender dysphoria?</p>

A

<p>Transgenderism involves an incongruence between one's gender and one's sex. Gender dysphoria involves an incongruence between one's gender and one's sex PLUS clinically significan distress and/or impairment.</p>

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27
Q

<p>Give an example of two depressants, stimulants, hallucinogens, dissociate anesthetics, and multi-phasic (sub. with multiple effects).</p>

A

<p>depressants: alcohol (also multiphasic, initially it's a stimulant), opioids, sedatives // stimulants: caffeine, ampehtamines, cocaine // hallucinogens: LSD, salvia, mescaline, Psilocybin // dissociative anesthetics: PCP (phenylcyclohexyl piperidine), Ketamine // Multi-phasic Substances: Nicotine/Cannabis/Inhalants/Ecstasy (MDMA), GHB (gamma hydroybutyrate)</p>

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28
Q

<p>What's the name of the disorder characterized by having at least 1 psychotic symptom for more than a day and less than a month?</p>

A

<p>Brief Psychotic disorder</p>

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29
Q

<p>What brain structure/functioning changes do we see in older adults?</p>

A

<p>Volume reduction in hippocampus and PFC, affecting memory and executive control. In terms of functioning, there is also increased default network "stuckness"—have a hard time attending to the world. Also, oldder adults show signs of compensation: they recruit more brain parts to do the same tasks compared to younger individuals.</p>

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30
Q

<p>What characterizes dependent personality disorder? How can it be viewed through the dimensional approach? What are its causes, and therefore, how is it treated?</p>

A

<p>It is characterized by extreme lack of self-confidence leading to desparate need to rely on others to take care of you, and a fear of abandonment that manifests as submissiveness. It can be seen in the dimensional approach as a disturbance in the self-other relationship, self-world, as well as the self-self relationship. The pathological trait at its root is Negative Affectivity. It is thought to be caused by learning mechanisms (rewarded for dependence, punished for independence), combined with distorted cognitions about the self. It is therefore treated through gradual development of confidence.</p>

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31
Q

<p>How is gender dysphoria displayed throughout the lifespan?</p>

A

<p>In children, it might look like differences in play behaviour: who the child plays with, and with what kind of toys (preference for one, rejection of the other). In adults, the diagnostic criteria includes internal experiences of one's gender, desire to change their gender, feelings about their relationship to the world. So, the criteria looks different across the lifespan.</p>

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32
Q

<p>What is antisocial personality disorder? How is it distinguished from psychopathy? How can it be viewed through the dimensional approach?</p>

A

<p>It is characterized by pervasive patterns of disregard for others and norms, occuring since age 15, although not diagnosable until age 18. Is is preceded by conduct disorder. It can be distinguished from psychopathy in that APD is in the DSM-5 (not psychopathy), it is characterized by behaviours (as opposed to psychopathy, which is characterized by what's going on inside a person - calousness, impulsivity, lack of empathy, etc.). It can be seen in the dimensional approach as a disturbance in the self-other relationship, primarily, with unhealthy self-self relationship as well. The pathological trait at its heart is antagonism, and disinhibition.</p>

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33
Q

<p>What do dissociative anesthetics do?</p>

A

<p>They produce hazy, dream-like states characterized by dissociation, i.e. detachment.</p>

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34
Q

<p>When/how do individuals with paraphilic disorders get treatment?</p>

A

<p>They usually don't seek treatment. If they do, it's at the behest of the court, a spound/partner, or when the person seeks treatment due to fears/anxiety of acting on their urges/impulses. Sometimes medication is used like SSRIs to reduce sexual drive, but they're not often very effective. Behaviour approaches focus on extinction or aversive conditioning, while reinforcing appropriate sexual behaviours. There's also attempts made to improve appropriate social skills. CBT is also used, and focuses on how a person think/behaves in the company of others, and aims to modify them to be adaptive.</p>

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35
Q

<p>What is the name of the disorder characterized by eating non-foods? regurgitating and rechewing food? severely avoiding/restricting certain foods? What is the name of the class of disorder these three can fit into?</p>

A

<p>Pica. Rumination Disorder. Avoidant/Restrictive Food Intake Disorder. CLASS: Feeding Disorders.</p>

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36
Q

<p>What are the psychological causes, generally, of sexual disfunctions? What about the sociocultural? What does self determination theory have to say about this?</p>

A

<p>PSYCHOLOGICAL: stress in relationships and quality/intimacy of/satisfaction with relationship // abuse, especially in childhood // perfomance anxiety // self-conscious, especially in relation to one's body - 30% of women report issues here affect their sex, with 51% report hiding parts of their body // maladaptive cognitions: prohibitive beliefs, expectancies, catastrophizing, etc. // psychological conditions/disorders: depression, anxiety, etc. SOCIOCULTURAL: nature of relationship (intimacy, care, amount of conflict/resentment), gender/cultural scripts (MALES: potency related to status in some cultures // FEMALES: sexual restraint related to status in some cultures) Self Determination Theory says that sex is most satisfactory when we fulfill the needs of AUTONOMY, COMPETENCY, and AFFILIATION.</p>

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37
Q

<p>What are the biological causes, generally, of sexual disfunctions?</p>

A

<p>Often they are related to lifestyles. Cardiovascular issues, medical conditions and some medications (cardiovascular medications or antidepressents such as SSRIs), alcohol and nicotine, obesity/lack of exercise.</p>

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38
Q

<p>What is the name of the disorder characterized by gradual cognitive decline that does interferes with daily functioning?</p>

A

<p>major neurocognitve disorder (previously labelled dementia)</p>

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39
Q

<p>What are the 4 (5) phases of the human sexual response cycle? What broadly characterizes each phase?</p>

A

<p>According to Masters & Johnson, they are 0.) The Desire Phase (not in masters & johnson's theory) 1.) EXCITEMENT Phase (Increase in bloodflow to genitals (vasocongestion), increase muscle tension throughout body (myotonia), increased sensitivity leading to pleasure. 2.) PLATEAU (decreased attention to irrelevant stimuli - the feeling that orgasm is inevitable is tightly focused on. increase in heart rate and breathing.) 3.) ORGASM (Contraction of muscles in the genitals and the rest of the body) 4.) RESOLUTION: (decrease blood flow to the genitals (reduction in vasocongestion), reduction muscle tension in genitals and rest of body (reduction in myotonia))</p>

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40
Q

<p>What happens in neurocognitive disorder due to frontotemporal lobar degeneration? What are its symptoms, and what are they associated with?</p>

A

<p>There is a loss of neuronal mass in the frontal and temporal areas. Associated with frontal losses, are changes in behaviour, personality, social skills (ex: impulsivity, apathy, loss of empathy, overeating, stereotyped behavioural patterns). Associated with temporal damage, is increasing dificulties with fluent speach or word meaning.</p>

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41
Q

<p>What is lewy body disease? What does neurocognitive disease disorder due to lewy body disease look like?</p>

A

<p>Lewy bodies are abnormal bundles of proteins found INSIDE the neuron, which leads to Lewy Body Dementia characterized by: parkinson's-like symptoms, alterations in attention and alertness (wherewithall), visual hallucinations, impaired mobility. IMPORTANTLY: language and memory usually remain intact.</p>

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42
Q

<p>What is the name of the disorder characterized by gaining sexual gratification by being humiliated, made to suffer, or made to feel helpless?</p>

A

<p>sexual masochism disorder</p>

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43
Q

<p>What characterizes Bulimia Nervosa? What causes binges? What are its risks? When does it usually develop? What distinguishes bulimia from anorexia nervosa?</p>

A

<p>Bulimia Nervosa is characterized by binge-eating (which is compulsive, i.e. subject lacks control) followed by compensatory behaviour, such as fasting/purging/extreme exercise/or laxative/diuretic use. The binges are often characterized by interpersonal stressors, such as those found in relationships. There are many physical complications, such as dehydration and electrolyte imbalances, tooth enamel erosion, salivary gland swelling. excessive laxative use can lead to odema: swelling of tissue due to fluid aggregation. It usually develops in late adolescence/early adulthood. Bulimics have normal body weight, and their bingeing episodes are actually characterized by eating A LOT of food, not just a relatively high amount of food (anorexics call their eating binges even though the quantity is relatively low).</p>

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44
Q

<p>What do all treatments that target sexual dysfunction have in common?</p>

A

<p>They all involve psychoeducation.</p>

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45
Q

<p>Describe roughly how a threatening stimulus activates the bodies HPA axis and sympathetic nervous system, and describe the regulatory mechanisms built into the response. What roles do epinephrine and cortisol play?</p>

A

<p>A threat is detected, which is processed cortically and then the amygdala fires in response. This triggers the hypothalamus to release corticotrophin releasing hormone (CRH). This, however, can be inhibited by the hippocampus if relavent memories/learning apply to the situation. The CRH tells the pituitary gland to release adrenocorticotropic hormone (ACTH) which binds to the adrenal glands on the kidneys, which then release cortisol and epinephrine. The hypothalamus can also use the sympathetic nervous system to tell the adrenal glands to release epinephrine directly, without going through the pituitary gland signalling process. Cortisol increases glucose supply, enhances the use of glucose by your brain, and suppresses immune system's inflammator response. Epinphrine also releases glucose, but acts primarily on your cardiovascular system (heart, lungs and vasculature) to prepapre for movement. There is a negative feedback loop from the adrenal glands to the pituitary gland and to the hypothalamus mediated by cortisol. This causes less ACTH and CRH to be released and REDUCES the stress reponse. There is also a positive feedback loop from the adrenal gland to the pituitary gland to release ACTH mediated by epinephrine, which INCREASES the stress response.</p>

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46
Q

<p>What is the disorder characterized by little or no sexual activity in imagination or reality, causing significant distress and/or impairment, that has been present for at least 6 months in MALES? and in females?</p>

A

<p>In MALES: Male hypoactive sexual desire disorder. // FEMALES: Female sexual interest/arousal disorder.</p>

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47
Q

<p>What role does the cortex play in addiction?</p>

A

<p>The PFC helps regulate the reward circuit (VTA-NAc), but in addiction is not able to. Additionally, the cortex plays an important role in expectancies (PFC and Orbitofrontal. The cingulate gyrus plays an important role in learning (tied closely to the limbic system, i.e. hippocampus and amygdala), emotional processing, and and memory.</p>

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48
Q

<p>What is the estimated approximate prevalence of Alzheimer's disease in 2030? What is the symtpom course?</p>

A

<p>It is expected to be 13.8%. The symptom course begins by initial memory and learning problems that worsen. You get irritability, and social withdrawal, along with aphasia, apraxia, agnosia, anomia. You also see difficulties with planning, organizaing, sequencing, or abstracting information. The early signs are often missed by professionals.</p>

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49
Q

<p>During what age range is binging most prevalent? Why is binge drinking so dangerous?</p>

A

<p>21-29, but especially 21-25. Rapid consumptoin of alcohol can lead to alcohol poisoning, which (b/c of major CNS depression), might cause you to stop breathing, cause a coma, or kill you outright.</p>

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50
Q

<p>What are the systems (loose) that respond to stress inside the mind and body?</p>

A

<p>There is the brain response, hormonal response, autonomic nervous system reponse, immune response, cognitive response, behaviour response, and emotional response.</p>

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51
Q

<p>What is male hypoactive sexual desire disorder?</p>

A

<p>Characterized by little or no sexual activity in imagination or reality, causing significant distress and/or impairment, that has been present for at least 6 months.</p>

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52
Q

<p>What is the name of the disorder characterized by inflicting suffering to gain sexual gratification?</p>

A

<p>sexual sadism disorder</p>

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53
Q

<p>What does the cognitive perspectives say about the etiology of schizophrenia? Give an important example of a difference in processing between people with schizophrenia and healthy individuals.</p>

A

<p>The cognitive perspective says that people with schizophrenia show important differences in attention, working memory, and perception measures. An example speaking to this difference is in the Charlie Chaplin mask illusion. In healthy individuals, top-down processing overrides bottom-up processing so that the mask appears concave even though it should appear convex. In people with schizophrenia, bottom-up processing dominates, so the mask appears to be concave. This shows difficiencies in top-down processing of sensory stimuli.</p>

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54
Q

<p>In the context of substance abuse, what are the levels of involvement?</p>

A

<p>Starting from the lowest: 1.) Substance use (ingestion of psychoactive substance in amounts that don't cause impairments of functioning) 2.) Intoxication (injesting enough of a psychoactive substance in order to impair judgement, cause mood disturbances, or cause dificiency in motor ability) 3.) Substance Abuse (repititive behaviour of substance use that causes significant impairments in functioning) 4.) Substance Dependence / Addiction (Compulsive substance-seeking behaviour, loss of control, distress when substance is unobtainable. Involves both a psychological and physical dependence.)</p>

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55
Q

<p>What are the 6 common themes of delusions?</p>

A

<p>Grandeur (I'm great or famous), Persecution (others are plotting against me), Control (Something else is trying to take control of me), Reference (The world secretly revolves around me), Thought Broadcasting (Other people are listening to my thoughts), Thought Withdrawal/Insertion (Someone/something is removing/inserting thoughts from my mind). The latter 3 are called "Delusions of Influence".</p>

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56
Q

<p>What is schizoid personality disorder? How can it be viewed through the dimensional approach?</p>

A

<p>It is characterized by a pervasive detatchment from social relationships, and a inhibited/restricted range of emotion, causing clinically significant distress or impairment. Individuals come off as cold, or uninterested. This involves disturbances in the self-other relationship, and the pathological trait of Detachment and Negative Affectivity (restricted affectivity).</p>

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57
Q

<p>Which researchers conducted the early work on human sexuality? Briefly describe their work.</p>

A

<p>Kiney's early work sampled people's sexual behaviour and revealed how people had sex, that women were interested, that people had sex outside of committed relationships, and that homosexuality was relatively common. This was surprising at the time. Masters & Johnson invited uninhibited people to perform sexual acts in their lab, either masturbating or with more people. They reportedly witnessed over 4000 orgasms. From this was born their theories about the sexual response cycle.</p>

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58
Q

<p>How is adaptation and allostasis though about vis-a-vis the autonomic nervous system?</p>

A

<p>Allostasis is brought about via the parasympathetic nervous system, where as adaptation is brought about via the sympathetic branch.</p>

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59
Q

<p>What kind of abnormalities do we see in the endophenotypes more likely found among schizophrenics?</p>

A

<p>We see more motor and physical anomalies, metabolic abnormalities (how cells use glucose), neuropsychological abnormalities, neuromotor abnormalities (smooth pursuit problems—how eyes track something moving), sensory processing abnormalities (EEGs are diff)</p>

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60
Q

<p>What are the ABCs of self-control strategies in substance-abuse treatment?</p>

A

<p>They are: control the ANTECEDENTS: What are my triggers and how do I avoid/remove them. control the BEHAVIOURS: don't walk by the liquor store, replace behaviours (chew gum when craving hits), buy one beer at a time, hide your matches in somewhere annoying. control the CONSEQUENCES: reward nonuse behaviour and punish use behaviour. swtich to a branch of beer/cigarrettes that aren't your favourite. Reminding yourself of the benefits/downside of the quitting/continuing.</p>

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61
Q

<p>What are the psychological causes of trauma/stress-related disorder and what are its risk-factors?</p>

A

<p>From the learning perspective, there is conditioning. The traumatic event is an unconditioned stimuli, and all the things that become triggers associated with it become conditioned stimuli. Additionally, there is negative reinforcement which can trap people in cycles of avoiding the stimuli that trigger them. There is also cognitive AND learning generlization, which can expand the triggering stimuli further. Risk factors include pre-existing conditions like anxiety or depression, negative emotions like anger/hositility, and negative cognitive styles like those seen in mood disorders (beliefs about myself, world, the future, overgeneralization, personlization, black or white thinking, etc.)</p>

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62
Q

<p>What characterizes Binge-Eating Disorder? How is it (dis)similar to bulimia? What are its risks? When does it usually develop?</p>

A

<p>Binge-eating disorder is characterized by a loss of control over ones eating behaviours leading to the injestion of large quantities of food. Unlike in bulimia, there is no compensatory behaviour. This can lead to obesity, which is where the primary health risks are, however, psychologically it can be uncomfortable due to feelings of disomfort (from being disgustingly full), shame, loss of control, embarrassment. It usually develops in late adolescence/early adulthood like in bulimia.</p>

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63
Q

<p>What are cluster A, B, C personality disorders?</p>

A

<p>Cluster A is made paranoid, schizoid, and schizotypal personality disorder. This cluster is characterized by difficulty interacting socially, and thoughts that don't line up with reality. Cluster B is on the opposite end of cluster A's limited affective expression: narcissistic, histrionic, antisocial, and borderline personality disorder. These disorder are characterized by highly emotional and unpredictable behaviours. Cluster C personality disorders are about fear and/or anxiety: avoidant, dependent, and obsessive-compulsive personality disorders.</p>

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64
Q

<p>What is a psychosis?</p>

A

<p>It is a loss of contact with reality.</p>

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65
Q

<p>What is the mesocortical pathway, and what role does it play in addiction?</p>

A

<p>They mesocortical pathway connects dopaminergic cell bodies in theVTA extensively to the cortex, especially the frontal lobe, including the PFC and the cingulate gyrus. The PFC is involved in impulse control, executive function, and decision making. The cingulate gyrus is part of the limbic system, and plays an important role in memory formation, learning, and emotional processing. The PFC in turn projects back to the VTA. It is thought that dopamine disregulation in the VTA and NAc causes the PFC to be unable to regulate dopamine release. That, combined with the increased emotionality and compulsiveness (cingulate gyrus?)</p>

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66
Q

<p>What are the biological causes of eating disorders?</p>

A

<p>Genetic: there is a relatively moderate to strong heritability, often involving genes related to serotonin and dopamin regulation. Pubertal weight gain: people that put on a lot of weight at puberty have a higher incidence of eating disorders. Disregulation is grehlin (more causes more hunger) and leptin (more causes more satiation). brain differences revolving around endogenous opiods which are involved in positive reinforcement after binges, which cause that euphoria brought about by them.</p>

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67
Q

<p>Generally, what are the psychological treatments for sexual disfunctions?</p>

A

<p>They always involve psychoeducation. They often concentrate anxiety reduction (mindfulness, tackling expectancies, CBT for distorted thinking process), relaxation technques (for genito-pelvic pain, for instance), and communication training. Therapies often invovle structured behaviour exercises that involve graded tasks that gradually increase the intensity/quality of sexual interaction. For example: for genito-pelvic pain, you might start by learning to masturbate, then work your way up to stimulation using just one finger, then something larger. For premature ejaculation, you might learn to start slow, and pay attention to how close you are to climax so you can stop and squeeze. For performance anxiety, you might begin by stipulating the sexual interaction without any performance goals, such as beginning by just touch, then just genital touch, then play, but without any goals, etc. working your way up.</p>

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68
Q

<p>What are the treatments for acute stress disorder and PTSD?</p>

A

<p>They are medication, usually SSRIs and SNRIs (which themselves are not very helpful <6%), but you might also get anxiolotics, mood stabilizers, and prazosin (reduces nightmares). Psychologically there is psychotherapy: CBT and Trauma-focused CBT, which involves psychoeducation (what is PTSD?), helping individual challenge negative cognitive styles vis-a-vis their PTSD, helping people find solutions to their practical problems to counter helplessness, as well as exposure to trauma-related stimuli, and coping techniques like mindfulness. There is also Prologned Exposure Therapy, which involves exposure to trauma-related stimuli in-person or in imagination, to create extinction of learning.</p>

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69
Q

<p>What is adjustment disorder? How can it be distinguished from other trauma/stress-related disorders?</p>

A

<p>It is characterized by severe reactions in response to a identifiable stressor within the 3 months before the onset of symptoms. The reaction is one of anxiety/depression in addition to impairing behaviour changes. Important to distinguishing this from other trauma/stress-related disorders is that it be related to a specified stressor, so that the onset corresponds to the stressor, and the symptoms go away/reduce dramatically when the consequences of the stressor go away.</p>

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70
Q

<p>What is the name of the disorder characterized by gaining sexual gratification through inanimate objects or nongenital body parts?</p>

A

<p>Fetishistic Disorder</p>

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71
Q

<p>What is the dopamine-glutamate hypothesis? What's going on in the brain in relation to this hypothesis?</p>

A

<p>Glutamate regulates dopamine activity in the brain. In the frontal cortex, normally, glutamate increases dopamine signalling, but b/c there's not enough glutamate there in schizophrenia, and b/c dopamine there increases cell firing, there's both underactivity in cell firing and dopamine. On the other hand, in the basal ganglia, glutamate still promotes cell firing, but dopamine inhibits it. Because there's not enough glutamate, there's dopamine overactivity, causing an inhibition of cell firing. So dopamin isn't doing what it's supposed to because glutamate isn't correctly regulating it.</p>

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72
Q

<p>What are the three main medication treatments for substance-abuse?</p>

A

<p>They are: AGONIST SUBSTITUTION (like methadone): this creates a similar affect, but is safer. downside: you can become addicted to this new substance. ANTAGONIST: take something that blocks the effects of the drug you're taking (ex: Naltrexone) These often act by binding to the receptor sites drugs work on. Lastly, AVERSIVE TREATMENT: this creates extinction by producing a aversive sensation/effect when the drug is ingested (ex: Disulfiram)</p>

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73
Q

<p>In the context of stress, what is adaptation, allostasis, and allostatic load?</p>

A

<p>Adaptation is the internal and behavioural response needed to address a threat/challenge that uses up resources. Allostasis describes the processes that bring about adaptation to stress, as well as those the down-regulate that adaptation when the stress no longer exists. "Allo" means "variable" and stasis means "standing still". The idea is that we make large changes to get ahead of stresses to maintain our homeostasis. Allostatic load is the wear and tear put on the body in the process of going about with allostasis, i.e. meeting the stressors of the world.</p>

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74
Q

<p>What are the causes of substance-related disorders? What about personality?</p>

A

<p>They are genetic (like gene influencing alcohol breakdown enzyme (ALDH), or those affecting dopamin receptors) and epigenetic. There are biological factors that involve the nature and function of the reward circuit (NAc), as well as dopamine activity. A MAJOR lense to see the cause of addiction is through the learning model. Think OPERANT and CLASSICAL CONDITIONING (as well as OBSERVATIONAL). Think about how cues prompt addiction, how euphoria is a positve reinforcer, how the reduce of stress/anxiety/withdrawal symptoms is a negative reinforcer. From the cognitive perspective, positive expectancies play a large role, as do maladaptive coping strategies/beliefs about the self, etc. From a personality perspective, THERE IS NO SUCH THING AS AN ADDICTIVE PERSONALITY, however, there ARE personality traits associated with an increased risk of substance-related disorders. Personality traits associated: rebeliosness, risk-taking, impulsivity. From a social and sociocultural perspective, culture plays an important roll. Family and friend attitudes are huge. Childhood maltreatment, and victimization play important roll. Also going back to observational learning, models in environment model behaviour and normalize behaviours and beliefs. Another important cause, is timing of first use - the earlier the worse. Also, the addictive potential of the substance.</p>

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75
Q

<p>What is the second most frequent cause of neurocognitive disorders after Alzheimers? What are the proximate and distal causes?</p>

A

<p>Nerucognitive disorder due to vascular diease. Atherosclerosis is a thickening of arteries that narrows them and reduces blood flow, which can lead to brain damage. This can be a one-time thing (as in a stroke, which causes numbness in the face/arm, confusion, trouble speaking, walking dizziness etc.) or a gradual thing. This ist he proximate cause. The distal cause is obviously lifestyle factors.</p>

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76
Q

<p>What cognitive ability does not decline with age? Which do?</p>

A

<p>Vocabulary basically doesn't decline with age. Perceptual speed (ex: quickly comparing patterns), Episodic Memory, Spatial Visualization (ex: rotating something in your mind), and Reasoning declines with age</p>

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77
Q

<p>What characterizes fetishistic disorder and transvestic disorder? What is common between them?</p>

A

<p>Both diagnoses require a paraphilia that has lasted at least 6 months + impairment/distress and/or risk of harm to others. Additionally, they're both involve non-human objects for sexual gratification. Fetishistic disorder is when sexual impulses are targeted at inanimate objects, or nongenital body parts (partialism) (in imagination or reality). Transvestic disorder is when sexual gratification is targeted at cross-dressing (in imagination or reality)</p>

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78
Q

<p>What are the causes of personality disorders, generally, and what does the treatment appraoch involve?</p>

A

<p>The causes are genetics (40%-60% heritable) and environemental: early abuse, upbringing. Because personality disorders are very difficult to treat, treatment must involve: structure (coordinated approach to treatment applied at many levels), treatment alliance (a secure positive relationship with a therapist/clinician/doctor), consistency (regularity to create habits and dependibility), Validation (accept as true and sensical the feelings others have, but don't necessarily endorse or support them), Motivation (create in them a desire to change), Metacognition (create in them tools to analyze their own thoughts and behaviours).</p>

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79
Q

<p>According to the DSM, what is delayed ejaculation disorder and premature ejaculation disorder? What are their prevalence rates?</p>

A

<p>Delayed ejaculation is a disorder characterized by in ability to ejaculate or long delays to achieve ejaculation which causes distress at least 3/4 of the time. DSM is not clear about what a "delay" is. Its prevalence is <1%. Premature ejaculation is a disorder characterized by a male cumming in 1 minute or less AND distress about that fact. By this strict definition, only 1-3% of men meet this criteria, but if its loosened a bit, 21-30% of men meet this criteria. For both of these these disruptions need to be persistent, and last for at least 6 months AND cause distress/impairment.</p>

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80
Q

<p>What is the major difference between 1st generation (typical/conventional) and 2nd generation (atypical) schizophrenia medication?</p>

A

<p>1st generation works by block dopamine receptors. It reduces positive symptoms, but doesn't change negative symptoms. It has severe side-effects, like tardive-dyskinesia and weight gain. It's also less expensive. 2nd generation work dopamine receptors (diff dopamamine receptors?) and other neurotransmitters. They reduce the positive AND negative symptoms, generally have fewer side-effects (but not in everyone), and are MORE expensive.</p>

81
Q

<p>What is the name of the personality disorder characterized by a persistent pattern of detachment from others and restricted affecivity? In what cluster of personality disorders is this found?</p>

A

<p>schizoid personality disorder. Cluster A</p>

82
Q

<p>What is the name of the disorder characterized by gaining sexual gratification by furtively watching unsuspecting strangers?</p>

A

<p>Voyeuristic Disorder</p>

83
Q

<p>What are the main types of non-pharmeceutical treatments for schizophrenia and what are their aims?</p>

A

<p>The two types are CBT and family interventions. In CBT, the main goal is to give schizophrenics coping tools, psycho-education, and different perspectives to help them deal. With the family internvetions, the goal is to provide practical emotional support, and support/educate the family in their struggles with the disorder.</p>

84
Q

<p>What are the adaptive benefits of short-term stress? What are the maladaptive disadvantages of long-term stress?</p>

A

<p>Short-term stress increases glucose avilability and mobilizes the metabolism to use it, increases blood pressure (so blood moves quicker through veins), increases immunity, increases vigilance, temp, diminishing of interest in sex, improves memory and cognition, and improves blood clotting. Long-term stress can cause hyperclysemia (too much glucose), hypertension, increased thickness of coronary artery walls, loss of immune function, hypervigilance (oversensitivity to threat-detection), increased focus on the negative, global loss of sex interest.</p>

85
Q

<p>What are the treatments for neurocognitive disorders?</p>

A

<p>Rehabilitation (physical, occupational, social, speech, and language therapies to help relearn new skills or compensate for dificient ones), Biological (medications, surgeries, etc. that address underlying biological issues), Physchological (psychoeducation, emotional, and cognitive approaches to improve self-understand, create more adaptive thining processes, and to address underlying emotional issues), Lifestyle (address the lifestyle changes that are causing or worsening symptoms), Environmental Support (create a safe and functional environment, eg. rails and chairlifets, as well as a safe and functional social environment, eg. support groups, aids, family-education, etc.)</p>

86
Q

<p>What are the phases of the development of schizophrenia? when do they typically develop? and are the features of each phase?</p>

A

<p>The phases are: premorbid, prodromal, psychotic, stable, and residual. The premorbid phase generally occurs in childhood, and is expressed in its cognitive, motor, and social dificiencies which are often missed. In the prodromal phase, the individual might show signs of brief less intense positive symptoms and/or a functional decline in their life. This is when people will generally start to socially withdraw, develop peculiar beliefs, show innapropriate affect, and neglect their personal grooming habits, and it typically occurs in early adolescence. At some point, the individual will have a psychotic episode, which marks the beginning of the psychotic phase. In this phase the individual will experience intense repeating episodes of full-blow psychotic symptoms. This occurs in late adolescence / early adulthood. Then comes the stable or residual phase, which is characterized by fewer positive symptoms, but lots of negative symptoms and cognitive/social/functional dificiencies occuring in early adulthood / late adolescence. Or, if they're lucky, is the residual phase. This is when symptoms are no longer prominent, and psychotic behaviour and symptoms severely decline.</p>

87
Q

<p>What is personality?</p>

A

<p>It is a consistent pattern of of inner experience and behaviours in the environment (including the interpersonal environment) that originates from an individual, and usually coellesces and stabilizes by adolescence.</p>

88
Q

<p>What are the objective indicators of sexual arousal? What are the subjective indicators of sexual arousal?</p>

A

<p>Objective indicators: blood flow in genitals, skin conductance, changes brain activity. Subjective indicators: changes in cognition, emotional processing, outward behaviour, and interpretations thereof.</p>

89
Q

<p>In the context of sexual dysfunctions, what is partialism?</p>

A

<p>It describes a situation in which a person has a fetish specifically towards a nongenital body part.</p>

90
Q

<p>What is delusional disorder? How does it differ from schizophrenia? What is required for a diagnosis? How is it treated?</p>

A

<p>Delusional disorder is characterized by a delusionary theme with accompanying strange patterns of behaviour. However, in delusional disorder, the strange patterns of thought and behaviour are limited only to the delusionary themes, rather than penetrating into all sphere of life. For a diagnosis, the delusion must kick around for at least 1 month. It is treated, like in schizophrenia, with antipsychotics and CBT.</p>

91
Q

<p>What are hallucinations?</p>

A

<p>They are the perception of non-existent stimuli through one or more sense modalities, the most common of which is hearing (hearing voices).</p>

92
Q

<p>What are the psychological causes of schizophrenia?</p>

A

<p>There are cognitive and social-cognitive problems that make people vulnerable to schizophrenia—a diathesis. For instance, there are theory of mind deficits (often seen in childhood during the premorbid phase), early cognitive deficits, and then there are negative attitudes and misattributions that don't CAUSE schizophrenic symptoms, but play a role in MAINTAINING them. ("if i show my real feelings other people will hate me" or "they're all talking and laughing about me, so I need to control my expressoin")</p>

93
Q

<p>What is the mesolimbic pathway, and what role does it play in addiction?</p>

A

<p>The mesolimbic dopamine path connects dopaminergic cell bodes in the Ventral Tegmental Area (VTA) to the limbic system: amygdala, nucleus accumbens (NAc), and the hippocampus. The effect of most drugs is to stimulate the dopamine receptors in the NAc. This can happen by blocking reuptake of DA in the NAc (like in Cocaine), acting on the DA receptors in the NAc (opiates mimic DA action in NAc), releasing more dopamine by the VTA (nicotine and cocaine), blocking inhibiting neurons in the VTA leading to release of more DA (acohol and opiates), or altering regulatory neurotransmitters causing the action of DA to be more intense (altering GABA or Glutamate in NA or VTA). The end effect of all of these is to INREASE DOPAMINE IN THE NAc.</p>

94
Q

<p>What is the name of the personality disorder characterized by self-aggrandizement and a lack of empathy? What cluster is it in?</p>

A

<p>Narcissistic personality disorder. It is in cluster B</p>

95
Q

<p>What are the psychomotor abnormalities in schizophrenia?</p>

A

<p>They are 1) Disorganized behaviour and 2) Catatonia. Catatonia is characterized by pseudo-paralysis, being mute, withdrawal/refusal from eating, staring, negatavism (refusing what is asked), rigidity, waxy flexibility, purposeless repititive movements or phrases, repeating what is said or acted out (echolalia/echopraxia)</p>

96
Q

<p>What is the name of the personality disorder characterized by a persistent pattern of odd, eccentric, or paranoid thoughts and behaviours combined with a detchment from, and dificiency in, social engagement? In what cluster of personality disorders is this found?</p>

A

<p>schizotypal personality disorder. Cluster A</p>

97
Q

<p>What is schizophreniform disorder? How is it treated? How does it differ from schizophrenia and brief psychotic disorder?</p>

A

<p>It is the presence of 2 or more psychotic symptoms for between 1 and 6 months. It's very similar to schizophrenia (similar age of onset, and anotomical and neural dificiencies), but does not require that there be negative symptoms for >6 months. This usually develops (66%) into full-blown schizophrenia. It is treated with antipsychotics and CBT.</p>

98
Q

<p>What is histrionic personality disorer? How can it be viewed through the dimensional approach?</p>

A

<p>It is characterized by attention-seeking behaviour, regardless of whether its good or bad, that manifests in dramatic emotionality. It can be seen in the dimensional approach as a disturbance in the self-other relationship, as well as the self-self relationship. The pathological trait at its root is likely Negative Affectivity.</p>

99
Q

<p>What is narcissistic personality disorer? How can it be viewed through the dimensional approach?</p>

A

<p>It is characterized by an inflated ego and ego-inflating behaviour, along with a lack of empathy/concern. It can be seen in the dimensional approach as a disturbance in the self-other relationship, as well as the self-self relationship. The pathological trait at its root is Antagonism.</p>

100
Q

<p>What is the name of the personality disorder characterized by social fear, social withdrawal, and hypersensitivity to negative social evaluation? What cluster is it in?</p>

A

<p>avoident personality disorder. It is in cluster C.</p>

101
Q

<p>What are the genetic contributors to trauma/stress-related disorders?</p>

A

<p>Genes don't contribute that much. However, remember the short-allele version of the seratonin transporter gene that was implicated (if you have two of them) in depression, is relavent here. It makes you more sensitive to stress and stressors.</p>

102
Q

<p>What is the name of the disorder characterized by modest cognitive decline that does not yet interfere with daily functioning?</p>

A

<p>mild neurocognitve disorder</p>

103
Q

<p>What are some the long-term consequences of heavy drinking?</p>

A

<p>Thiamine (Vitamin-B1) dificiency leading to Wernicke-Korsakoff syndrom (motor impairments, memory problems, horrible cognitive effects), cirrhosis of liver (scar tissue caused by liver damage), oral cancer, esophogeal cancer, tracheal cancer, and cardiomyopathy (heart muscle disease characterized by dilation and impaired contraction of one or both myocardial ventricles)</p>

104
Q

<p>What are opiodes/opiates? What effect do they have? What risks do they carry? How do they create addiction?</p>

A

<p>Opiates are naturally-derived (poppy) depressant chemicals, whereas opiods are synthesized. They produce drowsiness, euphoria, analgesia (pain-relief). Opiates create quick Tolerance. They usually create withdrawal symptoms, and even after the physical withdrawal is complete, cravings can persist for years. Also there are risks of overdose, and bloodborne illness from sharing needles. They work by preventing inhibition in the VTA, creating more DA in the NAc.</p>

105
Q

<p>What is cue-exposure training, social skills training, and motivational enchancement therapy in the context of substance-abuse?</p>

A

<p>Cue-exposure helps extinguish learning by pairing conditioned stimulie (cues) with non-use behaviours. Social skills training helps people navigate social spaces in order to maintain abstinence. For example, training in assertiveness to say no, or helpful ways of communicating in marriages to avoid conflict which creates need for alcohol. Motivational enchancement therapy targets a lack of desire to change. Abusers typically have ambivalent or negative attitudes towards change, and hence aren't self-motivating. Motivational enchancement therapy aims to change that.</p>

106
Q

<p>What is the name of the personality disorder characterized by attention-seeking behaviour that manifests in dramatic emotionality? What cluster is it in?</p>

A

<p>histrionic personality disorder. it is in cluster B</p>

107
Q

<p>What is gender identity?</p>

A

<p>It is the internal experience of one's gender.</p>

108
Q

<p>What are feeding disorders? What are the three major ones? How are they treated?</p>

A

<p>It is a disorder usually diagnosed in infancy, childhood, and adolescence characterized by a disturbance in feeding behaviour that has the risk of leading to nutritional dificiences, weight loss, or harm. The three major disorders are PICA, which is characterized by the persistent eating of non-foods, RUMINATION DISORDER, which is the repeated regurgitation of food, and AVOIDANT/RESTRICTIVE FOOD INTAKE DISORDER, characterized by a lack of interest in certain foods (colour/texture/type/ etc.) which can lead to a loss of body mass and vitamin and nutrient dificiencies. They're treated broadly in a two-step way: 1.) Correct any nutritional dificiences and 2.) Behavioural psychotherapy (thinking learning approaches with reinforcement/punishment)</p>

109
Q

<p>Generally, what are the biological treatments to sexual disfunction?</p>

A

<p>They might involve drugs (ex: viagara, cialis for erection issues, creams for genito-pelvic pain/penetration disorder: antifungal, corticosteroids, or estrogen), you might get surgery, hormose replacement, penile implans, vaccuum pumps, supposetories, SSRIs (for premature ejaculators). NOT very effective on their own, usually, or only produce temporary solutions. BEST when combined with psychotherapy.</p>

110
Q

<p>What is erectile disorder and what is its prevalence?</p>

A

<p>It characterized by problems achieving or mainting erections during sexual activity AND being distressed about it. Its prevalence is 10%.</p>

111
Q

<p>What are the biological and psychological causes/risk factors of paraphilic disorders?</p>

A

<p>Biologically, there is evidence of limbic system dysfunction, and PFC dysnfunction—emotional and impulse control. Psychological causes/risk factors may involve a fear of castration (exhibitionistic men show their dicks to prove its still there), learning (accidental associations between sexual gratification and non-target stimuli like hanging socks, for instance. Especially in juveniles, but in other sex offenders as well, there is often low self-esteem, anxiety, early exposure to sex (in pornography/witnessing sexual violence), or sexual abuse. There are also often impulse control problems: either trait impulsivity or just difficulty with impulse control in certain situations.</p>

112
Q

<p>What is schizophrenia?</p>

A

<p>It is not just a single disorder, but rather, a group of disorders that have simliar characteristics, including at least some degree of reality distortion.</p>

113
Q

<p>What is the name of the disorder characterized by an inability to orgasm, long delays to orgasm, or marked reduction in orgasm intensity in women?</p>

A

<p>female orgasmic disorder</p>

114
Q

<p>What are the major brain structure differences underlying PTSD? What about neurotransmiter differences?</p>

A

<p>The amygdala is oversensitive, and triggers threat responses when there is no real threat. The hippocampus is also over-responsive, in that it plays a part in conjuring up intense emotions and flashbacks in response to trauma stimuli. Additionaly, the PFC seems to be under-responsive. The PFC plays a role in emotional regulation and executive function. This happens because the person can't process the event/stimuli/internal situation. There is also serotonin disregulation in PTSD.</p>

115
Q

<p>What are the psychological causes of eating disorders?</p>

A

<p>body-image distortion and body-image dissatisfaction, often linked with low self-worth, and maladaptive ideas about the role of weight/looks in ones worth. also, there is learning involved (think reinforcement, observational learning). The personality trait of Perfectionism is also potentially a cause.</p>

116
Q

<p>What is vaginismus?</p>

A

<p>It is characterized by involuntary spasms/contraction or the vaginal wall and/or pelvic floor caused by penetration attempts, preventing penetration, and causing pain and potentially fear of pain.</p>

117
Q

<p>What is schizotypcal personality disorder? How can it be viewed through the dimensional approach?</p>

A

<p>It is characterized by odd, eccentric, or paranoid thoughts/behaviours combined with a disomfort with, and reduced ability for, engaging socially, causing clinically significant distress or impairment. Although associated with, and similar to, schizophrenia, IMPORTANTLY, it does not include breaks from reality. This involves disturbances in the self-other relationship, and the pathological trait of Detachment and Negative Affectivity (restricted affectivity). Through the dimensional approach it might be seen as a disturbance in the self-self relationship (muddy borders separating the self from others/world), the self-other relationship (again, muddy borders), and the self-world relationship (seeing patterns that aren't there). The pathological traits involved would be Psychoticism, and Detachment.</p>

118
Q

<p>How does Parkinson's disease develop, and what are its symptoms? How does this relate to schizophrenia and substance abuse disorders?</p>

A

<p>It develops because there is damage to dopamine neurons in the substantia nigra (SN) and an accumulation of Lewy bodies. FOUR main symptoms: 1.) tremor of hands, arms legs jaw or face 2.) rigidity of the limbs and trunk 3.) slowness in initiating movements 4.) drooping posture or impaired balance and coordination. // The SN is a part of the mesolimbic dopamine pathway. This pathway is involved in substance abuse (remember, the NAc in the reward circuitry). Also remember, the side-effects of schizophrenia meds that reduce dopamine in schizophrenia are parkinson-like symptoms, whereas the side-effects of L-Dopa (used to treat parkinson's) was schizophrenia-like symptoms.</p>

119
Q

<p>What are the non-brain physiological contributors to trauma/stress-related disorders?</p>

A

<p>There is a sensitized nervous system, which is highly reactive it is chronically over-firing. There is also disfunction/disregulation in the HPA axis, like cortisol remaining at chronically high levels.</p>

120
Q

<p>What are the three main steps to treating anorexia nervosa?</p>

A

<p>step 1: weight gain. step 2: developing habits and attitudes that support long-term weight gain. step 3: creating healthy identity/attitude towards weight, and cultivating a sense of control. Anorexia is comorbid with other things like depression and substance-abuse, so addressing those is key, as is involving the family, since people with anorexia have a high rate of relapse, so having a safety net is really important.</p>

121
Q

<p>If someone has Alzheimers disease and shows cognitive decline as a result, what diagnoses can be applied?</p>

A

<p>If their cognitive degeneration precludes their independence, its MAJOR neurgocognitive disorder due to Alzheimer's Disease. If they can still function, it's MILD neurocognitive disorder due to Alzheimer's Disease.</p>

122
Q

<p>What are the treatments for adjustment disorder?</p>

A

<p>There isn't much study of this. Usually it's a form of psychotherapy focused on more adaptive ways of being with respect to the stressor and or mitigating exposure to stressor and or limiting its consequences on ones life.</p>

123
Q

<p>What is general adaptation syndrome (GAS)?</p>

A

<p>General adaptation syndrome describes in terms of 3 phases how an organism fails (or succeeds) to adapt to a NONSPECIFIC stressor. In the model, the organism begins in a state of homeostasis, until a stressor occurs. In an attempt to mobilizie resources in the body to the stressor, the organisms "resistance" to the stressor (i.e. it's ability to meet the challenge/demands/threat of the stressor) is initally reduced. this happens in the "alarm" stage. After the brief reduction in resistance, the organism rebounds and mounts it's resistance. This leads to the resistance stage, during which the organism has mobilized its resources towards addressing the threat, depleting resources in the process. In the end, there is the exhaustion stage (or, conversely, the recovery stage if all goes well), in which the organism can no longer mobilize its resources effectively to address the stressor. Seyle developed this model in light of the fact that all the patients he saw, regardless of what thing they were suffering from, all looked "sicked" and had similar physiological markers of illness.</p>

124
Q

<p>What are the pathological personality traits used in the dimensional approach to personality disorders?</p>

A

<p>Remember the acronym: AND DP (double penetration). Antagonism (vs. aggreeableness - manipulativeness, deceitfulness, grandiosity, attention seeking, calousness, hostility. Negative Affectivity (vs emotional stability (rougly neuroticism) - emotional lability, anxiousness, separation insecurity, submissivness, hostility, perseveration, depressivity, restricted affectivity), Detatchment (vs. extraversion - withdrawal, intimacy avoidance, anhedonia, depressivity, restricted affectivity, suspiciousness), Disinhibition (vs. Conscientiousness - irresponsibility, impulsivity, distractibility, risk-taking, rigid perfectionism), Psychoticism (vs. Lucidity (roughly oppeness) - unusual belifs and experiences, eccentricity, cognitive perceptual dysregulation)</p>

125
Q

<p>What characterizes sexual sadism disorder and sexual masochism disorder? What is common between them? What is important for a clinician to consider specifically in the case of sexual masochism disorder?</p>

A

<p>Both diagnoses require a paraphilia that has lasted at least 6 months + impairment/distress and/or risk of harm to others. Additionally, they're both involve pain and/or humiliation. Sexual sadism disorder is when someone achieves sexual gratification by urges, acts, or fantasies involving inflicting pain/suffering, either psychological or physical, and do not always involve coitus. Sexual masochism disorder is when someone achieves sexual gratification by urges, acts, or fantasies involving being humiliated, bound up, or made to suffer. Helplessness is often at the root of the pleasure. Clinicians should specifically figure out if asphyxiation is present, since this may involve harm and is important to consider in the context of the diagnostic criteria.</p>

126
Q

<p>What is a stressor? What is stress? What is trauma? How is trauma specified in the DSM?</p>

A

<p>A stressor is an external or internal event that puts a physiological or psychological demand on an individual. Stress is that demand. Trauama is SIGNIFICANT psychological stress. The DSM specified under which kinds of conditions a person can experience trauma, so that you cannot be said to have trauma unless you have encountered one of those conditions.</p>

127
Q

<p>What treatments are available? How available are they? Are their any treatments effective on changing gender identity/sexual orientation?</p>

A

<p>Main treatments are hormone therapy and gender confirmation surgery (sex change). Treatment for gender dysphoria are scarce to come by—these people lack support. To date, there is no treatment that can change someone's gender identity or sexual orientation.</p>

128
Q

<p>Discuss the "alcoholism is a disease" perspective?</p>

A

<p>According to AA, alcoholism is a life-long disease. You can't get rid of it, and therefore you must admit you have it and commit to a lifelong program of avoiding it. According to the "controlled use" perspective, alcoholism is NOT a disease, and you can learn to control your drinking and eventually do it in moderation. There is evidence for both, meaning for some people moderation IS possible.</p>

129
Q

<p>What characterizes obsessive-compulsive personality disorder? How can it be viewed through the dimensional approach? What are its causes, and therefore, how is it treated?</p>

A

<p>It is characterized by a pervasive pattern of control with regard to items, tasks, and situations, manifesting in a preoccupation with details at the expense of the bigger picture, combined with rigidness and inflexibility. It can be seen in the dimensional approach as a disturbance in the self-world, as well as the self-self relationship. The pathological trait at its root is Disinhibition. It cooccurs with OCD in families, but its etiology is not well understood. It is treated with CBT.</p>

130
Q

<p>What characterizes exhibitionistic disorder and voyeuristic disorder? What is common between them?</p>

A

<p>Both diagnoses require a paraphilia that has lasted at least 6 months + impairment/distress and/or risk of harm to others. Additionally, they're both involve non-consenting persons. Exhibitionistic disorder is when someone achieves sexual gratification by exposing their genitals to an unsuspecting stranger, often with the intention of shocking or impressing. Voyeuristic disorder is when someone achieves sexual gratification by observing an unsuspecting person that is naked, disrobing, or engaged in sexual activity. IT IS ONLY DIAGNOSED in people 18 YEARS OR OLDER. The voyeurs are typically not interested in looking at their partners, typically have no interest in interacting with the victim, and typically are interested interested in settings in which nudity or display of nudity is not normal/acceptable.</p>

131
Q

<p>What does the developmental perspective say about the etiology of schizophrenia?</p>

A

<p>It says that schizophrenia is a neurodevelopmental disorder. It can be caused realy early by fetal infections and fetal malnutrition, but also later in life by early and heavy use of cannibis, early life trauma, growing up in an urban environment. It also says that a child might begin life with bad synnaptic connections early in life that are reinforced. Later, when synaptic pruning occurs, other adaptive connections are pruned away leaving only the bad ones, which is why we see schizophrenia developing into the psychotic phase in adolescence.</p>

132
Q

<p>What are the negative symptoms of schizophrenia? Give some examples. Why are negative symptoms important?</p>

A

<p>They are the inability to act, talk, express how you feel, and feel pleasure. They're called "negative" symptoms because they constitute a loss of normal functioning. Ex: avolition (inability to start or keep up goal-oriented activity), alogia (a lack of thinking inferred from terse or short speech), asociality (min. interest in social relationships), anhedonia (reduced enjoyment or inability for enjoyment), flat affect (diminished facial expression, intonations, and gestures). These negative symptoms are less responsive to medications, and they affect the progrnosis for schizophrenia much more strongly than the positive symptoms.</p>

133
Q

<p>What is sexual orientation made up of?</p>

A

<p>It is made up of romantic attraction (people you want to be romantic with), sexual arousal (people that prompt physical arousal), sexual attraction (psychological attraction, which includes fantasies, feelings, lust), sexual behaviour (actual sexual interactions), and sexual identity (how a person understands and labels their sexual attract to, and interaction with, others)</p>

134
Q

<p>What are the social and sociocultural causes of schizophrenia?</p>

A

<p>Social environment isn't a large contributor to the CAUSE of schizophrenia, but may increase the risk. Things like accidents, maltreatment, and unhealth upbringing increase the risk. There's also an association in some cultures between Expressed Emotion (EE)—a negative communication pattern in which others put negative outside pressure on the schizophrenic person, like overconcern, criticism, resentment, etc—and schizophrenia. Although the behaviour might look the same between cultures, the interpretation might be different, like for instance, in one culture putting someone down might be a sign that you really care about them. Sociocultural causes include immigration. Immigrants have highest rates of schizophrenia in Western Europe, although that might be caused by a clinician bias b/c of the unintelligibility of actions from another culture.</p>

135
Q

<p>What is a paraphilic disorder and what distinguishes it from paraphilia?</p>

A

<p>Paraphilic disorder is diagnosed when an individual has a paraphilia that causes clinically significant distress/impairment or when acted upon, harms or risks harming others, that has existed for at least 6 months.</p>

136
Q

<p>What does the immune system do? What are the two major types of immunity and give a little description? Give an example of each.</p>

A

<p>The immune system removes or destroys harmful bacteria, viruses, and toxic chemicals (including cellular waste) from the body. The two major kinds of immunity are innate immunity (present at birth), and adaptive immunity created by experience of pathogens. Innate immunity in a non-specific response, that can be seen in such responses as inflammation and fever. Adaptive immunity is a SPECIFIC response targeting particular issues, such as anitbodies that target specific pathogens.</p>

137
Q

<p>What are caffeine, amphetamines, and cocaine? What do they do? How do they have their effects? What are their risks?</p>

A

<p>These are all CNS stimulants. Caffeine works on the serotonin and adenosine system. Can produce tolerance and withdrawal (caffeine headaches). Amphetamines (like adderal) produce emotional and cognitive effects such as euphoria, change in desire for sex, increased wakefulness, and improved cognitive control. They do so by working on the norephinephrine and dopamine system, increasing activity. Can cause tolerance and withdrawal. Cocaine works by—among other things—increasing dopamine. It causes euphoria, increases blood presure and heart rate, promotes wakefulness, reduces appetite, and can create paranoia. It has a high potential to create tolerance, withdrawal, and psychological dependence, which for cocaine looks like depression + anxiety.</p>

138
Q

<p>In the context of the sexual response cycle, what is the refractory period?</p>

A

<p>It is the period after a male orgasm during which it is physiologically not possible to have another orgasm.</p>

139
Q

<p>What are the causes of gender dysphoria?</p>

A

<p>Not much is known since its so rare. Some believe prenatal exposure to testosterone plays a role. There also seems to be some difference in fibre pathways and cortical thickness.</p>

140
Q

<p>What is genito-pelvic pain/penetration disorder?</p>

A

<p>It is a disorder characterized by fear and anxiety in relation to the pain experienced during intercourse caused by penetration (vulvovaginal or pelvic), thrusting, or the tensing or tightening of the pelvic floor muscles that has occured persistently for at least 6 months, causing distress and/or impairment.</p>

141
Q

<p>What are the strategies aimed at treating bulimia nervosa and binge-eating disorder?</p>

A

<p>CBT and other cognitive approaches aimed at controlling antecedants: the things that cause binge-episodes, controlling behaviours: how do i eat regular amounts of food, how do i control my food shopping, etc. AND controlling consequences: reward/punishment, changing how you view the consequences of your actions (ex: this will cause long-term harm). also, it's important to address the cognitive components, like expectencies and distortions of thinking, as well as coping mechanisms.</p>

142
Q

<p>What is the name of the disorder characterized by distress caused by an inability to achieve or maintain an erection during sexual activity?</p>

A

<p>Erectile disorder.</p>

143
Q

<p>What is the name of the disorder characterized by psychological and physiological dependence on a substance that induces significant impairment and/or distress?</p>

A

<p>Substance-related Disorder</p>

144
Q

<p>What major thing is required for a diagnoses of acute stress disorder or PTSD? In additiion to that, symptoms in which clusters must be present for a diagnoses? What do these symptom clusters look like?</p>

A

<p>Both require that a person directly or indirectly experience a traumatic event. Additionally, individuals must be experience symptoms in the clusters of: intrusion (intrusive thoughts, distressing recollections, nightmares, flashbacks, physical symptoms like sweating or heart rate or panic attack that seem ill-adapted to the surroundings. they're intrusive because you can't stop them), *dissociative symptoms* (this is a symptom category only for acute stress disorder, but not PTSD—in PTSD it is subsumed in other clusters like negative alterations in mood or cognition, as in the case of dissociative amnesia. other examples include depersonalization and derealization), avoidance (of thoughts, places, similar situations, people, feelings or other reminders of trauma) , negative alterations in mood or cognition (anhedonia, detachment from self and others, frequent negative emotions, difficulty remember details of event, distorted cognititions leading to persistent negative view of self and world or self/other-blame), and "arousal and changes in reactivity" (irritability that can lead to anger or physical aggression, reckless or self-destructive behaviour, hypervigilance, heightened physiological reactivity, difficulty concentrating/focusing and sleeping)</p>

145
Q

<p>What does the iRISA model say about why addiction? What does the acronym stand for? Describe the cycle it specifies.</p>

A

<p>It stands for impaired Response Inhibition and Salience Attribution. The cycle begins with INTOXICATION, during which you have an impaired self-awareness (your attention is focused on things that don't have to do with your goals, with things that might stop your behaviour, on your problems, etc). The next part is BINGEING, which is a loss of control, which comes about by the repeated bad decisions in the intoxication phase. After you're glutted, you come down, eventually leading to WITHDRAWAL. Here, you feel like shit. You have no motivation and potentially, lyou can't feel pleasure. Other goals are not sailient to you, and not worth pursuing. Eventually, memories from the substance enter your mind, either bidden or unbidden (intrusive). This leads to CRAVING, which is characterized by biased thinking aimed at consuming the drug, and positive expectencies about its effects, which leads to move thinking about it. This eventaully leads back to getting the drug, getting INTOXICATED, and starting again.</p>

146
Q

<p>How does the DSM-5 define what a traumatic event is?</p>

A

<p>It defines it as actual or threatened death, serious injury, or sexual violence. It can be experienced directly by an individual (either acutely, or repeatedly in an indirect way, the way a paramedic sees it), witnessed as occuring to another individual, or learned about occuring to someone that's very close.</p>

147
Q

<p>What is Major Neurocognitive Disorder?</p>

A

<p>It is characterized by the GRADUAL DETERIORATION of brain function in a way that PRECLUDES INDEPENDENCE (interferes with) in practical day-to-day life.</p>

148
Q

<p>What is the name of the personality disorder characterized by an extreme reliance on others, unusual submissiveness, and separation anxiety? What cluster is it in?</p>

A

<p>dependent personality disorder. It is in cluster C.</p>

149
Q

<p>What are the cognitive symptoms of schizophrenia? How are these symptoms measured/accounted for?</p>

A

<p>They are 1) Difficulty focusing, 2) Speaking unintelligibly, 3) Replying tangentially, 4) Loosening of association/cognitive slippage (shifting from topic to topic without meaningful connection) . These symptoms can be seen in 1) Trouble abstracting 2) Categorization problems causing overinclusiveness 3) Memory issues 4) Decision-making problems 5) Problems in Attention (distraction, focus, sustaining attention) 6) Social-cognitive problems (including issues about theory of mind)</p>

150
Q

<p>What are sedatives, hypnotics, and anxiolytics? Give two examples. How do they work? What risks do they carry?</p>

A

<p>Sedatives are depressents that calm, reduce muscle tension, tranquilize, often causing rest/sleep. Hypnotics are depressents that cause sleep. Anxiolitics are depressents that reduce anxiety/calm/reduce muscle tension, without the hypnotic effect of sedatives, although there is overlap between sedatives and anxiolytics. Baribturates are CNS depressents that are hypnotics/sedatives/anxiolytics. At low doses the relax muscles and produce mild feelings of well-being. At higher doses, the depressent effects cause slurred speech, motor issues, and concentration problems, eventually potentially causing the diaphrag muscle to relax and cause death by suffocation. Benzodiazepines (valium and xanax) have largely replaced barbiturates, since they are safer, since barbiturates have a narrow therapeutic dosage range, that is easily exceeded, causing overdose. Like barbiturates, they can cause tolerance and withdrawal, and produce similar effects. Both Barbiturates and Benzos act on the GABA neurotransmitter system, causing inhibition. They're especially dangerous when combined with alcohol.</p>

151
Q

<p>What are the two main steps involved in treatment of substance-abuse disorders? Where are the two broad places these treatments occur?</p>

A

<p>The two main steps are: 1.) Remove drug from the body and 2.) Increase understanding and skillset regarding addiction, and develop a plan. Treatments broadly take place in either inpatient (residential) settings or out-patient settings (ex: AA or NA).</p>

152
Q

<p>What causes delerium? How is it treated? What is key in treatment?</p>

A

<p>Delerium seems to be caused by a lack of resiliency in the brain to stressors, so it can be caused by fever, malnutrition, dehydration, infection, medication, intoxication or withdrawal, trauma, and environmental stressors generally. Treatment aims to isolate the cause and remove it. Psychosocial treatment is often given to help poeople cope with stressors generally, and sometimes antipsychotics if delerium is really acute (often cause of substance withdrawal). The key to treatment is to figure out the causes EARLY and get treatment SOON. This leads to better long-term outcomes.</p>

153
Q

<p>What is the prevalence rate of schizophrenia?</p>

A

<p>It is about 1% worldwide.</p>

154
Q

<p>What is the name of the personality disorder characterized by a preoccupation with narrow details at the expense of the big picture, combined with inflexibility and rigidity? What cluster is it in?</p>

A

<p>obsessive-compulsive personality disorder. It is in cluster C.</p>

155
Q

<p>What is brief psychotic disorder? How does it differ from schizophrenia, schizophreniform disorder, and delusional disorder? What usually causes it? How is it usually treated?</p>

A

<p>It is diagnosed by the presence of at least 1 psychotic symptom that continues for at least 1 day but no longer than a month. It's usually caused by a major stressor, and is usually treated with antipsychotics. It differs from delusional disorder from the fact that it doesn't necessarily require delusion, just psychosis. It differs from schizophreniform disorder in that it requires less psychotic smptoms (only requires 1), and in its short duration (<1 month), rather than between 1 and 6 months for schizophreniform disorder and >6 months for schizophrenia. It is usually treated with antipsychotics.</p>

156
Q

<p>What is the name of the disorder characterized by revealing yourself to unsuspecting strangers?</p>

A

<p>Exhibitionistic disorder</p>

157
Q

<p>What are four important features of the DSM criteria for subtance-related disorder?</p>

A

<p>The DSM-5 requires TWO symptoms from a list of symptoms (PSYCHOLOGICAL symptoms, and PHYSIOLOGICAL symptoms), that cause significant impairment or distress over the course of a 12-MONTH PERIOD. Additionally, the level of SEVERITY of the diagnoses depends on HOW MANY symptoms you're presenting: 2-3 (mild), 4-5 (moderate), >=6 (severe)</p>

158
Q

<p>What characterizes Anorexia Nervosa? What are its subtypes? What are its risks? When is its typical onset?</p>

A

<p>anorexia nervosa is characterized by extreme fear of weight gain, body image distortion (not dysmorphia, which is its own disorder), AND VERY LOW BODY WEIGHT, which is its central feature. It has two subtypes: Restricting Subtype: losing weight occurs by extreme dieting and/or exercise and/or Binge-Eating/Purging Subtype: losing weight occurs by vomiting, laxitives, or diuretics, and is often combined with bingeing. (NOTE: bingeing in anorexia is diff from bulemia in that although anorexics binge, that term is relative to the normal amount of food they injest, meaning it still may be a relatively small amount) The most important risk of anorexia is that it has a high mortaility rate (10-15%), brought about by suicide, substance abuse, and the health impacts of the disorder. Another risk is that it has a high rate of relapse, and takes years to treat: 6-7 years. Those with the binge-eating/purging subtype are more at risk of negative outcomes. It's typical onset is betweetn 13-20 y.o.</p>

159
Q

<p>What constitutes succesfull aging? What is a reserve in this context?</p>

A

<p>good mental health, physical health, independence, social net + positive relationships. It involves finding strong optimization and compensatory strategies to make use of what one is good at, and compensating with things where one falls short: use social support, physical training, or mental training. Building intelligence/mental resources, a strong body, and a strong social net constitutes a RESERVE, that can be drawn upon to compensate for dificiencies in neural functioning.</p>

160
Q

<p>What characterizes a personality disorder?</p>

A

<p>It is characterized by a stable and inflexible pattern of behaviours and inner exeperiences that is maladaptive and at odds with the world and the culture an individual finds themself in, so much so, that it causes significant impairment and/or distress. It is diagnosed later in life since personality does not coalesce until adolescence.</p>

161
Q

<p>What are the broad symptom categories of schizophrenia, and what is required for a diagnoses according to the DSM-5?</p>

A

<p>The broad symptom categories include positive symptoms, cognitive symptoms, psychomotor abnormalities, and negative symptoms. A diagnoses of schizophrenia requires that for at least 1 month an individual has had either 1) Delusions, 2) Hallucinations, or 3) Cognitive Symptoms. Additionally, they must have impairment(s) that have lasted for at least 6 months.</p>

162
Q

<p>What is female sexual interest/arousal disorder?</p>

A

<p>Characterized by little or no sexual activity in imagination or reality, causing significant distress and/or impairment, that has been present for at least 6 months.</p>

163
Q

<p>What characterizes frotteuristic disorder and pedophilic disorder? What is common between them? What does pedophilic onset usually begin and what's so special about treating it?</p>

A

<p>Both diagnoses require a paraphilia that has lasted at least 6 months + impairment/distress and/or risk of harm to others. Additionally, they're both involve non-consenting persons. Frotteuristic disorder is when someone achieves sexual gratification by urges, acts, or fantasies involving sneaking a touch of someone else body in a nonconsenting way, usually men, and often in crowded situations. Pedophilic disorder involves achieving sexual gratification through urges, acts, or fantasies targeted as prepubescent or early pubescen children. Its onset is typically in adolescence and lasts the course of a lifetime. There's no effective treatment for it.</p>

164
Q

<p>What is female orgasmic disorder and what is its prevalence?</p>

A

<p>It is characterized by long times to reach orgasm, or inability to do so, or unstatisfying orgasms with low intensity. Its prevalence is 10%-42%.</p>

165
Q

<p>What is the dopamine hypothesis of schizophrenia? What was wrong about it?</p>

A

<p>The dopamine hypothesis of schizophrenia was born out of the observation that medications that increased dopamine levels in the brain often produced schizophrenia-like symptoms. Further studies, however, showed that individuals with schizophrenia often did not have higher levels of dopamine.</p>

166
Q

<p>What are prions? What role do they play in neurocognitive disorder?</p>

A

<p>They are malfolded proteins that are capable of boring holes in brain tissue, causing swiss-cheese like damage, producing a sponge-like appearence.</p>

167
Q

<p>What is the name of the personality disorder characterized by a enduring patterns of volatility, instability, and implsivity? What cluster is it in?</p>

A

<p>borderline personality disorder. It is in cluster B.</p>

168
Q

<p>What are the DSM-5 Trauma and Stressor-related disorders?</p>

A

<p>They are: adjustment disorder, acute stress disorder, post-traumatic stress disorder, reactive attachment disorder, and disinhibited social engagement disorder</p>

169
Q

<p>What are the genetic and environmental causes of neurocognitive disorder due to Alzheimer's disease?</p>

A

<p>genes play an important in onset time, which will determine how bad symptoms get. Genes implicated in tau tangles, as well as thoughs coding for enzymes that break down beta amyloid plaques. Environmental dangers are those that increase the risk of cardiovascular disease and stroke: ex: lack of exercise, high LDL, low HDL.</p>

170
Q

<p>What risks does marijuana carry?</p>

A

<p>Marijuana can cause dependence and withdrawal. Withdrawal causes a wind-ranging lack of care and concern about outcomes/consequences. It also causes irritability, anxiety, insomnia, and depression. Also, it increases the chance of psychosis at large doses.</p>

171
Q

<p>What happens most often in substance-induced neurocognitive disorder? What else can contribute besides substances? What can control it?</p>

A

<p>Delirium! Associated with extreme sub intoxication, withdrawal, use of multiple substances, or inhalent use. Heavymetal in environment might be a contributor to substance-induced .neurcognitive disorder. Abstaining and maintaining abstinence can control or even make cognitive disorder totaly subside.</p>

172
Q

<p>What are the positive symptoms of schizophrenia?</p>

A

<p>They are delusions and hallucinations.</p>

173
Q

<p>What is aphasia, apraxia, agnosia, anomia?</p>

A

<p>aphasia: language propblems (listening, reading, speaking) // apraxia: motor problems that prevent you from correctly producing learned behaviours // agnosia: inability to recognize objects // anomia: inability to name objects</p>

174
Q

<p>What happens in the female/male genitals in each phase of the sexual response cycle?</p>

A

<p>1.) EXCITEMENT (MALES: testes rise, errection begins (myotonia - muscle firming) // FEMALES: Uterus rises, vagina lubricates, labia engorge, clitoris becomes erect) 2.) PLATEAU (MALES: Bulbourethral gland contracts, testicles engorge and fully elevate, scrotal skin thickens, penis full erect // FEMALES: Upper part of vagina expands, orgasmic platform forms, clitoris retracts under hood) 3.) ORGASM (MALES: Vas deferens contracts, seminal vesicles contract, prostate gland contracts, anal sphincter contracts, urethra contracts // FEMALES: Uterus conctracts, anal sphincter contracts, orgasmic platform contracts) 4.) RESOLUTION (MALES: loss of erection, testicles descend,</p>

175
Q

<p>What is the name of the personality disorder characterized by a pervasive pattern of systematic disregard for others and social norms? What cluster is it in?</p>

A

<p>antisocial personality disorder. It is in cluster B.</p>

176
Q

<p>What is common to adjustment disorder, acute stress disorder, post-traumatic stress disorder, reactive attachment disorder, and disinhibited social engagement disorder?</p>

A

<p>They are all stressful event or trauma-related.</p>

177
Q

<p>What is delerium? When does it occur?</p>

A

<p>It is an intense state of confusion that comes on abruptly, and has a short duration. In this state, peole are disoriented. This can occur randomly, often after surgeries, and seomtimes even at particular times of day: "sundowning"</p>

178
Q

<p>What are the phsyiological and brain acitivity differences between schizophrenic people and healthy controls? What brain regions are associated with the disorganized behavioural, negative, and positive symptoms of schizophrenia?</p>

A

<p>People with schizophrenia tend to have larger ventricles, which is thought to be caused by a lost of neuroligcal mass around the ventricles, allowing them to expand into that area. Schizophrenic people show abnormal activity in the PFC, ACC, and thalamus. Disorganized symptoms are associated with abnormal activity in the dorsolateral PFC. Ventrolateral PFC dysfunction is associated with negative symptoms. Medial PFC dysfunction is assocaited with the postive symptoms.</p>

179
Q

<p>What characterizes avoidant personality disorder? How can it be viewed through the dimensional approach? How is it treated?</p>

A

<p>It is characterized by low self-worth manifesting in extreme social anxiety, social inhibition, and a hypersensitivity to negative evaluation. It can be seen in the dimensional approach as a disturbance in the self-other relationship, as well as the self-self relationship. The pathological trait at its root is Detachment. It is treated through behaviour therapies which address maladaptive cognitive distortions, social skills, and social fear through desensitization and exposure.</p>

180
Q

<p>What counts as moderate vs heavy drinking for men/women? What counts has a binge for men/women?</p>

A

<p>Moderate: <=1/day (woman), <=2/day (man) // Heavy: >1/day (woman) >2/day (man) // A binge is defined as >= 4 (woman) and >=5 (man).</p>

181
Q

What are the four sources of canadian law?

A

Statute law, civil law, common law, constitution of canada

182
Q

What rights does the bill of rights give you with respect to treatment, and how is this balanced against the duties of the government has to the public?

A

Bill of rights: right to treatment, right to be informed about the reasons for hospital detention, right to refuse treatment, right to treatment in the least restrictive setting possible, right to legal council, right to apply to a review panel that can grant discharge from detention. These rights are balanced against the two types authorities the state has in regards to maintaining public safety: 1) Police Power: protect public safety, health, welfare, and create new laws if need be to do so, and 2) parent patriae (“state as parent”): government can act in situations in which citizen are not likely to act in their own best interest.

183
Q

What are the two broad ways a person can be committed? What are the two ways of being committed against your will?

A

Voluntarily, and involuntarily. You can be involuntarily committed by Criminal commitment, and civil commitment.

184
Q

What criteria must be met in all Canadian jurisdictions for civil commitment to be legal? What additional criteria must be met in SOME Canadian jurisdictions for civil commitment to be legal?

A

1) person is dangerous to himself or other people (all)
2) the person has a mental disorder (all)
3) the person is in need of treatment (some)

185
Q

In the context of commitment, what does the law mean by “mental disorder” and “dangerous”?

A

It’s not always clear, and varies from jurisdiction to jurisdiction.

186
Q

Who can commit someone civilly, and by what means?

A

Doctors, police officers can apply for a form 1/form 2 respectively, can have people involuntarily committed for a SHORT period of time for an assessment. Individuals can present evidence to a justice of the peace to apply for the same using a form 2.

187
Q

What do forms 1/2/3/4 do?

A

They’re for civil commitment. Form 1/2 is for short assessment commitment. Form 3 is for involuntary ADMISSION into institution for longer periods of time (ex: ON:2 weeks). Form 4 is for an EXTENSION for LONG periods of time. (ex: ON: 3 months)

188
Q

What is hidden homelessness?

A

It is homelessness that looks like living in a car, couch to couch, or being in jail.

189
Q

What are the 2 main goals of disinstutionilization?

A

1) downsize or close large prov. and territ. mental hospitals, and 2) transinstitutionalization: create a network of smaller community mental hospitals and services

190
Q

What are the two parts in this process by which someone gets criminally committed?

A

1) They must be ACCUSED, detained in a mental health facility, then deemed fit to stand trial. 2) Tried in court and found not criminally responsible on account of a mental disorder (NCRMD)

191
Q

What does being fit to stand trial mean?

A

it means you 1) understand the object and nature of the proceedings 2) understand the consequences of sentencing and outcomes 3) can communicate with your legal council

192
Q

What is required for the prosecution to prove in order to establish culpability?

A

1) mens rea (guilty mind) and 2) actus rea (guilty act)

193
Q

What is required for a conviction of not guilty by reason of insanity? (NGRI)

A

1) presence of a mental disorder 2) lack of of understanding why you’re doing, or why its bad.

194
Q

What changes from NGRI to NCRMD?

A

1) “not guilty” goes to “not criminally responsible” 2) “insanity” changes to “mental disorder” 3) Instead of rotting in an asylum, you get turned over to prov. review boards that automatically review your mental status.

195
Q

What is a supervening defense?

A

Even if mens rea and actus rea can be proven, you can still show that they did not understand the nature of what they did, or why its bad.

196
Q

What are the ethical considerations researchers must acknowledge to do research? What are the rights of participants?

A

1) Do no harm - minimize likelyhood of harm (ethical review board). 2) Participation must be voluntary (they can leave whenever for any reason) 3) Participation must be informed (informed of risks and nature. if cover story/deception was involved, full debrief reveals why)

197
Q

What are the legal and ethical considerations of treatment?

A

Informed voluntary consent, boundaries (don’t be business partner, relationship), Do no harm* (avoid power/sex and misuse of information), Recognize limits of competence* (don’t work outside your training), Confidentiality (shit you say is private, unless client says otherwise, or the law)

198
Q

What is the duty to warn? What criteria should we keep in mind when deciding?

A

In america, clinician have a LEGAL duty to warn 3rd parties at risk. In Canada, they have an ETHICAL duty to do so. 1) Clear risk to an identifiable person or group? 2) Is there a risk of bodily harm and/or death? 3) Is the danger imminent?