Exam 1: Obesity and GI Flashcards

1
Q

What is the BMI of pre-obese, Class I Obesity, Class II Obesity, and Class III Obesity

A

Pre-obese - 25-29.9

Class I - 30-34.9

Class II - 35-39.9

Class III greater than equal to 40

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2
Q

We measure obesity though ___

A

BMI

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3
Q

BMI

A

body mass index

a measure of an adult’s weight in relation to his or her height, calculated by using the adult’s weight in kilograms divided by the square of his or her height in meters

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4
Q

What are some chronic conditions we are at risk for when obesity

A

Alzheimer Disease, Anxiety, Depression, Stroke

Asthma, Obstructive Sleep Apnea, resp infections

Non alcoholic fatty liver disease, liver cancer

thyroid cancer

CAD, MI, heart failure , HTN

Renal cancer

Type II Diabetes and Pancreatic cancer

colorectal cancer

hypercholesterolemia

prostate cancer

osteoarthritis

cholecystitis, cholelithiasis, gallbladder cancer

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5
Q

Treatment Options for Obesity

A

1 is Behavioral Modifications (diet and exercise)

  1. Pharmacological management
  2. Bariatric Surgery
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6
Q

What is the problem with pharmacology management for obesity

A

rarely do patients lose more than 10% of total body weight

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7
Q

Standard Treatment post Behavioral Modification for Obesity

A

Bariatric Surgery

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8
Q

How is bariatric surgery trending over time

A

it is trending upward (13000 in 98 to 278000 in 19)

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9
Q

What are the 2 mechanisms of Bariatric Surgery

A
  1. Restriction
  2. Malabsorption depending on the type of surgery / Affect Absorption
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10
Q

Benefits of Bariatric Surgery

A

Total Body Weight Loss

Recovery of Chronic Illnesses

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11
Q

Criteria for Bariatric Surgery

A
  1. BMI greater than or equal to 40 kg/m^2

OR

  1. Patients with BMI greater than or equal to 35 and one or more severe obesity associated comorbid conditions

OR

  1. Patients with BMI greater than or equal to 30 with type 2 diabetes or metabolic syndrome
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12
Q

4 Major Types of Bariatric Surgery

A
  1. Biliopancreatic Diversion w/ Duodenal Switch
  2. Roux En Y Gastric Bypass
  3. Sleeve
  4. Gastric Banding
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13
Q

Biliopancreatic Diversion w/ Duodenal Switch

A

“Sleeve Gastrectomy w/ Duodenal Switch”

Half of stomach is removed, leaving a small area that holds about 60 mL

Jejunum is excluded from the GI tract and connected to the start of the duodenum and then the ileum on the other side

Pyloric Valve is still intact in this one

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14
Q

Is there risk for dumping syndrome with biliopancreatic diversion w/ duodenal switch

A

No there is no dumping syndrome since the pyloric valve is intact

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15
Q

Roux En Y Gastric Bypass

A

horizontal row of staples across fundus of stomach makes a pouch with a capacity of 20-30 mL - the jejunum is then divided and brought to the small pouch and then brought through roux limb

The pyloric valve is bypassed

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16
Q

Is there risk for dumping syndrome in Roux En Y Gastric Bypass

A

yes, the pyloric valve is bypassed entirely

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17
Q

Sleeve

A

Sleeve Gastrectomy

Stomach is incised vertically and up to 85% of the stomach is surgically removed, leaving a “sleeve” shaped tube that retains intact nervous innervation and dose not obstruct or decrease the size of the gastric outlet

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18
Q

The Gastric Sleeve can hold up to __ mL

A

20

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19
Q

Will a pt get dumping syndrome with a sleeve

A

no pyloric valve is left intact

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20
Q

Gastric Banding

A

a prosthetic device is used to restrict oral intake by creating a small pouch of 10-15 mL that empties through the narrow outlet into the remainder of the stomach

the band hangs outside the stomach for adjustment

many pts not successful with this procedure

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21
Q

What kind of bariatric surgery is being phased out

A

Gastric Binding

It is generally unsuccessful and also has lowest level of weight loss

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22
Q

What bariatric surgery leads to most excess weight loss

A

bilopancreatic diversion with DS

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23
Q

What is important to know about fluids and bariatric surgery

A

No fluids with meals, and avoid fluid intake 30 min before a meal and 30-60 min after a meal

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24
Q

Bariatric surgery postop care is similar to gastric resection but …

A

greater risk for complications due to obesity

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25
What is a very important thing to do preop before bariatric surgery?
PSYCHOSOCIAL INTERVENTIONS to modify eating behaviors
26
What is contraindicated following a bariatric surgery
an NG tube - risk for perforation from disrupting surgical suture line
27
Nursing Diagnoses for Bariatric Surgery
Deficient knowledge about dietary limitations Anxiety related to impending surgery Acute pain related to surgical procedure risk for deficient fluid volumes related to nausea, gastric irritation and pain risk for infection related to anastomotic imbalanced nutrition disturbed body image risk for constipation and/or diarrhea
28
Goals Pre and Post Op for Bariatric Surgery
preop and postop knowledge, manage anxiety post op: manage pain, maintain homeostatic fluid balance, prevent infection, adhere to dietary instructions, vitamin supplements, lifelong follow up, positive body image, and normal bowel habits
29
T/F: After bariatric surgery, the average pt loses between 25-35% of presurgical body weight within the first 18-24 months post-procedure
True - pt will expect to see weight loss following bar surgery if following instructions
30
T/F: After bowel sounds have returned and oral intake is resumed follow bariatric surgery, 6 small feedings consisting of a total of 600 to 800 calories per day should be consumed
True
31
T/F: Traditionally, the term morbid obesity applies to adults whose body mass index (BMI) exceeds 40 kg/m^2
True
32
Risk Factors for GI Disorders
Family Hx Lifestyle - stress, poor diet, alcohol, tobacco, smoking can all lead to these disorders - many of the disorders are associated with lifestyle behaviors Domino Effect Previous abdominal surgeries or trauma Neurologic disorder
33
What can GERD lead to?
Barret's esophagus --> predisposition for esophageal cancer
34
What can chronic gastritis lead to?
predisposition of gastric cancer
35
What can previous abdominal surgeries lead to
potential adhesions --> potential obstructions
36
What can neurological disorders like MS/Parkinsons can impair what
pt ability to move and have peristalsis which impairs movement of waste products
37
GERD
Backward movement of gastric or duodenal contents makes pt feel like they are having heartburn and these episodes occur more than 2 times a week
38
What is the major cause of GERD
relaxation or weakness of LES (lower esophageal sphincter) obesity can also cause GERD
39
Things that Trigger LES Relaxation
Fatty Food Caffeinated Beverages Carbonation Chocolate Milk Chocolate Milk Tobacco Spearmint Progesterone therapy and in surgery NG tube also permanently impairs function Medications: NSAIDS, Calcium Channel Blockers, Blood Pressure Meds, Nitroglycerine for chest pain Pyloric Stenosis Mucosal irritants - tomato's and citrus
40
What to do prior to laying down for the night when you have GERD
do not eat 3 hours prior to laying down - no supine if you do
41
What is a classic symptom of GERD
"Waking up in the middle of the night feeling a pain in their throat or feeling heartburn"
42
Clinical Manifestations of GERD
Pyrosis Dyspepsia Sour Taste Hypersalivation Dysphagia Ordynophagia Eructation Fullness (even when eating a v small amount of food) - Early Satiety Nausea
43
Pyrosis
burning in the esophagus / heartburn
44
Dyspepsia
pain in the upper abdomen
45
Dysphagia
difficulty swallowing
46
Ordynophagia
Painful swallowing
47
Eructation
belching
48
When do symptoms worsen for GERD
worsens lying down, bending over, and occurs 30 min to 2 hours post meal
49
Non Surgical Interventions
Dont let the sphincters relax Promote gastric emptying and avoid gastric distention Watch those acidic foods Medications (eat small meals, lose some weight, stop smoking, keep HOB elevated after a meal, avoid tight clothing, avoid lying down after eating)
50
Surgical Intervention for GERD
Nissen Fundoplication
51
Nissen Fundoplication
takes the fundus and wraps it around the LES to reinforce the closing function of the sphincter
52
Risks of surgery of Nissen Fundoplication
Hemorrhage, Bleeding, Infection, Obstruction (If too tight) Short bouts of temporary dysphagia Bloating and gas buildup
53
Does Nissen Fundoplication cure GERD
no they still need to follow non surgical recommendations
54
Barretts Esophagus
w/ prolonged GERD acid erodes lining of the esophagus and turns cells of esophagus to look like the lining of the intestines alterations can lead to esophageal cancer
55
Hiatal Hernia
when the opening through the diaphragm where the esophagus passes becomes enlarged and part of upper stomach moves into lower portion of the thorax
56
Risk Factors for Hiatal Hernias
Age Obesity Women more at risk
57
Concerns of Hiatal Hernias
Obstructions and Strangulations
58
What are the two types of Hiatal Hernias
1. Sliding 2. Rolling
59
Sliding Hiatal Hernia
Gastroesophageal jxn is compromised the stomach sits right in the esophagus
60
Rolling Hiatal Hernia
Gastroesophageal junction is intact or compromised the stomach is pushed through the diaphragm and sits next to esophagus
61
How does a Sliding Hiatal Hernia present
can be asymptomatic GERD symptoms
62
How does a Rolling HIatal Hernia Present
can be asymptomatic GERD symptoms breathlessness after eating chest pain that mimics angina feeling of suffocation worse lying down (SOB) *since it is pushing on resp tract*
63
Which type of hiatal hernia has a higher risk for strangulation?
Rolling Hiatal Hernia Piece of stomach can be strangulated - leading to higher risk for strangulation
64
Interventions for Hiatal Hernias
limit or eliminate foods that relax LES promote gastric emptying or avoid gastric distention (this also helps prevent movement of the hernia) limit or eliminated foods that add fuel to the acid fire d/t acidic content (tomato and citrus) medication reconcilliation sleep in low fowlers position Reglin to promote mobility and peristalsis
65
Gastritis
lining of the stomach is inflamed or swollen - disrupted stomach lining over time the mucosa can erode due to this
66
Gastritis can be ___ or ___
acute or chronic
67
How long is acute gastritis compared to chronic gastritis
Acute = few hours to days chronic = repeated exposure/recurrent episodes
68
What is the cause of non erosive acute gastritis
H pylori
69
What is the cause of erosive Gastritis
NSAIDS, motrin, ASA< Alcohol use
70
Why can H Pylori lead to pernicious anemia
Chronic Gastritis caused by H Pylori can destroy the parietal cells of the stomach leading to a lack of intrinsic factor production which is needed for VitB12 absorption --> therefore chronic gast. pts may need VitB12 injections lifelong
71
What makes gastritis worse
stress caffeinated beverages any triggers for GERD: smoking, spicy or highly se4asoned food, alcohol etc
72
What are some s/s of gastritis
acute: anorexia, epigastric pain, hemtaemesis, hiccups, melena or hematochezia, NV Chronic: belching, early satiety, intolerance to fatty or spicy foods, NV, pyrosis, sour taste in mouth, vague epigastric discomfort relieved by eating
73
What are the goals for patients hospitalized for gastritis
relieving pain (abdominal) promote fluid balance reduce anxiety promote optimal nutrition
74
Why is nutrition balance and fluid balance impaired with gastritis
they become essentially NPO and are not consuming enough calories so they arnt getting the food they need or are drinking and risk dehydration
75
Interventions to Treat Chronic Gastritis
If caused by H Pylori --> Combo of antibiotics NSAIDS/Alcohol --> collaborate with health care team, educate patient, refer smoking cessation stress management avoid trigger foods *focus on the mind-gut connection*
76
Peptic Ulcer Disease (PUD)
sores in the lining of the GI system and these sores can erode at the mucosa
77
How does gastritis and PUD differ
Gastritis only effects the stomach lining while peptic ulcers are localized sores that can erode past the mucosal layer at least half a centimeter (deeper than gastritis)
78
A patient with H Pylori induced chronic gastritis is at high risk for developing
PUD
79
What are the 4 locations peptic ulcers can be found
Duodenum Stomach Pylorus Esophagus
80
___ is the most common location for a peptic ulcer, and ___ is the second most common
Duodenum; Stomach
81
Risk Factors for PUD
age genetics stress NSAID use diet
82
Main Underlying Cause of PUD
H Pylori and Excessive secretion of hydrochloric acid
83
PUD Chief Complain
Dull gnawing burning pain in the mid epigastric area that can radiate into the back *due to radiation to the back rule out other potential causes*
84
Nursing Management and Interventions for PUD
Dietary Modification Smoking cessation Pharmacologic therapy surgical management
85
How is PUD diagnosed
via an upper endoscopy - provider will visualize the ulcer, lesion and inflammation
86
What is the drug regimen like for H Pylori infection
triple or quadruple therapy (with quadruple adding bismuth salts)
87
What is the timing of pain like for PUD depending on if it is duodenal or gastric
Duodenal (farther down so takes longer): 2-3 hours after a meal, occurs at night, relieved by food Gastric - Immediately after a meal or 30-60 min after a meal, rarely at night, worse with food
88
What is the stomach acid secretion like for PUD depending on if it is duodenal or gastric
Duodenal - Hypersecretion Gastric - Hypo or normal
89
What is weight change like with PUD depending on if it is duodenal or gastric and why?
Duodenal - Weight Gain - since food relieves the pain Gastric - Weight Loss - since it becomes worse with food
90
4 Types of Surgical Interventions for PUD
Vagotomy Pyloroplasty Biliroth I Biliroth II
91
When is surgical intervention for PUD done
if the obstruction or perforation or ulcer wont heal over 12-16 weeks
92
Vagotomy
Surgical Intervention for PUD Involves severing the vagus nerve to decrease gastric acid making them less responsive to gastrin which can help prevent PUD
93
Pyloroplasty
Surgical Intervention for PUD widens the opening of the lower part of the stomach so contents pass easier into the duodenum
94
Biliroth I (Gastroduodenostomy)
PUD Surgery Lower portion of stomach (gastrin release area) and a small part of the duodenum and pylorus are removed and then what remains is resewn to the duodenum Removes the pylorus so risk for dumping syndrome
95
Biliroth II (Gastrojejunostomy)
PUD Surgery Removes lower portion of stomach and connects it to the jejunum Can have dumping syndrome here
96
Nursing Dx for PUD
Pina Fluid and Nutrition Balance Anxiety Home and Community Based Care
97
What are some common complications of PUD
Hemorrhage Perforation and Penetration Gastric Outlet Obstruction
98
___% of PUD pts hemorrhage and present with bloody stool or emesis
15%
99
What does perforation and penetration with PUD cause
erode the serousa --> gastric contents leak into peritoneum (peritonitis) --> EMERGENCY
100
When does gastric outlet obstruction from PUD occur
Area near pyloric sphincter is scarred and stenosed from healing ulcers over time meaning the sphincter cannot function right leading to scar tissue and obstruction
101
T/F: Most pepetic ulcers result from infection with the gram negative bacteria H pylori which may be acquired through ingestion of food and water
True
102
Currently the most commonly used therapy for peptic ulcers is a combination of ___, proton pump inhibitors, and bismuth salts that suppresses or eradicates H Pylori
Antibiotics
103
Chronic Constipation
Fewer than 3 BMs weekly or hard, dry, small, and difficult to pass based on normal BM schedule
104
Clinical manifestations of chronic constipation
straining pain or pressure sensation of incomplete evacuation lumpy hard stools fewer stools
105
Causes of Chronic Constipation
diet - low fiber holding in poop inadequate fluid intake (<8 glasses) being a couch potato / lack of exercise too active leading to being too busy and forgetting or not having time to BM medications: pain meds, chronic laxative use Hypothyroidism and Spinal Cord Injuries
106
Nursing Management for Chronic Constipation should focus on what
education and controlling any pain
107
Ways to prevent constipation
high residue high fiber diet making sure pt is consuming enough fluids unless contraindicated exercising diet
108
Diarrhea
increased frequency of BM (more than 3 / day) and alternative consistency of the stool
109
When is diarrhea considered chronic
when changed consistency and 3/day stools persist 2-3 weeks or more
110
Clinical Manifestations of Diarrhea
Urgency Perianal discomfort from frequency of BM and skin irritation around anus abdominal cramping and distention rumbling in the stomach or intestinal region
111
Causes of Diarrhea
stool softeners antibiotics tube feedings C Diff diabetic neuropathy or pancreatic insufficiency inflammation
112
Complications of Diarrhea
dehydration!! cardiac dysrhythmias low potassium skin irritation around anus
113
What is nursing management of diarrhea focused on
Dehydration!!! But also: Lyte Balance Skin Integrity Accurate Health Hx Exploring Diet and IV Hydration / Lyte Replacement
114
Small bowel disorder leads to what stool characteristics
watery
115
Large bowel disorders leads to what stool characteristics
loose, semi solid
116
Malabsorption syndrome leads to what stool characteristics
voluminous, greasy
117
Inflammatory disorders leads to what stool characteristics
blood, mucus, pus
118
Pancreatic Insufficiency leads to what stool characteristics
oil droplets
119
Diabetic neuropathy leads to what stool characteristics
nocturnal frequency
120
C Diff leads to what stool characteristics
diarrhea, unexplained, and they are on antibiotics which can alter things
121
Diarrhea is defined as the increased frequency of more than 3 bowel movements per day
true
122
Inflammatory Bowel Disease (IBD)
A group of chronic disorders: Ulcerative colitis and Crohns disease
123
Ulcerative colitis
IBD recurrent ulcerations that affect the mucosa and submucosa layers of the colon and rectum (particularly the transcending and descending colon ulcers are often continuous/contiguous and are connected to one another
124
Crohn's disease
IBD - AKA: Regional enteritis Subacute and chronic inflammation of the GI tract that spreads deep into the tissue layers (deeper than UC) of the affected bowel tissue Can happen anywhere mouth to anus but is typically found in the ileum and ascending colon Has a cobblestone appearance because it does go deeper into the bowel layers
125
IBD is most common in what age group
15-30 year olds Young people!: HS Students, College Students, Young Adults Some links to smoking and active smoking for UC but needs more research
126
Location of UC v CD
UC - Colon CD - Mouth to anus
127
What are the lesions like in UC v CD
UC - Contiguous CD - Cobblestone / Not contiguous
128
What are the exacerbations like in UC v CD
UC - Exacerbations and remissions CD - Prolonged bouts
129
What is the diarrhea like in UC v CD
UC: More severe (10-20 bouts QD) CD: Less severe (5-6 bouts of QD)
130
Symptomology of UC v CD
UC: LLQ pain (where descending colon is), passage of mucus and pus, tenesmus (ineffective painful straining), rectal bleeding, anorexia CD: RLQ crampy pain (ileum here), eating stimulates cramps, anorexia, steatorrhea, fever
131
Bleeding of UC v CD
UC: Common and severe CD : not common and mild
132
Fistulas in UC v CD
UC: Rare CD: Common
133
Other Complications in UC v CD
UC - Perforation, Toxic Megacolon --> Bowel perforation CD: Bowel obstruction, abscesses, colon cancer
134
Surgery in UC v CD
UC: Curative (since removal can cure) CD: Non curative (since it can be anywhere it cannot be cured)
135
What sort of pharmacologic treatments are done for IBD
corticosteroids and antibiotics
136
Big concern with IBD is ....
nutritional imbalance the anorexia - IBD often underweight, malnutrition, malnourished - so its common (esp in CD) to see Parenteral nutrition - GI will need rest and anorexia
137
Biggest complication concerns of IBD
electrolyte imbalance cardiac dysrhythmias related to electrolyte imbalances GI bleeding with fluid volume loss perforation of the bowel
138
Nursing Goals of IBD
bowel elimination pain management fluid volume nutrition fatigue anxiety (v bad they are young) coping skin (frequent BM) knowledge (deficit about IBD) self health management complications
139
Nursing Interventions for IBD
Diet, activity and stressors - nutritional therapy ready access to restroom pain management fluid volume and low residue diet --> low gas diet easy to digest rest anxiety and coping skin understanding and self care
140
Irritable Bowel Syndrome (IBS)
chronic functional disorder associated with pain and disordered BMs diagnosed s/s
141
What differentiates IBD and IBS
IBD - the doctor can do an endoscopy and visually see the ulcers IBS - functional disorders means there is no diagnostic finding on colonoscopy (scope shows nothing) - diagnosed based on s/s
142
Clinical Manifestations of IBS
Disorder of frequency and consistency of stool - diarrhea to constipation back and abdominal pain/pain assoc with change in stool and stool appearance and frequency
143
Interventions for IBS
Education Dietary Habits Chew and Dont Drink with Meals - Fluid cause distention Stress Management
144
T/F: The patient with IBS should select foods low in fiber in order to minimize intestinal irritation
False - want them to have high fiber foods
145
In Crohn's disease, the clusters of ulcerations on the intestinal mucosae have a ___ appearance
Cobblestone
146
What are the 3 subclasses of Intestinal Obstructions be
Mechanical v functional small bowel v large bowel partial v complete
147
Mechanical Intestinal Obstruction
Caused from pressure on the intestinal wall and the pressure leads to adhesions, intussusception, inguinal hernia, hernia, or tumor
148
Functional Intestinal Obstruction
"Paralytic Obstruction" When intestinal musculature cannot propel food, cannot do peristalsis, cannot propel weight
149
Common causes for Intestinal Obstructions
Endocrine Disorders and Neurological Disorders
150
What is the difference between partial and complete intestinal obstructions
Parial means only part of the movement is occluded; complete means nothing can move
151
A patient with intestinal obstruction is at significant risk for what
fluid imbalance - critically imbalanced We want to maintain the fluid and lyte balance, insert and NG tube as orders, and be NPO
152
S/S of Intestinal Obstruction
Pain May or may not have BM reported potential mucus of blood in stool abdomen distended!!!! (large and firm) emesis weakness potential weight loss
153
Nursing Interventions for Bowel Obstructions
IV fluids NG tube decompression fluid and lyte replacement surgery - if tissue is strangulated fix root cause - ex: hernia anti nausea meads- not PO, IV or suppository's
154
T/F: Decompression of the bowel through a nasogastric tube is necessary for all patients with a small bowel obstruction
True - if the pt is obstructed they are getting an NG tube
155
General Nursing Considerations Post GI Surgery
1. Resuming enteral intake (PO) - get them back up and moving 2. Dysphagia 3. Gastric Retention 4. Bile Reflux (when pylorus removed/broken) 5. Dumping Syndrome (when pylorus removed/broken) 6. Vit and Min Deficiencies
156
Intestinal Diversion
Allows stool to leave the body when there is disease or injury It is a pouch with a stoma that is from the wall of the colon or ileum v- brought to surface and fused with it
157
Ostomy location depends on...
disease and condition location - depends on where in the GI system is affected
158
What changes based on ostomy location
stool consistency
159
Colostomies
Sigmoidostomy Descending Colon Ostomy Transverse Colon Ostomy Ascending Colon Ostomy
160
Ileostomy Stool
ostomy that bypasses the entire large intestine, so stools are liquid, frequently contain digestive enzymes, and must be pouched at all times has lots of digestive enzymes so can be irritating to skin
161
How do the colostomy stools compare
Sigmoid - stool may be more solid - water absorbed Descending - semisolid, less solid than sigmoid Transverse - more mushy than descending Ascending - liquid stool
162
Ileostomy byupasses what
colon, rectum, and anus
163
Which ostomy has fewest complications
Ileostomy
164
Colostomy
diverts colon to a stoma
165
Ileoanal Reservoir
essentially a "new rectum" large intestine removed but anus remains intact and disease free colon like pouch from last several inches of ileum stool collects and exits during bowel movement
166
Continent Ileostomy (K Pouch)
For pts, with rectal or anal damage who do NOT want ostomy pouch large intestine removed and a Kock pouch is made from the end of the ileum effluent is then drained by inserting a catheter into a valve
167
Ostomy Care education should include
basic assessments size strict I&O effluent monitoring skin care and pouch care diet and medications monitor and report increase or decrease of effluent, stomal swelling, abdominal cramping and distention
168
When does effluent post ostomy surgery appear
not until 24-48 hours after surgery
169
Nursing Dx for Ostomy Care
Disturbed body image Risk for impaired skin integrity r/t to irritation of the peristomal skin by the effluent Imbalanced nutrition: less than body requirements r/t avoidance of foods Anxiety r/t to the loss of bowel control Risk for deficient fluid volume Sexual dysfxn Deficient Knowledgeo
170
Ostomy Irrigation
to stimulate emptying at scheduled times note always in routine care but can help stop unplanned bowel movements or fecal drainage in social situations gives pts control
171
T/F: The pt with an ileostomy with a Kock Pouch will not need to use an external collection bag
True
172
What is the main risk factor for esophageal cancer
barrets esophagus
173
what gender is more likely to get esophageal cancer
men
174
what race is more likely to get esophageal cancer
African American
175
Risk factors for esophageal cancer
smoking ETOH use gender age comorbidities
176
One of the number one complaints about esophageal cancer is what
dysphagia - trouble swallowing sensation in throat or something is getting stuck *also weight loss and weakness
177
by the time esophageal cancer symptoms appear
the cancer has advanced
178
Diagnostics for Esophageal Cancer
biopsy and endoscopy
179
Treatments for Esophageal cancer
chemo radiation re-sectioning esophagus with part of small intestine
180
What gender and races are more likely to get gastric cancer
men > women native america, hispanic, african american > caucasian
181
Risk Factors for Gastric Cancer
poor diet smoking alcohol use gastritis
182
How does gastric cancer present
clinical manifestations present like PUD undiagnosed until CT scan
183
Diagnostic for Gastric Cancer
CT Scan
184
Treatments for Gastric Cancer
chemo and radiation - may be palliative not curative total gastrectomy if it hasnt spread and is caught early
185
Duodenal Tumors
Usually benign and diagnosed incidentally present asymptomatic if severe, intermittent pain and occult bleeding occurs can be removed with surgery
186
3rd most common cause of cancer death is via ____ cancer
colorectal
187
chief sign for colorectal cancer
change in bowel habits!!!' *second most common manifestation is blood in stool
188
____ is the most prevalent cancer diagnosis in colorectal cancers
adenocarcinomas
189
How is colorectal cancer diagnosed
via colonoscopy and biopsy
190
Tenesmus
recurrent inclination to evacuate bowels - can be painful or spasming sensation
191
Risk Factors for colorectal cancer
increasing age - >50 yo family hx of colon cancer or polyps high consumption of ETOH cig smoking obesity hx of gastrectomy hx of inflammatory bowel disease high fat, high protein (with high intake of beef), low fiber genital cancer (endometrial CA< ovarian CA) or breast CA (in women)
192
S/S of Colorectal Cancer
Right Sided Lesions - Dull abdominal pain and melena Left Sided Lesions - abdominal pain, cramping, narrowed stools, constipation, distention, bright red blood Rectal lesion - tenesmus, rectal pain, feeling of incomplete evacuation after a BM. alternating constipation and diarrhea, bloody stool
193
The etiology of cancer of the colon and rectum is predominantly (90%) ____, a malignancy arising from the epithelial lining of the intestine
adenocarcinoma