Exam 1 Flashcards

1
Q
A

Normal Sinus Rhythm

Rate 60-100 bpm

Regular Rhythm

QRS w/ each P-wave

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2
Q
A

Sinus Bradycardia

Rate < 60 bpm

Causes: MI, surgical procedure, medications (digoxin, beta-blockers, morphine, vagal stimulation

Symptoms: some pts. maybe asymptomatic (athletes, etc.); syncope, dizziness, lightheadedness, confusion, dyspnea, n/v, decreased UOP, cool & clammy skin

Treatment (if symptomatic): provide O2, increase intravascular volume (IV fluids), atropine is the drug of choice (increases HR), pacing may be necessary if other interventions unsuccessful

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3
Q
A

Sinus Tachycardia

Rate > 100 bpm

Causes: Sympathetic nervous system stimulation, drugs (caffeine, nicotine, epinephrine, dopamine, atropine), compensatory response to decreased BP or CO (shock, infection, MI, HF), pain, fear, anxiety

Symptoms: fatigue, weakness, SOB, palpitations, chest pain

Treatment: treat the underlying cause (fever = antipyretics, anxiety = provide reassurance, pain = administers meds as ordered, hypovolemia = replace fluids or blood), medications (digoxin, beta-blockers, diuretics)

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4
Q
A

Supraventricular Tachycardia

P-wave present but difficult to identify, PR interval not measurable, rate 150-250 bpm

Causes: emotions, stimulants, rheumatic heart disease, digoxin toxicity, MI

Symptoms: chest pain, palpitations, fatigue, anxiety, SOB, hypotension, dizziness, syncope

Treatment: vagal stimulation (have patient bear down), administer adenosine, cardioversion, ablation

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5
Q
A

Atrial Fibrillation

Most common dysrhythmia, risk increased with age, increases the risk of stroke

NO distinct p-wave (may be mistaken for fibrillation beats), PR interval absent, irregular rhythm, rate varies

Cause: multiple rapid-firing impulses from atria (350-600x/min.); HTN, CAD, DM, HF, mitral valve disease, obesity, caucasian, thrombolytic event

Treatment: GOAL = anticoagulation and rate control, medications (anticoagulants & antidysrhythmics), cardioversion, ablation, pacemaker for rate control

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6
Q
A

Atrial Flutter

P-wave absent (can be mistaken for flutter waves which appear “saw-toothed”), PR interval not measurable, rhythm can be regular or irregular, rate varies

Causes/Symptoms/Treatment: same as A. fib

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7
Q
A

3rd Degree Heart Block

LIFE-THREATENING - can progress to asystole

Electrical impulses blocked btwn. atria and ventricles

P wave sam size and shape but no correlation to QRS, no true PR interval, QRS usually widened, rate varies

Causes: MI, severe heart disease

Treatment: temporary pacing until a permanent pacemaker can be placed once the patient is stable

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8
Q
A

Premature Ventricular Contractions

Not medical emergency but could be a red flag

P-wave not present before PVC, QRS widened, irregular rhythm, the rate varies

Causes: MI, heart failure, caffeine, alcohol, nicotine, stress, infection, surgery, electrolyte imbalance, digoxin toxicity

Treatment: If patient medically stable then continue to observe, begin treatment if the patient shows s/s of poor cardiac output (fatigue, hypotension, cool extremities, dizziness, thready pulse, etc.) or if any “danger signs” ( > 6 PVCs/min, multifocal PVCs, run of V. tach), treat underlying cause, antiarrhythmics

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9
Q
A

Ventricular Tachycardia

May be intermittent or sustained

LIFE-THREATENING, significantly decreased cardiac output

Pulseless V. tach is a SHOCKABLE RHYTHM

No p-wave, no PR interval, wide QRS, rate > 150 bpm

Causes: ischemic heart disease, MI, cardiomyopathy, valvular heart disease, HF, drug toxicity, electrolyte imbalance

Treatment: depends on the severity of the patient, cardiovert (pulse)/defibrillate (pulseless), medications, ablation, pacemaker or ICD placement

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10
Q
A

Torsades De Pointes

LIFE-THREATENING

No p-wave, no PR interval, QRS usually same shape (widened, varied amplitude), rhythm & rate vary

Causes: MI, severe heart disease, low magnesium, drugs that prolong QT

Treatment: Magnesium sulfate, treat underlying cause (correct electrolyte imbalance, remove med that is prolonging QT), cardioversion

** if pulseless, begin CPR and follow treatment guidelines for V. Fib and pulseless V. tach

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11
Q
A

Ventricular Fibrillation

Results from electrical chaos w/i cardiac tissue

LIFE-THREATENING

SHOCKABLE RHYTHM

No p-wave, no PR interval, no QRS, irregular rhythm, cannot measure the rate

Causes: CAD, MI, electrolyte imbalance, medications, SVT, shock, surgery, trauma

Symptoms: LOC, loss of pulse, apnea, pt. becomes faint

Treatment: check pulse, initiate CPR, maintain airway, defibrillate, medications

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12
Q
A

Idioventricular/Agonal

Dying heart, final attempts of heart to make electrical impulse, usually seen in end-stage heart disease

NOT a shockable rhythm

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13
Q
A

Asystole

Lethal dysrhythmia, pt. will NOT have a pulse, immediate interventions necessary

NOT a shockable rhythm

No p-wave, no PR interval, no QRS, no rhythm, no rate

Treatment: treat cause (H’s and T’s), CPR, medications, airway maintenance

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14
Q

Temporary Pacemaker

Types

A
  • Transvenous - through a vein (usually r. femoral), lead wire threaded through the skin and large vein into the right atrium, electrical impulse stimulates atria to produce a contraction, can also be used for ventricular pacing
  • Transcutaneous - electrical impulse sent through skin and body to the heart, stimulating a contraction
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15
Q

Permanent Pacemaker

A
  • Necessary when heart is unable to maintain a normal rate or cardiac output
  • Surgically implanted under the skin in the upper right or left chest
  • lead wire inserted in heart through large vein
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16
Q
A

Atrial Pacing

Inserted in the right atrium, stimulates atria and then follows normal conduction through the heart

17
Q
A

Ventricular Pacing

Inserted through the left or right ventricle, stimulates depolarization of ventricular muscle

18
Q
A

Sequential Pacing

Most common permanent pacemaker

Stimulates depolarization of atria AND ventricles

Leads usually placed in right atrium and ventricle

AKA “dual-chambered pacemaker”

19
Q

Biventricular Pacing

A

Used when ventricles contract at different times, decreasing cardiac output

Three leads are placed: one in atria, one in right ventricle, one in left ventricle

20
Q

__________ is the ability of cardiac cells to depolarize in response to the electrical impulse generated by a pacemaker.

A

Capture

21
Q

What indicates capture?

A

P wave or QRS after every pacer spike

22
Q

What is the difference between capture and mechanical capture?

A

Capture indicates electrical impulse but does NOT always mean there is a contraction

Contraction = pulse present = mechanical capture

23
Q

Percent of Capture

A

Number of pacer spikes followed by a complex / Total number of pacer spikes

24
Q

Failure to Capture

A

Complex does not follow pacer spike meaning, cardiac cells did not depolarize in response to stimulation from pacemaker

25
Q

Implantable Cardioverter Defibrillator

A

Used for a hx of lethal dysrhythmias

Surgically implanted under skin w/ lead wires inserted into atria and one or both ventricles

Programmed to deliver shock when HR becomes too rapid ( > 150)

May increase voltage and shock again if the first shock does not convert the rhythm

Patient CAN feel shock

26
Q

Patient education for pt. with Pacemaker or ICD

A
  • Do not shower for 24 hours following the procedure
  • No baths for 2 weeks
  • Need to wear affected arm in a sling
  • Do not raise the affected arm above head for 2 weeks
  • No lifting > 10 lbs for 2 weeks
  • Always wear a medical alert card
  • NO MRIs
  • NO contact sports
27
Q

What are shockable rhythms?

A

V. fib

Pulseless V. tach

Pulseless torsades

28
Q

What is the ratio of compressions to breaths during CPR?

A

30:2

29
Q

What is the order of steps taken during a code situation?

A
  1. Assess patient (pulse?)
  2. Call for help
  3. If pulseless begin CPR & attach to AED
  4. If shockable rhythm, defibrillate immediately, deliver shock and resume CPR for 2 minutes
  5. Reassess pt., check pulse and rhythm
  6. Repeat step 3

**administer meds as necessary:

Epinephrine every 3 min. - 1 gram

Amiodarone every 3 min. - 300 mg

Vasopressin ONCE to replace wither 1st or 2nd dose of epi

Meds to treat the underlying cause (H’s and T’s)

30
Q

H’s and associated treatments

A
  1. Hypovolemia = fluid resuscitation
  2. Hypoxia = supplemental O2
  3. Hydrogen Ions (acidosis) = treat cause, sodium bicarbonate
  4. Hypothermia = rewarm
  5. Hyperkalemia = calcium gluconate
  6. Hypoglycemia = check glucose and replace if indicated (D10)
31
Q

T’s and associated treatments

A
  1. Tablets or toxins = treatment for the specific agent
  2. Trauma = pericardiocentesis
  3. Thrombosis = treat per cause
  4. Tension pneumothorax = needle decompression w/ eventual chest tube
  5. Tamponade = surgical evaluation
32
Q

S/S of Deterioration that Warrant Calling Rapid Response

A

Shortness of breath with O2 sat lower than 92% or RR very low or high

Dysrhythmias

BP – severely hypotensive or severely hypertensive

Altered level of consciousness

Stroke – slurred speech, unilateral weakness

Urine output

Wacky electrolyte levels

Abnormal ABGs

Nursing judgement - patient just doesn’t look right