EXAM 1 Flashcards
tx of anaphylactic shock
Epinephrine, antihistamines, H2-blockers (Zantac), Bronchodilators (albuterol), Steroids
be fast
balance eyes facial dropping arm /extremity speech time
major cause of TIA
atherosclerosis
clot formation = blocks passage of blood through artery
thrombotic ; most common
blockage in the brain
ischemic
bleeding in the brain
hemorrhagic
major cause of hemorrhagic
htn
what do you not give a patient with hemorrhagic stroke
anticoag and antiplatelet
bleeding INSIDE brain caused by RUPTURE OF VESSEL
intracerebral hemorrhagic
s/s of intracerebral hemorrhagic
headache and nausea / vomitting
patient reports the worst headache of their life
subarachnoid hemorrhagic
receptive aphasias- LANGUAGE COMPREHENSION
Wernicke’s
expressive aphasia- SPEECH CONTROL
Brain stem- breathing
broca
PERSONALITY MOTOR FUNCTION HIGHER LEVEL SOLVING SKILL
frontal lobe
elevated bp in ischemic is what
this is ok because body is trying to maintain perfusion
platelet inhibitors to give
Aspirin 325 mg
clopidogrel (Plavix)
iicp can be caused by primary or secondary
primary: iicp happens at time of injury, trauma from a car accident
secondary: follows a primary injury. Hypoxia, ischemia, hypotension, edema and ultimately IIICP
normal icp
5-15
elevated icp
greater than 20
how do we check if there is icp
pressure transducer
1 cranial nerve
olfactory
2 cranial nerve
optic
3 cranial nerve
oculomotor
4 cranial nerve
trochlear
5 cranial nerve
trigeminal
6 cranial nerve
abducens
7 cranial nerve
facial
8 cranial nerve
acoustics
9 cranial nerve
glossopharyngeal
10 cranial nerve
vagus
11 cranial nerve
accessory
12 cranial nerve
hypoglossal
cerebral perfusion pressure
60-100
less than 50 is neuro death and ischemia
what to monitor for icp
glasglow coma scale best score 15
seizure safety
sensory/motor impairment
speech changes
priorities for icp
airway
fluids and electrolytes
most reliable indicator of patients neuro status
change in loc
what is assessed is glascow coma scale
eyes
verbal
motor
icp hallmark signs
cushings triad : systolic hypertension with a widening pulse pressure, bradycardia with a full and bounding pulse, and irregular respirations
posture that is worse
decerebrate
what do you not want to do for a person suspected of icp
no lumbar puncture because it can cause herniation
what is the golden standard for monitoring icp
ventriculostomy
pao2
greater than or equal to 100
paco2
35-45
characterized by decreased reflexes, loss of sensation, and flaccid paralysis below the level of the injury.
This syndrome lasts days to months and may mask post injury neurologic function.
spinal shock
what do you look at for a spinal cord injury
dermatomes tell you the level of impairment
loss of vasomotor tone caused by injury and is characterized by hypotension and bradycardia.
Loss of sympathetic nervous system innervation causes peripheral vasodilation, venous pooling, and a decreased cardiac output.
neurogenic shock
neurogenic shock is usually…
t6 or higher injury
gold standard in diagnosing stability of the injury, location and degree of injury, and degree of spinal canal compromise.
ct scan
assess for soft tissue and neurologic changes and when there is unexplained neurologic deficit or worsening of neurologic status.
MRI
rules out any vertebral artery damage
ct angiogram
what can a neurogenic bladder lead to
autonomic dysreflexia
if no gag reflex….
plan for intubation
above t6 injury
decrease in SNS, bradycardia, atropine to increase hr
prevention of dvt
enoxaparin
used to maintain the mean arterial pressure at a level greater than 90 mm Hg so that perfusion to the spinal cord is improved.
dopamine / intropin
what to monitor with patient who has a SCI
monitor bp; high bp but low heart rate
intervention for their bp
sit them up to prevent postural hypotension
nursing management for sci
maintain ventilation; 2) Intact skin; 3) Bowel and bladder management program; 4) no episodes of autonomic dysreflexia
med given for spinal cord tumors
dexamethasone for edema
glascow coma scale
less than 8 intubate
loss of csf
rhinorrhea and otorrhea
wbc
3-12
hemoglobin
12-18
bun
10-20
creatinine
0.6-1.2
fasting glucose
70-100
a1c
4-5.6
a1c for diabetics
less than 7.0
total cholesterol
less than 200
ptt
20-35 seconds
pt
11.2-13.2 seconds
inr therapeutic
2-3.5
why is last known normal with a stroke patient important
tpa time frame is up to 4.5 hours
testing labs for stroke
CT scan (#1), weight (tpa), glucose a1c, lipids, coagulants, ekg (looking for dysrhythmias), echo, mri, carotid ultra sound, dysphasia screening
two anti platelet
aspirin and clopitagril
what does NIHSS tell us
tells us their disability - higher the # the worse they are. neuro checks (orientation, LOC, speech, and extremity testing such as pronator drift)
diabetes, hyperlipidemia, previous stroke or tia, dysrhythmia, smoking, hypertension, sedentary lifestyle, cardiac abnormalities
risk factors for stroke
black label warning medication for headaches
imitrex given for cad patients
pill rolling (tremors), shuffle gait (bradykinesia), cog wheel (rigidity)
parkinson disease
what is issue with dopamine
parkinsons
tx for parkinsons
sinamet (levodopa/carvidopa)
what are we worried about with sinamet (levodopa/carvidopa)
- Worried about adherence, dosage changes and it is divided throughout the days
- Need consistent amount of moderate protein
- Can be a wearing off of medications
increased dopamine; genetic
hunting tons disease
what do we want to do with pt for huntingtons
they experience chorea- increased movements so we need to increase their caloric needs
progressive muscle weakness that happens in the upper body
myastenia gravis
what are we worried about w MG patient
respirations so do a respiratory assessment
interventions for patient with ALS
Care planning Cognitively intact Communication Passive/ ROM Skin assessment Respiratory assessment
signs and symptoms of multiple sclerosis
bladder or vision disturbances
sclerotic plaques on myelin sheath
multiple sclerosis
MS paralysis
it can happen during a flare and tx w steriods to decrease the inflammation
intervention for MS
try to get ADLs is at once and quickly
intervention for epilepsy/seizure
find out what triggers are so they can stay away from them
lights, stress, smells, lack of sleep, caffeine, exercise, alcohol
some triggers for a seizure patient
tx for status epi
benzos; ending in pam
unwitnessed seizure what are we worried about
head and spinal cord injury so we would do assessments to rule this out
tx for seizures
gabapentin, keepra, Dilantin, Topamax
what is important for seizure meds
taper them down because abrupt stopping can cause seizures
trigeminal neuralgia
cranial nerve 5
tx for trigeminal neuralgia
antiepileptic drugs
bells palsy
cranial nerve 7
Cause of it being HSV or herpes, meningitis, tooth infection or any upper infection
bells palsy
what does bells palsy mimic
stroke
can you recover from bells
yes
Autoimmune response to something such as Vaccine, viral illness, bacterium
o Guillain-Barre
Guillain-Barre is paralysis
ascending which means it works its way up so we are worried about respiratory muscles
can you recover from guillain barre
yes
interventions for Guillain-Barre
assessments, respiratory, psychosocial
how do you treat viral meningitis
symptomatically
how do you treat bacterial meningitis
antibiotics
droplet isolation for which meningitis
bacterial
Stiff neck, nuchal rigidity, headache, photophobia, change in LOC, nausea and vomiting, papilledema
meningitis
what can cause icp
trauma, edema, fluid build up, tumor
interventions for patient with icp
having bowel movements, coughing, and turning patient
bruising over the mastoid process as a result of a brain bleed
battle sign
what to look out for with pt with icp
rhinorrhea, battles sign, halo sign
spinal cord injuries
autonomic dysreflexia
how do you know if a person with MOD is getting better
LOC improve, I and o is good, BUN, organ labs improve
dysphasia
difficulty speaking
mannitol
used as a diuretic to reduce cerebral edema
what are we checking for MODs
LOC, temp, color
management for mod
preventing dic; put them on heparin or lovanox, monitor for bleeding, bruising on skin
• Need to know when it started, stopped, what it looked like, how long, new onset
seizures
phases of seizure
- 1) prodromal (preceding seizure)
- 2) aural (sensory warning before seizure)
- 3) ictal (full seizure)
- 4) postictal (recovery after seizure)
what kind of diet can we recommend for seizure patient
ketogenic; high fat low carb
drug given for seizures and what do we want to monitor with it
Phenytoin (Dilantin)- liver complications
diagnostic lab for seizures
eeg
drugs given for symptoms with migraines
aspirin, acetaminophen, analgesic combo, imitrex
what to be careful about with imitrex
*(avoid with heart disease/constriction of arteries)
prophylactic drugs for migraines
topamax; be careful with abruptly stopping
which is worse decorticate or decerebrate
decerebrate more severe (brainstem and midbrain damage)
Kernigs sign
lifting knee up
Brudinski
lifting head up while supporting
modifiable risk factors associated with stroke
HTN, DM, hyperlipidemia, smoking, excessive alcohol, sleep apnea untreated, drug use (risk for hemorrhagic-cocaine), obesity, sedentary lifestyle, heart disease
non modifiable risk factors with stroke
age, gender, race (African American-higher risk), family history
o BUT REALLY: A fib, smoking, sedentary lifestyle, DM, excessive alcohol use
most common stroke
ischemic Thrombotic
which will you see collateral circulation
thrombotic because it is slower onset ; this will show up on a ct scan
rapid occurrence and lack of collateral circulation
embolic
major cause of hemorrhagic strokes
htn
any stroke patient…
is made NPO until they have receive dysphagia screening
what can you give for stroke patient and how is it given
aspirin asap unless they fail screening then it can be given rectal
total inability to communicate; massive stroke
global aphasia
loss of production of language
expressive aphasia
loss of comprehension
receptive aphasia
impaired ability to communicate; used interchangeably with aphasia
dysphagia
ok high bp in
ischemic stroke
how is NIHSS score
want it to be 0; higher the number the worse patient is
drug approach for restless leg syndrome
Sinemet (also used for PD), Requip, Mirapeto increase dopamine in brain
• Gabapentin: to decrease sensory sensations
disseminated demyelination of nerve fibers in brain and spinal cord; relapsing/remission disease
multiple sclerosis
• Weakness, paralysis of limbs, speech patterns delayed/interrupted, patchy blindness (early S&S), tinnitus, vertigo, sex dysfunction, bowel/bladder impairments, dysphagia, ataxia, dysarthia (weak speech muscles)
multiple sclerosis
“shock” sent down leg when neck flexed
• Lhermitte’s sign in MS
tx pt with MS
do adl in morning because they fatigue, may have to straight cath themselves
lack of dopamine in the brain
parkinson
unusual clumps of proteins; found in brains; contribute to dementia
lewy bodies with parkinson
what to take for parkinson and why do some people not take it
sinomet- carvidopa; it takes weeks for effect so some people think it is not working
weakness in certain skeletal groups (UPPER body); antibodies attack acetylcholine (ACh) receptors
myasthenia gravis
intervention for MG
rr, pulmonary assessment
in MG crisis they are high risk for
aspirating
drug for mg
Mestinon
what slows the process of also / drug
riluzole (Rilutek)
limb weakness, dysarthria (facial muscle weakness), dysphagia (difficulty swallowing), muscle wasting, fasciculations (involuntary muscle twitching), drooling, depression, sleep disorder, esophageal reflux
als
genetic, autosomal dominant
huntingtons disease
excessive dopaminergic(DA), excessive involuntary movements (CHOREA)
hunt
interventions for hunt
increase caloric needs due to increase in movement
cpp needs to be
60-100
eyes GCS
4-1
verbal GCS
5-1
motor GCS
6-1
• Minor head injury: > 13 Moderate: 9-12 Severe: <8
GCS
Pt comes in with cerebral edema…. what do we want to look out for
pt complaining of a headache
cushings triad tells us about
icp
increased SBP, decreased HR and RR
icp
decreased BP, increased HR and RR
shock
abnormal with basilar skull fracture
temp being high
what is the risk of otorrhea and rhinorrhea
risk for meningitis with high csf leak
bruising of brain tissue
contusion
brain hitting front or back of skull;worried about a c spine injury
Coup-Contrcoup injury with contusion
what to ask pt with a contusion
are they on anticoags ? increase risk of hemorrhage
sudden transient mechanical injury with disruption of neuronal activity and change in LOC
concussion