Exam 1 Flashcards
Otitis externa etiologies
P. aeruginosa
S. aureus
Influenza etiologies
Type A > Type B»_space;> Type C
Pertussis etiologies
Bordetella pertussis
Atypical pneumonia etiologies
Mycoplasma pneumoniae
Legionella pneumophila
Chlamydiae pneumoniae
Croup etiologies
PIV 1 > PIV 2»_space;> RSV
Diphtheria etiologies
C. Diphtheriae
CAP etiologies
Strep pneumoniae
H. influenzae
K. pneumoniae
S. aureus
ARD etiologies
Adenovirus or unknown virus > Rhinovirus > Coronavirus
Otitis media/sinusitis etiologies
S. pneumo
H. influenzae
M. catarrhalis
SARS/MERS etiologies
Novel coronavirus
HAP etiologies
P. aeruginosa»_space;> S. aureus, K. pneumoniae
Infant pneumonia
C. trachomatis
Alpha hemolytic
Strep pneumo
Catarrhal vs. paroxysmal vs. convalescent stage
Bordetella pertussis
“Palisades” or V shaped
C. diphtheriae
Currant jelly sputum
Klebsiella pneumoniae
Beta hemolytic
Staph aureus
Quadrivalent vaccine
Influenza
Aquatic and pleomorphic
Legionella pneumophila
Attachment organelle which causes ciliostasis
Mycoplasma pneumoniae
Non-segmented
PIV
Oxidase positive
Beta lactamase producer
M. catarrhalis
Coagulase positive
Staph aureus
Pyocyanin vs. Pyoverdin
Pseudomonas aeruginosa
Loeffler’s medium + cysteine-tellurite agar
C. diphtheriae
Alginate capsules = biofilms
Pseudomonas aeruginosa
Sensitive to optochin
Bile sensitive
Strep pneumo
Nonencapsulated virulent strains
H. influenzae
Sxs due to inflammatory response
RSV
Acute flaccid myelitis is a complication of _____.
ARD
Likes to live in amoeba/macrophages
Legionella pneumophila
Pneumolysin
Strep pneumo
LOS instead of LPS
H. influenzae
Pneumonia, Reyes syndrome, and Guillain Barre are complications of _____.
Influenza
Bull neck
Pseudomembrane
C. diphtheriae
Elek test
C. diphtheriae
Has 4 exotoxins apart of their virulence
Bordetella pertussis
MacConkey agar
Klebsiella pneumoniae
Obligate intracellular parasite
Viruses
C. trachomatis and C. pneumoniae
Generates ROS
Pyocyanin (P. aeruginosa)
Elastases (Las A and B) = tissue damage
Pseudomonas aeruginosa
Bordet Gengou and Regan Lowe agar
B. Pertussis
Neuramidinase
H. influenza and influenza (neuraminidases are different for each one though)
FAB test
Pseudomonas aeruginosa
Associated with atherosclerosis
C. pneumoniae
Fried egg appearance on culture
Mycoplasma pneumoniae
Pontiac fever
Legionella pneumoniae
No peptidoglycan
Mycoplasma pneumoniae
Elementary vs. Reticulate bodies
Chlamydia (both)
Natural competence (Griffith’s experiment)
Strep pneumo
NEVER treat with B-lactams
Mycoplasma pneumoniae
Treat with penicillins
Strep neumo
Tx w/ monoclonal immune globulin
High risk pts with RSV
Removes ATP from host
Chlamydia
Secretoy IgA
Strep pneumo (prevents organism from getting stuck in mucus)
Otitis media and parotitis are complications of _____.
PIV
WTA and/or LTA are a component of _____ bacteria. What is their function?
Gram +
Attachment
Mycolic acid layer is a component of ____ bacteria.
Acid fast
LPS is a component of _____ bacteria. Describe its 2 components.
Gram -
Lipid A: toxic moiety
O antigen: attachment
Porin proteins
Gram - or acid fast cell walls
Sexi pili are a component of some _____ bacteria
Gram -
not in +
Optical density measures:
Absorbance of light
measures viable AND nonviable
Colony forming unit measures
viable cells (only living cells will form colonies)
Biomass measures
viable and nonviable cells
takes dry weight of all cells in culture
Pathogens produce virulence factors in the _____ phase of the bacterial growth curve.
Log
Describe enriched media
complex + growth factors
Describe selective media
selects against unwanted organisms
Describe differential media
differentiates between organisms
The most pathogenic type of bacteria are
mesophiles
Endotoxins exist in the:
LPS of gram - organisms (not toxic until released)
Accumulation of genetic mutations
antigenic DRIFT
Reassortment of viral genome
antigenic SHIFT