Exam 1 Flashcards
What all is part of the Cognitive-Affective Domain?
Level of consciousness Communication Orientation Attention Memory Perception/Judgment Mood and Pain
Alert
Eyes open, interacting with their environment
Lethargic
Drowsy, in and out of consciousness, not attending to tasks
Glasgow Coma Scale
Level of consciousness and severity of injury with trauma to head/brain
Assessed by MD in ER/ICU
What levels of consciousness may PT be done?
Active participation in alert and lethargic states
PROM and positioning if unconscious
Difference between Orientation x3 and Orientation x4
x3: Person, place, and time
x4: Person, place, time, and diagnosis
Aphasia
Inability to comprehend written or spoken language, formulate written or spoken language, or use written or spoken language
Expressive Aphasia
Non-fluent Broca's Aphasia Limited vocabulary Slow, hesitant speech with good auditory/reading comprehension (communication board) May have impaired written skills
Receptive Aphasia
Fluent
Wernicke’s Aphasia
No problem speaking, no comprehension
Global Aphasia
Inability to produce or comprehend language
Dysarthria
Lesion in brain that mediates speech production
Labored speaking skills, but understand communication and can express in written
Aphonia
Inability to produce speech due to paralysis of vocal cords or diaphragm
Broca’s Aphasia Physiology
Lesion of frontal lobe (anterior motor association - inferior frontal gyrus)
Usually seen with CVA/TBI
Refer to SLP for assessment
Wernicke’s Aphasia Physiology
Lesion in posterior portion of temporal lobe and part of parietal lobe
Reading and writing are impaired
Dysarthria Physiology
Acquired
Impairment of speech production from damage to the PNS/CNS causing weakness of the motor/speech system (respiration, phonation, articulation, resonance)
Production is not altered, but melody and rhythm changed
CP, TBI, CVA, MS, PD, ALS
May coexist with aphasia in CVA/TBI
Attention
Ability to select and attend to a specific stimulus while suppressing extraneous stimuli
Ability to remain on task
Amnesia
State of memory dysfunction
Perception
Ability of an individual to recognize and interpret sensory information, high order function as it requires the individual to integrate auditory, sensory, visual, and somatosensory information
Should be screened for any type of brain injury
Clinical indicators of perceptual difficulty
Brain Damage Unable to perform self ADLs/simple tasks Difficulty initiation/completing a task Unable to identify common onbjects Unable to follow simple directions
Unilateral Neglect
Not aware of one half of their body/plate/environment
Typically right-sided brain injury that impairs left side
Brain damage over eye damage
Lesion located in Right parietal lobe (parietal sensory association cortex)
Often right middle cerebral artery CVA
Clinical measures of neglect
Line Bisection: draw horizontal line and ask to draw line bisecting (bisect line to the right of the center in neglect)
Cancellation tests: cross out all objects they see on piece of paper
Hemianopia
Defective vision in half of the visual field
Lesion in optic pathway or tract
Check visual fields
May present with hemianopia and neglect
Apraxia
Movement disorder resulting in inability to perform movements without underlying cause
Cognitive deficit that interferes with motor planning
Lesion in frontal and posterior parietal lobes
i.e. when given scissors, try to use as a comb
Types of Apraxia
Ideomotor
Ideational
Constructional
Childhood apraxia of speech
Ideomotor Apraxia
Disconnect between idea of a movement and the execution
Knows what to do but can’t
Can’t walk on command, but can get up and get a coffee
Lesion in frontal and/or posterior parietal lobe
Ideational Apraxia
Inability to figure out how to perform a task
Given a hairbrush and told brush hair, but doesn’t know how to
Lesion in parietal lobe
Constructional Apraxia
Faulty spatial analysis and conceptualization of task
Unable to understand relationship of parts to whole
Can’t put sandwich together even though they are given the ingredients; all parts of clock are there but not correct
Lesion in posterior parietal lobe
Childhood Apraxia of speech
Developmental challenge in the brain and can’t form speech correctly
2-4 years: issues getting the wrong sounds/words
Won’t just grow out, refer to SLP
Judgment
Part of executive function
Impairments lead to impulsiveness, socially inappropriate behavior
Assess executive function if they can figure out how to fold/open a walker, find number in phone book, differentiate photos
Mood
Affects participation and treatment
What are the emotional states?
Fatigue Anxiety Depression - impacts recovery Delusion Disinhibition - inappropriate expression of emotion Aggressiveness Apathy
Components of Musculoskeletal Domain
Strength: need at least 3/5 for most functional activities
ROM: visual screening followed by goni in limited range (AROM and PROM)
Posture: sitting and standing
Pressure Sores
Most common preventable secondary condition PTs need to assess for in neurologic dysfunction
Bony prominences at risk for pressure ulcers
Calcaneus
External Occipital Meatus
Sacrum
Causes of Ulcer
Immobility Moisture Lack of Sensation Poor Oxygenation Poor Nutrition Shearing Force
Trophic skin exam changes
Skin color: venous stasis (bluish/purple in LE)
Skin texture: smooth and shiny, absence of hair
Skin Temp: cool = impaired; hot=infected
Edema
Calf pain + increased temp + edema may = DVT
Braden scale: administered by nursing to determine risk
Upper Motor Neuron Lesions (cortex, brainstem, corticospinal tracts, spinal cord)
Spasticity/Hypertonia Exaggerated reflexes Babinski and Clonus Synergy Weakness Disuse Atrophy
Lower Motor Neuron Lesions (cranial nerves/nuclei, spinal cord - AHC, spinal root, peripheral nerve)
Flaccidity/Hypotonia Decreased or absent reflexes No primitive reflexes Weakness Atrophy due to lack of innervation (severe wasting)
Reflexes
Muscle
Developmental: ATNR; STNR; postural synergy patterns: observed muscle hypertonicity and gross movement patterns
Primitive Reflexes: babinski, clonus
Cranial Nerve Testing
UE Synergy Patterns
Flexor component: elbow, wrist, and finger
Extensor component: shoulder ADD and IR, and forearm pronation
LE Synergy Patterns
Extensor component: Hip IR and ADD, knee extension, plantarflexion and inversion
Tonal Abnormalities
Flaccidity Hypotonia Normal Spasticity Dystonia Rigidity
Spasticity
Resistance to external stretch increases with increasing speed of stretch and varies with direction of movement
Resistance to external stretch rises above threshold speed
Dystonia
Involuntary sustained or intermittent muscle contractions cause twisting and repetitive movements, abnormal postures, or both
Rigidity
Resistance to external stretch is present at very low speeds of movement, does not depend on speed, and doesn’t exhibit speed or angle threshold
Spasticity Physiology
Injury to pyramidal tracts (corticospinal)
CVA, CP, TBI
UMN Syndrome (babinski and clonus)
Chronic spasticity leads to contractures, deformity, activity limitations
Clasp knife response: feeling of letting go of resistance when stretching spastic muscle (autogenic inhibition)
Modified Ashworth Scale
Grades tone from:
0 (no increased tone)
4: Extremely stiff
Rigidity Physiology
Basal Ganglia lesion
Leads to contractures and activity limitations
Types of Rigidity
Cogwheel: alternating letting go and increased resistance (PD)
Leadpipe: constant
Decorticate: UE flexed and LE extended; above superior colliculus, reduce stimuli and position (TBI)
Decerebrate: UE and LE extended; between vestibular nucleus and superior colliculus; reduce stimuli and posture (TBI)
Positions that affect tone
UE: antigravity: UE flexed, adducted, wrist flexed = spasticity
LE: antigravity: LE extended, adducted, plantarflexed: spasticity
Dynamic components of tone
Volitional effort Arousal Stress Position Medications Temperature
Functional Balance Tests
Berg/PBS Tinetti TUG and TUDS Dynamic Gait Index; Functional Gait Assessment BOT-2 Pediatric Functional Reach
Coordination
Ability to execute smooth, accurate, controlled motor responses
Based on intact and functioning nervous system
Fluidity of movement
Coordination characterized by:
Appropriate speed Distance (dysmetria) Direction Timing Accuracy
Dysdiadochokinesia
Problems with alternating pronation/supination
Dysmetria
Problem with pointing and past pointing
Tremor
Either a resting tremor or intention worsens
Bradykinesia
Slowed gross and fine motor movement
Sensation Testing
Results from sensory testing => clinical judgments about diagnosis, prognosis, and outcomes
Sensory input impacts motor output
Sensory Testing Principles
Position Patient: informal with supine Socks off Strict about eyes closed Simple instructions Remove clothing Obscure vision
Sensory System classification
Receptors
Spinal Pathways
Receptors
Mechano, thermo, chemo, and pain
Cutaneous: nociceptors, Meissner’s corpuscles (touch), Pacinian corpuscles (deep pressure); Merkel’s disks (light pressure, 2 point discrimination)
Deep Sensory: Ruffini corpuscles (muscle and joint receptor)
Spinal Pathway
Spinothalamic: pain and temperature
Dorsal Column: discriminative touch
Spinothalamic
Nondiscriminative
Small, slow conduction
Skin origin
From dorsal root horn, cross in spinal cord, lower brainstem, thalamus
Dorsal Column
Discriminative
Large, fast conduction
Skin, joint, tendon origin
Dorsal column, cross in medulla to contra thalamus, sensory cortex
Dermatome Testing
Cutaneous area supplied by single dorsal root
Less likely to be dermatomal and more likely to to homunculus, but still test
Sensory Exam
Light touch (sharp/dull)
2 point: dorsal column
Kinesthesia/proprioception: dorsal column
Vibration: early screening in diabetic cases
Pain and temperature: lateral spinothalamic tract
Cranial nerve screening
Cranial Nerve Testing
CN I: Smell CN II: Read nametag and check visual fields CN II, IV, and VI: eye movement and pupillary reflexes; Ptosis, diplopia, and strabismus CN V: Open and close jaw CN VII: Smile CN VIII: hearing and scratch test CN XI: MMT of SCM and trap CN XII: stick tongue out
Cerebellum Function
Regulates coordinated movements
Rapid movement
Maintain posture, muscle tone, and equilibrium
Cerebellar Dysfunctions
Ataxia: uncoordinated
Hypotonia: Gamma input determined by cerebellum, so less function
Dysmetria: can’t judge distance or range
Dysdiadochokinesia: can’t perform rapid alternating movements
Tremor
Cerebellar Dysfunction Features
Movement decomposition: inability to perform smooth, coordinated movements
Gait Disorder: wide BOS, high guard
Rebound: loss of check reflex
Dysarthria: inability to perform motor component of speech articulation
Coordination Exams
Nonequilibrium: finger to nose, pronation/supination, tapping, heel to toe, heel on shin
Equilibrium: standing, feet together, tandem, perturbations, eyes open/closed
Basal Ganglia Function
Regulates initiation of movement, planning of complex motor tasks, automatic and postural adjustments
Basal Ganglia Dysfunctions
Bradykinesia
Rigidity: lesion in basal ganglion and extrapyramidal tracts
Resting Tremor
Akinesia
Chorea: ongoing, involuntary, discrete movements, rapid, jerky motions
Athetosis: continuous, flowing motions, writhing, twisting
Hyperkinetic Disorders
Unwanted excess movement (involuntary or voluntary)
Chorea
Athetosis
Dystonia
Cerebral Cortex Function
Perceiving and interpreting sensory information
Controlling voluntary movement
Controlling co-contraction
Cerebral Cortex Dysfunction
Spasticity
Activity Limitations Domain
Bed Mobility
Transfers
Locomotion
MOST IMPORTANT ASPECT OF EXAM
What are the core dimensions of PT Expert Practice?
Knowledge
Clinical Reasoning
Movement
Virtues
Knowledge
Multidimensional
Patient-Centered