Exam 1 Flashcards

1
Q

What is laryngeal penetration?

A

When the bolus is above or at the level of the vocal folds.

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2
Q

What is aspiration?

A

When the bolus goes below the level of the vocal folds.

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3
Q

What is residue?

A

Food left behind after we complete a swallow

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4
Q

What is a feeding disorder?

A

One that deals with the process of eating

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5
Q

What is a swallowing disorder?

A

A disorder that deals with the swallowing process in the pharynx and esophagus

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6
Q

Which type of swallowing disorder do SLPs treat?

A

oropharyngeal

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7
Q

What type of function is swallowing?

A

neuro-motor

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8
Q

Most children and adults with ______ have an accompanying swallowing disorder.

A

neuro-motor speech disorder

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9
Q

Speech production and swallowing involve the same _____ ____.

A

anatomical structures

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10
Q

What other professionals help with swallowing disorders?

A

ENT, Neurologist, radiologist, GI physician, Respiratory therapist, OT, dietitian, dentist, nurse, neuro-developmental specialist, patient’s family

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11
Q

What healthcare settings treat swallowing disorders?

A

in patient (sub-acute and acute care), skilled nursing facility, inpatient rehab, outpatient clinics, home health

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12
Q

Why are our lips important in swallowing?

A

They help build up pressure and keep the bolus from spilling out of the mouth

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13
Q

What causes swallowing to occur?

A

Higher pressure in the mouth pushes the bolus to the back of the throat for swallowing

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14
Q

Why are our tongue and jaw important in swallowing?

A

Mastication and bolus formation

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15
Q

Why does the velopharyngeal port close?

A

to prevent naso-regurgitation

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16
Q

What is pharyngeal stripping?

A

contraction of the pharyngeal wall to push food down the esophagus

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17
Q

What does “inversion of epiglottis mean”

A

the epiglottis is closing

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18
Q

Which structures are needed for the oral stage?

A

lips, jaw, tongue, hard palate, soft palate, cheek, mandible, and maxilla

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19
Q

Which structures are needed for the pharyngeal stage?

A

base of tongue, soft palate, larynx, cricopharyngeus

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20
Q

Which structures are needed for the esophageal stage?

A

esophagus

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21
Q

What does the cricopharyngeus muscle do?

A

opens the UES

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22
Q

What are the five cranial nerves needed for swallowing?

A

Trigeminal, facial, vagus, hypoglossal, glossopharyngeal

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23
Q

What are the muscles of mastication? (4)

A

Temporalis, Masseter, Lateral pterygoid, medial pterygoid

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24
Q

Which nerve innervates the muscles of mastication?

A

Trigeminal

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25
Q

Which muscle of mastication is important for projection of the jaw?

A

Lateral pterygoid

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26
Q

What will occur if there is a lesion on the trigeminal nerve?

A

the mastication process will be impaired

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27
Q

What are the 10 muscles of the face? (that we need to know)

A
  1. Orbicularis oris
  2. Zygomaticus minor
  3. Levator labii superior
  4. Zygomaticus major
  5. Levator anguli oris
  6. Depressor anguli oris
  7. Depressor labii inferior
  8. Mentalis
  9. Risorius
  10. Buccinator
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28
Q

What nerve innervated the muscles of the face?

A

Facial

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29
Q

The facial nerve controls the ____ of the tongue.

A

anterior 2/3

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30
Q

Which muscle closes, opens, protrudes, inverts and twists lips?

A

orbicularis oris

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31
Q

Which muscles elevate the upper lip?

A
  1. Zygomaticus minor

2. Levator labii superior

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32
Q

Which muscle elevates the oral cavity?

A

Levator anguli oris

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33
Q

Which muscle lowers the oral cavity?

A

Depressor anguli oris

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34
Q

Which muscle depresses the lower lip?

A

depressor labii inferior

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35
Q

Which muscle pushes up the lower lip and raises the chin?

A

Mentalis

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36
Q

Which muscle gives the expression of strain and angles the mouth outward?

A

Risorious

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37
Q

Which muscle flattens the cheeks?

A

Buccinator

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38
Q

Why are the facial/ lip muscles important?

A

They help prevent anterior spillage.

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39
Q

What are the 5 muscles of the palate?

A
  1. Levator veli palatini
  2. Tensor veli palatini
  3. Palatoglossus
  4. Palatopharyngeus
  5. Uvulae
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40
Q

What nerve innervates 4/5 of the palatal muscles?

A

Vagus

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41
Q

What is the only palatal muscle not innervated by the vagus nerve?

A

tensor veli palatini

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42
Q

Which nerve innervates the tensor veli palatini?

A

Trigeminal

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43
Q

Which palatal muscle elevates the soft palate?

A

Levator veli palatini

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44
Q

Which muscle stretches out the soft palate?

A

Tensor veli palatini

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45
Q

Which muscle raises the back of the tongue during the first stage of swallowing?

A

Palatoglossus

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46
Q

Which muscle shuts of the nasophrynx during the second stage of swallowing?

A

Palatopharyngeus

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47
Q

Which muscle raises the uvula?

A

Uvulae

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48
Q

What are the 4 supra-hyoid muscles?

A
  1. Mylohyoid
  2. Diagastric
  3. Geniohyoid
  4. Stylohyoid
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49
Q

Which 2 supra-hyoid muscles are innervated by the Trigeminal nerve?

A

Mylohyoid and Digastric

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50
Q

Which 1 supra-hyoid muscle innervated by the Hypoglossal nerve?

A

Geniohyoid

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51
Q

Which supra-hyoid muscle is innervated by the facial nerve?

A

Stylohyoid

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52
Q

Which tongue muscle is innervated by the glossopharyngeal and vagus nerve?

A

Palatoglossus

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53
Q

Which muscle controls the anterior and superior movement of the hyoid bone?

A

Geniohyoid

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54
Q

What is impaired if there is a lesion of the facial nerve?

A

Motor:
-Control of the lips- causing anterior spillage
- Control of facial muscles
- Movement of the stylohoid
Sensory: Taste on anterior 2/3rds of tongue

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55
Q

What is impaired if there is a lesion on the hypoglossal nerve?

A

anterior and superior movements of the hyoid bone and tongue movements

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56
Q

What is impaired if there is a lesion on the trigeminal nerve?

A

Motor:
- stretching of the palate (tensor veli palatini)
- mastication
- use of the mylohypid and digastric muscles
Sensory:
- Sensation on the anterior 2/3rd of tongue

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57
Q

What is impaired if there is a lesion on the vagus nerve?

A
Motor:
-laryngeal, pharyngeal, and velum movements
- some esophageal movements
Sensory
- impaired ability to sense aspiration
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58
Q

What happens in the oral prep phase?

A

labial seal –> mastication–> bolus formation

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59
Q

What happens during the oral phase?

A

Tongue pushes the bolus to the back of the mouth

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60
Q

What happens during the pharyngeal phase?

A
  • velopharyngeal port closes
  • base of tongue retraction
  • posterior pharyngeal wall retraction
  • hyolaryngeal excursion
  • epiglottic inversion
  • squeeze of the laryngeal vestibule
  • approximation of the true and false vocal folds
  • pharyngeal stripping
  • UES opening
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61
Q

What negatively affects the oral prep phase?

A

poor salivary function, poor dentician, surgical defects, and neurological disorders

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62
Q

Why is saliva important?

A

It triggers the sensory receptors which trigger saliva production

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63
Q

Which gland produces the majority of our saliva?

A

sub-mandibular

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64
Q

What are the two kinds of saliva?

A

thick mucous and thin serum-like

65
Q

What kind of saliva does the parotid gland produce?

A

thin serum-like

66
Q

What kind of saliva does the sublingual gland produce?

A

thick mucous like

67
Q

What kind of saliva does the sub-mandibular gland produce>

A

a combo of thick and thin

68
Q

What is Xerostomia?

69
Q

Which nerve controls oral sensation in the posterior 1/3rd of the tongue?

A

Glossopharyngeal

70
Q

Which nerve controls oral sensation in the anterior 2/3rds of the tongue?

71
Q

What happens during the esophageal stage?

A

Peristalsis propels the bolus from the UES to the LES–> secondary peristalsis if needed

72
Q

What would patient’s with an insula abnormality present?

A

delayed swallowing

73
Q

Where is the Central Pattern Generator for swallowing housed?

A

In the brainstem

74
Q

The CPG is made up of ___ nuclei

75
Q

Which CPG nucleus is responsible for sensory?

A

nucleus tractus solitarius

76
Q

Which CPG nucleus is responsible for motor?

A

nucleus ambiguous

77
Q

The severity of the dysphagia is ____ when someone has a stroke in the brain stem?

A

higher because that is where the CPG is located.

78
Q

What happens if there is a lesion on the glossopharyngeal nerve?

A
Motor: 
- impaired pharyngeal stripping
- impaired velopharyngeal port closure
Sensory: 
- impaired taste function in posterior parts of tongue
- impaired initiation of the swallow
79
Q

The ________ in the medulla is the principle motor nucleus that provides input to the cranial nerves.

A

nucleus ambiguous

80
Q

Sensory information from the pharynx is relayed by afferent fibers that terminate in the _______

A

nucleus tractus solitarius

81
Q

Describe pathway 1.

A

Sensation –> nucleus tractus solitarius –> thalamus–> sensory strip –> motor strip –> nucleus ambiguous –> swallowing movements

82
Q

Pathway 1 is….

A

More complex, controlled and voluntary

83
Q

Describe pathway 2.

A

Sensation–> nucleus tractus solitarius–> nucleus ambiguous –> swallowing movements

84
Q

Pathway 2 is….

85
Q

What function does the basal ganglia play in swallowing?

A

coordination of movement of the finer elements of swallowing

86
Q

What function does the thalamus play in swallowing?

A

it is the relay station for motor movement

87
Q

What function does the insula play in swallowing?

A

coordinated the interaction of oral musculature, sense of taste and the digestive tract

88
Q

What happens if there is a lesion on the right anterior insula?

A

delayed swallow and sensations of taste, volume, and temperature due to reduced sensory input

89
Q

What is proprioception?

A

awareness with respect to height and movement

90
Q

What function does the cerebellum play in swallowing?

A

timing, coordination, and sequencing of movements–> uses proprioceptive, vestibular and motor planning to create smooth movements

91
Q

Describe the normal anatomy of an infant’s oral cavity.

A
  • Jaw provides stability and neg pressure
  • Large tongue that creates a central groove
  • hard palate and tongue provide pos pressure
  • velum provides posterior seal
92
Q

Describe the normal anatomy of an infant’s pharynx.

A
  • high hyoid position
  • naso-pharynx and hypo-pharynx are curved
  • larynx grows and descends
93
Q

What is non-nutritive sucking?

A

rhythmic movements on a pacifier or finger

NO apneic period

94
Q

What is nutritive sucking?

A
  • for nutrition purposes
  • apneic period present
  • starts around 32 weeks gestation
  • suck swallow ratio of 1:1
95
Q

What is suckling?

A

backward and forward movement of the tongue

96
Q

What is sucking?

A

up and down movement of the tongue

97
Q

What is the rooting reflex?

A

When you touch a infant’s mouth or cheek and the infant turns towards the object and opens mouth

98
Q

What is the suck/swallow reflex?

A

When infant’s mouth is open and lips/ mouth are touched the infant begins suckling or sucking. When a liquid enters, the tongue moves it to the back of the mouth for swallowing

99
Q

What is the tongue thrust reflex?

A

When the lips are touched, the infant’s tongue moves out of the mouth

100
Q

What is the gag reflex?

A

When an object is placed into the back of the mouth, the object is quickly moved back out on the tongue

101
Q

By what age do the reflexes usually diminish in infants?

102
Q

What are the mouth patterns during feeding of an infant birth -5 months?

A
  • suck/swallow reflex
  • tongue thrust reflex
  • rooting reflex
  • gag reflex
103
Q

What are the mouth patterns during feeding of an infant 4-6 months

A
  • draws in upper or lower lip when eating with a spoon
  • up and down munching movements
  • can transfer food from front to back of tongue to swallow
  • tongue thrust and rooting begin to disappear
  • gag reflex diminishes
104
Q

What are the mouth patterns during feeding of an infant 8-11 months?

A
  • moves food from side to side in mouth
  • curves lips around cup
  • begins to chew in rotary pattern
105
Q

What are general physiological changes in aging individuals?

A
  • atrophy
  • decreased range of motion, speed and strength
  • delayed swallowing initiation
  • coordination of breathing and swallowing may be off
106
Q

Is aspiration ever normal?

107
Q

What impact does aging have on the oral stage?

A
  • loss of muscle mass in the tongue
  • decreased tongue pressure
  • prolonged mastication
  • poor dentition
108
Q

What impact does aging have on the pharyngeal stage?

A
  • delayed swallow initiation
  • inadequate hypolaryngeal excursion
  • Cricopharyngeal muscle may be impaired
  • Poor laryngeal adductor reflex
  • sensory and motor impairments leading to residue
109
Q

What terms do we use to define the level of laryngeal penetration?

A

shallow, middle and deep

110
Q

What impact does aging have on the esophageal stage?

A

decreased movement

resting pressure impaired

poor peristalsis

111
Q

Name the symptoms if there is impaired oral bolus prep.

A
  • prolonged mastication
  • longer oral transit time
  • anterior or posterior spillage
  • residue in oral cavity
112
Q

Name the symptoms if there is delayed swallow initiation.

A
  • pre-swallow pooling
  • laryngeal penetration
  • aspiration BEFORE the swallow
113
Q

What is pre-swallow pooling?

A

bolus falls back into the pharyngeal/ laryngeal area before patient actually wants to swallow

114
Q

Name the symptoms associated with impaired epiglottic inversion and laryngeal vestibule squeeze

A

laryngeal penetration/ aspiration DURING the swallow

115
Q

Name the symptoms associated with tongue base retraction.

A
  • residue in the valleculae
116
Q

If bolus is past the level of the vallecular spaces, then there is an issue with ____________.

A

pharyngeal stripping

117
Q

What are the symptoms of impaired tongue base retraction?

A

residue left in the valleculae due to a lack of pressure

118
Q

What are the symptoms of an impaired velopharyngeal port closure?

A

nasal regurgitation

119
Q

What are the symptoms of impaired UES opening and hyoid excursion?

A
  • residue in the pyriform sinuses

- laryngeal penetration/ aspiration AFTER the swallow

120
Q

What are the symptoms of impaired pharyngeal propulsion and pharyngeal stripping?

A
  • diffuse pharyngeal residue in pharyngeal walls, valleculae, and pyriform sinuses
  • prolonged pharyngeal transit time
121
Q

Patient has laryngeal penetration DURING the swallow. What is impaired?

A

epiglottic inversion

122
Q

Patient has residue in the posterior pharyngeal wall. What is impaired?

A

Poor pharyngeal stripping

123
Q

Patient has pre-swallow pooling of bolus to the pyriform sinuses. What is impaired?

A

delayed swallow initiation

124
Q

Patient has residue in the base of the tongue. What is impaired?

A

tongue base retraction

125
Q

Patient aspirates AFTER the swallow of this liquids. What is impaired?

A

Could have multiple causes

  • impaired UES opening
  • impaired hyoid excursion
126
Q

Patient takes 3 seconds to propel pudding into the oropharynx. What is impaired?

A

the oral stage leading to extended oral transit time

127
Q

What is bolus flow?

A

movement of the bolus from the oral cavity to the esophagus

128
Q

Describe volume in terms of bolus flow.

A

Higher the volume, more is the hyoid displacement.

Prolonged UES open times with higher volume.

129
Q

Describe viscosity in terms of bolus flow.

A

Increased oral pressures for thicker consistencies.
Higher chances of laryngeal penetration/aspiration for thinner consistencies (Better airway protection for thicker consistencies).
Chances of residue are more for thicker consistencies.

130
Q

Where do cranial nerves 1 and 2 originate?

A

In the cerebral cortex

131
Q

Where do the cranial nerves 3-4 originate?

132
Q

Where do the cranial nerves 5-8 originate?

133
Q

Where do the cranial nerves 9-12 originate?

134
Q

What happens if there is a lesion on the UMN?

A

spasticity

135
Q

What happens if there is a lesion on the LMN?

A

flaccidity

136
Q

What is it called if there is a lesion on the nerve itself?

A

neuropathy

137
Q

What is it called if there is a lesion on the muscle?

138
Q

What is it called when there is a lesion where the nerve and muscle connect?

A

myoneuropathy

139
Q

What are common swallowing symptoms associated with a stroke?

A
  • sensing the residue
  • sensing penetration/ aspiration
  • sensing swallowing initiation
140
Q

What are the stages of a stroke?

A

Acute- first few days
Sub-acute- up until 6 weeks
chronic - after 6 weeks

141
Q

What sensory deficits are associated with a stroke?

A

delayed pharyngeal initiation

silent aspiration

poor sensation of residue

poor oral sensation

142
Q

What motor deficits are associated with a stroke?

A

poor oral coordinations

poor VPC

poor base of tongue contraction

poor pharyngeal stripping

poor hyoid movement

poor UES opening

poor coordination of swallowing and breathing

143
Q

Bilateral lesions lead to ____ forms of dysphagia

144
Q

Representation of swallowing in the brain is ______.

145
Q

What is Parkinson’s disease?

A

slowly progressive neuro movement disorder caused by lack of dopamine in the substantia naigra

146
Q

Why is dopamine importnat?

A

it inhibits movements so that they are not overactive

147
Q

What is hypokinetic dysarthria?

A

reduced movement of the muscles of speech and swallowing

148
Q

What are the 3 classic symptoms of Parkinson’s

A

rigidity, tremors, and hypokinesia (slow movements)

149
Q

What are the swallowing related symptoms in Parkinson’s disease?

A
Aspiration
Pocketing/pooling of food or saliva.
Nasal /oral regurgitation
Drooling. (reduced muscle movements or sensation)
Excessive secretions.
Pre-swallow pooling
Incomplete airway protection
limited pharyngeal stripping
150
Q

_______ is one of the biggest things effected by Parkinson’s

A

lingual propulsion

151
Q

What is ALS?

A

A degenerative and progressive motor neurom disease that affects the UMN and LMN

152
Q

What are swallowing symptoms of ALS?

A

More motor deficits than sensory

  • aspiration/ penetration after the swallow
  • weakened muscles of larynx and mastication
  • shortness of breath
153
Q

What is Myasthenia Gravis?

A

a myo-neura; junction disease where the body makes antibodies to ACH

154
Q

How does Myasthenia Gravis impact swallowing?

A
  • muscle weakness

- prolonged pharyngeal transit time

155
Q

What is MS?

A

Destruction of the myelin sheath that causes sensory and motor deficits

156
Q

How does MS impact swallowing?

A
SENSORY
pooling
residue
delayed swallow initiation
MOTOR
weak muscle movements
poor airway protection

poor UES opening

157
Q

What is the main stage impacted by swallowing apraxia?

A

the oral stage because it is the only one that can be voluntarily controlled

158
Q

What is swallowing apraxia?

A

lack of coordination of movements during the oral stage of swallowing