Exam 1 Flashcards
Initiation: Bud Stage
Tooth # determined in this stage.
Disturbance → missing or supernumerary teeth
Proliferation: Cap Stage
Tooth # determined in this stage
Disturbance → missing or supernumerary teeth
Histodifferentiation: Bell Stage
Structure of dentin & enamel.
Ameloblasts and odontoblasts form.
Disturbance → Amelogenisis Imperfecta
Morphodifferentiation: Advanced Bell Stage
Tooth size and shape formed at this stage
Apposition
Structure of enamel & dentin.
Enamel & dentin matrix form. This accounts for the layered appearance of enamel & dentin.
Enamel hypoplasia - most common
Calcification
Structure of enamel & dentin.
Sensitive and lenghty process. 4+ years needed for calcification process.
Distrubances → enamel hypocalcification, molar incisor hyperplasia (MIH)
Primary Tooth Eruption
Central → Lateral → 1st Molar → Canine → 2nd Molar
Timing: starts at 7 mo (4+ rule)
Perment Tooth Eruption
MN 1st Ms → MX 1st Ms → MN CIs → MX CIs → MN LIs → MX LIs → MN Cs → MX 1st PMs → MN 1st PMs → MX 2nd PMs → MN 2nd PMs → MX Cs → MN 2nd Ms → MX 2nd Ms → MN 3rd Ms → MX 3rd Ms
MN: canine erupts before premollars
MX: canine erupts after the premolars
What is the first primary tooth to erupt?
MN Central Incisor
7 months
When do primary first molars erupt?
After the lateral incisors and prior to canines
How can delayed tooth eruption (DTE) be determined?
- Biologically: eruption has not occurred despite formation of 2/3 or more of the root
- Chronologically: 2 standard deviations from average expected eruption time based on population studies
What is the most common deviation in eruption?
Delayed Tooth Eruption (DTE)
What causes delayed eruption?
- Genetic disorders (amelogenisis imperfecta, Down Syndrome, etc.)
- Environment (trauma, radiation damage, etc.)
- Systemic conditions (nutrition, rickets, HIV infection, celiac disease, anemia, drugs - phenytoin, low birth weight, etc.)
Primate Space
- MX diastema: b/t primary lateral & canine
- MN diastema: b/t primary canine & 1st molar