Exam 1 Flashcards

1
Q

what are the 5 tissue densities on an x-ray?

A
From dark to light:
Air
Fat
Soft tissue
Bone
Metal
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2
Q

CT scan v MRI

A
* ct
		x rays
		blood
		fast
		use for stroke
			if negative could be ischemic 
*MRI
		radiation
		spinning electrons
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3
Q

dexa scan

A

bone density

shows: thin bones –> easy fracture

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4
Q

why would you choose a bone scan over an x-ray?

A

it shows inflammation from fracture, infection, and

cancer

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5
Q

DDx

A

list of conditions you think would most likely be causing problems
ordered from most to least likely

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6
Q

what is a growth plate fracture called?

A

salter-harris fracture

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7
Q

what is a growth plate called?

A

epiphyseal plate–kids; epiphyseal line–adults

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8
Q

what is adhesive capsulitis?

A

a condition of uncertain etiology characterized by significant restriction of both active & passive shoulder motion that occurs in the absence of a known intrinsic shoulder disorder
capsule becomes inflamed
worse at night
3 phases
pain
stiffness + partially/completely unmoveable
resolves on its own after ~2 years

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9
Q

what motion is most severely limited in adhesive capsulitis?

A

external rotation

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10
Q

what comorbidity is most associated with adhesive capsulitis?

A

diabetes

pre-diabetes/impaired glucose tolerance

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11
Q

what is tos and what causes it?

A

caused by compression of neurovascular structures passing thru one of the spaces in the thoracic outlet

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12
Q

where are the places the neuromuscular structures involved in TOS be impinged?

A
  • interscalene triangle–bordered by anterior scalene muscle anteriorly, middle scalene muscle posteriorly, and the *subcoracoid space/subpectoralis minor space–beneath the coracoid process and just under the minor tendon
  • costoclavicular triangle–middle third of clavicle anteriorly, first rib posteromedially, and upper border of scapula posterolaterally
  • cervical rib
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13
Q

TOS is diagnosed with what tests?

A
  1. physical exam–ADSON & Allen Test
  2. imaging studies:
    x ray
    ultrasound
    venogram
    artereorgram
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14
Q

what is CRPS and why was it previously called RSD?

A

2 types
1 – no specific nerve injury (RSD)
2 – Causalgia obvious nerve damage
OR Warm (acute–swelling) v. Cold (chronic)

Complex Regional Pain Syndrome (CRPS) is a chronic progressive disease characterized by severe pain, swelling and changes in the skin. [1]
It often affects an arm or a leg and may spread to another part of the body.
The pain of CRPS is continuous, and it is widely believed it can be heightened by emotional or physical stress.
Though treatment is often unsatisfactory, early multimodal therapy can cause dramatic improvement or remission of the syndrome in some patients.
unknown etiology of causation but some associated diseases

RSD generic term used to describe post-traumatic pain
us. regarded as a type of sympathetic overactivity and this injury was thought of as a reflex sympathetic disease

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15
Q

allodynia

A

perception of pain from a non-painful stimulus

elicited by a doctor

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16
Q

Hyperalgesia

A

exaggerated sense of pain

17
Q

Hyperesthesia

A

non-noxious stimulus causes the sensation of pain

i.e. not palpation–water, sunlight, heat

18
Q

1st stage of CRPS

A
acute:
**swells, red, burning **
increased diaphoresis of affected limb
symptoms near site of injury
muscle spasm
jt stiffness
restricted mobility
rapid hair & nail growth
vasospasm
19
Q

2nd stage of CRPS

A

dystrophic:
cool & diaphoretic
Sudden’s atrophy of bone on x=ray
pain occurs throughout limb, not just site of injury
skin buildup
more intense pain
hair growth diminishes
hyperkeratosis (skin buildup)
nails cracked, brittle, grooved, spotty
joints thicken
muscles atrophy

20
Q

3rd stage of CRPS

A

atrophic:
pale & shiny skin
atrophy of muscle & bone in affected limb
pain may be constant even w/ treatment
mottling= alterations in blood flow
irreversible changes in skin & bones
pain-unyielding, may involve the entire limb
severly limited mobility of the affected area
flexor tendon contractures
makes bone softening & thinning + muscle atrophy